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1            The most frequent SSNVs shared by carcinomatous and sarcomatoid elements were in known ccR
2 heory, with a single progenitor resulting in carcinomatous and sarcomatous components, or alternative
3 into definitive uterine carcinosarcomas with carcinomatous and sarcomatous elements including heterol
4 g that both telomere subtypes contribute to 'carcinomatous catastrophy' under immunosuppression by se
5 rticularly in immunosuppressed individuals ('carcinomatous catastrophy').
6 oid elements arise from dedifferentiation of carcinomatous ccRCCs and implicate specific genes in thi
7 lmonary sarcomatoid carcinoma (PSC) contains carcinomatous component (CaC) and sarcomatous component
8 ous component always expresses ZEB1, and the carcinomatous component can also be positive.
9 he sarcomatous component developing from the carcinomatous component through epithelial-to-mesenchyma
10 y slowly compared to their non-myoepithelial carcinomatous counterparts and accumulate an abundant ex
11 47 x 10(-17)); TP53 mutations were absent in carcinomatous elements in nonhypermutated tumors and rar
12 ributed to the metaplastic transformation of carcinomatous elements into sarcoma.
13            In the remainder, sarcomatoid and carcinomatous elements shared 42% of somatic single-nucl
14 investigations has shifted from mutations in carcinomatous epithelium to disturbances of tissue organ
15  Laser capture microdissection of normal and carcinomatous glands, in conjunction with RNA amplificat
16 g staining was seen predominantly within the carcinomatous glandular epithelial cells.
17 ted as an "umbrella" term for dysplastic and carcinomatous lesions of the cornea and conjunctiva.
18 files were subtracted from the corresponding carcinomatous lesions, and a leave-one-out cross-validat
19  rate of apoptosis of both preneoplastic and carcinomatous mammary epithelial cells.
20 BM remodeling program that distinguishes the carcinomatous phenotype.
21                     Treatment of symptomatic carcinomatous pleural effusions is primarily directed at
22 using anti-L-plastin antiserum on normal and carcinomatous prostate tissues showed a very striking di
23 , and 0 to 28 in the normal, dysplastic, and carcinomatous samples, respectively (Kruskall-Wallis tes
24 performed exome sequencing of matched normal-carcinomatous-sarcomatoid specimens from 21 subjects.
25 ocking monoclonal antibodies (mAbs) in human carcinomatous states have been hampered by the limited a
26 y alpha2beta1 as a potential target in human carcinomatous states.
27 f *IUdR was observed in the hyperplastic and carcinomatous urothelium but not in the normal urotheliu