ライフサイエンスコーパス: cardiac cycle

1 stole (5.1 mm +/- 1.8) (32% variation during cardiac cycle).

2 during atrial contraction (40% variation per cardiac cycle).

3 rief muscle contractions ( approximately 4-7 cardiac cycles).

4 The TA was traced during a cardiac cycle.

5 data at &75% of the R-R interval during the cardiac cycle.

6 n with sympathetic nerve activity and/or the cardiac cycle.

7 anical, and electrical properties during the cardiac cycle.

8 the channel during the resting phase of the cardiac cycle.

9 red with one respiratory cycle performed per cardiac cycle.

10 of each two successive image frames over the cardiac cycle.

11 equally incremented time points through the cardiac cycle.

12 n be imposed during a specified phase of the cardiac cycle.

13 the period of minimal cardiac motion in the cardiac cycle.

14 hase) or last third (diastolic phase) of the cardiac cycle.

15 ons of influx vs. efflux via NCX during each cardiac cycle.

16 d in the plane of acquisition throughout the cardiac cycle.

17 wall during the predetermined phases of the cardiac cycle.

18 es in length and force that occur during the cardiac cycle.

19 culation eddies oscillate in size during the cardiac cycle.

20 cardial isopotential mapping during a single cardiac cycle.

21 values are quantified, for all frames in the cardiac cycle.

22 be attributed to the cortical control of the cardiac cycle.

23 ty of 30 miles per hour and was timed to the cardiac cycle.

24 haracterizing contractile changes during the cardiac cycle.

25 uced by impacts at any other time during the cardiac cycle.

26 delivered during the vulnerable phase of the cardiac cycle.

27 appearance of the myocardium throughout the cardiac cycle.

28 ver the entire epicardial surface during the cardiac cycle.

29 delivered during the vulnerable phase of the cardiac cycle.

30 8 versus 3.6 +/- 0.7 cm2, P = NS) during the cardiac cycle.

31 netium-99m sestamibi, using eight frames per cardiac cycle.

32 essment of phasic events associated with the cardiac cycle.

33 extremes of the pressure waveform during the cardiac cycle.

34 on at the best theoretical moment within the cardiac cycle.

35 ting cardiac wall deformation throughout the cardiac cycle.

36 ates changes in PLB-SERCA binding during the cardiac cycle.

37 cording compositional changes throughout the cardiac cycle.

38 pulsation of the arterial wall caused by the cardiac cycle.

39 g in three spatial dimensions throughout the cardiac cycle.

40 ntricular pressure and volume throughout the cardiac cycle.

41 ivalent spatial resolution and timing in the cardiac cycle.

42 show morphological changes during the entire cardiac cycle.

43 try and its metabolic activity vary over the cardiac cycle.

44 ates of segmental wall displacement during a cardiac cycle.

45 this myosin ATPase cycle and the macroscopic cardiac cycle.

46 s acquired during diastole of the subsequent cardiac cycle.

47 ts in a cyclical manner, consistent with the cardiac cycle.

48 and magnitude of calcium reuptake during the cardiac cycle.

49 fails to demonstrate similar modulations by cardiac cycle.

50 ges of luminal Ca(2+) that occur through the cardiac cycle.

51 ponents (PCs) representing breathing and the cardiac cycle.

52 changes in flow over different phases of the cardiac cycle.

53 were monitored during balloon deflation and cardiac cycle.

54 ), with dynamics severalfold slower than the cardiac cycle.

55 , and change in vascular blood volume over a cardiac cycle.

56 cast short-lived LLSTs in human blood during cardiac cycle.

57 perties of the aortic annulus throughout the cardiac cycle.

58 RVOT diameters (RVOTD max and min) during a cardiac cycle.

59 was reconstructed in 10% increments over the cardiac cycle.

60 trocardiogram at specific time points in the cardiac cycle.

61 re exposed to shear/fluid forces during each cardiac cycle.

62 arteriolar endothelium throughout the entire cardiac cycle.

63 and does not occur on the time scale of the cardiac cycle.

64 direction observed during each phase of the cardiac cycle.

65 unaffected by calcium during the course of a cardiac cycle.

66 observed contracting during one or multiple cardiac cycles.

67 ergy FLASH frames destroyed bubbles every 15 cardiac cycles.

68 ingle image from data acquired over multiple cardiac cycles.

69 ary views only with one frame every multiple cardiac cycles.

