ライフサイエンスコーパス: cardiorespiratory

1 These results suggest that persistent cardiorespiratory abnormalities caused by LT-IH are medi

2 vance of this dual chemoreceptor feedback to cardiorespiratory abnormalities present in diseases in w

3 rainstem DC potential and neuronal activity, cardiorespiratory activity and local tissue oxygen were

4 mbedded in the brainstem networks regulating cardiorespiratory activity and the response to glucopriv

5 er into the medullary raphe had no effect on cardiorespiratory activity or the chemoreflex.

6 n vivo, ATP injection into the NTS increased cardiorespiratory activity; however, injection of a P2-r

7 spiratory control and may underlie important cardiorespiratory adjustments for gas exchange improveme

8 rimary termination and integration point for cardiorespiratory afferents in the brainstem.

9 In short, C1 neurons orchestrate cardiorespiratory and arousal responses to somatic stres

10 Cardiorespiratory and arterial blood gases were collecte

11 hly polluted air resulted in weight gain and cardiorespiratory and metabolic dysfunction.

12 deaths but also may increase mortality from cardiorespiratory and other causes.

13 Our study shows persistent WTC-Cardiorespiratory and Vascular Dysfunction (WTC-CaRVD),

14 n severe toxicities, which can lead to rapid cardiorespiratory and/or neurological deterioration.

15 rse with numerous immunological, infectious, cardiorespiratory, and psychological events, he was disc

16 , unknown cause [n=2], cardiac arrest [n=1], cardiorespiratory arrest [n=1]) and two (1%) were report

17 tween postictal generalized EEG suppression, cardiorespiratory arrest and sudden death following a se

18 Preventable cardiorespiratory arrest from underlying cardiac dysrhyt

19 he suffered a pulseless electrical activity cardiorespiratory arrest from which he could not be resu

20 at RRSs are associated with reduced rates of cardiorespiratory arrest outside of the intensive care u

21 d unrelated to study treatment was reported (cardiorespiratory arrest).

22 e significantly for tachycardia, seizure, or cardiorespiratory arrest.

23 al hemorrhage, and 30% of patients following cardiorespiratory arrest.

24 en death at infant age by brainstem-mediated cardiorespiratory arrest.

25 photobleaching, was not affected just after cardiorespiratory arrest; and (iii) Aqp4 gene deletion d

26 ized rates for medicine, surgery/gynecology, cardiorespiratory, cardiovascular, and neurology cohorts

27 During hypoxia, PB resulted in increased cardiorespiratory coherence at the PB frequency.

28 arization (SD) in dorsal medulla, leading to cardiorespiratory collapse.

29 , in some cases, individuals present serious cardiorespiratory complications with possible long-term

30 encephalitis (n = 21), and encephalitis with cardiorespiratory compromise (n = 11).

31 rticipants (2.0%) were found to have serious cardiorespiratory conditions that had been previously mi

32 Arch (PRSx8-ArchT-EYFP-LVV) and measured the cardiorespiratory consequences of Arch activation (10 s)

33 and peripheral feedback mechanisms governing cardiorespiratory control and may underlie important car

34 he carotid body (CB) chemoreceptors improves cardiorespiratory control and survival during heart fail

35 EAAT2 modulates cardiorespiratory control and tempers excitatory cardior

36 also noted in ventral areas associated with cardiorespiratory control, including the gigantocellular

37 tus solitarii (nTS), an important nucleus in cardiorespiratory control.

38 information to brainstem nuclei involved in cardiorespiratory control.

39 ation has a significantly stronger effect on cardiorespiratory coupling than healthy aging.

40 ration increased ICU admissions triggered by cardiorespiratory criteria, whereas admissions triggered

41 n the late 2000s, with a larger increase for cardiorespiratory deaths than for deaths from other caus

42 rlier decades, for older populations and for cardiorespiratory deaths.

43 Glu5 signaling (i.e. seizures), or affecting cardiorespiratory defects in RS model mice.

44 was assessed using a home-based multichannel cardiorespiratory device.

45 Chronic cardiorespiratory disease is associated with low birthwe

46 assessed the impact of HAP on mortality and cardiorespiratory disease.

47 r cooking is a risk factor for mortality and cardiorespiratory disease.

