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2 controls received an intrasocket mineralized cancellous allograft (socket group), and 12 patients rec
4 ine if PRP combined with a rapidly resorbing cancellous allograft would enhance the regenerative resu
5 s treated with BG, such as mineralized human cancellous allograft, were more likely to have greater v
6 te that solvent-preserved, mineralized human cancellous allograft, with or without collagen membrane,
9 DBM formulated with hyaluronic acid (HY) and cancellous and cortical bone granules from the same dono
10 antibodies increased bone formation and thus cancellous and cortical bone mass in skeletally mature r
11 Notch2(tm1.1Ecan) mutant mice exhibit severe cancellous and cortical bone osteopenia due to increased
12 In conclusion, Notch2(Q2319X) mice exhibit cancellous and cortical bone osteopenia, enhanced osteoc
17 orphometric analysis of tetracycline-labeled cancellous bone and dual-energy x-ray absorptiometry, re
18 effects of microstructure on fatigue life in cancellous bone and lattice structures are described emp
22 We show that the more ductile surfaces of cancellous bone are a result of reduced accumulation of
23 creased bone density, serum osteocalcin, and cancellous bone area along with trabecular narrowing.
25 ts osteoclastogenesis and bone resorption in cancellous bone but increases intracortical remodeling a
26 Y, 32:68, wt/wt; DBM mixed with cortical and cancellous bone chips 1:4 (DBMC) (11 mg total, of which
27 ociated with enhanced bone resorption in the cancellous bone compartment and with suppressed endocort
34 ls in postnatal mice dramatically stimulated cancellous bone formation via marked expansion of the os
36 is study, data suggests GTR using allogeneic cancellous bone graft and absorbable collagen membrane t
37 defects treated with GTR using an allogeneic cancellous bone graft and covered by an absorbable membr
41 Histomorphometric parameters characterizing cancellous bone in the distal radius can be derived from
46 ontribute to the increase in osteoclasts and cancellous bone loss that occurs after loss of estrogen.
47 ion in mediating estrogen deficiency-induced cancellous bone loss was investigated in ovariectomized
51 s suggest that CMA plays a role in vertebral cancellous bone mass accrual in young adult mice and tha
52 age female L2ACgKO mice had lower vertebral cancellous bone mass compared to wild-type (WT) controls
54 e, but not in adult mice, whereas epiphyseal cancellous bone mass decreased with loading in both youn
55 likely to accumulate in strut centers making cancellous bone more tolerant of stress concentrations a
58 creased prevalence of apoptosis in vertebral cancellous bone osteocytes and osteoblasts that follows
59 n conclusion, Notch2(tm1.1Ecan) mice exhibit cancellous bone osteopenia that can be ameliorated by sy
64 evised a method for obtaining information on cancellous bone structure from iliac bone histomorphomet
68 tes is essential for osteoclast formation in cancellous bone under physiological conditions, and RANK
70 Notch in the skeleton causes an increase in cancellous bone volume and enhanced osteoblastic differe
71 reatment was associated with preservation of cancellous bone volume and inhibition of osteoclast form
73 microstructural abnormalities such as lower cancellous bone volume and reduced trabecular thickness.
74 mined by dual-energy densitometry; decreased cancellous bone volume and trabecular width and increase
75 ob/ob mice pair-fed to WT mice had normal cancellous bone volume fraction (BV/TV) in distal femur,
76 content (BMC) and density (BMD), and higher cancellous bone volume fraction in lumbar vertebra (LV).
80 e characterized by a significant decrease in cancellous bone volume in the tibial and femoral metaphy
82 usly over the calvaria of mice and increased cancellous bone volume when orally administered to rats.
83 7(-/-)) exhibit higher bone mineral density, cancellous bone volume, and mechanical strength compared
84 iblings, demonstrated a striking decrease in cancellous bone volume, connectivity, and trabecular num
85 m, and normalization of bone markers such as cancellous bone volume, trabecular number, osteoblast su
89 ature osteocytes in mineralized cortical and cancellous bone was positive for sclerostin with diffuse
94 palpable difference between the cortical and cancellous bone, both of which have different material p
95 anatomical structures including cortical and cancellous bone, intervertebral discs, ligaments, and ca
97 fiber reinforcement reached the strength of cancellous bone, which was much stronger than previous i
98 The ability to create mechanically strong 'cancellous bone-like' printable implants for tissue repa
109 tructed and registered, and the cortical and cancellous bones of the mandible and the maxilla were se
110 n derivative (EMD) and particulate anorganic cancellous bovine-derived bone xenograft (BDX) have both
111 In summary, both the particulate anorganic cancellous bovine-derived bone xenograft used alone and
113 or the protective effect of estrogens on the cancellous, but not the cortical, bone compartment that
114 sayed demineralized bone matrix and cortical cancellous chips uniformly dispersed in a thermoplastic
116 garding use of a combination 50%/50% cortico-cancellous FDBA compared with a 100% cortical or 100% ca
118 neralized CT/other material was found in the cancellous FDBA group compared with the cortical FDBA gr
121 ion in non-molar sites using 50%/50% cortico-cancellous FDBA versus 100% cortical and 100% cancellous
122 ancellous FDBA, 100% cortical FDBA, and 100% cancellous FDBA when used in ridge preservation of non-m
124 or dimensional changes among 50%/50% cortico-cancellous FDBA, 100% cortical FDBA, and 100% cancellous
128 he following: 1) 100% cortical FDBA; 2) 100% cancellous FDBA; or 3) 50%/50% cortico-cancellous FDBA.
129 years would result in measurable changes to cancellous features and the biochemical markers compared
130 d ridge preservation with either cortical or cancellous freeze-dried bone allograft (FDBA) in non-mol
132 ative concentration of HSPCs and BVFs within cancellous marrow was observed to diminish with increasi
133 ralized bone matrix (GDBM) by comparing with cancellous mineralized bone matrix (CMBM) and anorganic
136 Surprisingly, we found no effect on either cancellous or cortical bone, even following mechanical l
137 orticoid-induced osteoporosis, the number of cancellous osteoclasts increased, even though osteoclast
138 dministration of Notch2 ASOs ameliorates the cancellous osteopenia of Notch2(tm1.1Ecan) mice, and bon