ライフサイエンスコーパス: cardiomegaly

1 s ablated and W(sh) mice develop symptoms of cardiomegaly.

2 s morphological findings or mild to moderate cardiomegaly.

3 ase was progressive, causing weight loss and cardiomegaly.

4 trophic signals and exhibit stress-dependent cardiomegaly.

5 eading secondarily to hepatosplenomegaly and cardiomegaly.

6 effects, with cardiac VDR activation causing cardiomegaly.

7 quently than the overfitted model, including cardiomegaly (153 [76.5%] of 200 vs 64 [32%] of 200, res

8 ibrosis (4.43%), pneumothoraces (2.71%), and cardiomegaly (2.26%).

9 A chest radiographic image suggested cardiomegaly and a computed tomographic scan demonstrate

10 The presence of both cardiomegaly and CHF in the two affected males and atria

11 e creatine kinase-Cre;Vps34(f/f) mice led to cardiomegaly and decreased contractility.

12 Chest x-ray revealed cardiomegaly and pulmonary vascular congestion.

13 nd of the first month of life, she developed cardiomegaly and signs of cardiac failure.

14 e autopsies of SCD cases consistently showed cardiomegaly and ventricular hypertrophy.

15 ce of severe myocarditis, with dysrhythmias, cardiomegaly, and cardiogenic shock.

16 failure, stroke, peripheral artery disease, cardiomegaly, and cardiomyopathy.

17 etardation, skeletal deformities, blindness, cardiomegaly, and die prematurely.

18 on are abnormal apical impulse, radiographic cardiomegaly, and q waves or left bundle branch block on

19 s mild morphogenetic defects and progressive cardiomegaly, and that Lamb1a functions to limit heart s

20 isted mobility, presence of heart failure or cardiomegaly any time before discharge, presence of peri

21 majority of Galphaq/beta2ARH mice died with cardiomegaly at 5 weeks.

22 AdTNFRI did not reverse cardiomegaly but abrogated myocardial inflammation.

23 All FK506 and CsA children and adults had cardiomegaly by HW, HW/BW (P(FK506 peds) <0.024, P(CsA p

24 Cardiomegaly by HW, HW/BW, and histology was uniformly p

25 An exemplary cardiomegaly case-control analysis showed significantly

26 methods show promising results in improving cardiomegaly classification accuracy, while they are sti

27 ectual disability (ID), atrial fibrillation, cardiomegaly, congestive heart failure (CHF), some somat

28 Automatic cardiomegaly detection has great significance in helping

29 f the central pulmonary arteries and massive cardiomegaly due to right ventricular and right atrial e

30 thoracic abnormalities included atelectasis, cardiomegaly, effusion, infiltration, mass, nodule, pneu

31 vascular hemolysis, exuberant hematopoiesis, cardiomegaly, glomerulosclerosis, visceral congestion, h

32 x 320 pixels) resolution inputs, emphysema, cardiomegaly, hernia, and pulmonary nodule detection had

33 inary decision networks targeting emphysema, cardiomegaly, hernias, edema, effusions, atelectasis, ma

34 by a range of anatomical defects, including cardiomegaly, hyperflexibility of the joints, hypertrich

35 Male metformin-exposed offspring also showed cardiomegaly, increased cardiac collagen and vascular sy

36 Hypertension, cardiomegaly, increased creatinine, overdose (primarily

37 riteria variables (dyspnea, pulmonary rales, cardiomegaly, interstitial or pulmonary edema on chest r

38 Radiographic evidence of cardiomegaly is common in patients with congenital compl

39 iovascular phenotypes and show that neonatal cardiomegaly is exclusively dependent on increased Igf2.

40 overexpression of IGF-1 in myocytes leads to cardiomegaly mediated by an increased number of cells in

41 ciated with increased likelihood of SCD from cardiomegaly (odds ratio, 1.08 [95% CI, 1.05-1.11]; P<0.

42 oled sensitivity, 76%; 95% CI, 62%-90%), and cardiomegaly on chest radiograph (pooled sensitivity, 89

43 cardia, elevated jugular venous pressure, or cardiomegaly on chest radiograph.

44 ourth heart sound on examination, absence of cardiomegaly or signs of congestive heart failure on che

45 ratio (p < 0.001), subjective impressions of cardiomegaly (p < 0.01), and increased VPW (p = 0.02).

46 ochondrodysplasia, patent ductus arteriosus, cardiomegaly, pericardial effusion, and lymphoedema.

47 eft ventricular dysfunction, increasing age, cardiomegaly, peripheral vascular disease, chronic renal

48 The most common radiological findings are cardiomegaly, pleural effusion, signs of heart failure,

49 e used to train a neural network to identify cardiomegaly, pulmonary congestion, pleural effusion, pu

50 her stratification of reports by presence of cardiomegaly, pulmonary edema, pleural effusion, infiltr

51 werful overall predictors, with only age and cardiomegaly showing a better independent association wi

52 ed blood cells, consistent with the observed cardiomegaly, splenomegaly, elevated bilirubin levels an

53 ation, increased vascularization, and causes cardiomegaly through persistent addition of wall myocard

54 Cardiomegaly, urinary incontinence, non-epithelial skin

55 to deposition of immunocomplexes followed by cardiomegaly with ventricular dilation and hypertrophy,