ライフサイエンスコーパス: capsaicin

1 ain or improve the antioxidant properties of capsaicin.

2 cyanate (AITC; also known as mustard oil) or capsaicin.

3 tive following id histamine, chloroquine, or capsaicin.

4 mpared with the prototypical soluble agonist capsaicin.

5 nced the internalisation of nanoencapsulated capsaicin.

6 ting the redox reaction for the detection of capsaicin.

7 to better understand the redox mechanism of capsaicin.

8 arge cells, most of which did not respond to capsaicin.

9 mal similar to that of the aromatic group of capsaicin.

10 let and mechanisms of binding site access by capsaicin.

11 eal a preferred interfacial localization for capsaicin.

12 .3-fold by the addition of the TRPV1 agonist capsaicin.

13 TRPV1 worms exhibit an aversive response to capsaicin.

14 ed by inhalation of the nociceptor stimulant capsaicin.

15 activated by vanilloid compounds, including capsaicin.

16 e, as were the effects of the application of capsaicin.

17 fibres, 70% of which were also activated by capsaicin.

18 d by sweat-triggered ejection of menthol and capsaicin.

19 y responses to intralaryngeal application of capsaicin (10 microg/ml, 50 microl), a selective stimula

20 increasing rheobase, decreasing responses to capsaicin (2 mum) and reducing action potential discharg

21 d samples at different concentration ranges: capsaicin (28.23-2322.35microg/g), vitexin (2.93-33.46mi

22 epper, garlic and mountain pepper, were K(a, capsaicin) = 3.5206 x 10(-16) mol/L, K(a, allicin) = 5.0

23 was expressed in hADSC, and the TRPV1 ligand capsaicin (5uM) stimulated proliferation, which could be

24 Chili peppers produce capsaicin (a vanilloid) that activates the transient rec

25 ONALE: Heightened cough responses to inhaled capsaicin, a transient receptor potential vanilloid 1 (T

26 Capsaicin, a TRPV1 agonist, can initiate activity in mye

27 The ingestion of food containing capsaicin-a natural component of chili peppers that can

28 e naked mole-rat with the painful substances capsaicin, acid (hydrogen chloride, pH 3.5), and allyl i

29 (2+) dynamics revealed that the TRPV1 ligand capsaicin activated CaCCs.

30 Noradrenaline or clonidine inhibited the capsaicin-activated current by approximately 60%, and th

31 Inhibition of capsaicin-activated current by noradrenaline was mediate

32 Inhibition of the capsaicin-activated current by SKF 81297 was mediated by

33 radrenaline and clonidine were tested on the capsaicin-activated current recorded from acutely dissoc

34 opamine receptor agonists were tested on the capsaicin-activated current recorded from acutely dissoc

35 3, the inhibitory effect of SKF 81297 on the capsaicin-activated current was greatly reduced, suggest

36 he inhibitory effect of noradrenaline on the capsaicin-activated current was greatly reduced, suggest

37 he inhibitory effect of noradrenaline on the capsaicin-activated current was not affected either by b

38 The inhibitory effect of SKF 81297 on the capsaicin-activated current was not affected when the pr

39 receptors) had no significant effect on the capsaicin-activated current.

40 e preassociated in resting live cells, while capsaicin activation induced both the formation of more

41 only validate our current working model for capsaicin activation of TRPV1 but also should help guide

42 exosomal fraction of cultured DRG following capsaicin activation of TRPV1 receptors.

43 elated with responses to the algesic markers capsaicin, AITC and alpha, beta-methylene ATP.

44 ults showed that the activation constants by capsaicin, allicin and sanshool, the active components o

45 of mean force (i.e., free energy profile) of capsaicin along the bilayer normal confirms that it pref

46 Capsaicin also ameliorates cisplatin-induced nephrotoxic

47 Menthol, AITC, and capsaicin also elicited robust calcium responses that di

48 orometry, and we assessed the sensitivity to capsaicin, an agonist of the TRPV1 receptor.

