ライフサイエンスコーパス: caloric

1 found between the clock hour of food intake, caloric amount, meal macronutrient composition, activity

2 ad libitum buffet was used to measure total caloric and macronutrient intakes.

3 a predilection for conserving energy during caloric and protein deprivation and a profligate respons

4 The co-primary outcomes were proportion of caloric and protein prescription received enterally over

5 lyculture agroforestry systems fulfilled the caloric and protein requirements of Late Holocene pre-Co

6 aviors that function to maintain homeostatic caloric balance.

7 Decreasing the amount of caloric beverages consumed and simultaneously increasing

8 Caloric compensation ability decreases between 11 and 15

9 dy was to describe changes in the short-term caloric compensation ability of infants around the age o

10 As hypothesized, the caloric compensation ability significantly decreased wit

11 better understand the early determinants of caloric compensation ability.

12 re] related to interindividual variations in caloric compensation ability.

13 We also included a range of caloric compensation in the model (0%, 39%, and 100%).

14 A 20% reduction in SSB consumption with 39% caloric compensation is projected to result in 26,200 (9

15 -2024 period, a 10% reduction in SSBs with a caloric compensation of 39% is projected to reduce incid

16 l in real time during rewarding experiences (caloric consumption and social interaction).

17 intervention, such as the number, timing and caloric content of snacks, could be considered further i

18 ronutrient-fortified flour without increased caloric content of the general food basket was introduce

19 ate consummatory behaviors regardless of the caloric content or biological relevance of the consumed

20 unt of iodine in infant formulas is based on caloric content, and the label must provide the iodine c

21 plications.We aimed to quantify the systemic caloric contribution of acid-citrate-dextrose regional a

22 Quantitative estimates of the caloric contributions from food groups and their relativ

23 of POMC neurons is decreased selectively by caloric deficit and not altered by high-fat diet or stre

24 increase in AgRP neuron firing with mounting caloric deficit in afternoon vs morning recordings.

25 sed daily energy expenditure, resulting in a caloric deficit of approximately 1 kcal/day; however, to

26 The nutritional characteristics and caloric deficit of the two diets were similar.

27 (sIPSCs) onto POMC neurons increases during caloric deficits.

28 tudies we have demonstrated that a high fat, caloric dense maternal diet structures the offspring's e

29 As food and caloric-dense liquid solutions were used, the data repor

30 More happy tweets and lower caloric density of food tweets in a zip code were associ

31 ded insights into physiological responses to caloric deprivation or to specific macronutrients.

32 ate-day fasting, and other forms of periodic caloric desistance are gaining popularity in the lay pre

33 (HFD) (20% of calories from FA) or a normal caloric diet (C group) (10% of calories from FA) for 16

34 whether a 5-HT(2C)R agonist can reduce high caloric diet (HCD) or methamphetamine (METH) intake and

35 The cellular processes that cause high caloric diet (HCD)-induced infertility are poorly unders

36 metabolism is associated with high-fat high-caloric diet (HFD) feeding, ageing and obesity; however,

37 antly more compared with animals on a normal caloric diet (P < 0.05).

38 here is no evidence regarding whether a high-caloric diet increases survival.

39 n body composition under a high fat and high caloric diet, although the weight of the mutant mice was

40 ibit any fertility decline compared with low caloric diet-fed males despite their resistance to the s

41 remarkable difference in resistance to high caloric diet-induced weight gain of the dab2-deleted mic

42 ecreased consumption of a HCD, but not a low caloric diet.

43 Caloric effects are currently under intense study due to

44 rigeration technologies based on solid-state caloric effects have been attracting attention in recent

45 inexpensive and practical method of driving caloric effects in brittle magnetocaloric materials, whi

46 pharmacotherapeutic approach that decreases caloric efficiency by mitochondrial uncoupling.

47 Day-to-day clock timing of caloric events had poor stability within individuals (~

48 ulation is a common manifestation of chronic caloric excess and obesity that is strongly associated w

49 sly unrecognized transcriptional response to caloric excess by reducing PNC1 expression.

