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1  a carcinogenicity predictor, InCa (Index of Carcinogenicity).
2  human host, which may have implications for carcinogenicity.
3 ivity of NQO2-/- mice skin to benzo(a)pyrene carcinogenicity.
4 the liver is an organ susceptible to tobacco carcinogenicity.
5 ating evidence in the determination of human carcinogenicity.
6  for a chemical to be used for prediction of carcinogenicity.
7 ein may predispose human populations to lead carcinogenicity.
8 ossible effects of PKCI on susceptibility to carcinogenicity.
9 udy tobacco use as a model for environmental carcinogenicity.
10 ancer risk associated with estrogen-mediated carcinogenicity.
11  factor in protection against benzo(a)pyrene carcinogenicity.
12 ancer risk associated with estrogen-mediated carcinogenicity.
13  provides a basis for their mutagenicity and carcinogenicity.
14 nce of a Cr(III)-dependent pathway in Cr(VI) carcinogenicity.
15 ect DNA damage may also be involved in their carcinogenicity.
16  telomere length and the different stages of carcinogenicity.
17  by AA, - the cause of AA nephrotoxicity and carcinogenicity.
18 he mechanistic roles of HSA in modulating AA carcinogenicity.
19 f human serum albumin (HSA) in modulating AA carcinogenicity.
20  and eventually bring about mutagenicity and carcinogenicity.
21 y may not be involved in the safrole-induced carcinogenicity.
22 on to define the mechanisms underlying their carcinogenicity.
23 tic mechanisms may contribute to FA-mediated carcinogenicity.
24 t role in chemical metabolism, toxicity, and carcinogenicity.
25 rch for reliable parameters expressing their carcinogenicity.
26 g agent ezetimibe corroborates its potential carcinogenicity.
27  role for mutagenicity in TCE-induced kidney carcinogenicity.
28 aromatic amines, which may account for their carcinogenicity.
29 e-centric predictors of chronic toxicity and carcinogenicity.
30 underlying formaldehyde-induced toxicity and carcinogenicity.
31 as banned in children's sleepwear because of carcinogenicity.
32 l effects contribute to Acr mutagenicity and carcinogenicity.
33 us (HPV) types differ profoundly in cervical carcinogenicity.
34 dentify the genetic basis for HPV16's unique carcinogenicity.
35 n of signatures that predict mutagenicity or carcinogenicity.
36 gether, the data suggest PET-NPLs' potential carcinogenicity after extended exposure, highlighting se
37 s: (1) over 150 plastic additives have known carcinogenicity and (2) the majority (~90%) of plastic a
38 rea Under the ROC Curve (AUC) for predicting carcinogenicity and 82.3% AUC for predicting genotoxicit
39 cal toxicology assessment including systemic carcinogenicity and additional developmental and reprodu
40 c data hold promise for predicting long-term carcinogenicity and also for its ability to extrapolate
41 +)Fbxw7(DeltaG) mice may be used for testing carcinogenicity and drug screening.
42  a screening process for predicting chemical carcinogenicity and genotoxicity and characterizing mode
43                         Predictive models of carcinogenicity and genotoxicity were built using a rand
44                                Signatures of carcinogenicity and genotoxicity were compared with exte
45  hundreds of chemical compounds with varying carcinogenicity and genotoxicity.
46 h using gene expression profiling to predict carcinogenicity and infer MoAs of chemical perturbations
47                    DNA damage is a source of carcinogenicity and is also the source of the cytotoxici
48 gment-based approaches for the prediction of carcinogenicity and mutagenicity in methods described in
49 -dG lesion and contribute to the much higher carcinogenicity and mutagenicity of (+)-BPDE-2 compared
50 ity of PAH o-quinones may play a role in the carcinogenicity and mutagenicity of the parent hydrocarb
51  raise concerns for health effects including carcinogenicity and neurotoxicity.
52                    Key issues related to the carcinogenicity and noncancer health hazards of biphenyl
53 es to public health, mainly because of their carcinogenicity and oxidative potential.
54 anscriptome sheds light on mechanisms of EBV carcinogenicity and provides new leads for translational
55 he importance of metabolic activation in HCA carcinogenicity and the finding that several HCAs are ro
56 eoplastic cell line previously developed for carcinogenicity and tumor promotion studies.
