ライフサイエンスコーパス: capsid protein

1 ty is increased with a larger, flatter major capsid protein.

2 the E2 cytoplasmic tail/endodomain with the capsid protein.

3 me encodes three proteins including the ORF2 capsid protein.

4 ficient nuclear import of both viral DNA and capsid protein.

5 ptide motif (AspSerSer)(6) onto the AAV9-VP2 capsid protein.

6 usions that are formed by loops of the major capsid protein.

7 e of the infectious particle-associated ORF2 capsid protein.

8 severity, is a soluble variant of the viral capsid protein.

9 enome encodes 3 proteins, including the ORF2 capsid protein.

10 dissociated pb10 to the 120 hexamers of the capsid protein.

11 ntre of the 120 hexamers formed by the major capsid protein.

12 Vs from a KSHV mutant defective in the small capsid protein.

13 his case, it is the chlorovirus PBCV-1 major capsid protein.

14 ns, structure, and interactions of the viral capsid protein.

15 y/tertiary structures that interact with the capsid protein.

16 nt RNA polymerase (RdRP), capping enzyme and capsid protein.

17 he adenosine deaminase ADAR2 fused to an MS2 capsid protein.

18 ies mapping to the P domain of the norovirus capsid protein.

19 nthesis of the glycans attached to its major capsid protein.

20 analysis of peptides generated from the VP1 capsid protein.

21 ific modification of the BPV-1 and HPV-16 L2 capsid proteins.

22 osahedral viruses with the double jelly-roll capsid proteins.

23 in regulating lytic replication, but lacked capsid proteins.

24 ct, and no degradation was observed on VLPs' capsid proteins.

25 nner shell is different from all other known capsid proteins.

26 contain RNA genomes protected by a shell of capsid proteins.

27 and subsequently UV-crosslinked to adjacent capsid proteins.

28 llennia and which include changes within the capsid proteins.

29 by binding to a hydrophobic pocket in viral capsid protein 1 (VP1).

30 on-dividing cells through the ability of the capsid protein(1) to engage the cellular nuclear import

31 sticity among the 20 conformers of the major capsid protein, 2 conformers of the small capsid protein

32 monomeric mNeonGreen protein fused to viral capsid protein 26 (VP26) permitted detection of reactiva

33 For purity analysis, only 25 ng of total AAV capsid proteins (4.3 femtomole virus particles) were loa

34 Multiple peptides were identified from HAV capsid proteins (53.7% coverage), but none from nonstruc

35 ructure consists of 5040 copies of the major capsid protein, 60 copies of the penton protein and 1800

36 rkable global conformational polymorphism of capsid proteins, a network formed by extended N arms, mo

37 We show key residues in outer capsid proteins, a pH-sensing histidine of a zinc finger

38 A mutation in the HIV-1 capsid protein abolishes the ability of the virus to inf

39 crystal structure of a complex of the HAstV capsid protein and a virus-neutralizing antibody.

40 by VLP is insensitive to pH, but unassembled capsid protein and ARM peptide exhibit diminished activi

41 me that OATP1A2 interacts with the avian HEV capsid protein and can influence viral infection in host

42 The capsid protein and envelope proteins are both arranged i

43 molecule that disrupts the functions of HIV capsid protein and is amenable to long-acting therapy ow

44 rus (KSHV) mutant that is defective in small capsid protein and is unable to produce mature virions.

45 rion structure and the identity of the major capsid protein and other predicted virion proteins, incl

46 in complex known as retromer binds to the L2 capsid protein and sorts incoming virions into the retro

47 es, without affecting the integrity of viral capsid protein and the viral particle.

48 u hybridization were used to determine viral capsid protein and viral DNA respectively.

49 ith genetically encoded fluorescence-labeled capsid protein and/or glycoprotein to visualize viral pa

50 human adenovirus (HAdV) in vitro by binding capsid proteins and blocking endosomal escape of virus.

51 es specific human adenoviruses by binding to capsid proteins and blocking endosomal escape of virus.