49 The hydrophilic capsaicin analog Cap-EA activated a cTRPV1-A578E mutant,

50 these questions, we designed a pair of novel capsaicin analogs to increase or decrease the ligand siz

51 Capsaicin and AITC increased the numbers of warm-respond

52 and -T469I variants were more responsive to capsaicin and coal fly ash.

53 AT3 expression and also with the content of capsaicin and dihydrocapsacin during fruit development.

54 The Caco-2 cells incorporated capsaicin and dihydrocapsaicin (8.4% and 10.9%, respecti

55 and IBS were used in a mixed linear model of capsaicin and dihydrocapsaicin content and fruit weight

56 tem has been tested for the determination of capsaicin and dihydrocapsaicin from fresh chilli and swe

57 ietary fat increased the bioaccessibility of capsaicin and dihydrocapsaicin in digestions with red pe

58 ocessing on the in vitro bioaccessibility of capsaicin and dihydrocapsaicin was studied in the absenc

59 Capsaicin and four flavonoids were quantified in studied

60 H. armigera, but insignificantly inhibited (capsaicin and gossypol) or induced (nicotine) it in H. a

61 Our BRET study also confirmed that: (1) capsaicin and heat promoted distinct transitions, indepe

62 d varicosities that were also seen following capsaicin and menthol stimulation.

63 tion in palpebral opening and an increase in capsaicin and menthol-evoked responses only in female mi

64 ptive responses after corneal application of capsaicin and menthol.

65 channel activated by painful stimuli such as capsaicin and noxious heat, and enriched in sensory neur

66 mmation, we stimulated lung nociceptors with capsaicin and observed increased neuropeptide release an

67 linositol lipids occupy the binding site for capsaicin and other vanilloid ligands, suggesting a mech

68 dents, and antinociceptive properties in the capsaicin and partial sciatic nerve ligation models in m

69 essfully introduced high-affinity binding of capsaicin and resiniferatoxin to the vanilloid-insensiti

70 The application of both capsaicin and RTX on the L4 nerve significantly reduced

71 r only to capsaicin (COR neurons) or to both capsaicin and the endogenous TRPV1 and CB1 receptor liga

72 ultured DRG neurons also sensitive to TRPV1 (capsaicin) and/or TRPA1 (AITC) agonists.

73 ociception induced by formalin, acetic acid, capsaicin, and cinnamaldehyde.

74 PM8-positive small neurons also responded to capsaicin, and had significantly larger menthol-induced

75 tivated by heat and pungent agents including capsaicin, and has been extensively studied in nocicepti

76 We used concentrations of menthol, capsaicin, and hypertonic saline that evoked comparable

77 lymodal cellular sensor for heat, acidic pH, capsaicin, and other noxious stimuli.

78 The capsaicin- and heat-activated transient receptor potenti

79 Capsaicin application for 48 hours induces cutaneous fib

80 tive primary sensory neurons by burn injury, capsaicin application or sustained electrical activation

81 Surprisingly, capsaicin application to the isolated L6 dorsal root gan

82 sensory neuron-derived exosomes released by capsaicin are readily phagocytosed by macrophages in whi

83 Using capsaicin as a sensitizing stimulus, we show that behavi

84 tion of TRPV1 abolished all these effects of capsaicin as well as vasoconstriction triggered by lysop

85 ult in increased cough reflex sensitivity to capsaicin associated with an increase in 24-hour spontan

86 We used this capsaicin axotomy technique to examine the effects of ex

87 nd 30-day cutaneous regeneration after thigh capsaicin axotomy were compared for participants with ty

88 but surmountable barrier to the flipping of capsaicin between opposing leaflets of the bilayer.