57 xpression that may have implications for the carcinogenicity and/or therapeutic activity of the drug.
58 cal effects, such as toxicity, mutagenicity, carcinogenicity, and endocrine disrupter activity, are g
59 r of high-quality databases on mutagenicity, carcinogenicity, and skin sensitisation.
60 into 17 groups, based on exposure potential, carcinogenicity, and structural similarity.
61 llenges of assessing the scale of AA-related carcinogenicity, and the obstacles that must be overcome
62  probable, possible, or unclassifiable as to carcinogenicity are found to have greater binding affini
63 , but the molecular mechanisms underlying AA carcinogenicity are unclear.
64 challenges in the development of alternative carcinogenicity assays is the prediction of non-genotoxi
65 gent need for a systematic and comprehensive carcinogenicity assessment of plastic additives and regu
66          Little is known about the potential carcinogenicity associated with routine application of d
67 nic in humans based primarily on evidence of carcinogenicity at two sites (liver and lung) in male an
68                      Most TPs with increased carcinogenicity belonged to sulfonamides.
69 ws the range of uncertainty on potential TFE carcinogenicity but cannot conclusively confirm or refut
70 large number of chemicals not yet tested for carcinogenicity but to which people are exposed, the lim
71 aimed to resolve the issue of azathioprine's carcinogenicity by conducting a systematic review of the
72                      Due to its toxicity and carcinogenicity, chromatographic methods have been devel
73  high grade or worse, consistent with strong carcinogenicity, compared with 10.7% in women infected w
74 ficant risk to human health because of their carcinogenicity; consequently, materials that can be use
75                                 Quantitative carcinogenicity data are less strongly correlated with l
76                     In the absence of rodent carcinogenicity data for novel N-nitrosamines derived fr
77                          We tabulated rodent carcinogenicity data from the 1994 PDR for all drugs lis
78 urally related surrogates with robust rodent carcinogenicity data is recommended.
79 s between OW and FF; (2) evidence of mammary carcinogenicity, estrogenicity, or genotoxicity; and (3)
80                            Dismissing animal carcinogenicity findings would lead to human cancer case
81 eriments may underestimate the potential for carcinogenicity for compounds that follow this pattern.
82 correlation between DNA adduct formation and carcinogenicity for these compounds.
83         However, the underlying mechanism of carcinogenicity for UVA is not fully delineated.
84         The parent PAHs represent a range of carcinogenicity from inactive to highly potent.
85 chemicals with similar functions and similar carcinogenicity/genotoxicity profiles.
86 rouping chemicals with similar functions and carcinogenicity/genotoxicity profiles.
87 fication of carcinogens and understanding of carcinogenicity has become a main focus of biomedical re
88 antiepileptic drugs appear to be safe, as no carcinogenicity has been demonstrated either during regu
89 dies suggest that it may be clastogenic, its carcinogenicity has not been demonstrated in rodents.
90  a role for genotoxicity in biphenyl-induced carcinogenicity has not been established.
91  involved in TiO(2)-induced genotoxicity and carcinogenicity have not been clearly defined and are po
92 nogenic additives and additives with unknown carcinogenicity impacted similar biological pathways.
93  not significantly associated with long-term carcinogenicity in doses up to [Formula: see text].
94                                              Carcinogenicity in humans and rodents is generally consi
95 r, recent public concerns over its potential carcinogenicity in humans have generated calls for glyph
96 or biphenyl provides suggestive evidence for carcinogenicity in humans, based on increased incidences
97 ed mechanism underlying arsenic toxicity and carcinogenicity in humans.
98 ays involved in arsenic-induced toxicity and carcinogenicity in humans.
99 nt hepatic tumor promoter, with inconclusive carcinogenicity in humans.
100 ndrial processes involved in arsenic-induced carcinogenicity in humans.
101  occupationally exposed to BD and its potent carcinogenicity in laboratory mice.
102 osure to UVR is a contributing factor to its carcinogenicity in mice, and possibly in humans.
103                                         This carcinogenicity in rats has been linked to the formation
104 get site for chemically-induced toxicity and carcinogenicity in rodents.
