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1 cinar cells and encodes the digestive enzyme carboxyl ester lipase.
2 se, pancreatic lipase-related protein 2, and carboxyl-ester lipase.
3 ms related to serine and steroid metabolism (carboxyl ester lipase 2), indicating tryptophan-induced
6 ic triglyceride lipase, but close to that of carboxyl ester lipase, after a long lag phase, which cou
7 triacylglycerol estolides were substrates of carboxyl ester lipase and 5-PAHSA-containing lipids were
10 This study evaluated the contributions of carboxyl ester lipase (CEL) and pancreatic triglyceride
12 y deletion mutations in the last exon of the carboxyl ester lipase (CEL) gene, resulting in a CEL pro
15 ble number of tandem repeat (VNTR) domain of carboxyl ester lipase (CEL) presents an opportunity to s
16 G2 cells and primary hepatocytes showed that carboxyl ester lipase (CEL), a broad-spectrum lipase sec
17 nes favoring the production of R-9-PAHSA and carboxyl ester lipase (CEL), a PAHSA degradative enzyme,
19 tic lipase-related protein 2 (PNLIPRP2), and carboxyl ester lipase (CEL), which may leak into the vis
27 ly reduced in lipid rafts from the livers of carboxyl ester lipase-null mice compared with control an
29 eriments were performed to determine whether carboxyl ester lipase plays a role in scavenger receptor
31 of cell-surface proteoglycans also prevented carboxyl ester lipase stimulation of cholesteryl ester u
32 ock scavenger receptor BI function prevented carboxyl ester lipase stimulation of selective uptake in