70 he transmission rate was once every multiple cardiac cycles.

71 Measurements were averaged over 10 cardiac cycles.

72 er diastolic pressures resulting from longer cardiac cycles.

73 sations originating from the respiratory and cardiac cycles.

74 R-R interval were tracked for 15 subsequent cardiac cycles.

75 by under 20% (p = 1) and 2% (p = 2) after 20 cardiac cycles.

76 -per-second (FPS) capable of resolving mouse cardiac cycles.

77 table biotic/abiotic interface during normal cardiac cycles.

78 orm from skin displacement images during the cardiac cycle.).

79 assessments were obtained in three different cardiac cycles (0.92-0.98), rather than in one (0.79-0.9

80 2.2 uW/mL 1.6; P = .03), and summed over the cardiac cycle (2.4 uJ/mL 1.0 vs 1.9 uJ/mL 0.6; P = .02)

81 m the left ventricle to end organs with each cardiac cycle (200 million litres of blood transported i

82 pattern in cardiogram waveform over a single cardiac cycle (~200 ms) of a mouse, which has not been o

83 All 82 subjects had 2 contrast-enhanced full cardiac cycle 4-dimensional computed tomography scans: a

84 ges with acquisition window that covered two cardiac cycles (acquisition time, 10-12 min; temporal re

85 edical imaging data, pulsatile flow to mimic cardiac cycles, adjustable parameters for various hydroc

86 and sustain vasodilatation for several more cardiac cycles after contraction.

87 , digestive activity, respiratory sounds and cardiac cycles, all with clinical grade accuracy and ind

88 of each marker every 16.7 ms throughout the cardiac cycle, allowing calculation of three-dimensional

89 gated MRI of the fetal heart throughout the cardiac cycle, allowing for immediate data reconstructio

90 Second, stimulus timing along the cardiac cycle also affected perception.

91 ration to simulate in vivo conditions of the cardiac cycle and found that I-O forces decrease with in

92 of EPSPs and spike potentials locked to the cardiac cycle and occurring at the end of diastole.

93 e WT-Parv sequesters Ca(2+) too early in the cardiac cycle and prematurely truncates sarcomere shorte

94 ped to detect five key frames throughout the cardiac cycle and respective dense deformation fields, a

95 vely synchronized, enabling compensation for cardiac cycle and respiratory phase.

96 onduction system in order to synchronize the cardiac cycle and steer cardiac pump function.

97 Flow velocity was measured over the whole cardiac cycle and the wave-free period.

98 ce-area product (RAP) were obtained for each cardiac cycle and their dynamic response to a step chang

99 o estimate all cardiac time intervals from 1 cardiac cycle and thereby obtain the myocardial performa

100 12 min; temporal resolution, 60 msec) or one cardiac cycle and time-of-flight (TOF) MR angiographic i

101 hase (45+/-15% of the R-R interval) of their cardiac cycle and underwent assessments of oxygen uptake

102 area of each burst were determined for each cardiac cycle and were placed into 3 mmHg intervals of d

103 paired both during the systolic phase of the cardiac cycle and when heartbeats evoke stronger cortica

104 ac function focuses on a limited sampling of cardiac cycles and has considerable inter-observer varia

105 ted length is maintained through consecutive cardiac cycles and that hysteresis is minimal.

106 ndividual characteristic ("core") pattern of cardiac cycles and then tracks the changes in the patter

107 e (i.e. number of sympathetic bursts per 100 cardiac cycles) and the area of each burst were determin

108 motion-artifact-free imaging throughout the cardiac cycle, and a fluorescent membrane staining proto

109 tically track the endocardium throughout the cardiac cycle, and show myocardial perfusion defects whe

110 ) greater variation in neural specificity to cardiac cycles, and (iii) neural network activity and ca

111 ge, binarizing and separating the image into cardiac cycles, and extracting data values from each of

112 ally induced vasodilatation peaks within 1-2 cardiac cycles, and thus is dissociated from the tempora

113 based cine sequences, acquisition over three cardiac cycles appears to be the optimal compromise, wit

114 ating or reducing the problems caused by the cardiac cycle are discussed, including electrocardiogram

115 d-point of the diagram (T50) at which 50% of cardiac cycles are associated with bursts, was inversely

116 ith a peak and trough that correlates with a cardiac cycle as revealed by a reference pulse oximeter

117 mensional blood-flow patterns throughout the cardiac cycle at the widest diameter of the Fontan pathw

118 However, the need to acquire multiple cardiac cycles at varying loads limits its applicability

119 Local blood flow and changes over the cardiac cycle before and after application of atropine w

120 ker coordinates were obtained throughout the cardiac cycle before and during each intervention.