50 Recent research indicates that chronic caloric excess can lead to hypothalamic microinflammatio

51 es on the gut-liver axis under conditions of caloric excess in C57BL/6 mice.

52 or adaptive thermogenesis and dissipation of caloric excess through the activity of uncoupling protei

53 both reduced daily food intake and increased caloric expenditure, driven by an increase in whole-body

54 by superior nutritional density or minimized caloric expenditure.

55 known as beiging or browning that regulates caloric expenditure.

56 we sought to evaluate the efficacy of a high-caloric fatty diet (HCFD) for increasing survival.

57 -derived signaling peptide AQEE-30 upon high caloric feeding.

58 ors (5-HT(2C)Rs) attenuates cocaine and high caloric food intake, but whether a 5-HT(2C)R agonist can

59 ception of the hedonic sensory properties of caloric foods is commonly believed to guide our dietary

60 s essential towards optimization of its spin-caloric functionality for spintronics applications.

61 The appropriate caloric goal for critically ill adults is unclear.

62 sed nutritional support to reach protein and caloric goals (intervention group) or standard hospital

63 sed nutritional support to reach protein and caloric goals reduces the risk of adverse clinical outco

64 During the hospital stay, caloric goals were reached in 800 (79%) and protein goal

65 Pavlovian cues associated with junk-foods (caloric, highly sweet, and/or fatty foods), like the sme

66 Obesity typically is linked to caloric imbalance as a result of overnutrition.

67 olic changes occurred even in the absence of caloric ingestion and were coupled to increased lipogene

68 ARC) promote homeostatic feeding at times of caloric insufficiency, yet they are rapidly suppressed b

69 -day study period, the percent of prescribed caloric intake (+/- SE) received by the enteral route wa

70 -19.8%, p = 0.49) and in mean per capita SSB caloric intake (-13.3%, p = 0.56) from baseline to post-

71 all patients with bvFTD had increased total caloric intake (mean, 1344 calories) compared with the A

72 In the whole cohort, after adjustment for caloric intake and cardiovascular disease risk factors,

73 -type mice, the lean AAV mice have increased caloric intake and do not develop age-related obesity or

74 t is the consequence of an imbalance between caloric intake and energy consumption.

75 Obesity originates from an imbalance between caloric intake and energy expenditure that promotes adip

76 matory pathways results in the uncoupling of caloric intake and energy expenditure, fostering overeat

77 cues, and that maintaining a balance between caloric intake and expenditure may reduce striatal, insu

78 lso imply that maintaining a balance between caloric intake and expenditure over time may reduce stri

79 iposity, the effect of FGF21 on body weight, caloric intake and fat oxidation were significantly atte

80 libitum breakfast test meal, and their total caloric intake and food preferences were measured.

81 e energy expenditure, GLP-1 action to reduce caloric intake and improve glucose control, and GIP acti

82 style intervention, which focused on reduced caloric intake and increased physical activity, or usual

83 These results reveal that caloric intake and liver energy sensors dictate the bloo

84 timescale to adapt to rapid fluctuations in caloric intake and on a chronic timescale to regulate bo

85 ure secondary to exposure to excessive daily caloric intake and overnutrition.

86 of LHA glutamatergic neurons increased daily caloric intake and produced weight gain in mice that had

87 hotosynthesis constitute much of human daily caloric intake and provide the basis for high-energy bio

88 dietary switch changed the pattern of daily caloric intake and suppressed HFD-induced adipose macrop

89 tritional source for humans due to their low caloric intake and their high content in carbohydrates,

90 low social rank is associated with increased caloric intake and weight gain.

91 crops that are vital to sustain the world's caloric intake are salt sensitive.

92 All groups decreased their daily caloric intake by 400 kcal.

93 1.5 fewer HFSS adverts per day and decrease caloric intake by 9.1 kcal (95% UI 0.5-17.7 kcal), which

94 ivity, other breast cancer risk factors, and caloric intake controlled for (false discovery rate <0.2

95 Increased caloric intake correlated with atrophy in discrete neura

96 or age, sex, education, body mass index, and caloric intake examined the associations between MedDiet

97 ct of less-healthy food advertising on acute caloric intake in children from a published meta-analysi

98 igenic gastric peptide hormone secreted when caloric intake is limited.