89 er confirm that Y511, known to be crucial to capsaicin binding, has a distribution along the bilayer

90 s in mice by interacting with the vanilloid (capsaicin)-binding pocket and promoting the stabilizatio

91 onal quantification revealed the location of capsaicin-binding site and critical residues mediating l

92 We found that menthol and capsaicin both caused a significant reduction in corneal

93 with COPD had heightened cough responses to capsaicin but reduced responses to prostaglandin E2 comp

94 obility decreased upon channel activation by capsaicin, but only in the presence of extracellular Ca(

95 Capsaicin (CAP) activates transient receptor potential v

96 TRPV1 agonists including capsaicin (CAP) and the eCBs anandamide and N-arachidono

97 HA diminishes heat, pH and capsaicin (CAP) responses, thus reducing the opening pro

98 a coarse characterization of the location of capsaicin (CAPS) and resiniferatoxin (RTX).

99 Consistent with these findings, capsaicin caused a greater number of coughs in CS-expose

100 Our data indicate that capsaicin causes a transient STAT1 activation via TRPV1

101 cacy and potency in preclinical and clinical capsaicin challenge studies; despite this improved pharm

102 Full-dose capsaicin challenge was performed at screening to determ

103 on neurones showed no changes in response to capsaicin, cholecystokinin and potassium chloride in TNX

104 -mediated pain perception leading to altered capsaicin consumption and sensitivity to heat.

105 of diverse accessions are in agreement with capsaicin content (CA) and fruit weight (FW) classificat

106 of catalase and peroxidase, chlorophyll and capsaicin content gradually decreased for all the treate

107 is considered as one of the major markers of capsaicin content in pepper fruits.

108 acologically, neurons respond either only to capsaicin (COR neurons) or to both capsaicin and the end

109 The pungent molecule capsaicin (CP) has a similar effect as AEA; however, CP

110 ocomotor adaptation task with pain on Day 1 (capsaicin cream around the ankle), while the task was pe

111 Surgical removal of the periosteum, capsaicin denervation of sensory nerves or knockdown in

112 glia, which activated even more neurons than capsaicin, did not produce myenteric activation or contr

113 TAC1 neuropeptide precursor and decreased in capsaicin-diet fed mice.

114 nornordihydrocapsaicin, nordihydrocapsaicin, capsaicin, dihydrocapsaicin, homocapsaicin and homodihyd

115 he contents of the four major capsaicinoids: capsaicin, dihydrocapsaicin, nordihydrocapsaicin, and ho

116 uscle contractions induced by application of capsaicin, direct colon stimulation, stimulation of ExPA

117 was performed at screening to determine the capsaicin dose inducing a half-maximal response, which w

118 ny concentration of capsaicin (Emax) and the capsaicin dose inducing half-maximal response (ED50).

119 tional analyses in Caenorhabditis elegans As capsaicin elicits heat and pain sensations in mammals, t

120 ough response evoked by any concentration of capsaicin (Emax) and the capsaicin dose inducing half-ma

121 Capsaicin evoked inward currents (current density ~10% o

122 nts with stable asthma exhibited exaggerated capsaicin-evoked cough responses consistent with neurona

123 We sought to investigate capsaicin-evoked cough responses in a group of patients

124 on awake cough frequency (primary outcome), capsaicin-evoked cough, and patient-reported outcomes.

125 these patients, despite small reductions in capsaicin-evoked cough.

126 There was also an increase in capsaicin-evoked coughs after allergen exposure compared

127 se from hindpaw skin biopsies, and increased capsaicin-evoked inward current and membrane expression

128 TRPV1 activity, as demonstrated by increased capsaicin-evoked nocifensive responses, increased calcit

129 1 receptor only reduces both anandamide- and capsaicin-evoked responses in ACR neurons.

130 of G618W and M644I were recapitulated in the capsaicin-evoked YO-PRO1 uptake assay.

131 n resulted in a loss, rather than a gain, in capsaicin-evoked YO-PRO1 uptake.

132 ochemical biosensor for the determination of capsaicin extracted from chilli fruits, based on a novel

133 Our analysis revealed that capsaicin failed to activate TRPV2_Quad likely due to st

134 We discuss the significance of capsaicin flipping from the extracellular to the intrace

135 s produced by intraplantar administration of capsaicin, formalin or complete Freund's adjuvant.