121 The change in MAA throughout the cardiac cycle before ischemia was 17+/-4% in control ani

122 lumes were thus reconstructed throughout the cardiac cycle by combining transverse cardiac image sect

123 fy the role of isovolumic intervals during a cardiac cycle by in vivo visualization of left ventricul

124 ity in muscle nerves are phase-locked to the cardiac cycle by the sinoaortic baroreflexes.

125 g into account valve plane motion during the cardiac cycle by using Fourier-guided, dual-ROI analysis

126 be reliably estimated in humans from single cardiac cycles by a new method that has a potential for

127 ere acquired from three independent complete cardiac cycles by using a 15L8-MHz US transducer.

128 nce (Y: 26 +/- 8 vs. A: 50 +/- 7 bursts (100 cardiac cycles (CC))(-1) , P </= 0.01) were greater in t

129 es) analysis showed that 7 of 14 neurons had cardiac-cycle-correlated rhythms.

130 dinates of 3DE and CMR geometries across the cardiac cycle decreased from 7 +/- 1 to 4 +/- 1 mm for t

131 baseline buffer perfusion, maximal dP/dt per cardiac cycle decreased on average by 30.4%, 40.9%, and

132 es and the plots of dV/dt values through the cardiac cycle demonstrated more gradual developments of

133 had a significant reduction in MRI-assessed cardiac cycle-dependent change in aortic area and disten

134 lder patients with isolated DHF have reduced cardiac cycle-dependent changes in proximal thoracic aor

135 The goal of this study was to determine if cardiac cycle-dependent changes in proximal thoracic aor

136 We hypothesized that a reduction in cardiac cycle-dependent changes in thoracic aortic area

137 ant changes in mean thickness throughout the cardiac cycle, despite the inherent unsteadiness of the

138 the link between cortical neural firing and cardiac-cycle duration (CCD).

139 aging at pulsing intervals (PIs) of <1 to 30 cardiac cycles during an intravenous infusion of microbu

140 Cardiac cycle dynamics reflect underlying physiological

141 This flow occurred during most of the cardiac cycle, except for 19.6 +/- 2.3% of the cycle whe

142 al regularities induced a phase shift of the cardiac cycle exclusively in the MCS group.

143 eir prediction error representations between cardiac cycles exhibited higher learning rates and great

144 racterize beat-by-beat changes in FVC for 15 cardiac cycles following each MSNA burst and a peak resp

145 terize changes in left and right ventricular cardiac cycles following induction of experimental, stre

146 imate the optimal duration and timing in the cardiac cycle for coronary MR angiography.

147 f local myocardial count variations over the cardiac cycle for each patient, and then unpaired t test

148 were gated to the vulnerable portion of the cardiac cycle for the induction of VF.

149 ional imaging of the heart tube over various cardiac cycles for the measurement of cardiac structural

150 erves) was intact, rhythms correlated to the cardiac cycle frequency were found in 20/34 (59 %) of un

151 as well as local area change throughout the cardiac cycle from a dataset of 24 patients.

152 quantify thoracic aortic area throughout the cardiac cycle from cardiac magnetic resonance images and

153 s of maximum elastic myofiber stretch over a cardiac cycle from its corresponding local homeostatic s

154 ariations, and (3) removing unrepresentative cardiac cycles from the final data set.

155 ion of LV endocardial surface throughout the cardiac cycle, from which global and regional LV volume

156 f the mouse heart and for the animal's rapid cardiac cycle has proven to be a formidable challenge, r

157 cardial perfusion images obtained from whole cardiac cycles have lower myocardial intensity and great

158 -PH, RA pressure was elevated throughout the cardiac cycle (HFpEF-PH: 10 [8-14] versus PAH: 7 [5-10]

159 d permits analysis of events during a single cardiac cycle; however, at present, RT3-D imaging has po

160 Indeed, phasic fluctuations within the cardiac cycle impact neural and information processing.(

161 at 14 time points evenly spaced through the cardiac cycle in 10 volunteers and eight patients with C

162 CCTA covered the whole cardiac cycle in 10% increments.

163 istent through all twelve time points of the cardiac cycle in all five experimental groups and agreed

164 tricle from 3D MR-tagged images for the full cardiac cycle in dogs with cardiac failure and a left bu

165 In vivo DT-CMR was acquired throughout the cardiac cycle in healthy swine, followed by in situ and

166 b) to measure the ASA and VSA throughout the cardiac cycle in healthy volunteers using cardiovascular

167 on mitral valve (MV) deformation during the cardiac cycle in man.

