ライフサイエンスコーパス: cancer tissue

1 eractivated in overproliferating cells (e.g. cancer tissues).

2 in estrogen receptor- positive (ER+) breast cancer tissue.

3 d kinase 4 (PAK4) is amplified in pancreatic cancer tissue.

4 ncer tissue array, we confirmed elevation in cancer tissue.

5 roximates a mitotic clock in both normal and cancer tissue.

6 es with an expression-based mitotic index in cancer tissue.

7 ptor EP2 was overexpressed in human prostate cancer tissue.

8 how Th17 cells are recruited to the cervical cancer tissue.

9 es revealed that Myo9b is upregulated in the cancer tissue.

10 maging of target metabolites in human kidney cancer tissue.

11 gion-specific lipid signatures in colorectal cancer tissue.

12 xis genes are methylated and dysregulated in cancer tissue.

13 ys are frequently found to be coactivated in cancer tissue.

14 tagged biomarker in a thin section of breast cancer tissue.

15 e as surrogate markers of DNA methylation in cancer tissue.

16 l tissue, adult cancer tissue, and pediatric cancer tissue.

17 ltures, in vivo mouse models, and human lung cancer tissue.

18 d based on CEACAM1 expression in the gastric cancer tissue.

19 nces are significantly altered within breast cancer tissue.

20 ive gastric cancer cell model and in gastric cancer tissues.

21 nature" that is distinct from primary breast cancer tissues.

22 ection of DNA samples taken from oesophageal cancer tissues.

23 ir protein and mRNA expression in normal and cancer tissues.

24 r absolute quantitation of AKT peptides from cancer tissues.

25 to efficiently differentiate cancer from non-cancer tissues.

26 st prevalent bacterial species in colorectal cancer tissues.

27 elated with Sox2 expression in human ovarian cancer tissues.

28 n of UBR5 and MYC in human basal-type breast cancer tissues.

29 expression in stromal cells in human gastric cancer tissues.

30 levels correlate with each other in prostate cancer tissues.

31 man metastatic castration-resistant prostate cancer tissues.

32 with CRC and a high somatic mutation rate in cancer tissues.

33 significantly overexpressed in human breast cancer tissues.

34 ased levels of both KLF4 and PRMT5 in breast cancer tissues.

35 l landscape of BRAF and other drivers across cancer tissues.

36 ular location annotations for bioimages from cancer tissues.

37 te for detection of MGA expression in breast cancer tissues.

38 levels of matriptase activation in prostate cancer tissues.

39 ession of Siah2 and AKR1C3 in human prostate cancer tissues.

40 nes were further validated in human prostate cancer tissues.

41 anscript in three of 122 primary endometrial cancer tissues.

42 R: 7.0-209.2; P = 0.001) relative to the non-cancer tissues.

43 lectively and significantly higher in breast cancer tissues.

44 up-regulated and co-localized in human renal cancer tissues.

45 nsistent dysregulated expression in multiple cancer tissues.

46 was then applied to MALDI-IMS of human lung cancer tissues.

47 llular carcinoma, breast cancer, and ovarian cancer tissues.

48 cans from human laryngeal cancer and ovarian cancer tissues.

49 anges of protein distribution from normal to cancer tissues.

50 ventional LAMP and nested PCR in 50 cervical cancer tissues.

51 iated with the nucleus of malignant prostate cancer tissues.

52 n can distinguish between normal and bladder cancer tissues.

53 highly expressed in human epithelial ovarian cancer tissues.

54 reduced in breast cancer cells and clinical cancer tissues.

55 wth, even though it was expressed by ovarian cancer tissues.

56 st cancer cell lines as well as human breast cancer tissues.

57 ate, and N-acetylaspartate on human prostate cancer tissues.

58 to impute lncRNA transcriptome of normal and cancer tissues.

59 natures and tumor mutational burden (TMB) in cancer tissues.

60 e novo biosynthesis are not defined in human cancer tissues.

61 isualization of EVs in a range of normal and cancer tissues.

62 ative analysis of clonal dynamics in grafted cancer tissues.

63 D co-expression was observed in human breast cancer tissues.

64 omain-containing receptor 2 (DDR2) in breast cancer tissues.

65 with beta-catenin and PKM2 levels in breast cancer tissues.

66 EG3 expression has been reported in multiple cancer tissues.

67 res regarding the intrinsic heterogeneity of cancer tissues.

