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1           Type I hair cells are contacted by calyceal afferent endings.
2 ost Sox2 immunoreactivity and the density of calyceal afferent nerve terminals (specific to Type I ha
3 al transmission from type I cells onto their calyceal afferent terminals.
4 synaptic cleft between type I hair cells and calyceal afferents, K(+) ions accumulate as a function o
5 synaptic cleft between type I hair cells and calyceal afferents, K(+) ions accumulate as a function o
6 ells and afferents, afferent transmission at calyceal and bouton synapses, and spike generation in re
7 system are innervated by afferents that form calyceal and bouton terminals, respectively.
8 nomalies, five urinary tract calculi, and 18 calyceal and/or papillary, 30 renal pelvic and/or ureter
9           We found that the stability of the calyceal AP waveform requires KCNQ (K(V)7) K(+) channel
10 outcome of patients with small, asymptomatic calyceal calculi.
11 ear period, 15 patients (26 kidneys) without calyceal dilatation at ultrasonography (US) who required
12                                              Calyceal diverticula are rarely diagnosed in children.
13                       Standard management of calyceal diverticula in children includes ultrasonograph
14 en should include particularly: simple cyst, calyceal diverticulum and the first demonstration of ADP
15                            The management of calyceal diverticulum has evolved from an open surgical
16                   A direct puncture into the calyceal diverticulum is recommended.
17                 Percutaneous ablation of the calyceal diverticulum is the most established minimally
18                                 Diagnosis of calyceal diverticulum should be confirmed by contrast st
19 hree (without reflux) had a pelvic kidney, a calyceal diverticulum, or a renal stone.
20  we present a case of pediatric patient with calyceal diverticulum, with initial ultrasonographic dia
21 algorithm for the patient with a symptomatic calyceal diverticulum.
22    Using a multidisciplinary approach to the calyceal endbulb of Held synapse that transmits auditory
23 e central zones of vestibular epithelia form calyceal endings around type I hair cells and have phasi
24 rom efferent terminals, directly depolarizes calyceal endings by activating nicotinic ACh receptors (
25 c spaces found at giant synapses such as the calyceal endings in the auditory and vestibular systems.
26 c spaces found at giant synapses such as the calyceal endings in the auditory and vestibular systems.
27 t encode timing with precision, some through calyceal endings.
28 he trapezoid body was unable to elicit large calyceal EPSCs in MNTB neurons of hyperbilirubinaemic ra
29 ganglia-thalamo-pallial loop that contains a calyceal GABAergic synapse in the thalamus.
30 ich suppress multiple AP firing, during each calyceal giant EPSC.
31 ure 1:1 innervation by selecting the winning calyceal input based on synaptic strength.
32 with glutamatergic conductances activated by calyceal inputs.
33 e, there are 15-20 ribbon synapses and 25-30 calyceal invaginations in each type I hair cell.
34                                              Calyceal invaginations per type I HC range from 19 in CZ
35 ly small in 2-week-old rats, an age by which calyceal maturation has reportedly neared completion.
36 hat a patterned alignment of proteins at the calyceal membrane resembles a type of intercellular junc
37 mbrane and appears diffused along the entire calyceal membrane.
38  This pharmacological profile suggested that calyceal nAChRs contain alpha6 and beta2, but not alpha9
39 tors potentiates glutamate release in mature calyceal nerve terminals of the rat medial nucleus of th
40 urotransmitter acetylcholine (ACh) activates calyceal nicotinic ACh receptors (nAChRs); however, it i
41 ncreases afferent input impedance by closing calyceal potassium channels.
42     By contrast, mouse photoreceptors lacked calyceal processes and had no USH1 proteins at the inner
43 n rods and (ii) between the microvillus-like calyceal processes and the outer segment basolateral reg
44 otein network, which was associated with the calyceal processes from the early embryonic stages of ou
45                                          The calyceal processes were virtually absent in cones and di
46  were presynaptic to type II cells, calyces, calyceal processes, and other nerve fibers in the macula
47                              We propose that calyceal processes, together with their associated links
48  The USH1 protein complex is associated with calyceal processes, which are microvilli of unknown func
49 ceptors and proper protein expression in the calyceal processes.
50 Multi-photon imaging of anterograde-labelled calyceal projections revealed axonal staining and presyn
51 ates microglia in the brainstem and disables calyceal pruning and maturation of astrocytes in the med
52 found that BLZ/PLX treated mice had impaired calyceal pruning, diminished astrocytic GFAP in the late
53 lly, HCN+ cells were shown to integrate into calyceal RPSM pacemaker tissues, and HCN channel block a
54 emaker depolarizations originate in proximal calyceal RPSM.
55     The influence of spike thresholds at the calyceal spike initiation stage sharpened tuning and adv
56 ed how stages from hair-cell transduction to calyceal spiking contribute tuning and timing to central
57 oreal shockwave lithotripsy (ESWL) for lower calyceal stones are generally disappointing.
58 of spatial anatomy in the clearance of lower calyceal stones by ESWL is needed.
59 ve lithotripsy for small, asymptomatic renal calyceal stones does not appear to offer any advantage t
60 d to determine treatment protocols for lower calyceal stones.
61 he reliability of signal transfer across the calyceal synapse and observed a potassium conductance me
62  using pre- and postsynaptic recordings of a calyceal synapse in the medial nucleus of the trapezoid
63                                         At a calyceal synapse, a profound temporal filtering of fast
64 ng the AMPA receptor EPSC were examined at a calyceal synapse.
65                                  Endbulb and calyceal synapses convey signals with a high degree of p
66 tance and the postsynaptic current at single calyceal synapses in rat brainstem.
67 293 cells or at presynaptic terminals of the calyceal synapses in the auditory brainstem are more vul
68 fast component of nonquantal transmission at calyceal synapses is indefatigable and responsible for u
69 e vestibular sensory epithelia form distinct calyceal synapses.
70 KCNQ4 at the postsynaptic membranes of these calyceal synapses.
71 n be explained by the high resistance of the calyceal synaptic cleft since it was significantly reduc
72 like junction provides structural support to calyceal synaptic contact with the vestibular hair cell
73 and vCAPs have been shown previously to make calyceal synaptic contacts with type I hair cells in the
74 manner by MNTB principal neurons receiving a calyceal synaptic input.
75 ions for conveying precise timing, including calyceal synaptic terminals and matching axonal conducti
76 ron microscopy confirmed loss of presynaptic calyceal terminals and supported the electrophysiologica
77                                          The calyceal terminals both respond to glutamate released fr
78 ive zones (AZs), previously possible only at calyceal terminals containing many AZs, where few channe
79     Here, we made whole-cell recordings from calyceal terminals in newborn rat pups.
80 in the axons of VCN bushy cells and/or their calyceal terminals rather than in the MNTB neurons thems
81             These aberrant branches ended in calyceal terminations in MNTB.
82 ng rate output without relying on one-to-one calyceal transmission.
83 gic inhibitory system capable of suppressing calyceal transmission.
84                        With the exception of calyceal-type endings in the contralateral ventral nucle