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1 cytosis of C. albicans by these cells, while candidal adherence to chelator-treated endothelial cells
2 B1 did not produce any detectable effects on candidal adherence to human endothelial or epithelial ce
3 irect fungicidal activity, and inhibition of candidal adherence.
4 ation with the recombinant N terminus of the candidal adhesin Als3p (rAls3p-N) protects mice from let
5 uvant plus the recombinant N-terminus of the candidal adhesin, Als3p (rAls3p-N), protects mice from d
6 hese results indicate that ALS1 may encode a candidal adhesin.
7 rotein of approximately 185 kDa, and inhibit candidal adhesion to human epithelium.
8 ccine based on the recombinant N-terminus of candidal Als3p (rAls3p-N) in preparation for future clin
9                                              Candidal Als3p also is structurally similar to the micro
10 s to both PPD and candida in TB patients and candidal antigen in contacts.
11  to PPD, partially restored blastogenesis to candidal antigen, and significantly increased PPD-stimul
12 uggest that IgG responses against a panel of candidal antigens might represent an accurate and early
13 osis (TB) patients to both mycobacterial and candidal antigens were suppressed by approximately 50% w
14 lated from crustacean exoskeletons, inhibits candidal biofilm formation in vivo.
15 o studies have addressed the effect of LE on candidal biofilm production.
16 nation demonstrated that chitosan penetrates candidal biofilms and damages fungal cells.
17 nd deletion of one of them, SOD5, eradicated candidal biofilms on venous catheters in a rodent model.
18 om HIV-infected patients with thrush against candidal cells grown in vitro and screened a C. albicans
19 gesting that this cationic peptide may enter candidal cells through native yeast polyamine transporte
20 ransporters used by Hst 5 for its entry into candidal cells.
21 ilable data on the efficacy of probiotics in candidal colonization of host surfaces.
22  spectrometry (PCR/ESI-MS) for bacterial and candidal detection using 83 formalin-fixed paraffin-embe
23                                          The candidal DNA fragment within this adherent/flocculent or
24  were recovered by quantitative culture, and candidal DNA was detectable by PCR.
25                    In order to determine the candidal effector(s) mediating enhanced virulence, an un
26 o peptide fragments obtained from a purified candidal enzyme displaying phospholipase activity.
27 s study, we investigated the effect of LE on candidal germination and its ability to form biofilm on
28           Moreover, Ca37 was able to inhibit candidal growth following 18 h incubation time and reduc
29                                              Candidal Hsp70 proteins are classical chaperone proteins
30                                              Candidal hyphae were present in the superficial layers o
31                                              Candidal induction of ICAM-1 expression was independent
32                      We compared the rate of candidal infection in 78 patients with New York Heart As
33     In a time-to-event analysis, the risk of candidal infection in patients receiving fluconazole was
34    Denture-associated stomatitis is a common candidal infection that may give rise to painful oral sy
35 implantation of LVAD, the risk of developing candidal infection was 28% in LVAD recipients, compared
36 a persistent protection against disseminated candidal infections and candidiasis-related death, resul
37 OPC and indicate the feasibility of treating candidal infections by targeting the host cell receptors
38 icans accounted for 51.5, 47.8, and 36.5% of candidal infections in North America, Europe, and Latin
39                                              Candidal infections often develop because of immune supp
40 age, who experienced recurrent bacterial and candidal infections with bronchiectasis, growth delay, a
41  exploration to prevent systemic and mucosal candidal infections.
42 ity, are unable to resist staphylococcal and candidal infections.
43 y ally in the battle against chronic mucosal candidal infections.
44  defense against hematogenously disseminated candidal infections.
45 eir use as long-term prophylactic agents for candidal infections.
46 rferon (IFN-gamma) protects these cells from candidal injury in vitro.
47                   These results suggest that candidal injury of endothelial cells is independent of t
48 ments demonstrated that maximal reduction in candidal injury of endothelial cells occurred after the
49 nor membrane-permeative antioxidants reduced candidal injury of endothelial cells.
50 hroline and deferoxamine protected them from candidal injury, even though the organisms germinated an
51 ch IFN-gamma protects endothelial cells from candidal injury.
52 l of this study was to determine the role of candidal mannan-specific human IgG antibodies in complem
53  in 183 (44.5%), peptic ulcer in 43 (10.5%), candidal oesophagitis in 38 (9.3%), tumour in 6 (1.5%),
54  released ATP in response to Hst 5 activates candidal P2X(7)-like receptors to cause cell death.
55      Another gene, LPD1, for which a role in candidal pathogenesis is unknown, encodes a protein homo
56 nfected hosts might advance understanding of candidal pathogenesis, but is impractical using existing
57 pecies-level identification of bacterial and candidal pathogens.
58                   Comparison of the putative candidal phospholipase with those of other proteins in d
59                     These studies identify a candidal protein that displays superantigen-like activit
60 Oxododecanoyl)-L-homoserine lactone, induces candidal resistance to fluconazole by reversing the anti
61 -2 and -3 were more effective at killing the candidal species at low micromolar concentrations than w
62 se of fluconazole alters the distribution of candidal species seen in the SICU and impacts measures o
63  to blastospores and germ tubes, recognize a candidal surface protein of approximately 185 kDa, and i
64 ut not Int1, suggesting that heparin changes candidal surface structures and may modify some antigens
65                                              Candidal susceptibility in patients with APECED syndrome
66 ctobacillus acidophilus in the prevention of candidal vaginitis.
67 bility to form filaments may be critical for candidal virulence by enabling the fungus to induce cell
68                                 LE-inducible candidal virulence determinants, such as germination and
69 zoles for empirical therapy of uncomplicated candidal vulvovaginitis.