68 ,714) and expressed by nearly half of breast cancers tissues.

69 In contrast, in matched cancer tissue, 5' tRH abundance was reduced, and relativ

70 ted in large-scale human genetic studies and cancer tissues, all of which occur close to two regulato

71 sequential Ga(3+)-Fe(3+)-IMAC to human lung cancer tissue allowed the identification of 2560 unique

72 h9 and Th17 cells was detected in human lung cancer tissue and correlated with poor survival.

73 obtain chemical images of cross sections of cancer tissue and hair samples sandwiched between infrar

74 mma was produced in HEp-2 cells derived from cancer tissue and in HEK293T cells derived from normal t

75 ant correlation with Met expression in colon cancer tissue and is highly prognostic for occurrence of

76 mined DDX21 protein expression in colorectal cancer tissue and its association with patient clinical

77 have determined its distribution in prostate cancer tissue and its role in prostate tumorigenesis usi

78 of histone modifications in primary bladder cancer tissue and provides an important resource for the

79 In total, 20 cases of primary breast cancer tissue and resected BrM (10 estrogen receptor [ER

80 hort harbored patient-matched primary breast cancer tissue and resected BrM.

81 This was clinically tested in prostate cancer tissue and shown that cytoplasmic PML and CRM1 co

82 nalase expression is increased in pancreatic cancer tissue and that it functions as a growth factor.

83 st highly suppressed gene in both colorectal cancer tissue and the L1-NF-kappaB pathway.

84 her characterize metabolic changes in breast cancer tissue and the tumor microenvironment.

85 st abundantly expressed galectin in prostate cancer tissue and was markedly upregulated during diseas

86 ng known proportions of ovarian and prostate cancer tissue and yeast, or control HEK293T cells.

87 iii) shareable regulatory information across cancer tissues and (iv) associations among multi-phospho

88 equence in the TGFBR2 gene of 32 MSI-H colon cancer tissues and 6 cell lines (HCT116, LS180, LS411N,

89 Utilizing resected human lung cancer tissues and a p21CIP1/WAF1-deficient, K-rasG12D-e

90 fibroblasts (CAFs) coexist within pancreatic cancer tissues and can both promote and restrain disease

91 tein expression are down-regulated in breast cancer tissues and cell lines compared with adjacent nor

92 Studies of both human liver cancer tissues and cell lines uncovered that DDD cleavag

93 alyzed proteomic and pharmacogenomic data in cancer tissues and cell lines using a global statistical

94 t Rig-G was frequently downregulated in lung cancer tissues and cell lines, and correlated with poor

95 ZNF367 was upregulated in breast cancer tissues and cell lines, and significantly correla

96 rect measurement of this biomarker in breast cancer tissues and cells might serve as a prognosis biom

97 dicted E-cadherin-targeting miRNAs in breast cancer tissues and cells showed that miR-221 was abundan

98 ger RNA in human pancreatitis and pancreatic cancer tissues and cells with normal pancreas, and measu

99 e cancer (CRPC) cell lines, primary prostate cancer tissues and circulating tumor cells (CTCs) to inv

100 hat PPARalpha is overexpressed in pancreatic cancer tissues and clofibrate-mediated PPARalpha activat

101 ymerase chain reaction in 915 primary breast cancer tissues and correlated with known clinicopatholog

102 e, we report that TOP1MT is overexpressed in cancer tissues and demonstrate that TOP1MT deficiency at

103 arcinogenesis by analyzing its expression in cancer tissues and different models of neoplastic transf

104 rrelated with TRIB2 expression in both liver cancer tissues and established liver cancer cell lines.

105 of CagA on HER-2 expression in human gastric cancer tissues and gastric mucosal tissues of H. pylori

106 s also found to be altered in human cervical cancer tissues and high expression of this lncRNA was as

107 signal was increased significantly in breast cancer tissues and highly correlated with ex vivo expres

108 , which was later corroborated in human lung cancer tissues and immortalized lung cancer cell lines v

109 PRMT1 expression is increased in pancreatic cancer tissues and is associated with higher tumor grade

110 etection of KRAS G12V mutation in colorectal cancer tissues and is presented herein.

111 that PIPKIgamma is highly expressed in lung cancer tissues and its expression level is critical for

112 e tissue, was reduced in both human prostate cancer tissues and LNCaP-B cells.