48 ed as a cause of hypertension in a number of cardiorespiratory diseases states and has therefore been

49 ion of autonomic morbidities associated with cardiorespiratory diseases, such as sleep-disordered bre

50 the role of the carotid body chemoreflex in cardiorespiratory diseases.

51 chemoreflex function is strongly related to cardiorespiratory disorders and disease progression in h

52 tion might be of therapeutic value to reduce cardiorespiratory dysfunction and improve survival durin

53 This ultimately results in cardiorespiratory dysfunction, which is the predominant

54 sessed short-term associations between daily cardiorespiratory ED visit counts and daily levels of 24

55 showed particularly strong associations with cardiorespiratory ED visit outcomes.

56 Their complex cardiorespiratory effects are presumably mediated by the

57 Neuronal excitation and cardiorespiratory effects following EAAT2 inhibition wer

58 These cardiorespiratory effects were prevented via ganglionic

59 e conducted a time-series study of PM2.5 and cardiorespiratory emergency department (ED) visits in th

60 s have significant associations with certain cardiorespiratory end points, such as asthma, congestive

61 netic evidence for the polygenetic nature of cardiorespiratory endurance and be used as genetic bioma

62 ization as well as severe cardiovascular and cardiorespiratory events.

63 ophysiological pathways linking exposure and cardiorespiratory events.

64 otected' genetic test result for obesity via cardiorespiratory exercise capacity (experiment 1, N = 1

65 ge of 66 years (range 26-86 years) underwent cardiorespiratory exercise testing before major hepatobi

66 s, the natural history is characteristically cardiorespiratory failure and death in the first year of

67 Cardiorespiratory failure is the leading cause of death

68 Cardiorespiratory failure is the most common cause of su

69 g the study (pulmonary artery thrombosis and cardiorespiratory failure); neither death was judged to

70 ures frequently led to apneas, brainstem SD, cardiorespiratory failure, and death.

71 nd/or sudden death due to apnea episodes and cardiorespiratory failure.

72 n is a rescue therapy used to support severe cardiorespiratory failure.

73 nd monitored cases, as well as human seizure cardiorespiratory findings related to SUDEP, and SUDEP a

74 cal care unit as younger people with similar cardiorespiratory fitness (13 vs 12; P = 0.08 and 1 vs 1

75 The primary outcome measures were weight and cardiorespiratory fitness (as measured with the 6-minute

76 The present review focuses on cardiorespiratory fitness (commonly measured by maximal

77 is study sought to determine the capacity of cardiorespiratory fitness (CRF) algorithms without exerc

78 t of a lifestyle intervention and changes in cardiorespiratory fitness (CRF) and body mass index on r

79 al factors influence the association between cardiorespiratory fitness (CRF) and cardiovascular disea

80 Cardiorespiratory fitness (CRF) and endurance performanc

81 A positive association between cardiorespiratory fitness (CRF) and white matter integri

82 ce has firmly established that low levels of cardiorespiratory fitness (CRF) are associated with a hi

83 Cardiorespiratory fitness (CRF) as assessed by formalize

84 features around the home and workplace with cardiorespiratory fitness (CRF) based on a treadmill tes

85 <.05) associated with BF%, diastolic BP, and cardiorespiratory fitness (CRF) for the Chu et al PT onl

86 has been shown to be effective in improving cardiorespiratory fitness (CRF) in patients with systoli

87 Poor cardiorespiratory fitness (CRF) is an independent risk f

88 It is well established that cardiorespiratory fitness (CRF) is inversely associated

89 Cardiorespiratory fitness (CRF) is not routinely measure

90 Evidence on the effect of cardiorespiratory fitness (CRF) on age-related longitudi

91 Cardiorespiratory fitness (CRF) refers to the capacity o

92 rength, muscular endurance, flexibility, and cardiorespiratory fitness (CRF) were performed in 22 int

93 ave examined the association between LTL and cardiorespiratory fitness (CRF), an enduring trait influ

94 the individual and joint associations among cardiorespiratory fitness (CRF), body mass index, and he

95 ted the association between overall diet and cardiorespiratory fitness (CRF).

96 d self-reported physical activity, predicted cardiorespiratory fitness (cycle ergometer test), obesit

97 Data were collected on cardiorespiratory fitness (e.g., peak oxygen uptake), di

98 s well known, there is a lack of data on how cardiorespiratory fitness (hereafter referred to as fitn

99 tive functioning predisposed to better adult cardiorespiratory fitness (neuroselection).

