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1 Magnetic resonance analysis confirmed cardiac enlargement.
2 gery were congestive failure and progressive cardiac enlargement.
3 at lower, at 89.0%, in the 167 patients with cardiac enlargement.
4 eft ventricular hypertrophy, and symmetrical cardiac enlargement.
9 (2) a large reversible defect (n=36), or (3) cardiac enlargement and either increased pulmonary uptak
10 trate that VSM KATP channel GoF underlies CS cardiac enlargement and that CS-associated abnormalities
12 lly silenced in the postnatal heart, induces cardiac enlargement because of cardiomyocyte hyperplasia
13 results in widespread cardiomyocyte mitosis, cardiac enlargement, contractile failure, and death befo
14 ing severe hypertriglyceridemia, hepatic and cardiac enlargement, growth retardation, and premature m
16 and Darling in 1899 through observations of cardiac enlargement in endurance athletes, has evolved f
19 a proapoptotic Bcl2 family protein, mediates cardiac enlargement, reshaping, and dysfunction in mice
20 Rc1 was activated in pregnancy to facilitate cardiac enlargement that was reversed after delivery in
22 The appropriate management of patients with cardiac enlargement will remain a matter of clinical jud