136 neurons express Fos after cheek injection of capsaicin, formalin, or chloroquine.

137 ptive responses to intraplantar injection of capsaicin, formalin, or complete Freund's adjuvant more

138 These results revealed that capsaicin forms hydrogen bonds with GLU355, GLU541, GLU5

139 gion of India evaluated showed variation for capsaicin from 0.27% (CHF-CA-1) to 3.03% (CHF-CA-21), ol

140 er of the sensitivity mediated by hTRPV1 was capsaicin > sanshool > allicin.

141 renal nerves, renal afferent disruption with capsaicin had no effect on blood pressure or renal infla

142 Capsaicin has also been used as an analgesic, and its to

143 oposal as second line for lidocaine patches, capsaicin high-concentration patches, and tramadol; and

144 lease and enacarbil; and 10.6 (7.4-19.0) for capsaicin high-concentration patches.

145 simulations were used to explore behavior of capsaicin in a 1-palmitoyl-2-oleoyl phosphatidylcholine

146 EN-D0501 and SB-705498 were profiled against capsaicin in a sensory nerve activation assay and in viv

147 highly improved the voltammetric process of capsaicin in comparison to the CNTs/GCE and bare GCE.

148 ic afferents, and sensitization responses to capsaicin in dorsal root ganglia (DRGs) following applic

149 r study comparing cough responses to inhaled capsaicin in patients with mild atopic asthma after alle

150 (CNTs) was applied for the determination of capsaicin in pepper samples.

151 n of submucosal neurons by the TRPV1 agonist capsaicin in rectal biopsy specimens collected from 9 pa

152 tein was seen for simulations initiated with capsaicin in the bilayer.

153 as successfully been applied for quantifying capsaicin in various pepper samples including Isot.

154 orresponded to reduced sensitivity to 100 nM capsaicin in vitro (IC50 = 230 +/- 20 nM, 76 +/- 4.4% wi

155 ntraduodenal capsaicin infusion (1.5 mg pure capsaicin) in healthy volunteers on hunger, satiety, and

156 Additionally, we found that capsaicin increased cannabinoid receptor (CB2) in the co

157 inal domain features an enhanced response to capsaicin, independent of the phosphoinositide lipid con

158 Consequently, capsaicin induced apoptosis in the cancer cells, but not

159 Investigations of Swiss Albino mice through capsaicin induced paw lickings and dextran induced infla

160 dependent signaling plays a dominant role in capsaicin-induced ablation of nociceptive terminals and

161 Ca(2+) influx through TRPV1 is necessary for capsaicin-induced ablation of nociceptive terminals.

162 ice showed that EGTA and MDL28170 diminished capsaicin-induced ablation.

163 of reactive oxygen species did not attenuate capsaicin-induced ablation.

164 The mechanisms underlying capsaicin-induced analgesia likely involve reversible ab

165 plasmic reticulum (ER) Ca(2+) stores reduced capsaicin-induced Ca(2+) increases and CaCC activation,

166 ombinant human TRPV1 channels, 4 antagonized capsaicin-induced Ca(2+) influx, with an IC50 value of 4

167 Moreover, EREG application potentiated capsaicin-induced calcium influx in a subset of sensory

168 as none of the mutations selectively altered capsaicin-induced changes in NMDG permeability, the loss

169 ssay and in vivo potency established against capsaicin-induced cough in the guinea pig.

170 In vivo XEN-D0501 completely inhibited capsaicin-induced cough, whereas 100 times more SB-70549

171 old more potent than SB-705498 at inhibiting capsaicin-induced depolarization of guinea pig and human

172 and primary afferent cultures, we monitored capsaicin-induced effects on afferent terminals in real

173 hibited nociceptive transmission by reducing capsaicin-induced internalization of NK-1 and phosphoryl

174 imilarly, clonidine reduced the frequency of capsaicin-induced mEPSCs by approximately 60%.