100 Move and OnTrack resulted in less decline in cardiorespiratory fitness (P < .001), better physical fu

101 >/=2 compared to those with METs gain <2 in cardiorespiratory fitness (p < 0.001 for both).

102 piratory fitness compared to adequate or low cardiorespiratory fitness (p < 0.001 for both).

103 g program (25+/-9 miles/wk) led to increased cardiorespiratory fitness (peak oxygen consumption, 44.6

104 ciation was found between sedentary time and cardiorespiratory fitness (r = -.13, p>.05).

105 Secondary outcomes measured were cardiorespiratory fitness (VO2 peak) and body compositio

106 Exercise improved cardiorespiratory fitness (VO2 peak) compared with the C

107 Cardiorespiratory fitness [measured by the predicted max

108 Cardiorespiratory fitness also declines with age, and th

109 udy aimed to define the relationship between cardiorespiratory fitness and age in the context of post

110 independent and linked associations between cardiorespiratory fitness and age on postsurgical mortal

111 nsity exercise training was found to improve cardiorespiratory fitness and attenuate cutaneous vasodi

112 mary aim of this study was to investigate if cardiorespiratory fitness and body composition are risk

113 Cardiorespiratory fitness and body mass index (BMI) were

114 ficant epidemiologic observations connecting cardiorespiratory fitness and cancer.

115 The association between cardiorespiratory fitness and covariate adjusted decline

116 M1, GMPS, COL18A1 and PRKCA) associated with cardiorespiratory fitness and endurance performance in C

117 elationships between hard physical activity, cardiorespiratory fitness and health parameters.

118 hese findings support the importance of high cardiorespiratory fitness and healthy body weight during

119 re is a graded, inverse relationship between cardiorespiratory fitness and incident AF, especially am

120 ave demonstrated strong associations between cardiorespiratory fitness and lower cardiovascular disea

121 2-hour glucose level (primary outcomes) and cardiorespiratory fitness and measures of insulin action

122 tive tests, there was no association between cardiorespiratory fitness and midlife cognitive function

123 inverse, dose-dependent association between cardiorespiratory fitness and mortality is well-establis

124 An inverse association between cardiorespiratory fitness and mortality was robustly dem

125 cacy of 2 exercise training dose regimens on cardiorespiratory fitness and patient-reported outcomes

126 ge, during and after treatment) and improves cardiorespiratory fitness and physical function.

127 een increased levels of physical activity or cardiorespiratory fitness and reduced coronary heart dis

128 nd no weight loss demonstrate that increased cardiorespiratory fitness and reduced intra-abdominal ad

129 The primary outcomes were cardiorespiratory fitness and skeletal muscle (vastus la

130 ned the effects of simvastatin on changes in cardiorespiratory fitness and skeletal muscle mitochondr

131 Simvastatin attenuates increases in cardiorespiratory fitness and skeletal muscle mitochondr

132 This study sought to evaluate the role of cardiorespiratory fitness and the incremental benefit of

133 y while promoting physical activity, ET, and cardiorespiratory fitness are needed throughout the heal

134 no evidence for a neuroprotective effect of cardiorespiratory fitness as of midlife.

135 oper Center Longitudinal Study who underwent cardiorespiratory fitness assessment at a mean age of 49

136 Improvement in cardiorespiratory fitness augments the beneficial effect

137 rences were observed in daily step counts or cardiorespiratory fitness between the groups.

138 d mild anemia might be associated with lower cardiorespiratory fitness but not with anaerobic fitness

139 Exercise training improved cardiorespiratory fitness by 5.0 ml kg(-1) min(-1) (95%

140 trategies was greatest in patients with high cardiorespiratory fitness compared to adequate or low ca

141 vity (>/=9 METs) holds greater potential for cardiorespiratory fitness compared to physical activity

142 s additional improvements in S(I), S(G), and cardiorespiratory fitness compared with a sedentary life

143 We review the prognostic utility of cardiorespiratory fitness compared with obesity and the

144 Higher cardiorespiratory fitness during childhood is associated

145 milar if not indeed superior improvements in cardiorespiratory fitness for a given training volume an

146 hard physical activity were associated with cardiorespiratory fitness for boys (F = 5.64, p<.01) whe

147 eficial effects of physical activity /ET and cardiorespiratory fitness for the prevention of chronic

148 ty decreased significantly in the group with cardiorespiratory fitness gain >/=2 METs as compared to

149 ts effect on AF recurrence or the benefit of cardiorespiratory fitness gain is unknown.