52 ainst autophagic destruction by sequestering capsid proteins and coordinating particle assembly and s

53 ted to model the cooperative effects between capsid proteins and genomic RNA that would occur in a pa

54 ificant structural rearrangements induced by capsid proteins and invite the application of time-resol

55 nfectious virions requires the production of capsid proteins and other late genes, whose production i

56 N-termini of two-fold symmetry-related VP4A capsid proteins and RNA, provides inter-pentamer stabili

57 RNA viruses can involve interactions between capsid proteins and secondary structures in the viral ge

58 (mReC), we find that the similarity of major capsid proteins and terminase large subunits further sug

59 del system to study the interactions between capsid proteins and the changes they undergo during entr

60 tide sequences at the interfaces between the capsid proteins and the genomic RNA of bacteriophage MS2

61 study reveals new insights into the roles of capsid proteins and their multiple functions during vira

62 containing the RNA genome surrounded by the capsid protein, and a viral envelope containing 80 spike

63 r therapies that target the functions of HIV capsid protein, and demonstrate the potential of GS-6207

64 tions in nsP3 HVD and clustered mutations in capsid protein, and tested the effects of these modifica

65 formed from a combination of minor and major capsid proteins, and are recurrent within viral lineages

66 chicken liver cells by a truncated avian HEV capsid protein (ap237) in which the host protein OATP1A2

67 igner" AAV, AAV2/Anc80L65, in which the main capsid proteins approximate the ancestral sequence state

68 Although the capsid proteins are all identical, they nevertheless arr

69 Capsid proteins are specific to each virus and can be us

70 , indicating that they are still composed of capsid proteins arranged in a hexagonal lattice.

71 oplasm as mRNAs encoding the Gag and Gag-Pol capsid proteins as well as genomic RNAs (gRNAs) packaged

72 le in the maturation of viral mRNAs encoding capsid proteins as well as in viral DNA replication.

73 tail attachment; however, bacteriophage HK97 capsid proteins assemble efficiently without a portal wh

74 igh rates of amino acid substitutions in the capsid proteins at exposed sites not previously identifi

75 adenovirus lineage, characterized by a major capsid protein bearing two beta-barrels.

76 have T = 1 symmetry with the characteristic capsid protein beta-barrels found in all the viruses in

77 studies have revealed that herpesvirus small capsid proteins bind to capsids via their amino terminus

78 F6 significantly decreased the expression of capsid protein but not that of DHX15.

79 ase to cleave the P1 polyprotein into mature capsid proteins, but the FMDV 3C protease is toxic to ho

80 ng inhibitors that block recognition of VEEV capsid protein (C) by the host importin (IMP) alpha/beta

81 The orthoretroviral capsid protein (CA) assembles into polymorphic capsids,

82 iven maturation process, the orthoretroviral capsid protein (CA) assembles to form the convex shell t

83 le spinning (MAS) spectra at 14.1 T on HIV-1 capsid protein (CA) assemblies.

84 The capsid protein (CA) content of the infectious viral core

85 The HIV-1 capsid protein (CA) facilitates reverse transcription an

86 The central cavity of the capsid protein (CA) hexamer reveals itself as a plausibl

87 sid consists of a variably curved lattice of capsid protein (CA) hexamers and pentamers.

88 Recent studies suggest that the viral capsid protein (CA) is a determinant for the sensitivity

89 surface of the viral membrane, to the mature capsid protein (CA) lattice, which encloses the viral RN

90 s study, the hexagonal lattice formed by the capsid protein (CA) of human immunodeficiency virus (HIV

91 The viral capsid protein (CA) partly determines the sensitivity of

92 we report the atomic-resolution structure of capsid protein (CA) tubes, determined by magic-angle spi

93 e found that HIV-1 carrying AA insertions in capsid protein (CA) undergoes aberrant CA degradation.

94 HIV-1, shares its binding site in the viral capsid protein (CA) with the host factors CPSF6 and NUP1

95 despite an anomalously low affinity for the capsid protein (CA).

96 capsid, which is made from individual viral capsid proteins (CA), is a target for a number of antivi

97 CD8(+) T-cell responses against the AAV capsid protein can, however, affect therapeutic efficacy

98 harbors HBoV1 nonstructural protein (NS) and capsid protein (Cap) genes.

99 portal and its interactions with surrounding capsid proteins, CATCs, and the terminal end of KSHV's d

100 ic promoter at levels similar to that of the capsid protein-coding mRNA and is essential for replicat

101 ngle-stranded RNA viruses with 180 copies of capsid protein comprising the T=3 icosahedral capsids.

