コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 le with that observed previously with potent cannabinoid receptor agonists.
2 esent study demonstrates that the endogenous cannabinoid receptor agonists 2-arachidonoylglycerol (2-
3 In Caenorhabditis elegans, the endogenous cannabinoid receptor agonist, 2-arachidonoylglycerol (2-
4 attenuated the inhibition produced by a full cannabinoid receptor agonist, (+)-[2, 3-dihydro-5-methyl
5 of several compounds as potent and selective cannabinoid receptor agonists (20, hCB(2)K(i) = 2.5 nM,
9 y examined whether systemically administered cannabinoid receptor agonists and a cannabinoid receptor
10 assessments of the physiological actions of cannabinoid receptor agonists and antagonists on adult h
11 m, 2) summarize animal models of exposure to cannabinoid receptor agonists and antagonists, 3) review
14 that WIN-55,212-2 or other non-habit-forming cannabinoid receptor agonists could be developed as nove
18 Testing peanut butter, we found that the cannabinoid receptor agonist CP55940 (0.5 mg/kg, IP) low
22 n the absence of exogenous agonists, and the cannabinoid receptor agonist desacetyllevonantradol fail
27 ed the effects and mechanism(s) of action of cannabinoid receptor agonists, including Delta9-THC, on
28 t not the undesirable side effects of direct cannabinoid receptor agonists, indicating that FAAH may
29 ndesirable side effects observed with direct cannabinoid receptor agonists, indicating that FAAH may
32 pies, such as the combined use of opioid and cannabinoid receptor agonists or selective activation of
33 acute administration of the potent synthetic cannabinoid receptor agonist R-(+)-WIN 55,212-2 (WIN; 5
35 rocannabinol (Delta(9)-THC) or the synthetic cannabinoid receptor agonist (R)-(+)-2,3-dihydro-5-methy
36 desensitized after prolonged exposure to the cannabinoid receptor agonist (R)-(+)-[2,3-dihydro-5-meth
40 The rapid proliferation of new synthetic cannabinoid receptor agonists (SCRAs) has initiated cons
41 trips, thought to contain opioids, synthetic cannabinoid receptor agonists (SCRAs), and other novel s
45 ion of central nervous system (CNS)-excluded cannabinoid receptor agonists to test the hypothesis tha
47 hibition produced by Win55212-2, a synthetic cannabinoid receptor agonist, was stereoselective and wa
48 ell EPSCs were unimpaired in the presence of cannabinoid receptor agonist when the postsynaptic membr
49 ffect were observed after treatment with the cannabinoid receptor agonist WIN 55,212-2 (0.2 mg kg(-1)
50 ffect were observed after treatment with the cannabinoid receptor agonist WIN 55,212-2 (0.2 mg kg-1 i
51 We used N1E-115 neuroblastoma cells and the cannabinoid receptor agonist WIN 55,212-2 (WIN) to exami
53 effects of close-arterial injections of the cannabinoid receptor agonist WIN 55,212-2 (WIN2) on uter
54 mate release is mimicked and occluded by the cannabinoid receptor agonist WIN 55,212-2, and is abolis
58 elf-administration (SA), using the synthetic cannabinoid receptor agonist WIN55,212-2 (WIN), in order
59 arboxamide] and mimicked and occluded by the cannabinoid receptor agonist WIN55,212-2 [(beta)-(+)-[2,
60 bolished by the exogenous application of the cannabinoid receptor agonist WIN55,212-2 [R-(+)-(2,3-dih
62 n of the prototypical opioid morphine or the cannabinoid receptor agonist WIN55,212-2 produced dose-d