戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 le with that observed previously with potent cannabinoid receptor agonists.
2 esent study demonstrates that the endogenous cannabinoid receptor agonists 2-arachidonoylglycerol (2-
3    In Caenorhabditis elegans, the endogenous cannabinoid receptor agonist, 2-arachidonoylglycerol (2-
4 attenuated the inhibition produced by a full cannabinoid receptor agonist, (+)-[2, 3-dihydro-5-methyl
5 of several compounds as potent and selective cannabinoid receptor agonists (20, hCB(2)K(i) = 2.5 nM,
6                                      Because cannabinoid receptor agonists act presynaptically to inh
7                                              Cannabinoid receptor agonists act presynaptically to inh
8                               The endogenous cannabinoid receptor agonist anandamide is present in ce
9 y examined whether systemically administered cannabinoid receptor agonists and a cannabinoid receptor
10  assessments of the physiological actions of cannabinoid receptor agonists and antagonists on adult h
11 m, 2) summarize animal models of exposure to cannabinoid receptor agonists and antagonists, 3) review
12          We designed AM1241, a selective CB2 cannabinoid receptor agonist, and used it to test the hy
13                                     Notably, cannabinoid receptor agonists as well as inhibitors of e
14 that WIN-55,212-2 or other non-habit-forming cannabinoid receptor agonists could be developed as nove
15 esia was markedly attenuated by morphine and cannabinoid receptor agonist CP 55940.
16                                 Finally, the cannabinoid receptor agonists, CP55,942 and WIN55,212-2,
17      Treatment with THC (4 mg/kg, IP) or the cannabinoid receptor agonist CP55940 (0.5 mg/kg) reduced
18     Testing peanut butter, we found that the cannabinoid receptor agonist CP55940 (0.5 mg/kg, IP) low
19          We found that administration of the cannabinoid receptor agonist CP55940 (2-[(1S,2R,5S)-5-hy
20                       Neither baclofen nor a cannabinoid receptor agonist, CP55940, affected all bout
21                                          The cannabinoid receptor agonist, Delta(9)-tetrahydrocannabi
22 n the absence of exogenous agonists, and the cannabinoid receptor agonist desacetyllevonantradol fail
23                                    We tested cannabinoid receptor agonists for effects on excitatory
24                                              Cannabinoid receptor agonists have gained attention as p
25                                    Exogenous cannabinoid receptor agonists impair hippocampus-depende
26 olecule is well established as an endogenous cannabinoid receptor agonist in the brain.
27 ed the effects and mechanism(s) of action of cannabinoid receptor agonists, including Delta9-THC, on
28 t not the undesirable side effects of direct cannabinoid receptor agonists, indicating that FAAH may
29 ndesirable side effects observed with direct cannabinoid receptor agonists, indicating that FAAH may
30                                              Cannabinoid receptor agonists induced a dramatic reducti
31                   We examined the effects of cannabinoid receptor agonists on forskolin-induced forma
32 pies, such as the combined use of opioid and cannabinoid receptor agonists or selective activation of
33 acute administration of the potent synthetic cannabinoid receptor agonist R-(+)-WIN 55,212-2 (WIN; 5
34                     Acute application of the cannabinoid receptor agonists R(+)-methanandamide, R(+)-
35 rocannabinol (Delta(9)-THC) or the synthetic cannabinoid receptor agonist (R)-(+)-2,3-dihydro-5-methy
36 desensitized after prolonged exposure to the cannabinoid receptor agonist (R)-(+)-[2,3-dihydro-5-meth
37                             We show that the cannabinoid receptor agonist, R+WIN55,212, ameliorates p
38                               With synthetic cannabinoid receptor agonist (SCRA) use still prevalent
39                                    Synthetic cannabinoid receptor agonists (SCRAs) are an evolving cl
40     The rapid proliferation of new synthetic cannabinoid receptor agonists (SCRAs) has initiated cons
41 trips, thought to contain opioids, synthetic cannabinoid receptor agonists (SCRAs), and other novel s
42                                    Synthetic cannabinoid receptor agonists (SCRAs), termed "Spice" or
43          Based on these data we suggest that cannabinoid receptor agonists should be considered as no
44                                              Cannabinoid receptor agonists significantly diminish pai
45 ion of central nervous system (CNS)-excluded cannabinoid receptor agonists to test the hypothesis tha
46  exposure on the neuroprotection afforded by cannabinoid receptor agonists was also studied.
47 hibition produced by Win55212-2, a synthetic cannabinoid receptor agonist, was stereoselective and wa
48 ell EPSCs were unimpaired in the presence of cannabinoid receptor agonist when the postsynaptic membr
49 ffect were observed after treatment with the cannabinoid receptor agonist WIN 55,212-2 (0.2 mg kg(-1)
50 ffect were observed after treatment with the cannabinoid receptor agonist WIN 55,212-2 (0.2 mg kg-1 i
51  We used N1E-115 neuroblastoma cells and the cannabinoid receptor agonist WIN 55,212-2 (WIN) to exami
52 s and conductance mechanisms affected by the cannabinoid receptor agonist WIN 55,212-2 (WIN-2).
53  effects of close-arterial injections of the cannabinoid receptor agonist WIN 55,212-2 (WIN2) on uter
54 mate release is mimicked and occluded by the cannabinoid receptor agonist WIN 55,212-2, and is abolis
55 rine, nicotine, baclofen, clonidine, and the cannabinoid receptor agonist WIN 55,212.
56               Treatment of neonates with the cannabinoid receptor agonist, WIN 55,212-2 (WIN), reduce
57                           The effects of the cannabinoid receptor agonist Win55,212 on Ca2+ channels
58 elf-administration (SA), using the synthetic cannabinoid receptor agonist WIN55,212-2 (WIN), in order
59 arboxamide] and mimicked and occluded by the cannabinoid receptor agonist WIN55,212-2 [(beta)-(+)-[2,
60 bolished by the exogenous application of the cannabinoid receptor agonist WIN55,212-2 [R-(+)-(2,3-dih
61                                          The cannabinoid receptor agonist WIN55,212-2 also reduced fi
62 n of the prototypical opioid morphine or the cannabinoid receptor agonist WIN55,212-2 produced dose-d
63                            Additionally, the cannabinoid receptor agonist WIN55,212-2 suppressed IPSC
64  of corticoid-receptor antagonist RU-486 and cannabinoid-receptor agonist WIN55,212-2.
65                                          The cannabinoid receptor agonist WIN55212-2 (10-30 ng/side),