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1 marked and genotyped field crickets (Gryllus campestris).
2 olved in the regulation of heat stress in B. campestris.
3 (Pseudomonas) solanacearum, and Xanthomonas campestris.
4 ith RXC4 confers digenic resistance to X. c. campestris.
5 s RXC1, a gene conferring tolerance to X. c. campestris.
6 a (Pseudomonas) solanacearum and Xanthomonas campestris.
7 from the Brassica pathogen X. campestris pv. campestris.
8 terial pathogens P. syringae and Xanthomonas campestris.
9 glycopeptide processing by X. campestris pv. campestris.
10 significant homology to RpfF in Xanthomonas campestris.
11 o a virulent bacterial pathogen, Xanthomonas campestris.
12 gae, Pseudomonas aeruginosa, and Xanthomonas campestris.
15 ra43 (Abra43) and Xanthomonas campestris pv. campestris 8004 (Xcc8004), on the structure and function
19 Ms of Pseudomonas aeruginosa and Xanthomonas campestris, albeit not in the enzymes from Pseudomonas f
22 2, conditions monogenic resistance to X. c.; campestris and was mapped to a 5.5 cM interval of chromo
23 hogens, Pseudomonas syringae and Xanthomonas campestris, and an oomycete, Peronospora parasitica.
24 E. coli and plant pathogens X. oryzae and X. campestris, as well as against human fungal pathogens C.
25 Recently, it was shown that the Xanthomonas campestris AvrBs2 protein can be delivered directly into
26 ound in Allium cepa, Beta vulgaris, Brassica campestris, Brassica oleracea, Pennisetum glaucum, Pinus
27 vars of Pseudomonas syringae and Xanthomonas campestris, but also enhanced the growth of the host pat
30 Our results evidenced that X. campestris pv campestris CN08 tales are relevant for symptom developme
31 cted vegetables (Spinacia oleracea, Brassica campestris, Coriandrum sativum, and Mentha spicata) were
32 notypes of a DeltarpfF strain in Xanthomonas campestris could be complemented by its own DSF, the DSF
35 eported that, the Xanthomonas campestris pv. campestris effector XopP compromises AtExo70B1, while by
36 The velvet longhorned beetle, Trichoferus campestris (Faldermann) ("VLB"; Coleoptera: Cerambycidae
37 . microcarpus) for p-coumaric acid, 20.3 (A. campestris) for ferulic acid, 561.9 (A. campestris) for
38 (A. campestris) for ferulic acid, 561.9 (A. campestris) for gallic acid, 38.7 (A. campestris) for p-
40 .9 (A. campestris) for gallic acid, 38.7 (A. campestris) for p-hydroxybenzoic acid and 7.08 (A. campe
45 amylovora, Pseudomonas spp., and Xanthomonas campestris has impeded the control of several important
46 regardless of host species and occurs in C. campestris haustoria produced in the absence of any host
50 ted, and pathogen (Xanthomonas campestris pv campestris)-infected plants, callus, roots, and young se
53 tryptophan 2,3-dioxygenase from Xanthomonas campestris into a monooxygenase for oxidative cyclizatio
54 ophan 2,3-dioxygenase (TDO) from Xanthomonas campestris is a highly specific heme-containing enzyme f
56 odis pv. vesicatoria or to X. campestris pv. campestris is associated with increased synthesis of the
57 erecta (Ler) with Xanthomonas campestris pv campestris isolate 2D520 results in extensive necrosis a
58 ality of flowering Chinese cabbage (Brassica campestris L. ssp. chinensis var. utilis Tsen et Lee).
63 s of the active site cysteine in Xanthomonas campestris OleA (Cys(143)) enabled trapping of two catal
64 two B. subtilis homologs of the Xanthomonas campestris organic hydroperoxide resistance (ohr) gene.
67 XopN is a virulence factor from Xanthomonas campestris pathovar vesicatoria (Xcv) that is translocat
68 t XopD, a type III effector from Xanthomonas campestris pathovar vesicatoria (Xcv), suppresses sympto
69 thogen fitness and its prevalence in many X. campestris pathovars suggests that the Bs2 gene may be d
70 omparative mapping approach between Brassica campestris plants homozygous for the S8 haplotype and Ar
71 e stably transformed and regenerated RUBY C. campestris plants produced haustoria, the signature orga
72 minants of the vascular pathogen Xanthomonas campestris pv campestris (Xcc) during infection of the c
76 scular phytopathogenic bacterium Xanthomonas campestris pv campestris (Xcc), the causal agent of blac
78 sion Landsberg erecta (Ler) with Xanthomonas campestris pv campestris isolate 2D520 results in extens
79 ted, heat-treated, and pathogen (Xanthomonas campestris pv campestris)-infected plants, callus, roots
80 gene are resistant to strains of Xanthomonas campestris pv vesicatoria (Xcv) expressing the bacterial
81 to (Lycopersicon esculentum) and Xanthomonas campestris pv vesicatoria (Xcv), to examine the interact
84 notypes with virulent bacterial (Xanthomonas campestris pv vesicatoria and Pseudomonas syringae pv to
85 rBsT is a type III effector from Xanthomonas campestris pv vesicatoria that is translocated into plan
86 effector from the plant pathogen Xanthomonas campestris pv vesicatoria, interacts with the proteasoma
88 from the Xcv library were conjugated into X. campestris pv. campestris (Xcc) and exconjugants were sc
89 e two signals in the Arabidopsis-Xanthomonas campestris pv. campestris (Xcc) compatible interaction.