168 -clamp technique to quantify I(f) during the cardiac cycle in mouse SAMs.

169 r distances remained constant throughout the cardiac cycle in normal hearts, during ischemia, and aft

170 changes in the left ventricle throughout the cardiac cycle in normal Wistar Kyoto rats (WKY) and also

171 stolic compared with systolic portion of the cardiac cycle in patients with CHF and CRT.

172 ed to measure arterial distension during the cardiac cycle in the brachial arteries of 361 children f

173 ation task, and effects of timing within the cardiac cycle) in twenty-six non-clinical participants a

174 e sequence (without DL, performed over 10-12 cardiac cycles) in regard to acquisition time, subjectiv

175 astolic cranial CSF flow velocity across the cardiac cycle, in HD (caudal flow: 0.17 +/- 0.07 mL/s, c

176 trasound transmission rate was one frame per cardiac cycle; in secondary and tertiary views, the tran

177 eformations during the various phases of the cardiac cycle, including aortoventricular and sinotubula

178 The cardiac cycle influences the blood flow velocity profile

179 ses are interrupted (delivered only once per cardiac cycle) instead of conventional 25- to 30-Hz fram

180 hat timing of sensory stimulation during the cardiac cycle interacts with perception.

181 We increased information by dividing the cardiac cycle into fewer bins, excluding cycles without

182 The cardiac cycle is composed of an anterograde beat in alte

183 NCX function during the cardiac cycle is determined by intracellular [Ca] and [N

184 The average blood velocity throughout the cardiac cycle is strongly correlated with pulmonary pres

185 resolved SL MR angiographic imaging over two cardiac cycles is a reliable clinical tool for cerebral

186 ements included blood pressure, body height, cardiac cycle length, carotid to femoral artery pulse wa

187 chords, which remain taut during the entire cardiac cycle, limit the motion of the anterior mitral l

188 A digital echocardiogram (often several one cardiac cycle loops) can be stored at one site and forwa

189 ior with intravascular ultrasound during the cardiac cycle may help to evaluate and quantify the seve

190 When the information for the cardiac cycle modulations and other consciousness-relate

191 rgely to the immediate vasodilatation (first cardiac cycle) observed in response to a brief, single c

192 tical flow analysis allows us to explore the cardiac cycle of the zebrafish and determine the changes

193 diminishes minimally or not at all during 3 cardiac cycles of the washout phase); and TMP grade 3 in

194 shout (blush is minimally persistent after 3 cardiac cycles of washout).

195 be the effects of ectopy and slow changes in cardiac cycles on the disturbances in the pattern.

196 that distinct time windows exist across the cardiac cycle, optimizing either perception or action.

197 By leveraging information across multiple cardiac cycles, our model can rapidly identify subtle ch

198 ith time-resolved SL MR angiography over one cardiac cycle (P < .001) and TOF MR angiography (P < .00

199 y wedge pressure at any point throughout the cardiac cycle (P=0.290).

200 Fifty per cent of IC neurons exhibited cardiac cycle periodicity, with activity occurring prima

201 eart rate (HR) and HR variability (HRV), and cardiac cycle phase shifts triggered by the processing o

202 and specificity of SG networked activity to cardiac cycle phases.

203 AVA) measured during the ejection phase of a cardiac cycle predicts the rate of hemodynamic progressi

204 ly applying this test to activity during the cardiac cycle proved ineffective because subjects-by-tre

205 resolved SL MR angiographic imaging over two cardiac cycles provided a median diagnostic confidence i

206 during tachycardia (mean trend, +15 ms/96.8 cardiac cycles; r(2)=0.92).

207 find that synchronizing imaging scans to the cardiac cycle reduces motion artifacts, significantly im

208 eart rate, a measure of the frequency of the cardiac cycle, reflects the health of the cardiovascular

209 tor neurones in the RVL, identified by their cardiac cycle-related probability of discharge, by the d

210 Here we investigate whether the cardiac cycle relates to learning-related internal repre

211 pression occurrence and magnitude during the cardiac cycle remain unknown.