175 rons from stg/stg mice, but the amplitude of capsaicin-induced mEPSCs from C-fiber synapses was unalt

176 itch in the current-voltage relationships of capsaicin-induced mEPSCs, from linear to inwardly rectif

177 d slices, clonidine reduced the frequency of capsaicin-induced miniature EPSCs in the presence of tet

178 Although capsaicin-induced mitochondrial Ca(2+) uptake was TRPV1-

179 responses in formalin-induced tonic pain, in capsaicin-induced neurogenic pain, and notably in oxalip

180 nduced pruritus (itch) and additionally in a capsaicin-induced nociception model of pain without any

181 chanical or heat withdrawal thresholds or in capsaicin-induced nocifensive behavior.

182 activating LJA5 neurons in male mice reduces capsaicin-induced pain and histamine-induced itch.

183 compound 30 rescued albino mice by 80% from capsaicin-induced paw licking and recovered it by 60% fr

184 Furthermore, capsaicin-induced spontaneous pain, inward currents in D

185 Moreover, MDL28170 prevented capsaicin-induced thermal hypoalgesia.

186 We hypothesized that capsaicin induces satiety through the release of gastroi

187 investigate the effects of an intraduodenal capsaicin infusion (1.5 mg pure capsaicin) in healthy vo

188 gallbladder volumes were observed after the capsaicin infusion compared with after the placebo infus

189 The intraduodenal capsaicin infusion significantly increased satiety (P-tr

190 Capsaicin inhalational challenge was performed, and coug

191 Sensory neuron ablation by neonatal capsaicin injection prevented perineural invasion (PNI),

192 f capsaicin-sensitive rat TRPV1 (rTRPV1) and capsaicin-insensitive chicken TRPV1 (cTRPV1) exposed to

193 est stimulus because the coughing induced by capsaicin interfered with measurements.

194 atory responses evoked by microinjections of capsaicin into the paratrigeminal nucleus were significa

195 Histamine, chloroquine, and capsaicin intradermally elicited similar distributions o

196 Capsaicin is an ingredient in spicy peppers that produce

197 As a consequence, the efficacy of capsaicin is unknown in a significant number of IR patie

198 -articular injection of HA in rats decreases capsaicin joint nociceptor fibres discharge.

199 w that activation of multiple terminals by a capsaicin-like current allows summation of the responses

200 In contrast, channel activations by capsaicin, low pH, divalent cations, and even heat are m

201 challenged recombinant rTRPV1 receptors with capsaicin, low pH, or heat.

202 Further, capsaicin markedly and dose-dependently reduced coronary

203 tantially reduced maximal cough responses to capsaicin (mean change from baseline, XEN-D0501, -19.3 +

204 Furthermore, capsaicin mediated protection is lost when CB2 antagonis

205 TLR5-mediated Abeta-fiber blockade, but not capsaicin-mediated C-fiber blockade, also reduced chemot

206 However, sensitization of capsaicin-mediated currents after the activation of PKC

207 timal ligand-binding pocket conformation for capsaicin-mediated TRPV1 activation gating, and reveals

208 orosensory afferent neurons also respond to capsaicin, menthol, and/or mustard oil (allyl isothiocya

209 We also observed a capsaicin molecule flipping from the extracellular to th

210 sts of nocisensitive TRP channels, including capsaicin, mustard oil, and noxious heat.

211 We have no idea about the efficacy of capsaicin nasal spray in these patients nor about the ti

212 We report here that capsaicin nasal spray is effective in a broader group of

213 itis (IR) is a prevalent condition for which capsaicin nasal spray is the most effective treatment.

214 We sought to study the effects of capsaicin nasal spray on the afferent innervation of the

215 nd, placebo-controlled randomized trial with capsaicin nasal spray was performed involving 33 patient

216 ture is convincing regarding the efficacy of capsaicin nasal treatment in idiopathic rhinitis (IR).