113 C5 was significantly hypermethylated in lung cancer tissues and lung cancer cell lines compared with

114 tient survival in examinations of colorectal cancer tissues and paired normal colorectal mucosa tissu

115 , POSTN was highly enriched in the stroma of cancer tissues and produced mainly by CAFs.

116 In lung cancer tissues and single cells, p66(Shc) expression inv

117 We performed RNA-Seq on T1 and T2 bladder cancer tissues and used publicly available bladder cance

118 fy epialleles that differ between normal and cancer tissue, and define a measure of global epigenetic

119 l for identifying clusters of cancer and non-cancer tissue, and identifying a cancer CpG island hyper

120 ets consisting of adult normal tissue, adult cancer tissue, and pediatric cancer tissue.

121 rmal colorectal tissue is subtle compared to cancer tissue, and the simultaneous FP/HW Raman endoscop

122 c networks corresponding to four healthy and cancer tissues, and analysed the healthy-cancer transiti

123 ion was detected in 17 (56.7%) of 30 gastric cancer tissues, and eight (47%) of them showed HER-2 DNA

124 was decreased in the majority of human liver cancer tissues, and its reduced expression was significa

125 n both cancer cell lines and advanced breast cancer tissues, and the levels of TRIM28 and TWIST1 are

126 ariable, 2) most somatic L1 insertions in GI cancer tissues are absent from normal tissues, and 3) un

127 Increases in stem cell population in breast cancer tissues are associated with tumor recurrence and

128 wed that higher levels of sGCbeta1 in breast cancer tissues are correlated with greater survival prob

129 of cancer, but iron regulatory mechanisms in cancer tissues are largely unknown.

130 carcinoma (NSCLC), especially in those whose cancer tissues are unavailable or in those that have acq

131 immunohistochemical analysis of 162 prostate cancer tissue array samples representing a range of Glea

132 Using a human breast cancer tissue array, we confirmed elevation in cancer ti

133 In human prostate cancer tissue arrays, Gal-1 expression correlated with t

134 f amplified FRET to determine pAkt status in cancer tissues as candidate biomarker for the identifica

135 p54(nrb)/Nono is highly expressed in breast cancer tissues as compared with the adjacent normal tiss

136 TS but negatively with YAP/TAZ in pancreatic cancer tissues as well as pancreatic and breast cancer c

137 positive immunostaining for nuclear STAT3 in cancer tissues, as compared to non-cancer histologically

138 tantly, CDK20 is overexpressed in human lung cancer tissues, as determined by immunostaining.

139 In the ex vivo breast cancer tissue assay, WISP1 promoted the growth of cancer

140 itive and resistant cells and between breast cancer tissues (available from The Cancer Genome Atlas p

141 d is developed for predicting the stage of a cancer tissue based on the consistency level between the

142 protein in skin, normal cervix, and cervical cancer tissues, but not in larynx.

143 oxin (Prx) enzymes are overexpressed in most cancer tissues, but their specific signaling role in can

144 on of an RNA cancer biomarker on fresh human cancer tissues by topical application (spraying) of a mo

145 in human chronic pancreatitis and pancreatic cancer tissues, Cad-11 expression was significantly incr

146 how variations in the cellular properties of cancer tissue can influence cancer biomarker ion images.

147 Remarkably, human lung cancer tissues can only be distinguished from adjacent n

148 SCD1 was highly expressed in ovarian cancer tissue, cell lines, and a genetic model of ovaria

149 are colocalized in plasma membrane of breast cancer tissue cells and breast cancer cell lines BT474 a

150 have been charted between normal and breast cancer tissues, changes in higher-order chromatin organi

151 ed cell-free DNA and eight frozen metastatic cancer tissues collected during rapid autopsy was perfor

152 and CYSLTR2 gene expression was decreased in cancer tissue compared to control at 0.26-fold and 0.23-

153 rotein levels are decreased in human gastric cancer tissue compared with surrounding normal gastric t

154 ere highly overexpressed in about 36% of 136 cancer tissues compared to adjacent tissues, in which FE

155 mRNA isoform, SIGIRR(DeltaE8), in colorectal cancer tissues compared to paired nontumor tissues.

156 ave significantly lower expression in breast cancer tissues compared to paired normal breast tissue.

157 is upregulated in tamoxifen-resistant breast cancer tissues compared to their primary counterparts.