105 ve been previously implicated as a source of carcinogenicity in tobacco and cigarette smoke.
106 eas of biomedicine, concerns regarding their carcinogenicity, inefficient dispersion in aqueous solut
107 ive, the rate-limiting step in N-nitrosamine carcinogenicity, involving cytochrome P450-mediated alph
108                                          BaP carcinogenicity is believed to occur mainly through its
109 both antiepileptic drugs, evidence for human carcinogenicity is not consistent and both are considere
110   The underlying mechanism of action for UVB carcinogenicity is well defined; however, the mechanisti
111 ion and human evidence of lung disorders and carcinogenicity, it is imperative to understand the effe
112 espread use, general toxicity, and potential carcinogenicity, its biodegradation has garnered signifi
113                    A pragmatic threshold for carcinogenicity may therefore exist for such genotoxins.
114                             Postinduction of carcinogenicity, mutations further propagate and drastic
115 st that LOH contributes significantly to the carcinogenicity of a variety of mutagens and raises the
116 nd current evidence for the mutagenicity and carcinogenicity of AA, and the effect of removing source
117  associated with both the nephrotoxicity and carcinogenicity of AAs, further increasing their disease
118  on Cancer but epidemiologic evidence on the carcinogenicity of acrylamide from dietary sources is li
119  that could explain an important step in the carcinogenicity of aflatoxin B1.
120                            Owing to the high carcinogenicity of aflatoxins, these toxic secondary met
121 dG adducts, which may be responsible for the carcinogenicity of alcoholic beverage consumption.
122           However, little is known about the carcinogenicity of arsenic at levels commonly observed i
123  best places in the world to investigate the carcinogenicity of arsenic.
124                                    Thus, the carcinogenicity of arsenite can be explained at least in
125            The mechanism responsible for the carcinogenicity of asbestos is not known.
126 ological and molecular data on the pulmonary carcinogenicity of benzene.
127                                          The carcinogenicity of BKPyV integration was proven to distu
128 xicology relies on animal testing to predict carcinogenicity of chemicals, often with limited human r
129 anism-based biomarker for rapid screening of carcinogenicity of CNTs and related nanomaterials for th
130 exposure, supporting its contribution to the carcinogenicity of combustion-derived PAHs in humans.
131 the antidiabetic activity of Cr(III) and the carcinogenicity of Cr(VI) compounds arise from similar m
132                                          The carcinogenicity of Cr(VI) is related to its solubility,
133                                The potential carcinogenicity of crystalline silica to humans remains
134                      Rationale: Although the carcinogenicity of diesel engine exhaust has been demons
135 lusively at extrahepatic sites, mediates the carcinogenicity of DMBA.
136 ation of MCF-10F cells in vitro confirms the carcinogenicity of E2, supporting the concept that this
137                           In this study, the carcinogenicity of ECS was tested in mice.
138 cer cases as the only means of demonstrating carcinogenicity of environmental agents.
139 ant role with regard to the genotoxicity and carcinogenicity of formaldehyde.
140 ic variation in xenobiotic metabolism in the carcinogenicity of hair dye use needs to be confirmed in
141        Our findings do not indicate that the carcinogenicity of high sun exposure can be counteracted
142 ole of this phenomenon in the regulation and carcinogenicity of HPV-16, 115 clinical samples were stu
143 , and these data further lend support to the carcinogenicity of HPV35 despite limited sequence hetero
144                                  Evidence of carcinogenicity of lipid-lowering drugs from clinical tr
145 s of compounds notorious both for the potent carcinogenicity of many of its members and for their wid
146                                  Because the carcinogenicity of N-nitrosamines depends on formation o
147 ferase (MGMT) is a major defense against the carcinogenicity of N-nitroso compounds and other alkylat
148                                The potential carcinogenicity of naphthalene (NA), a ubiquitous enviro
149                                          The carcinogenicity of nickel compounds has been well docume
150 volved in the essentiality, toxicity, and/or carcinogenicity of nickel compounds, a mRNA differential
151 red or less biased approach in assessing the carcinogenicity of night-shift work.
152                                    Given the carcinogenicity of NNN and the frequent use of nicotine
153 nism for the genotoxicity, mutagenicity, and carcinogenicity of PAHs.
