ライフサイエンスコーパス: cardiac sympathetic nerve

1 lts in imbalanced activity in right and left cardiac sympathetic nerves.

2 enous NGF on ischemia-induced dysfunction of cardiac sympathetic nerves.

3 substantially decreased vesicular storage in cardiac sympathetic nerves.

4 arterial pressure (AP), heart rate (HR) and cardiac sympathetic nerve activity (CSNA) in urethane-ch

5 ion curves for changes in heart rate and non-cardiac sympathetic nerve activity (SNA, thoracic chain

6 to explore the relationship between EAT and cardiac sympathetic nerve activity in patients with hear

7 ation of histamine H(3) receptors (H(3)R) on cardiac sympathetic nerves also reduces carrier-mediated

8 on the relation between the distribution of cardiac sympathetic nerves and the occurrence of spontan

9 The condition of cardiac sympathetic nerves can be evaluated by (123)I-me

10 sensitive quantitative measures of regional cardiac sympathetic nerve densities in human hearts.

11 de robust and sensitive measures of regional cardiac sympathetic nerve densities.

12 an provide quantitative measures of regional cardiac sympathetic nerve density in human hearts using

13 ional relationship between HED retention and cardiac sympathetic nerve density.

14 amine infusion or by stimulation of efferent cardiac sympathetic nerves did not induce significant re

15 ed to the 8- to 13-Hz rhythm in the inferior cardiac sympathetic nerve discharge (SND) of urethane-an

16 amine H(3) receptors (H(3)Rs) are present in cardiac sympathetic nerve endings (cSNE) of animals and

17 s, at concentrations likely to be reached at cardiac sympathetic nerve endings in advanced congestive

18 Partial reinnervation of cardiac sympathetic nerves has been observed after heart

19 iac vagal (parasympathetic) and the inferior cardiac (sympathetic) nerves identify the cardiac parasy

20 The role of cardiac sympathetic nerves in regulating coronary blood

21 the Postn+ CFs from P0 to P6 led to altered cardiac sympathetic nerve patterning and a reduction in

22 uanidine ([(18)F]4F-MHPG, [(18)F]1) is a new cardiac sympathetic nerve radiotracer with kinetic prope

23 Most cardiac sympathetic nerve radiotracers are substrates of

24 lete atrioventricular (AV) block, we induced cardiac sympathetic nerve sprouting by infusing nerve gr

25 Chronic excessive norepinephrine (NE) causes cardiac sympathetic nerve terminal abnormalities, myocar

26 Noninvasive mapping of cardiac sympathetic nerve terminals reveals regionally i

27 inephrine uptake and storage capacity in the cardiac sympathetic nerve terminals which may reflect th

28 own-regulates norepinephrine exocytosis from cardiac sympathetic nerve terminals, in both normal and

29 nd can be associated with loss of functional cardiac sympathetic nerve terminals.

30 n norepinephrine reuptake and storage in the cardiac sympathetic nerve terminals.

31 h the regional norepinephrine content in the cardiac sympathetic-nerve terminals.

32 NE to ACh and we provide evidence that mouse cardiac sympathetic nerves transiently produce ACh after

33 e of norepinephrine that is transported into cardiac sympathetic nerve varicosities by the neuronal n

34 11C]phenylephrine (PHEN) is transported into cardiac sympathetic nerve varicosities by the neuronal n

35 (123)I-metaiodobenzylguanidine that measure cardiac sympathetic nerve varicosities.

36 oped as a radiotracer for imaging studies of cardiac sympathetic nerves with PET.