150 Impact of cardiorespiratory fitness gain was ascertained by the ob

151 Participants with better cardiorespiratory fitness had higher cognitive test scor

152 atory fitness and the incremental benefit of cardiorespiratory fitness improvement on rhythm control

153 Further adjustment for cardiorespiratory fitness in a subset of 572610 men with

154 chronic diseases and is associated with low cardiorespiratory fitness in adults.

155 ive breastfeeding has a beneficial effect on cardiorespiratory fitness in children and adolescents.

156 ysical activity subcomponents are related to cardiorespiratory fitness in children.

157 se relationship between cancer incidence and cardiorespiratory fitness in large population studies.

158 Higher cardiorespiratory fitness in middle age is strongly asso

159 tion of health care costs in later life with cardiorespiratory fitness in midlife after adjustment fo

160 e, is associated with modest improvements in cardiorespiratory fitness in patients previously treated

161 ctivity, exercise training (ET), and overall cardiorespiratory fitness in the prevention and treatmen

162 Cardiorespiratory fitness increased by 10% (p < 0.05) in

163 Cardiorespiratory fitness is an independent predictor of

164 Poor cardiorespiratory fitness is an independent risk factor

165 On the other hand, high cardiorespiratory fitness is known to be a strong predic

166 training (ET) leading to improved levels of cardiorespiratory fitness is needed in all age groups, r

167 ramming effect of exclusive breastfeeding on cardiorespiratory fitness is of public health interest.

168 Although cardiorespiratory fitness is protective against incident

169 Cardiorespiratory fitness levels were assessed between 1

170 Energy metabolism, BC, and cardiorespiratory fitness may be associated with weight

171 well as the effect of physical activity and cardiorespiratory fitness on cardiovascular diseases.

172 , there are few data regarding the effect of cardiorespiratory fitness on health care costs independe

173 activity/exercise training, and increases in cardiorespiratory fitness on the prognosis of obese pati

174 Cardiorespiratory fitness predicts arrhythmia recurrence

175 Patients older than 75 years with low cardiorespiratory fitness spent a median of 11 days long

176 Older people with normal cardiorespiratory fitness spent the same number of days

177 nsitions revealed little variability between cardiorespiratory fitness tertiles.

178 axial accelerometers and undertook a maximal cardiorespiratory fitness test.

179 Adult cardiorespiratory fitness was assessed using a submaxima

180 To determine a dose response, cardiorespiratory fitness was categorized as: low (<85%)

181 Cardiorespiratory fitness was estimated by maximal metab

182 To determine whether cardiorespiratory fitness was longitudinally associated

183 Low cardiorespiratory fitness was strongly associated with l

184 sociations of habitual physical activity and cardiorespiratory fitness with IHTG and the prevalence o

185 The implications of cardiorespiratory fitness with prognosis are discussed,

186 cent studies investigated the association of cardiorespiratory fitness with white matter microstructu

187 muscle (low percentage of lean mass and low cardiorespiratory fitness) are likely to contribute thes

188 ody mass index, systolic blood pressure, and cardiorespiratory fitness).

189 r insulin sensitivity and secretion, greater cardiorespiratory fitness, and a predominantly lower bod

190 ther, and to what extent, physical activity, cardiorespiratory fitness, and obesity at age 16 mediate

191 factors influence physical activity levels, cardiorespiratory fitness, and risk of death.

192 djustment or not for key confounders such as cardiorespiratory fitness, and to the lack of consensus

193 hanges in myocardial structure and function, cardiorespiratory fitness, and traditional cardiac risk

194 ardial infarction, type 2 diabetes mellitus, cardiorespiratory fitness, body mass index, systolic blo

195 re made for height, weight, body mass index, cardiorespiratory fitness, cognitive ability, and socioe

196 demonstrated benefits in muscular strength, cardiorespiratory fitness, functional task performance,

197 Cardiorespiratory fitness, homeostasis model assessment

198 improvements in SI and that only EX improved cardiorespiratory fitness, mitochondrial respiration and

199 Low cardiorespiratory fitness, obesity, and the combination

200 Exercise training has been shown to improve cardiorespiratory fitness, physical capacity, and qualit

201 It is also possible that low cardiorespiratory fitness, rather than overweight or obe

202 included physical activity (steps per day), cardiorespiratory fitness, self-efficacy, healthy living

203 ow-up duration between the groups defined by cardiorespiratory fitness.