158 els were significantly downregulated in lung cancer tissues compared with adjacent non-cancerous tiss

159 promoter is highly methylated in pancreatic cancer tissues compared with non-cancerous tissues.

160 els of PPARD were increased in human gastric cancer tissues, compared with nontumor tissues, and asso

161 RSK1 and MSK2 were overexpressed in colon cancer tissues confirmed by western blot and IHC.

162 Cancer tissue contained significantly increased levels o

163 data from a single time point of normal and cancer tissues contains information on single-cell divis

164 macrophage-associated signatures from large cancer tissue datasets.

165 cer samples, our method ranked first in both cancer tissues, demonstrating its robustness and general

166 Remarkably, pDCs isolated from human colon cancer tissues displayed a strong surface expression of

167 e rapid intraoperative detection of residual cancer tissue during breast-conserving surgery.

168 e abundance of molecular profiling of breast cancer tissues entailed active research on molecular mar

169 in real time for accurate identification of cancer tissues, especially these without any obvious str

170 cally classify sample regions in human renal cancer tissue ex-vivo into tumor or benign tissue based

171 hat the d-spacing of the EMT positive breast cancer tissue (FFPE (dewaxed)) is within the range 64.5-

172 sue Bank (BCCTB), a vital resource of breast cancer tissue for researchers to support and promote cut

173 r and pSmad2 immunohistochemically in breast cancer tissue from 1,045 patients in the Shanghai Breast

174 the activation of JNK signalling in ovarian cancer tissue from human patients, suggesting that it ma

175 Here, we show that in cancer tissue from primary GIST patients as well as in c

176 of CD44v6 messenger RNA (mRNA) in pancreatic cancer tissues from 136 patients.

177 was performed on adjacent normal and breast cancer tissues from 96 premenopausal women with known cl

178 and overexpression of BCL-2 were observed in cancer tissues from metabolic organs.

179 s in APC(min/+) mice and in human colorectal cancer tissues, functioned redundantly to promote the Wn

180 Cancer tissues harbor thousands of mutations, and a give

181 Because up-regulation of Siglec ligands in cancer tissue has been observed, the characterization of

182 lf-renewal and tumor growth in heterogeneous cancer tissues, has stimulated interests in developing n

183 However, most DW-MRI studies of cancer tissues have relied on simplistic mathematical mo

184 A genomic analysis of colorectal cancer tissues identified a somatic mutation (P301S) in

185 , the detection and localization of prostate cancer tissue in patients with primary or recurrent dise

186 nstrated a high prediction power to diagnose cancer tissue in real time during routine surgical proce

187 e unrestricted multiple 'omics data for each cancer tissue in The Cancer Genome Atlas as ready-to-ana

188 um (F.) nucleatum was abundant in colorectal cancer tissues in patients with recurrence post chemothe

189 maging and therapeutic agents selectively to cancer tissues in vivo.

190 ly, immunohistochemical analysis of prostate cancer tissue indicated that nuclear localization of fat

191 mRNA compared with other genotypes in breast cancer tissues, indicating that rs17506395 may be a func

192 Modulation of immunologic interactions in cancer tissue is a promising therapeutic strategy.

193 Recently it has been observed that cancer tissue is characterised by an increased variabili

194 redominant 3'-terminal 2'Ome of miR-21-5p in cancer tissue is confirmed by qRT-PCR and northern blot

195 Cancer tissue is stiffer than its normal counterpart.

196 release of potent anticancer products within cancer tissues is a promising approach in chemotherapy.

197 ing formalin-fixed, paraffin-embedded (FFPE) cancer tissues is becoming the standard for identifying

198 uencing profiles from bulk DNA sequencing of cancer tissues is fueling the application of evolutionar

199 Furthermore, PRMT5 expression in prostate cancer tissues is significantly higher than that in beni

200 Although the stroma of cancer tissues is the main source of POSTN, it is still

201 ich has been detected in almost all kinds of cancer tissues, is considered as an important tumor mark

202 ibutors to hyaluronan degradation in bladder cancer tissue, leading to accumulation of inflammatory a

203 eakdown of extracellular HA in human bladder cancer tissue, leading to the accumulation of small HA f

204 One mechanism by which cancer tissues limit the host immune response is via upr

205 n patient tissues was examined using a multi-cancer tissue microarray approach (TMA).