154 d with log(k(az)/k(s)), so prediction of the carcinogenicity of PhIP and other HCAs or AAs based prim
155 lon, yet the role of colon metabolism on the carcinogenicity of PhIP is not clearly understood.
156 (CYP) enzymes are involved in activating the carcinogenicity of polycyclic aromatic hydrocarbons (PAH
157 g steroid hormones and may contribute to the carcinogenicity of polycyclic aromatic hydrocarbons (PAH
158              One potential mechanism for the carcinogenicity of PPs is epigenetic modulation of growt
159 ines (HAAs) has been related to the declared carcinogenicity of processed meats.
160      Shortcomings are length of protocol and carcinogenicity of retrorsine.
161                         The genotoxicity and carcinogenicity of tamoxifen have been attributed to met
162                                          The carcinogenicity of tamoxifen is ascribed to its genotoxi
163                      A MEDLINE search on the carcinogenicity of tamoxifen was conducted and the liter
164 gues that may provide an explanation for the carcinogenicity of the altered insulin protein?
165                                          The carcinogenicity of the more abundant UVA (320-400 nm) is
166 rpretation of mechanisms contributing to the carcinogenicity of these helminth infections, including
167 rpretation of mechanisms contributing to the carcinogenicity of these helminth infections, including
168             A strong correlation between the carcinogenicity of these isomers and methyl substitution
169 by ER would help in better understanding the carcinogenicity of these steroidogenic compounds and ass
170  of the clinical efficacy and, possibly, the carcinogenicity of these widely used chemotherapeutic ag
171 ta-estradiol (E2) has been implicated in the carcinogenicity of this hormone.
172 ullin (ADM) significantly contributes to the carcinogenicity of tobacco-activated AHR.
173 n a long-term rodent bioassay evaluating the carcinogenicity of triethanolamine, there was equivocal
174  growth and provide a new perspective on the carcinogenicity of ZEA.
175 EHS) reasons such as toxicity, mutagenicity, carcinogenicity, or for practical handling reasons such
176 ch as arachidonic acid, and to the toxicity, carcinogenicity, or teratogenicity of foreign compounds,
177 ts: Among agents with prior evidence of lung carcinogenicity, overall risk was elevated for cobalt (5
178 lay between all pathway responses leading to carcinogenicity predictions.
179 and interface properties we have developed a carcinogenicity predictor, InCa (Index of Carcinogenicit
180 nce of these two adduct groups for tamoxifen carcinogenicity remains to be established.
181 it a similar or higher level of mutagenicity/carcinogenicity, respectively, than their parent compoun
182 n- and off-target associated adverse events, carcinogenicity risk assessment, and guiding translation
183 ver 8000 studies including all available NTP carcinogenicity, short-term toxicity and genetic toxicit
184  also reviewed methods and interpretation of carcinogenicity studies in rodents and results of clinic
185 ghtly worsens sputum cytology, and in animal carcinogenicity studies, in which beta-carotene has esta
186                               A formaldehyde carcinogenicity study was conducted in which a major end
187 of 51 liver tissue samples from the original carcinogenicity study was conducted to determine the inc
188 ucts is crucial for delineating toxicity and carcinogenicity that might be caused by xenobiotic expos
189 estyle exposure that causes cancer, exerting carcinogenicity through >60 chemicals that bind and muta
190              The role of CYP1B1 in estradiol carcinogenicity thus depends on the identity of this sin
191 ly popular theory ascribes UVA (>320-400 nm) carcinogenicity to the ability of this wavelength to tri
192  to its widespread occurrence, toxicity, and carcinogenicity-to bioconcentrate in periphyton and thus
193             Transplacental inorganic arsenic carcinogenicity, together with postnatal exposure to die
194 of considerable interest because of its high carcinogenicity, was identified and quantified in the fo
195 emplified by chromosome instability-mediated carcinogenicity, we discuss the need to combine the stre
196 hanism by which folate deficiency influences carcinogenicity, we have analyzed the effect of folate d
197 TLE protocol suggest minimal genotoxicity or carcinogenicity with long-term hydroxyurea exposure.
198           Nickel compounds have demonstrated carcinogenicity without any associated mutagenesis, sugg

 
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