102 A single mutation, R143A, in the capsid protein conferred resistance to high concentratio

103 f Balb/c mice with different combinations of Capsid protein containing VLPs either as monovalent, biv

104 suggesting that phosphorylation of the viral capsid proteins contributes to infectious entry.IMPORTAN

105 sly packaged into spherical capsids by viral capsid protein (CP) because it is a more compact and fle

106 wo major open reading frames (ORFs): Rep and capsid protein (CP) characteristic of a type IV genome o

107 ck HBV (DHBV) as a model although DHBV has a capsid protein (CP) comprising ~260 rather than ~180 ami

108 The dimeric capsid protein (Cp) has multiple functions during the vi

109 e mosaic virus (BMV) is packaged by a single capsid protein (CP) into three types of morphologically

110 ding site bridging over three domains of one capsid protein (CP) monomer.

111 Previous work has shown that purified capsid protein (CP) of cowpea chlorotic mottle virus (CC

112 The alphavirus capsid protein (Cp) selectively packages genomic RNA (gR

113 lding protein (SP) for a binding site on the capsid protein (CP), and works by altering the angle bet

114 F sequence showed homology with the putative capsid proteins (CPs) of mycoviruses.

115 can be expressed as a free protein and as a capsid protein-delta (CP-delta) fusion protein.

116 show that MoCBP binds preferentially to MS2 capsid proteins demonstrating that specific molecular in

117 during the budding that must be overcome by capsid proteins diffusing along the membrane prior to in

118 mixture of intermediates extending from the capsid protein dimer to the icosahedral T = 4 capsid wit

119 and overgrown, containing more than the 120 capsid protein dimers needed to form a perfect T = 4 ico

120 diates on the assembly path from hepatitis B capsid protein dimers to the 120-dimer capsid.

121 However, the structure of the viral major capsid protein, elucidated at near-atomic resolution usi

122 particle composed of the Qbeta bacteriophage capsid protein encapsulating an immunostimulatory CpG-A

123 We show that VP2, a minor capsid protein encoded by all caliciviruses(1,2), forms

124 f NP1, and plays a role in the maturation of capsid protein-encoding mRNAs in the nucleus.

125 plasma by performing a paper-based HIV-1 p24 capsid protein enzyme-linked immunosorbent assay, which

126 cipally target conserved epitopes within the capsid protein, escape from which results in a significa

127 A VP2 (major capsid protein) EVE sequence from a mouse genome assembl

128 -measure protein and some of the other minor capsid proteins exist in other NCLDVs.

129 Virus capsid protein expression is confined to the upper epith

130 smic cellular membranes to acquire the outer capsid proteins for infectious particle assembly.

131 The minor capsid proteins form a hexagonal network below the outer

132 ic nanoparticles (MNPs) to extract the virus capsid protein from complex biological media and subsequ

133 ing a conserved ribosome-binding site in the capsid protein from exposure.

134 virus-like particles (VLPs) of the L1 major capsid protein from HPV-2, -4, or -9, including the AS04

135 ntified this PC as a previously unidentified capsid protein from multiple uncultivated tailed virus f

136 Analysis of 58,023 major capsid proteins from large and giant viruses using metag

137 previously determined, and show that all 10 capsid proteins (from four heterodimers and two homodime

138 ificially tethering viral mRNAs encoding Gag capsid proteins (gag-pol mRNAs) to distinct non-PM subce

139 s on the noncoding control region, the major capsid protein gene VP1, and the large T antigen gene.

140 A mutation in the major capsid protein, gp23, produced T=13 icosahedral capsids.

141 The major capsid protein, gp27, is identified and found to share t

142 These results demonstrate that the pUL25 capsid protein has a critical role in releasing viral DN

143 s in the nuclear localization signal in EEEV capsid protein have an additional negative effect on vir

144 sists of an ordered and interfacing array of capsid protein hexamers and pentamers that are recognize

145 he delayed protein E4orf6 and the major late capsid protein hexon is compromised.

146 ormed by residues from three neighboring VP2 capsid proteins.IMPORTANCE Parvovirus B19 is a common hu

147 ystal structure (1.5 angstrom) of the DENV-2 capsid protein in complex with an inhibitor that potentl

148 synthesis of the N-glycan of the viral major capsid protein in PBCV-1 and establishes that a single p

149 ty and strongly interacted with HIV immature capsid protein in pull-down experiments.