206 by immunohistochemistry in a unique ovarian cancer tissue microarray constructed with paired primary

207 Furthermore, analysis of a human lung cancer tissue microarray revealed a significant, positiv

208 Analysis of a comprehensive breast cancer tissue microarray revealed a tight correlation be

209 In an ovarian cancer tissue microarray, TR3 protein expression was ele

210 Here, using human lung cancer tissue microarrays and fresh frozen tissues, we f

211 The analysis of prostate cancer tissue microarrays revealed that tumors with redu

212 Immunohistochemical analysis of prostate cancer tissue microarrays showed significant P-AR S213 e

213 sification of breast cancer cells and breast cancer tissue microarrays with this system correlated wi

214 Analysis of human gastric cancer tissue microarrays, showed high levels of DARPP-3

215 Using data mining and human breast cancer tissue microarrays, we found that Ctr9, the key s

216 other cancer cell lines and human pancreatic cancer tissue microarrays.

217 y immunohistochemical staining on colorectal cancer tissue microarrays.

218 We investigated normal and breast cancer tissue microbiota from NHB and non-Hispanic White

219 pes, whereas organoids derived from prostate cancer tissue mimic the histology of the tumor.

220 Recent genomic studies on cancer tissues obtained during rapid autopsy have provid

221 Cancer tissues of 139 patients with CRLM were used for N

222 mmune checkpoint blockade, regardless of the cancers' tissue of origin.

223 e cancer metabolic phenotypes are defined by cancer tissue-of-origin and environment and that these f

224 oblastoma cells (U87), which formed 3D brain cancer tissues on the chip, and used the GBM chip to per

225 for additional surgeries to excise unremoved cancer tissue or alternatively in the removal of excess

226 ed to a number of effects off their intended cancer tissue or molecular targets.

227 gical specimens confirmed that most cervical cancer tissues overexpressed STIM1 and Orai1, accompanie

228 that PER2 was significantly up-regulated in cancer tissues (p < 0.005).

229 ataset comprising 155,165 cells from patient cancer tissues, patient-derived breast cancer xenografts

230 in vivo observation that different prostate cancer tissues polarize macrophages with distinct gene-e

231 Moreover, our findings for cancer tissues presenting concomitant cytidine deaminase

232 DNA methylation profiling performed on cancer tissues prior to chemo/radiotherapy identified on

233 s and differentiation, with implications for cancer, tissue regeneration, and inflammation.

234 ession analysis in human metastatic prostate cancer tissue revealed that beta1 was markedly upregulat

235 The genotyping of 805 breast cancer tissues revealed a genotypic and allelic frequenc

236 urity', or the percentage of cancer cells in cancer tissue sample, will bias the clustering results i

237 tative histological data of human colorectal cancer tissue samples (n = 20) including tumor cells, ly

238 analysis of >27,000 CpG sites in endometrial cancer tissue samples (n = 64) and control samples (n =

239 is associated with caveolin-1 in pancreatic cancer tissue samples and cell lines, and predicts the m

240 erexpression of miR-135b-5p in human gastric cancer tissue samples compared with normal tissue sample

241 unohistochemical staining of lung and breast cancer tissue samples demonstrated that increased S6K2 e

242 methylation are recapitulated in endometrial cancer tissue samples obtained from patients treated wit

243 itor the differences in PSA concentration in cancer tissue samples which holds great promise in clini

244 detected with the proposed immunosensor for cancer tissue samples which holds great promise in clini

245 or source of HB-EGF production in pancreatic cancer tissue samples, and that macrophages are present

246 d proteomics, which allow analysis of breast cancer tissue samples, leading to the first large-scale

247 tween miR-218 levels and Slug/ZEB2 levels in cancer tissue samples.

248 8 cancer proteins was performed on a breast cancer tissue section to illustrate the potential of thi

249 ould be identified from a FFPE mouse ovarian cancer tissue section.

250 s well as MS imaging of N-glycans from human cancer tissue sections.