99 Excess caloric intake is linked to weight gain, obesity, and re

100 In this paradigm, daily caloric intake is restricted to a consistent window of 8

101 However, the mechanisms by which reduced caloric intake modulates inflammation are poorly underst

102 t [Triticum aestivum]) providing most of the caloric intake of contemporary humans and their livestoc

103 the food, is costly, because it reduces the caloric intake of the benefactor vis-a-vis the beneficia

104 rves, independent of changes in body weight, caloric intake or adiposity.

105 ned from stores contribute to disparities in caloric intake over time.

106 Caloric intake tended to decrease after DB administratio

107 CD subjects had increased caloric intake versus controls, but no alterations in to

108 the women were nulliparous, and median daily caloric intake was 1,840 cal (IQR 1,487-2,222).

109 at stool calories expressed as percentage of caloric intake would increase with underfeeding compared

110 respond differentially to various aspects of caloric intake(8-13) and social stimuli(14,15).

111 (500-kcal/d deficit from weight-maintaining caloric intake) and then randomly assigned to pioglitazo

112 ypercaloric diets (in 75% excess of habitual caloric intake) for 3 days, enriched in unsaturated FA (

113 Homeostatic feeding (i.e. titration of caloric intake), is typically associated with hypothalam

114 time of scan, gender, ethnicity, education, caloric intake, and apolipoprotein genotype.

115 on cycle, provides a percentage of our daily caloric intake, and is a major driver in the renewable c

116 to support informed consumer choice, reduce caloric intake, and potentially encourage restaurant ref

117 ural systems regulating natural and adaptive caloric intake, and those regulating social behaviours,

118 These focused on reducing caloric intake, increasing physical activity, and behavi

119 Secondary outcomes included self-reported caloric intake, walking, and moderate physical activity.

120 Evaluating approximately 15% of Americans' caloric intake, we estimate that annual microplastics co

121 Decreases in caloric intake, weight, and BMI correlated with activati

122 reasing de novo lipogenesis independently of caloric intake.

123 gher ambulatory blood pressure and increased caloric intake.

124 er a meal and shows potential for decreasing caloric intake.

125 s of lactoferrin being partly independent of caloric intake.

126 ession, with adjustment for age, gender, and caloric intake.

127 gth z scores were negatively correlated with caloric intake.

128 eppers, is able to induce satiety and reduce caloric intake.

129 lation of LHA glutamatergic neurons enhances caloric intake.

130 eted their fat stores, despite having higher caloric intake.

131 ed weight gain and adiposity despite similar caloric intake.

132 d with accelerated aging due to chronic high caloric intake.

133 ghrelin plasma concentrations, satiety, and caloric intake.Women (n = 39) were more sensitive toward

134 ries that help in balancing food choice with caloric intake; however, this metabolic learning or memo

135 estricted feeding (TRF) regimen in which all caloric intakes occur consistently within </= 12 h every

136 Neither saturated fat nor total caloric intakes were independently associated with obesi

137 Sucralose is widely used as non-caloric intense artificial sweetener.

138 an studies, we examine the relative roles of caloric load and perceived sweetness in driving metaboli

139 demonstrate a non-linear association between caloric load and reward and describe an unanticipated ro

140 Whereas caloric load was manipulated using the tasteless carbohy

141 es and (2) when sweetness is proportional to caloric load, greater metabolic responses are observed.

142 demonstrate a non-linear association between caloric load, metabolic response, and reinforcement pote

143 extent to which sweetness is proportional to caloric load.

144 volving food anticipatory activity (FAA) and caloric loading following food access.

145 Here, we report that optimization of caloric loading in B6 mice subject to HFS, characterized

146 urons are also sufficient to restore FAA and caloric loading of B6 mice subjected to HFS.