150 nt studies have observed the presence of the capsid protein in the nucleus during infection(5-8), rev

151 p150 tegument protein, we initially detected capsid proteins in association with endosomes but later

152 The crystal structure of the major capsid protein, in combination with cryo-electron micros

153 We identified several additional capsid proteins, including a candidate for the prohead p

154 m patients, HEV produced 3 forms of the ORF2 capsid protein: infectious/intracellular ORF2 (ORF2i), g

155 hough with relatively fewer intra- and inter-capsid protein interactions and less stably associated t

156 Fitting of the capsid protein into densities shows the presence of a he

157 and inhibition of human Dicer enzyme by the capsid protein is a potential mechanism for this unique

158 of the HIV-1 life cycle.IMPORTANCE The HIV-1 capsid protein is an attractive but unexploited target f

159 human immunodeficiency virus type 1 (HIV-1) capsid protein is an attractive therapeutic target, owin

160 Each capsid protein is comprised of three domains: R (RNA bin

161 ain (CTD) of hepatitis B virus (HBV) core or capsid protein is highly dynamic and plays multiple role

162 Most, but not all, of the capsid protein is rapidly shed in tissue culture and pri

163 Circoviral capsid protein is the only structural protein of the vir

164 The capsid protein is unusual both for its rare fold and tha

165 t in a dissociation of a subset of the major capsid protein L1 from the minor capsid protein L2, whic

166 particle (VLP) formed by the self-assembling capsid protein L1.

167 lattice comprising 72 pentamers of the major capsid protein (L1) and the associated minor capsid prot

168 facilitate the release of most of the major capsid protein, L1, from the minor capsid protein L2 and

169 the major capsid protein, L1, from the minor capsid protein L2 and the viral genome.

170 ral protein in infectious entry is the minor capsid protein L2, which engages different components of

171 f the major capsid protein L1 from the minor capsid protein L2, which remains in complex with the vir

172 capsid protein (L1) and the associated minor capsid protein (L2).

173 into the host cell by retention of the minor capsid protein, L2, and the viral genome instead of traf

174 is dependent upon the presence of the minor capsid protein, L2, but independent of encapsidated DNA.

175 ttice and its interactions with the internal capsid protein lattice.IMPORTANCE Alphaviruses include i

176 tarts with the assembly of a curved layer of capsid proteins lining the interior of the plasma membra

177 Moreover, the major capsid protein locus of pacmanvirus appears to be differ

178 ture consisting of the herpesvirus-conserved capsid proteins MCP, Tri1, Tri2, and SCP and the HCMV-sp

179 8,280 copies of the double jelly-roll major capsid protein (MCP) p72, arranged in trimers displaying

180 (SP) drive assembly by chaperoning the major capsid protein (MCP) to build an icosahedral lattice.

181 ge T5 is composed of 775 copies of the major capsid protein (mcp) together with portal, protease, and

182 h potentially representing a divergent major capsid protein (MCP) with a predicted double jelly-roll

183 ilarly sized capsid, consisting of 955 major capsid protein (MCP), 900 small capsid protein (SCP), 64

184 machinery required to glycosylate its major capsid protein (MCP).

185 s composed of three copies of a single major capsid protein (MCP).

186 In the case of ZIKV, capsid protein-mediated ribosomal stress may contribute

187 d protein monomers, where it interferes with capsid-protein-mediated interactions between proteins th

188 the self-assembly of a coarse-grained virus capsid protein model.

189 of capsid assemblies (ranging from two to 42 capsid protein molecules) that recreate the various surf

190 ind tightly at a conserved interface between capsid protein monomers, where it interferes with capsid

191 eviously unknown role for the reovirus outer capsid protein mu1 in limiting the induction of a necrot

192 The reovirus outer capsid protein mu1 negatively regulates reovirus-induced

193 The reovirus outer capsid protein mu1 regulates cell death in infected cell

194 Virus capsid proteins must perform a number of roles.

195 e, we characterize the functional effects of capsid protein mutations that result in the loss of viru

196 ts suggest that the P17 protein is the minor capsid protein of Bam35 and P24 is the penton protein, w

197 Finally, capsid protein of Dengue virus, but not West Nile virus,

198 VP26, the small capsid protein of HSV and PRV, was one of the first herp

199 uses and two putative T cell epitopes in the capsid protein of the rubella and ruhugu viruses are mod

200 apsid is determined by a tape-measure, minor capsid protein of which there are 60 copies in the virio

201 By expressing the capsid proteins of different parvovirus EVEs that were f

202 tially vaccinated with DNA plasmids encoding capsid proteins of different RV A types.