90 cellular polysaccharide (EPS) in Xanthomonas campestris pv. campestris (Xcc) is regulated by a cluste
94 f apo Zur from the phytopathogen Xanthomonas campestris pv. campestris (XcZur), which reveals the mol
95 brassicicola Abra43 (Abra43) and Xanthomonas campestris pv. campestris 8004 (Xcc8004), on the structu
97 We previously reported that, the Xanthomonas campestris pv. campestris effector XopP compromises AtEx
98 thomonas axonopodis pv. vesicatoria or to X. campestris pv. campestris is associated with increased s
99 f the superfamily encoded by the Xanthomonas campestris pv. campestris str. ATCC 33913 genome (GI:212
104 thomonas wilt disease, caused by Xanthomonas campestris pv. musacearum (Xcm), is a major threat to ba
107 r Bs2 gene confers resistance to Xanthomonas campestris pv. vesicatoria (Xcv) pathogenic strains whic
108 Here, we provide evidence that Xanthomonas campestris pv. vesicatoria (Xcv) suppresses host autopha
110 outer protein S (XopS), a T3E of Xanthomonas campestris pv. vesicatoria (Xcv), interacts with and inh
113 ose to the tomato bacterial spot pathogen X. campestris pv. vesicatoria 85-10, with a completely diff
114 s most similar to hrp genes from Xanthomonas campestris pv. vesicatoria and Ralstonia solanacearum.
115 g the molecular basis for virulence of 20 X. campestris pv. vesicatoria field strains that were isola
116 ization of the avrBs2 locus from Xanthomonas campestris pv. vesicatoria has revealed that expression
117 tive tomato plants infected with Xanthomonas campestris pv. vesicatoria have greatly reduced disease
118 irulent and avirulent strains of Xanthomonas campestris pv. vesicatoria in tomato (Lycopersicon escul
119 o to those of X. axonopodis pv. citri and X. campestris pv. vesicatoria provides valuable insights in
121 at infection of pepper plants by Xanthomonas campestris pv. vesicatoria strains expressing the AvrBs2
122 confers resistance to strains of Xanthomonas campestris pv. vesicatoria that contain the correspondin
123 of the bacterial plant pathogen Xanthomonas campestris pv. vesicatoria triggers disease resistance i
125 resistance to B. cinerea infection and to X. campestris pv. vesicatoria, correlated with cuticle perm
126 type III secreted effector from Xanthomonas campestris pv. vesicatoria, is a desumoylating enzyme wi
132 Thus, the protocol for transformation of C. campestris reported here overcomes a major obstacle to C
134 ntitative and broad-spectrum resistance to X campestris RKS1-dependent gene expression was shown to i
135 ore conserved between B. oleracea S13 and B. campestris S8, two haplotypes that have been proposed to
140 L. edodes) and five wild (L. sulphureus, A. campestris, T. clypeatus, T. microcarpus and T. letestui
141 l and biochemical studies of the Xanthomonas campestris TDO and a related protein SO4414 from Shewane
143 riboswitch (SAM-I(Xcc)) from the Xanthomonas campestris that regulates methionine synthesis via the m
144 cholerae, and the plant pathogen Xanthomonas campestris The bioconjugated phanorods could selectively
145 defense responses against P. syringae and X. campestris The P. syringae T3SE HopZ1a is an acetyltrans
149 sponse to the bacterial pathogen Xanthomonas campestris To tackle this challenge, we first performed
153 a, like Pseudomonas syringae and Xanthomonas campestris, use the type III secretion system as a molec
155 To test this conjecture, rpfC and rpfF of X. campestris were replaced by those of X. fastidiosa, and
158 sequence similarity to GumC from Xanthomonas campestris, which is involved in exopolysaccharide expor
159 zobium meliloti (succinoglycan), Xanthomonas campestris (xanthan gum), and Salmonella enterica (O ant
160 tovorum, Ralstonia solanacearum, Xanthomonas campestris, Xanthomonas oryzae, and Xylella fastidiosa T
162 brary were conjugated into X. campestris pv. campestris (Xcc) and exconjugants were scored for an alt
164 vascular pathogen Xanthomonas campestris pv campestris (Xcc) during infection of the cauliflower hos
165 tic screen in the plant pathogen Xanthomonas campestris (Xcc) identified that XC_0250, which encodes
168 ccharide (EPS) in Xanthomonas campestris pv. campestris (Xcc) is regulated by a cluster of genes call
171 thogenic bacterium Xanthomonas campestris pv campestris (Xcc), the causal agent of black rot disease
176 O) indoleamine 2,3-dioxygenases, Xanthomonas campestris (XcTDO) tryptophan 2,3-dioxygenase, and the H
177 the phytopathogen Xanthomonas campestris pv. campestris (XcZur), which reveals the molecular mechanis
178 rysanthemi and carotovora (out), Xanthomonas campestris (xps), Pseudomonas aeruginosa (xcp), Aeromona