147 ntly investigated mucus function during high-caloric/low-fiber dietary interventions, thus making it

148 ver failure and 2 patients developed protein-caloric malnutrition treated by elongation of the common

149 ges outperform those observed in any type of caloric material, and that these changes are comparable

150 field-driven thermal changes in all of these caloric materials fall short with respect to their fluid

151 ricted by the limited performance of current caloric materials, owing to small isothermal entropy cha

152 aves a new pathway for developing novel spin-caloric materials.

153 sent activity and discuss recent advances in caloric measurements, materials, and prototypes.

154 y before and at 160 and 240 min after a high-caloric mixed meal.

155 han guo extracts, which are promoted as non-caloric natural sweeteners, are now incorporated into di

156 to the sweet taste of sugar, but due to the caloric nature of the sweetener.

157 -expressing neurons are acutely activated by caloric need, and this acute activation promotes increas

158 own to modify their behavior during times of caloric need, rapidly adapting to a consistently changin

159 ir mates benefit in ways that go well beyond caloric nutrition.

160 4% of products in the US food supply contain caloric or low-calorie sweeteners, or both.

161 and peripheral inflammatory responses due to caloric overload.

162 measurements reflect the sum of overlapping caloric processes involving binding-linked population sh

163 tate-of-the-art crop models show that global caloric production from maize, wheat, rice, and soybean

164 eneous sample of two groups with equal daily caloric reduction but different protein distributions be

165 daily cycles of feeding and fasting without caloric reduction, sustains robust diurnal rhythms and c

166 the quality of plant-based foods consumed as caloric replacements.

167 l per day, P<0.001; 46+/-14% vs. 71+/-22% of caloric requirements, P<0.001).

168 avioral pattern that promotes acquisition of caloric resources to compensate for low social resources

169 rat model to a greater extent than moderate caloric restriction (by comparison to the WM group).

170 rwent either 2 wk of eTRF (n = 8) or control/caloric restriction (CON:CR; n = 8) diet.

171 is unclear, but prior evidence suggests that caloric restriction (CR) can slow thymic aging by mainta

172 Importantly, caloric restriction (CR) extends lifespan in several org

173 Caloric restriction (CR) extends the lifespan of flies,

174 Caloric restriction (CR) improves health span and life s

175 Caloric restriction (CR) improves insulin sensitivity an

176 In lean mice, caloric restriction (CR) induces bouts of compulsive bin

177 Caloric restriction (CR) is commonly recommended for imp

178 e to adjust their activities when faced with caloric restriction (CR) to deal with reduced energy int

179 Caloric restriction (CR) without malnutrition extends li

180 (iePPARgammaKO) to a two-week period of 25% caloric restriction (CR), following which iePPARgammaKO

181 xtension of chronological life span (CLS) by caloric restriction (CR).

182 ction of resting energy expenditure (REE) to caloric restriction (CR).

183 s throughout the body that may be delayed by caloric restriction (CR).

184 low intestinal penetrance (n = 12), and (3) caloric restriction (n = 8).

185 Caloric restriction alone also led to significant decrea

186 ve caloric restriction and aerobic exercise, caloric restriction alone, aerobic exercise alone, or us

187 for potential factors that are regulated by caloric restriction and act as caloric restriction mimet

188 tested the hypothesis that implementation of caloric restriction and aerobic exercise is feasible and

189 ts consented, 111 were randomized to receive caloric restriction and aerobic exercise, caloric restri

190 l nutrient uptake and thus causes functional caloric restriction and allows improved anti-tumor immun

191 Diet can greatly impact health, while caloric restriction and fasting have putative benefits f

192 s using preconditioning protocols, including caloric restriction and hypoxic preconditioning, have be

193 ing strategies shows overlapping patterns in caloric restriction and hypoxic preconditioning, pointin

194 To identify genes and pathways shared by caloric restriction and hypoxic preconditioning, we used

195 he airway hyperresponsiveness preventable by caloric restriction and IL-1beta blockade.