217 aneous HA injection in mice reduces heat and capsaicin nocifensive responses, whereas the intra-artic

218 reflected in a reduced cortical response to capsaicin on fMRI.

219 lecular mechanisms underlying the effects of capsaicin on nociceptors.

220 st days, an intraduodenal infusion of either capsaicin or a placebo (physiologic saline) was performe

221 estricted to temperature and does not affect capsaicin or acid responses, thereby maintaining a role

222 Different concentrations of capsaicin or its analog, resiniferatoxin (RTX), were app

223 sponse to intracolonic application of either capsaicin or mustard oil, stimuli known to evoke sustain

224 cals of the tested hostplants significantly (capsaicin) or insignificantly (gossypol and nicotine) in

225 occurring pungent molecules such as allicin, capsaicin, or resiniferatoxin.

226 d) microinjection of histamine, chloroquine, capsaicin, or vehicle into the left cheek.

227 In conclusion, capsaicin otoprotection appears to be mediated by activa

228 -S4 transmembrane helices in the presence of capsaicin placed in the aqueous phase, in the lipid, or

229 ramers correlates with channel activation by capsaicin, providing an optical marker of conformational

230 vars is highly variable, with the content of capsaicin ranging from 31% to 71% of the total capsaicin

231 he cannabinoid type 1 (CB1) receptor and the capsaicin receptor (TRPV1) exhibit co-expression and com

232 ecture and domain assembly compared with the capsaicin receptor TRP vanilloid member 1 (TRPV1).

233 through the activation of the heat-sensitive capsaicin receptor TRPV1 by magnetic nanoparticles.

234 The capsaicin receptor TRPV1 ion channel is a polymodal noci

235 threshold temperature of the heat-sensitive capsaicin receptor TRPV1 ion channel, leading to its act

236 The capsaicin receptor TRPV1 is an outstanding representativ

237 le responses to capsaicin when the mammalian capsaicin receptor VR1 was ectopically expressed in thei

238 cules that serve as ligands of the "heat and capsaicin receptor" TRPV1.

239 The capsaicin receptor, TRPV1, is a key ion channel involved

240 The functional roles of the capsaicin receptor, TRPV1, outside of sensory nerves are

241 arting from the cytosolic aqueous phase, and capsaicin remained stable in the majority of simulations

242 - menthol, allyl isothiocyanate (AITC), and capsaicin, respectively - at concentrations found in foo

243 s elicited by id histamine, chloroquine, and capsaicin, respectively, 3.7%, 4.3%, and 4.1% were retro

244 id injection of histamine, chloroquine, and capsaicin, respectively.

245 (IC50 = 230 +/- 20 nM, 76 +/- 4.4% wild-type capsaicin responders vs. 56.9 +/- 4.7% HDAC4 cKO respond

246 without any effect on the overall number of capsaicin-responding cells.

247 tential vanilloid subtype 1 (TRPV1)-mediated capsaicin responses via Toll-like receptor 4 (TLR4) in m

248 d nocifensive responses and sensitization of capsaicin responses were significantly attenuated in the

249 f TRPV1 in different species globally alters capsaicin responses.

250 demonstrate that intra-arterial infusion of capsaicin results in a dose-related rise in MAP and HR t

251 rase inhibitor huprine Y and the antioxidant capsaicin results in compounds with nanomolar potencies

252 nsory nerves did not affect the responses to capsaicin revealing a vascular smooth muscle-restricted

253 f proline-rich protein and mucin 7 following capsaicin (rho = 0.71, p = 0.02, rho = 0.68, p = 0.04, r

254 ce, there was an increase in the fraction of capsaicin-sensitive cells relative to wild type (WT) con

255 We found that in small-diameter, capsaicin-sensitive dorsal root ganglia neurons correspo

256 show that GRP directly activates small-size capsaicin-sensitive DRG neurons, an effect that translat

257 imaging to characterize chimeras composed of capsaicin-sensitive rat TRPV1 (rTRPV1) and capsaicin-ins

258 residues responsible for differential TRPV1 capsaicin sensitivity among species, we used intracellul

259 ine or proline at residue A578 also elicited capsaicin sensitivity in cTRPV1.