203 espite very low sequence homology, the major capsid proteins of double-stranded DNA (dsDNA) bacteriop

204 ibodies against conserved regions located on capsid proteins of RV A viruses, mice were sequentially

205 urring variation on the structure of the HPV capsid proteins of vaccine-relevant oncogenic HPV genoty

206 copies of the penton protein and 1800 minor capsid proteins of which there are 13 different types.

207 on, which is necessary to properly enter the capsid protein open reading frame.

208 While we did not find any intact capsid protein open reading frames in the integrated vir

209 cterized HEV particles; we identified 3 ORF2 capsid proteins (ORF2i, ORF2g, and ORFc).

210 A-targeting CRISPR effectors and 167 AAV VP1-capsid-protein orthologues.

211 hibiting the final protease cleavage between capsid protein p24 and spacer protein-1, producing immat

212 ccessfully conjugated tHBcAg VLPs to the HIV capsid protein P24 in the cytosol.

213 with HIV viral replication, assayed by virus capsid protein p24 production.

214 multiple layers, and resolve the major outer-capsid protein p72 to higher resolution.

215 We show here that the HIV-1 Capsid protein plays a central role in protecting the vi

216 Results show the capsid protein plays a central role in the flavivirus as

217 tious entry.IMPORTANCE The papillomavirus L2 capsid protein plays an essential role in infectious ent

218 protein complex, along with its constitutive capsid protein, plays essential roles at virtually every

219 imate the impact of residue mutations on AAV capsid protein-protein interactions and thus predict cha

220 motifs in the parechovirus genome that bind capsid proteins, providing approximately 60 specific int

221 nts, we investigated the effect of the minor capsid proteins pUL17 and pUL25 on the structural stabil

222 uclear pore complex (NPC) is mediated by the capsid protein pUL25 and the capsid-tethered tegument pr

223 This structure illuminates capsid protein quaternary organization, including its or

224 The capsid protein residues involved in the RNA binding are

225 -1 isolates bearing defined mutations in the capsid protein revealed differences in their cytoplasmic

226 By contrast, the arrangement of the capsid proteins:RNA complex, which forms the core of the

227 n of a single RNA packaging signal (PS) with capsid protein(s) (most +ssRNA viruses so far studied);

228 , either (a) by specific recognition between capsid protein(s) and replication proteins (poliovirus),

229 or capsid protein, 2 conformers of the small capsid protein (SCP), 4 conformers of the triplex monome

230 of 955 major capsid protein (MCP), 900 small capsid protein (SCP), 640 triplex dimer (Tri2) and 320 t

231 Human papillomavirus (HPV) L1 and L2 capsid proteins self-assemble into virions capable of ef

232 to be serotype-specific, suggesting that the capsid protein sequence could be important.

233 gh proportions of nonforming or noninfective capsid protein sequences that reduce the effective depth

234 l neural network to identify candidate viral capsid proteins, several of which formed virus-like part

235 timely release of the HIV-1 genome from the capsid protein shell and efficient viral nuclear import.

236 ttice and its interactions with the internal capsid protein shell.

237 ine (G) difference at position 91 of the VP1 capsid protein shifts the profile of tumors induced by M

238 eviously unknown role for the reovirus outer capsid protein sigma3 in limiting the induction of a nec

239 To determine if the outer capsid protein sigma3, which interacts with mu1, also fu

240 sid is stabilized by 660 copies of the outer capsid protein, Soc, which clamp adjacent gp23 hexamers.

241 o interact with ap237, a truncated avian HEV capsid protein spanning amino acids 313 to 549, by a glu

242 We determined the crystal structure of the capsid protein spike domain from one of these HAstV stra

243 insic fitness constraints derived for vp1, a capsid protein subject to antibody responses, are compar

244 ought to occur by the sequential addition of capsid protein subunits to a nucleus, with the final ste

245 ne-rich motif (ARM) at the N-terminus of its capsid protein that appears to be in the interior of the

246 acid substitutions in the domain of the VP1 capsid protein that binds the sialic acid moiety of glyc

247 identify and characterize a mutation in the capsid protein that confers resistance to the inhibitor.