204 en childhood cognitive functioning and adult cardiorespiratory fitness.

205 itively associated (beta = .45, p<.001) with cardiorespiratory fitness.

206 ociated with 10-14 year-old schoolchildren's cardiorespiratory fitness.

207 relationship between sedentary behaviour and cardiorespiratory fitness.

208 1) when compared with patients with adequate cardiorespiratory fitness.

209 behaviour as a means to maintain or improve cardiorespiratory fitness.

210 ence interval = -0.039, -0.011), but not via cardiorespiratory fitness.

211 weight gain, cigarette smoking, and loss of cardiorespiratory fitness.

212 trics of physiological performance including cardiorespiratory function (heart rate [fH ] and ventila

213 Evidence of improved cardiorespiratory function after pectus excavatum repair

214 ng the constellation of factors that bear on cardiorespiratory function and that become intricately e

215 spiratory testing and techniques to preserve cardiorespiratory function before elective surgery in ol

216 Whereas cardiorespiratory function has long been applied by card

217 uronal activity and thereby basal and reflex cardiorespiratory function is unknown.

218 nistration to achieve supranormal indices of cardiorespiratory function, which has led to the advent

219 lays on buffering nTS excitation and overall cardiorespiratory function.

220 produced neuronal excitation to alter basal cardiorespiratory function.

221 operties, and ultimately on basal and reflex cardiorespiratory function.

222 Cardiorespiratory functions in mammals are exquisitely s

223 ry reflex response, with profound effects on cardiorespiratory functions.

224 channels in the spontaneous firing in these cardiorespiratory GABAergic neurons that possess a pacem

225 We aimed to investigate the cardiorespiratory health effects of particulate ambient

226 nception and Nov 28, 2016, investigating the cardiorespiratory health effects of particulate ambient

227 l in the nucleus tractus solitarii (nTS) for cardiorespiratory homeostasis and initiation of sensory

228 Maintenance of cardiorespiratory homeostasis depends on autonomic refle

229 for maintenance of carotid body function and cardiorespiratory homeostasis.

230 less than 10 mum (PM10) and daily emergency cardiorespiratory hospitalizations in Hong Kong, China,

231 Respiratory rates, wave morphology, and cardiorespiratory information were derived from the ECG

232 applied to address earlier identification of cardiorespiratory insufficiency and direct focused, pati

233 niques can be used to parsimoniously predict cardiorespiratory insufficiency.

234 This cardiorespiratory interaction may arise from interaction

235 f H is evidenced by a respiratory influence (cardiorespiratory interaction) on heart rate variability

236 rrhythmia represent different aspects of the cardiorespiratory interaction, and that key physiologic

237 ho did not panic displayed signs of impaired cardiorespiratory interoception, including a complete ab

238 ions: a panic induction and an assessment of cardiorespiratory interoception.

239 each breath to evaluate the significance of cardiorespiratory matching during diving.

240 nted and trained geese, collecting the first cardiorespiratory measurements of bar-headed geese flyin

241 Cardiorespiratory measures showed that apnea improved du

242 inary assessment of efficacy using automated cardiorespiratory measures, EEG, a set of RTT-oriented c

243 Cardiorespiratory monitoring is crucial for the diagnosi

244 ir pollution is associated with increases in cardiorespiratory morbidity and mortality in LMIC's, wit

245 articulate matter (PM2.5) is associated with cardiorespiratory morbidity and mortality, but the mecha

246 nnual PM(10) exposure with nonaccidental and cardiorespiratory mortality in the Latium region, even o

247 arction, stroke, heart failure) and COVID-19 cardiorespiratory ordinal severity score (worst to best:

248 ty of the preBotC, with a role in generating cardiorespiratory oscillations.

249 y cause or the first incidence of a nonfatal cardiorespiratory outcome.