260 lutamic acid was sufficient to endow it with capsaicin sensitivity in the micromolar range.

261 686 swapped into chicken receptors displayed capsaicin sensitivity, and that simply changing the alan

262 erone did not activate any A578 mutants with capsaicin sensitivity, suggesting that the vanilloid gro

263 sidue E570 with lysine or glutamine retained capsaicin sensitivity.

264 Capsaicin sensitizes warm trigeminal thermoreceptors and

265 An intraduodenal infusion of capsaicin significantly increases satiety but does not a

266 perception of sucrose, caffeine, menthol and capsaicin solutions.

267 er, interactions were seen between TRPV1 and capsaicin starting from the cytosolic aqueous phase, and

268 tidergic CGRP/somatostatin+ nociceptors upon capsaicin stimulation exert a tonic inhibitory control o

269 e Fos-positive neurons following pruritic or capsaicin stimuli, approximately 1-2% were retrogradely

270 Oral administration of the TRPV1 agonist, capsaicin, suppressed ligature-induced bone loss in mice

271 einstate channel expression and responses to capsaicin, temperature, and voltage.

272 acids are better tolerated for activation by capsaicin than for activation by hot temperature, sugges

273 ecent studies have shown a metabolic role of capsaicin that may be mediated via the transient recepto

274 d in pain sensation, and is the receptor for capsaicin, the active ingredient of hot chili peppers.

275 The best known agonist of TRPV1 channels is capsaicin, the pungent component of "hot" chili peppers.

276 (TRPV1) channel is activated by heat and by capsaicin, the pungent compound in chili peppers.

277 Capsaicin, the spicy component of hot chili peppers acti

278 Together with capsaicin, these ligands form a set of molecular rulers

279 des, is demonstrated in the determination of capsaicin through adsorptive stripping voltammetry.

280 e is relatively unstable, whereas binding of capsaicin to TRPV2_Quad antagonizes resiniferatoxin-indu

281 In Young MAs, capsaicin (to inhibit sensory neurotransmission) blocked

282 -316,243 were attenuated by 50% in intra-BAT capsaicin-treated mice.

283 showed a trend to being better responders to capsaicin treatment compared with patients with IR but w

284 patients with IR and symptom reduction after capsaicin treatment demonstrates the clinical relevance

285 The therapeutic action of capsaicin treatment in patients with idiopathic rhinitis

286 s and the increased threshold for AITC after capsaicin treatment in patients with IR demonstrate the

287 Capsaicin treatment of IR patients increased the thresho

288 However, the functional consequences of capsaicin treatment on nasal nerve activation and the as

289 nasal hyperreactivity (NHR) and response to capsaicin treatment remain unknown.

290 B2 antagonist AM630 is administered prior to capsaicin treatment.

291 rans-tympanic (TT) or oral administration of capsaicin (TRPV1 agonist) prevents cisplatin ototoxicity

292 When activated by capsaicin, TRPV1 recruits more glutamatergic, but not GA

293 ocatechuic acid, syringic acid, tyrosine and capsaicin used as controls.

294 Concentrations of flavonoids and capsaicin were simultaneously quantified for the first t

295 utants and WT showed comparable responses to capsaicin when the mammalian capsaicin receptor VR1 was

296 itide lipids display an enhanced response to capsaicin, whereas phosphoinositide lipid supplementatio

297 al rhizotomy nor an intrathecal injection of capsaicin, which completely eliminated spinal cord TRPV1

298 By contrast, capsaicin, which evokes a mix of itch and pain sensation

299 Capsaicin, which is the major pungent principle in chili

300 rdic response to intralaryngeal perfusion of capsaicin, which was associated with up-regulation of TR