248 n of the human immunodeficiency virus type 1 capsid protein that NMR and, in particular, residual dip

249 vage event found in approximately 10% of the capsid proteins that also was shown to alter capsid stab

250 s or specific residues within reovirus outer capsid proteins that impact the efficiency of cell entry

251 Compared with the capsid proteins, the oncogenes E7 and E6 had increased s

252 gets nonassembled and virus particle-forming capsid proteins to mediate their autophagy-dependent deg

253 stem of pegs and holes, and underlying minor capsid proteins, to assemble the capsid laterally from t

254 Unexpected findings included capsid protein-transcription factor chimeras; endonuclea

255 ost abundant SPN3US proteins to be the major capsid protein, two head ejection proteins, and the func

256 tron tomography, we show that binding of the capsid protein UL25 promotes the formation of NEC pentag

257 y that the closely related hepatitis B virus capsid protein undergoes similar structural changes, whi

258 We found that woodchuck hepatitis virus capsid protein undergoes structural transitions between

259 s) produced by recombinant expression of the capsid protein, using cryogenic electron microscopy.

260 e bocavirus genus controls expression of its capsid proteins via alternative RNA processing, by both

261 anines (MVC) governs production of the viral capsid proteins via its role in pre-mRNA processing.

262 plays an essential role in the maturation of capsid protein (VP)-encoding mRNAs and viral DNA replica

263 The major capsid protein VP1 of EV71 was selected as the biomarker

264 irus B3 (CVB3) differ by single mutations in capsid protein VP1 or VP3 and also differ in stability a

265 tified neutralizing antibodies that bind the capsid protein VP1 to block viral infection.

266 ted by the exposed apical loops of the major capsid protein VP1, a broad range of GAG oligosaccharide

267 cellular adhesion molecule 1 receptors via a capsid protein VP1-specific fivefold canyon feature, the

268 motif in the exposed, antigenic, GH loop of capsid protein VP1.

269 Peptide epitopes of the RV-A16 capsid proteins VP1 and VP2 were identified by peptide/M

270 Mutations in the major viral capsid protein, VP1, are common in JCPyV from PML patien

271 -nm particles with 60 copies of each of four capsid proteins, VP1 to VP4.

272 the less ordered, N-terminal region of their capsid proteins, VP1/2/3.

273 y conserved region in the N terminus of FMDV capsid protein VP2 (VP2N) was characterized using a pane

274 Here, we elucidate how outer capsid proteins VP2 and VP5 coordinate cell entry of BTV

275 res suggest that the detachment of the outer capsid proteins VP2 and VP5 during viral entry induces b

276 o acid substitutions at exposed sites in the capsid proteins VP2, VP3, and VP1 tend to be elevated in

277 Notably, most substitutions in the major capsid protein (VP2) gene are nonsynonymous, altering am

278 otein fusions to the amino terminus of small capsid protein VP26 are the most widely used method to v

279 amine-depleted cells generated a mutation in capsid protein VP3 at residue 234.

280 The C-terminal extension of capsid protein VP3 folds into a globular protruding (P)

281 HHs bind to a site on the top surface of the capsid protein VP3, which is hidden in the native virus.

282 undles formed from the extended C-termini of capsid protein VP4B and VP4C protrude from the capsid su

283 o viral particles measured by unglycosylated capsid protein VP5 content).

284 The glycans of the major capsid protein (Vp54) of Paramecium bursaria chlorella v

285 A minor capsid protein, VP6, unique for the orbiviruses, has bee

286 A limit of detection of 0.2 ng/mL (3.3 pM) capsid proteins was achieved with convenient UV absorban

287 RV is largely unknown, T cell epitopes of RV capsid proteins were analyzed, and cognate T cells were

288 The additional clustered mutations in capsid protein, which affect its ability to induce trans

289 Glycine 89 on the capsid protein, which is positioned within a nuclear loc

290 contacts mediated by N-terminal arms of VP2 capsid proteins, which result in the expansion of the ca

291 ized with the antibody against the HIV-1 p24 capsid protein, while it is powered via paper-based biof

292 ckles is dependent on the interaction of the capsid proteins with host cleavage and polyadenylation s

293 roviruses, is dependent on an interaction of capsid proteins with the multifunctional nonstructural p

294 The 2 capsid proteins with variant single jelly-roll folds for

295 ng in a subnanometer resolution structure of capsid proteins within the virus particle.

296 lieve that a deeper understanding of how the capsid protein works during infection will create opport

297 In both, the capsid proteins wrap around the DNA and maintain it in a

298 s are the most asymmetrically arranged major capsid proteins yet observed in virus structures.

299 Mutation of the viral capsid protein yielded SUN2-resistant HIV.

300 he latter system, nucleolar presence of ZIKV capsid protein (ZIKV-C) was associated with ribosomal st