251 ate utilization in purine synthesis in human cancer tissues should be considered when targeting one-c

252 Our recent data suggest that human breast cancer tissues show a redox imbalance (reducing) compare

253 A parallel analysis of colorectal cancer tissues showed differential expression of 83 miRN

254 Analysis of prostate cancer tissues showed that the presence of a TMPRSS2-ERG

255 Analyses of human breast cancer tissues showed that ZNF24 and VEGF levels were in

256 s frequently found in tumour cells and human cancer tissues showing high levels of phospho-FADD(Ser19

257 further confirmed from data in human breast cancer tissues showing that CNNM3 levels correlate posit

258 at elevated CDK11(p110) expression in breast cancer tissues significantly correlated with poor differ

259 ch by imaging Papanicolaou smears and breast cancer tissue slides over a large field-of-view of ~20 m

260 we developed a library of 76 healthy and 20 cancer tissue-specific reconstructions.

261 Examination of human prostate cancer tissue specimens shows a reduction of LZTS2 prote

262 In human colorectal cancer tissue specimens, a strong correlation of FOXM1 a

263 presence of iMPs was confirmed in colorectal cancer tissue specimens.

264 e-containing peptides identified in prostate cancer tissue studies carried out in our group were dete

265 demonstrated that IL-35 is produced in human cancer tissues, such as large B cell lymphoma, nasophary

266 SR-B1 is up-regulated in prostate cancer tissue, suggesting a possible role of this recept

267 higher expression of PPARalpha in pancreatic cancer tissues than in tumor-adjacent tissues and that t

268 lncRNA GAS5 had lower expression in stomach cancer tissues than the normal counterparts.

269 igher human FcRn (hFcRn) expression in human cancer tissue that provides the mechanistic basis for ex

270 nsive metabolic phenotyping method in breast cancer tissue that uses desorption electrospray ionizati

271 interleukin-22 (IL-22) in patient colorectal cancer tissues that was produced predominantly by CD4(+)

272 activity of a dual AI/SERM, while in breast cancer tissue the antiestrogenic SERM activity of a dual

273 In human breast cancer tissues, the levels of p54(nrb) and SREBP-1a prot

274 Moreover, in colorectal cancer tissues, the Sin1 protein but not mRNA was signif

275 tween cancer cell lines and fresh human lung cancer tissues; the latter preferred glucose to exogenou

276 thod to human cancer cell lines and biopsied cancer tissue, thereby illustrating its efficiency, repr

277 d specific ASE characteristics in normal and cancer tissues, thus indicating that cisASE has potentia

278 enabled the cellular architecture of breast cancer tissue to be characterized on the basis of cellul

279 f of principle study showing the response of cancer tissue to irradiation ex vivo in a bespoke system

280 In cancer tissues, TTT-RUVBL complex mRNAs were elevated an

281 Eight out of ten different human cancer tissue types screened for hFcRn expression by imm

282 tative SPECT imaging of preneoplastic breast cancer tissue using (111)In-labeled cCPE.

283 ia lesion (CIN2/3) samples and five cervical cancer tissues using rolling-circle amplification of tot

284 h much higher concentrations in secretome of cancer tissues vs. normal ones.

285 DOT1L expression and H3K79me2 in colorectal cancer tissues was a predictor of poor patient survival.

286 Most SIGIRR detected in human colon cancer tissues was cytoplasmic, whereas in nontumor tiss

287 188 normal breast and 1247 malignant breast cancer tissues, we observed the loss of KLLN in multiple

288 ormalin-fixed paraffin-embedded (FFPE) colon cancer tissues were collected from 53 stage II and 54 (5

289 High TERT levels in cancer tissues were independently associated with worse

290 tudy examines HER2 testing of primary breast cancer tissue when performed with immunohistochemistry (

291 RBP2 was overexpressed in human lung cancer tissues where its depletion impaired cell prolife

292 s commonly upregulated in primary human lung cancer tissues where its expression level correlates wit

293 expressed in primary and metastatic prostate cancer tissues, where it functions as an autocrine growt

294 R2+) but not in HER2-negative (HER2-) breast cancer tissues, whereas both HER2+ and HER2- tumors expr

295 1185-1 was downregulated in human colorectal cancer tissues, whereas expression of CD24 was increased

296 ting PIP2, is positively expressed in breast cancer tissues, which correlates intimately with the pro

297 re substantially decreased in human prostate cancer tissues, which positively correlated with disease

298 n of this pathway was detected in human lung cancer tissues with concomitant downregulation of BAMBI,

299 to differentiate between healthy and breast cancer tissues with high signal/noise ratios.

300 Using primary colorectal cancer tissues, xenograft models, and MAPK reporter cons