250 assays drives higher risk ratios for certain cardiorespiratory outcomes than PM mass, suggesting OP m

251 mergency department (ED) visits for selected cardiorespiratory outcomes were obtained for the five-co

252 ) was associated with ED visits for multiple cardiorespiratory outcomes, providing support for the ut

253 The burden of cardiorespiratory, paediatric, and maternal diseases ass

254 2, 2020, that evaluated the risk of adverse cardiorespiratory, paediatric, and maternal outcomes fro

255 Cardiorespiratory parameters and heart rate (HR) variabi

256 tional analyses excluded that measured basic cardiorespiratory parameters or interoceptive sensitivit

257 We investigate whether and how cardiorespiratory phase synchronization (CRPS) responds

258 enetic risk information changed individuals' cardiorespiratory physiology, perceived exertion and run

259 esponse to hypoxia and hypercapnia, and 24-h cardiorespiratory recording.

260 We evaluated autonomic and cardiorespiratory recovery following aerobic exercise in

261 of SNA is important - being recruited under cardiorespiratory reflex conditions and elevated in the

262 ence sympathetic nervous system activity and cardiorespiratory reflex function in health and disease.

263 wo classes of SPNs in situ to their roles in cardiorespiratory reflex integration and have shown that

264 late these characteristics to their roles in cardiorespiratory reflex integration.

265 nformation processing.SIGNIFICANCE STATEMENT Cardiorespiratory reflexes maintain autonomic balance an

266 ng than Wt in the hindbrain, most notably in cardiorespiratory regions of the nucleus tractus solitar

267 ifurcation and has a well-recognized role in cardiorespiratory regulation.

268 This study aimed to develop a cardiorespiratory-resolved 3D magnetic resonance imaging

269 of three types of feedback afferents on the cardiorespiratory response to voluntary, rhythmic exerci

270 hogenesis, how PNE affects the SLCF-mediated cardiorespiratory responses remains unexplored.

271 iorespiratory control and tempers excitatory cardiorespiratory responses to activation of the periphe

272 role of ORX in the anxiety-like behavior and cardiorespiratory responses to acute exposure to a thres

273 PNE rat pups at postnatal days 11-14: 1) the cardiorespiratory responses to intralaryngeal applicatio

274 The persistent cardiorespiratory responses to LT-IH were associated wit

275 Acute cardiorespiratory responses to O(2) deficiency are essen

276 ts continuously rated the intensity of their cardiorespiratory sensation using a dial.

277 terenol elicited dose-dependent increases in cardiorespiratory sensation, with all participants repor

278 y a role in dynamically detecting changes in cardiorespiratory sensation.

279 ympathetic arousal, heart rate increase, and cardiorespiratory sensation.

280 Asian patients had the highest COVID-19 cardiorespiratory severity at presentation (adjusted odd

281 Changes in cardiorespiratory status associated with postnatal CMV i

282 cemia, infection, and hypotension and elicit cardiorespiratory stimulation, adrenaline and adrenocort

283 imulation of these neurons produces vigorous cardiorespiratory stimulation, sighing, and arousal from

284 most effects of acute hypoxia, specifically cardiorespiratory stimulation, sighs, and arousal.

285 erentially respond during the peak period of cardiorespiratory stimulation.

286 nable period of time after the withdrawal of cardiorespiratory support (WCRS).

287 eath in less than 60 min after withdrawal of cardiorespiratory support conducted in 28 accredited int

288 Death within 60 minutes of withdrawal of cardiorespiratory support occurred in 377 (49.3%).

289 , including targeted temperature management, cardiorespiratory support, and percutaneous coronary int

290 th within 60 minutes following withdrawal of cardiorespiratory support.

291 ing death within 60 minutes of withdrawal of cardiorespiratory support.

292 ients die within 60 minutes of withdrawal of cardiorespiratory support.

293 e importance of therapeutic targeting of the cardiorespiratory system.

294 should consider both the prognostic value of cardiorespiratory testing and techniques to preserve car

295 Clinical assessment, diagnostic imaging, and cardiorespiratory testing of patients with pectus excava

296 Moderate-quality evidence supported use of cardiorespiratory training to improve maximum walking sp

297 Cardiorespiratory training, repetitive task training, an

298 ization and intensive medical care center on cardiorespiratory treatment, a growing number of cases p

299 tronic simulator for 6 hours while their own cardiorespiratory variables and the quality of the deliv

300 Cardiorespiratory variables were continuously recorded t