196 L-1beta mechanism, which can be prevented by caloric restriction and IL-1beta blockade.

197 Caloric restriction and intermittent fasting are emergin

198 lycemia and promoting survival during severe caloric restriction and the requirement for ghrelin cell

199 In rodents, caloric restriction and young blood-induced revitalizati

200 Caloric restriction can ameliorate celsr1a aging phenoty

201 In mammals, caloric restriction consistently results in extended lif

202 nth randomized controlled trial, comparing a caloric restriction diet arm (goal: 10% weight loss, N =

203 In this study we find that caloric restriction dramatically rescues the motor incoo

204 Gene expression and caloric restriction experiments in model organisms confi

205 treatment with 2.24 mg/kg.d rapamycin or 40% caloric restriction for 9 weeks partially rescued cardio

206 Biase et al. independently demonstrate that caloric restriction from fasting and pharmacologic inhib

207 Caloric restriction has been associated with increased l

208 rift is a determinant of lifespan in mammals.Caloric restriction has been shown to increase lifespan

209 sely, lifespan-extending maneuvers including caloric restriction impose beneficial pleiotropic effect

210 and includes peri-operative antibiotics and caloric restriction in addition to the altered anatomy.

211 , it is significantly prevented by long-term caloric restriction in aged mice.

212 ges in offspring fitness or plasticity under caloric restriction in B. manjavacas.

213 eplacement of ghrelin blocked the effects of caloric restriction in beta1AR-deficient mice.

214 tion drift correlates with lifespan and that caloric restriction in mice and rhesus monkeys results i

215 sized by the hypoglycemia that is induced by caloric restriction in mouse models of deficient ghrelin

216 iated with more weight loss during sustained caloric restriction in overweight subjects.

217 Caloric restriction increases lifespan in all offspring,

218 Caloric restriction inhibits hepatosteatosis, reduces Wn

219 Caloric restriction is known to improve inflammatory and

220 Caloric restriction is known to up-regulate expression o

221 ller reductions in energy expenditure during caloric restriction is not known.

222 Longevity-promoting caloric restriction is thought to trigger downregulation

223 Our data indicate that caloric restriction leads to a marked improvement in glu

224 mechanisms that prevent inflammation during caloric restriction may yield promising therapeutic targ

225 ccurring polyamine, has been recognized as a caloric restriction mimetic that confers health benefits

226 Caloric restriction mimetics (CRMs) mimic the biochemica

227 regulated by caloric restriction and act as caloric restriction mimetics.

228 "habit," supporting the view that persistent caloric restriction mimics some aspects of addiction to

229 The effects of caloric restriction on DNA methylation were detectable a

230 exercise, and aerobic capacity compared with caloric restriction on regulating intrahepatic lipids an

231 herefore determined the effects of aging and caloric restriction on the expression of FXR and TGR5 in

232 older patients with clinically stable HFPEF, caloric restriction or aerobic exercise training increas

233 normalization of weight was mediated not by caloric restriction or increased activity, but by increa

234 -dependent lifespan extension resulting from caloric restriction or loss of germline stem cells.

235 e on control diet or one of 2 diet regimens (caloric restriction or rapamycin) that altered protein t

236 eater decreases in energy expenditure during caloric restriction predict less weight loss, indicating

237 s are decreased in the aging kidney and that caloric restriction prevents these age-related decreases

238 We further show that caloric restriction rescues SIRT1 levels in transgenic M

239 red with depletions by exercise alone, acute caloric restriction results in rapid changes in appetite

240 how that both short-term fasting and chronic caloric restriction significantly reduce the percentage

241 to 30-year-old rhesus monkeys exposed to 30% caloric restriction since 7-14 years of age showed atten

242 seen in 2.7-3.2-year-old mice exposed to 40% caloric restriction starting at 0.3 years of age.

243 the weight-loss program included nutrition (caloric restriction) and psychological therapies.

244 nduced by dietary restriction (also known as caloric restriction).

