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1 atriuretic peptide expression, and sustained capillarization.
2  phosphorylation of Akt and increased muscle capillarization.
3 escence, and whether this effect was lost in capillarization.
4  an important determinant of skeletal muscle capillarization.
5 ed to overall measures of peripheral villous capillarization.
6 cle fiber type distribution, fiber size, and capillarization.
7 differences are associated with increases in capillarization.
8 t role in the maintenance of skeletal muscle capillarization across the life span.
9 hanisms for retinal oxygen supply, including capillarization and acid-induced haemoglobin oxygen unlo
10      The NO-dependent pathway is impaired in capillarization and activation of this pathway downstrea
11 animal models, inhibitory effects of FZHY on capillarization and angiogenesis were further confirmed
12 vestigated the effects of FZHY on sinusoidal capillarization and angiogenesis with mice challenged fo
13 phosphoprotein 1(-/-) mice leads to immature capillarization and blunted arteriolarization.
14 ipheral factors (skeletal muscle fibre type, capillarization and concentration of mitochondrial DNA [
15 combinant Emc10 enhanced infarct border-zone capillarization and exerted a sustained beneficial effec
16  phenotype with increased collateralization, capillarization and improved pericyte coverage.
17 Kupffer cells are responsible for sinusoidal capillarization and perisinusoidal matrix deposition, im
18 le vascular beds, promoting liver sinusoidal capillarization and portal hypertension, ischemic heart
19 ut mice were protected from arsenite-induced capillarization and protein nitration.
20 oximity to liver sinusoidal endothelial cell capillarization and stellate cell activation demonstrate
21 hat a Nox2-based oxidase is required for SEC capillarization and that it may play a central role in v
22 nvestigate if AMPK regulates skeletal muscle capillarization and the angiogenic responses to exercise
23 ationship was observed between type II fiber capillarization and the change in type II-fiber CSA with
24            Wheel running (28 days) increased capillarization and this response was AMPK independent.
25 date the nature of and mechanisms leading to capillarization and to determine how LSECs promote HSC q
26 cise training program for the measurement of capillarization and VEGF mRNA.
27  Vegfa from HS, enhanced infarct border-zone capillarization, and exerted sustained beneficial effect
28 PH patients (n = 4) to study RA hypertrophy, capillarization, and fibrosis.
29  proliferation, metabolic zonation, sinusoid capillarization, and hepatic stellate cell activation we
30 raining program improves muscle performance, capillarization, and mitochondrial content in both asymp
31 meliorated CCl(4) and DMN-induced sinusoidal capillarization, angiogenesis and expression of angiogen
32 correlations between VEGF protein and muscle capillarization are consistent with VEGF being an import
33 ion of both angiogenesis and pathogenic LSEC capillarization, as well as demonstrating a role for S1P
34 mentation and high-throughput, high-accuracy capillarization assessment in basement-membrane-immunost
35 ion model, in both instance segmentation and capillarization assessment.
36 (4) AMPK regulates basal VEGF expression and capillarization, but is not necessary for exercise-induc
37                                              Capillarization, characterized by loss of differentiatio
38 unofluorescence was used to determine muscle capillarization, fiber size, SC content, and activity.
39 es proliferation, contributing to sinusoidal capillarization, impaired endothelial regeneration, and
40           We have proposed the importance of capillarization in skeletal muscle for the satellite cel
41 ce, muscle mitochondrial function, and fiber capillarization in symptomatic and asymptomatic statin u
42  cells (SECs), including SEC defenestration, capillarization, increased junctional PECAM-1 expression
43 bryonic hepatic hemorrhage and microvascular capillarization induced by Smad6 deletion in endothelial
44                         The study shows that capillarization is due to repair of injured LSECs by BM
45                                              Capillarization, lack of liver sinusoidal endothelial ce
46 toration, portal inflammation and sinusoidal capillarization may not regress after viral eradication.
47                          We hypothesize that capillarization of sinusoidal endothelial cells (SEC) is
48                           After MI, however, capillarization of the infarct border zone was impaired
49  that affect liver function such as sinusoid capillarization or loss of metabolic zonation are common
50 ance to fatigue (P = 0.01), and muscle fiber capillarization (P < 0.01), with no differences between
51                                              Capillarization precedes hepatic fibrosis.
52 nd can be interpreted as a component of the "capillarization" process of the hepatic sinusoid.
53 ural demonstration of SEC defenestration and capillarization, quantitative immunofluorescence analysi
54                                Commonly used capillarization-related measurements can be derived from
55 60-2770 accelerated the complete reversal of capillarization (restored differentiation of LSECs) with
56 e processes of sinusoidal defenestration and capillarization that characterize liver fibrosis.
57 onal changes in SEC signaling for sinusoidal capillarization that may be initial events in pathogenic
58 signaling for angiogenesis or liver sinusoid capillarization, the mechanism for initiating these effe
59           Quantitative assessment of cardiac capillarization typically involves double immunostaining
60  increased sinusoidal endothelial cell (SEC) capillarization, vascularization of the peribiliary vasc
61                                              Capillarization was characterized by ectopic basement me
62  marrow (BM) endothelial progenitor cells to capillarization was identified using rats transplanted w
63                          Infarct border zone capillarization was impaired, scar size increased, and c
64                                      Resting capillarization was lower in AMPK DN compared to WT.
65 etraining, at a time point long after muscle capillarization was observed to be similar to pre-traini
66         Conversely, no changes in sinusoidal capillarization were observed after treatment, as assess
67             Prior to fibrosis, LSECs undergo capillarization, which is permissive for HSC activation,
68 ) mice revealed a special form of sinusoidal capillarization, with effacement of endothelial zonation