ライフサイエンスコーパス: carboxyhemoglobin

1 acteristic visible spectroscopic features of carboxyhemoglobin.

2 phoglycerate, binds to the central cavity of carboxyhemoglobin A (HbACO) at pH 6.35.

3 nhalation injury had higher plasma levels of carboxyhemoglobin and enhanced airway neutrophilia.

4 Tin protoporphyrin treatment normalized carboxyhemoglobin and improved arterial blood gases and

5 icotine constituents); 51% versus 55% (blood carboxyhemoglobin); and 29% versus 96% (nicotine exposur

6 Arterial carboxyhemoglobin, bilirubin, and standard biochemical,

7 ssion and CO production as measured by blood carboxyhemoglobin (COHb) levels increase in experimental

8 carbon monoxide (CO) loading to increase the carboxyhemoglobin (COHb) saturation to 15%.

9 CO binds to Hb, forming carboxyhemoglobin (COHb), and produces tissue damage.

10 formation of elevated, toxic blood levels of carboxyhemoglobin (COHb).

11 tion between estimated CO exposure and blood carboxyhemoglobin concentration in 708 pregnant western

12 In this subgroup, the median carboxyhemoglobin concentration increased 1.29% (95% CI:

13 rations (I-COHb) accurately predict arterial carboxyhemoglobin concentrations (A-COHb).

14 concentrations (V-COHb) and internal jugular carboxyhemoglobin concentrations (I-COHb) accurately pre

15 tudy tested the hypothesis that mixed venous carboxyhemoglobin concentrations (V-COHb) and internal j

16 Carboxyhemoglobin concentrations measured after terminat

17 lucose, lactate, inflammatory cytokines, and carboxyhemoglobin concentrations were measured at select

18 Arterial carboxyhemoglobin correlated with markers of the inflamm

19 mitigates CO toxicity by scavenging CO from carboxyhemoglobin, improving systemic oxygen delivery an

20 The small carboxyhemoglobin increase produced by uptake of a small

21 Arterial carboxyhemoglobin is elevated in patients with critical

22 s associated with a 1.12% increase in median carboxyhemoglobin level (95% CI: 0.54, 1.69).

23 The carboxyhemoglobin level (a measure of heme oxygenase 1 a

24 Abnormalities were variable depending on the carboxyhemoglobin level at study admission and duration

25 was to explore the hypothesis that arterial carboxyhemoglobin level is associated with inflammation

26 An increased carboxyhemoglobin level was associated with an increased

27 Carboxyhemoglobin level was correlated with estimated CO

28 of pulse co-oximetry revealed that the mean carboxyhemoglobin level was elevated in 29 Indonesian ad

29 re as a source of CO, loss of consciousness, carboxyhemoglobin level, arterial pH, and presence of en

30 iated with lower minimum and greater maximal carboxyhemoglobin levels (p < .0001 and p < .001, respec

31 Although it is clear that venous carboxyhemoglobin levels are similar to arterial levels,

32 Increased carboxyhemoglobin levels during severe malaria and sepsi

33 avascular monocytes/macrophages and arterial carboxyhemoglobin levels increased progressively from 3

34 ure to sidestream smoke, plasma nicotine and carboxyhemoglobin levels increased to 0.77 +/- 0.11 ng/m

35 Increased carboxyhemoglobin levels were consistently noted in Tg m

36 tions were sought between levels of arterial carboxyhemoglobin, markers of the inflammatory response,

37 Carboxyhemoglobin measurements were evaluated by analysi

38 er' instruments can estimate blood levels of carboxyhemoglobin, methemoglobin, and total hemoglobin (

39 ydralazine) and Sanguinate (pegylated bovine carboxyhemoglobin), on resting-state functional connecti

40 relevant variables, a lower minimum arterial carboxyhemoglobin was associated with an increased risk

41 Carboxyhemoglobin was measured in whole blood drawn arou

42 minimum and high maximum levels of arterial carboxyhemoglobin were associated with increased intensi

43 oncentrations of glucose, lactate, and blood carboxyhemoglobin were lower in the FO than in the contr

44 carbon monoxide and particulates and plasma carboxyhemoglobin were significantly higher in the 2 SHS