212 ns (i.e., three-dimensional strains over the cardiac cycle) remain elusive, especially in mice.

213 ve sensing is coupled and modulated with the cardiac cycle remains largely unknown.

214 ow is shifted into the latter portion of the cardiac cycle, resulting in apparent prolongation of sys

215 data obtained at multiple time points in the cardiac cycle show that the glycocalyx remains hydrodyna

216 olite ratios and pH remained stable over the cardiac cycle, signal amplitudes correlated strongly wit

217 filament and undergo a powerstroke once the cardiac cycle starts.

218 n, and protein synthesis is regulated by the cardiac cycle, suggesting that mechanotransduction at th

219 ith time-resolved SL MR angiography over two cardiac cycles than with time-resolved SL MR angiography

220 uron showed hyperpolarizations locked to the cardiac cycle that started during late diastole and ende

221 longation of the QT interval, a phase of the cardiac cycle that underlies myocyte repolarization dete

222 During the cardiac cycle, the angle alpha between mitral annulus an

223 During the cardiac cycle, the release of Ca(2+) from the sarcoplasm

224 During the cardiac cycle, the value of [Na(+)]sm is constrained wit

225 in the control condition were coupled to the cardiac cycle, their length did not vary as a function o

226 We explain cardiac cycle timing effects in a predictive coding acco

227 aves in the novel signals, and confirm their cardiac-cycle timings in consistency with established ca

228 d relaxation (expressed as percentage of the cardiac cycle to adjust for differences in heart rate) s

229 ex and commonly reported effects linking the cardiac cycle to affective behaviour.

230 cing Na+) within the time course of a single cardiac cycle to avoid slow secondary effects.

231 somatosensory stimuli is altered across the cardiac cycle to evoke differential changes in bodily st

232 ography was reviewed for CSF, defined as >=3 cardiac cycles to opacify distal vessels with contrast.

233 tes are subjected to shear stress during the cardiac cycle under physiological or pathological condit

234 atients showed minor abnormalities of the RV cardiac cycle unrelated to QRSd.

235 xel intensity fluctuations generated by each cardiac cycle using a robust image processing routine, a

236 gered and at four different times during the cardiac cycle using acoustic triggering.

237 nd the stimulation mechanisms throughout the cardiac cycle using the pinwheel protocol, and the resul

238 lows for nanoparticle tracking over multiple cardiac cycles using a conventional laptop, providing a

239 13 ms), with typically 12 time frames in the cardiac cycle, using a short echo time (5 ms) multislice

240 One or more S-L cardiac cycles, usually the result of a ventricular bige

241 The rest period for coronary arteries in the cardiac cycle varies substantially from patient to patie

242 Average KE throughout the cardiac cycle was calculated.

243 Change in blood velocity over the cardiac cycle was determined to be approximately 27%.

244 Pulsatility over the cardiac cycle was evaluated by sending a series of pulse

245 at systole and stretch variation during one cardiac cycle was greater in symptomatic group than thos

246 The cardiac cycle was not sensitive to the processing of loc

247 hese data, epicardial deformation during the cardiac cycle was quantified by computing finite strain

248 Peak tissue displacement over the cardiac cycle was quantified in the cerebellum and brain

249 Annular contraction during the cardiac cycle was similar in the 2 groups (xenograft 9.2

250 ed ECG sample, 5-minute history of preceding cardiac cycles was also obtained.

251 ed early event-related potentials across the cardiac cycle, we conclude that these effects are not a

252 Regarding the cardiac cycle, we confirmed previous findings that tacti

253 Time intervals of the RV cardiac cycle were measured from Doppler recordings.

254 Sheep-specific simulations of the cardiac cycle were performed using both incompressible a

255 their variation during balloon deflation and cardiac cycles were measured to describe the 3D strut, c

256 (25 to 130 microm), in focus throughout the cardiac cycle, were recorded after infusion of acridine

257 le-shape depth) were measured throughout the cardiac cycle with dedicated quantification software.

258 assess isovolumic PEC and PFR phases of the cardiac cycle with high-frame-rate cine MR images, and P

259 Optogenetic stimulation of cardiac cycles with in-animal control and induction of h

260 l arteries changed systematically during the cardiac cycle, with the flattest profile occurring durin

261 ain pulsations driven by the respiration and cardiac cycles, with more pronounced effects in gray and