245 lifespan, reproduction, and the response to caloric restriction, and investigate maternal investment

246 t stimulate AMPK and PGC1alpha via exercise, caloric restriction, and medications result in stimulati

247 BHB levels are elevated by starvation, caloric restriction, high-intensity exercise, or the low

248 energy deficit (kilocalories per day) during caloric restriction, incorporating energy intake and was

249 Starvation, or caloric restriction, is known to activate the transcript

250 Metabolic factors such as caloric restriction, ketogenic diet, and hyperglycemia i

251 nd nutritional factors, such as exercise and caloric restriction, may exert their known health benefi

252 While exercise, caloric restriction, metformin and many natural products

253 idative stress, exercise-induced adaptation, caloric restriction, osmotic stress, mechanical stress,

254 cations such as reduced carbohydrate intake, caloric restriction, structured exercise, and/or pharmac

255 by specific interventions: metabolic risk by caloric restriction, systemic inflammation by statins, p

256 In the context of caloric restriction, there seems to be no additional ben

257 e after bariatric surgery independently from caloric restriction, whereas the level of WAT TGR5 prote

258 ergic nature of AgRP neurons is increased by caloric restriction, whether the GABAergic phenotype of

259 be more effective for reducing body fat than caloric restriction, which is currently the treatment of

260 ) localized to ghrelin cells is required for caloric restriction-associated ghrelin release and the e

261 th the FXR-TGR5 dual agonist INT-767 induced caloric restriction-like effects and reversed age-relate

262 fitness benefits of lifespan extension under caloric restriction.

263 nisms promotes longevity in a manner akin to caloric restriction.

264 accumulation through a mechanism resembling caloric restriction.

265 own and delays age-related kidney disease is caloric restriction.

266 se and the changes in their capacities after caloric restriction.

267 ance to H(2)O(2) and extending lifespan upon caloric restriction.

268 glycemia and prevalent mortality upon severe caloric restriction.

269 e metabolite profile is mainly attributed to caloric restriction.

270 groups followed a Mediterranean diet without caloric restriction.

271 Volunteers then underwent 6 weeks of 50% caloric restriction.

272 uenced by the energy expenditure response to caloric restriction.

273 (r = 0.79), and was decreased in response to caloric restriction.

274 d metabolism were both increased, even under caloric restriction.

275 odulated by environmental challenges such as caloric restriction.

276 rfeeding and resistant to weight loss during caloric restriction.

277 reased the consumption of a palatable liquid caloric reward.

278 cortex that selectively responded to either caloric rewards or social stimuli, and found that activi

279 BNST GABA synapses was tightly linked to the caloric state of male rats, seesawing between long-term

280 interoceptive ARC(AgRP) neurons, respond to caloric state, and are sufficient and necessary to contr

281 ubtype 5 (mGluR5) in the VMH is regulated by caloric status in normal mice and reduced in brain-deriv

282 These different caloric strands have recently been unified to yield a si

283 sing a sugar alcohol, maltitol, a common low caloric sugar substitute that exploits the chemical exch

284 genic drugs and other precipitating factors, caloric support, and often heme therapy.

285 Our findings show how caloric suppression of the guanylin-GUCY2C signaling axi

286 vioside and rebaudioside A, are natural, non-caloric sweet-tasting organic molecules, present in extr

287 verage was associated with more purchases of caloric-sweetened desserts or sweeteners, which accounte

288 urchases, sugar, and fat, and purchased more caloric-sweetened desserts/caloric sweeteners compared w

289 (2020) compare intra-gastric sugar and non-caloric sweetener to investigate how post-ingestive effe

290 Non-caloric sweeteners (NCS) are food additives providing sw

291 013, 68% (by proportion of calories) contain caloric sweeteners and 2% contain low-calorie sweeteners

292 anges in the meat, poultry, fish; dairy; and caloric sweeteners categories.

293 nd purchased more caloric-sweetened desserts/caloric sweeteners compared with nonconsumers.

294 orie sweeteners and beverages sweetened with caloric sweeteners had poorer dietary quality, exhibited

295 ed, caused by adaptive mechanisms maximizing caloric uptake and increasing intestinal, villi, and mic

296 ore swiftly than a flower type with the same caloric value but without nicotine.

297 Accounting for the caloric value of RDAs for carbohydrate and fat, "flexibl

298 eostasis favored detection and comparison of caloric value.

299 olic steatohepatitis, despite its irrelevant caloric value.

300 Caloric vestibular stimulation did not alter perceived s