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1 e wasting in an inflammatory myopathy model (cardiotoxin).
2 es before and after muscle injury induced by cardiotoxin.
3 n was induced by injuring mouse muscles with cardiotoxin.
4 d dramatically from 1 to 3 d after injury by cardiotoxin.
5 doxorubicin-induced cardiotoxicity and other cardiotoxins.
6 generation, we injured wild-type muscle with cardiotoxin and found that Akt induced a faster regenera
7 protected muscle from both acute injury with cardiotoxin and from chronic muscle disease in the mdx o
8  between two different muscle injury models, cardiotoxin and glycerol.
9 ted in PDGFRbeta(+) cells are protected from cardiotoxin and laceration-induced skeletal muscle fibro
10 nin-alpha2-deficient muscle was damaged with cardiotoxin and muscle regeneration quantified.
11 Duchenne muscular dystrophy, and 2 myotoxin (cardiotoxin and notexin) muscle injury models.
12 ble disulfide-containing proteins, including cardiotoxin and prion aggregates.
13                                         Beta-cardiotoxin (B-CTX) from the king cobra venom (Ophiophag
14 kinetics of over 100 proteins in response to cardiotoxin (CTX) induced muscle damage and regeneration
15 eneration potential of pregnant mice using a cardiotoxin (CTX) injury mouse model.
16                                        After cardiotoxin (CTX) injury, regenerating myofibers express
17                                              Cardiotoxin (CTX) was used to induce acute injury.
18 ng and in regenerating muscle in response to cardiotoxin (CTX)-induced injury.
19 bustly in regenerating skeletal muscle after cardiotoxin (CTX)-induced muscle injury in vivo and diff
20 ythroid-derived-2)-like 2 (Nrf2), aggravates cardiotoxin (CTX)-induced tibialis anterior (TA) muscle
21                          We investigated the cardiotoxin (CTX)-mediated transient acute mouse model o
22                                        Cobra cardiotoxins (CTX) are a family of three-fingered basic
23                                              Cardiotoxins (CTXs) from cobra venom show cytotoxicity t
24 ffect skeletal muscle regeneration following cardiotoxin damage.
25 a precision medicine approach to the care of cardiotoxin-exposed cancer patients.
26 the discovery of a new insulinotropic agent, cardiotoxin-I (CTX-I), from the Naja kaouthia snake veno
27 rvival and wound healing in a mouse model of cardiotoxin induced muscle injury.
28  MyoR and miR-378 were anticorrelated during cardiotoxin-induced adult muscle regeneration in mice.
29 lt in impaired muscle regeneration following cardiotoxin-induced injury in mice.
30 ed cells and macrophages were assessed after cardiotoxin-induced injury of chimeric mice produced by
31 c conditions, regeneration was delayed after cardiotoxin-induced injury, with prolonged necrosis most
32 l when ablation occurred at the same time as cardiotoxin-induced injury.
33 wth and impaired regenerative capacity after cardiotoxin-induced injury.
34 o the tibialis anterior muscle 3 days before cardiotoxin-induced injury.
35                 Tubb6 is also upregulated in cardiotoxin-induced mouse muscle regeneration, in human
36                              Two hours after cardiotoxin-induced muscle damage, local activin A prote
37 dels, we studied regeneration consecutive to cardiotoxin-induced muscle injury and observed a signifi
38 has some impact on muscle regeneration after cardiotoxin-induced muscle injury in mice.
39 ers (MyoD, myogenin, and active-Notch) after cardiotoxin-induced muscle injury in vivo and in SCs cul
40                                         In a cardiotoxin-induced muscle injury model, lack of MKP-1 i
41 smaller size of newly formed myofibers after cardiotoxin-induced muscle injury, increased glycolysis,
42                       By studying a model of cardiotoxin-induced muscle injury, we identify a populat
43 splayed attenuated muscle regeneration after cardiotoxin-induced muscle injury.
44                               In vivo, acute cardiotoxin-induced muscle regeneration was enhanced in
45 licited by immunization with pVCL/MSP1a into cardiotoxin-induced regenerating muscle were evaluated i
46 ntiation and prolonged Pax7 expression after cardiotoxin-induced skeletal muscle injury, while single
47 ments revealed that Staufen1 increases after cardiotoxin injection before returning to the low levels
48                                    Following cardiotoxin injection, a known trigger for satellite cel
49 pha(-/-)) mice after injury by intramuscular cardiotoxin injection.
50 g in mouse tibialis anterior muscles after a cardiotoxin injection.
51 ersion of marrow cells to skeletal muscle in cardiotoxin-injured anterior tibialis muscle in a green
52 c gene expression and muscle regeneration in cardiotoxin-injured beta3-integrin-null mice are impaire
53 neration and that gene transfer of GEFT into cardiotoxin-injured mouse tibialis anterior muscle exert
54 ed that SP cells isolated from dystrophic or cardiotoxin-injured muscle fail to undergo myogenesis.
55 ute to muscle regeneration when grafted into cardiotoxin-injured muscle.
56 obust muscle regeneration when injected into cardiotoxin-injured skeletal muscle.
57 ransplantation of these committed cells into cardiotoxin-injured skeletal muscles of NOD/SCID mice re
58 d damage, exhibit delayed regeneration after cardiotoxin injury and suffer from defective myoblast fu
59 Rgamma-null mice in both critical defect and cardiotoxin injury models.
60                                Timing of the cardiotoxin injury relative to the transplantation was c
61           Muscle regeneration was induced by cardiotoxin injury, and we evaluated satellite cell acti
62 njury and improved muscle regeneration after cardiotoxin injury, as well as increased satellite cell
63  vivo model of muscle regeneration following cardiotoxin injury, ectopic miR-431 injection greatly im
64                             Within 3 days of cardiotoxin injury, GFP-positive mononuclear cells were
65 effect on muscle regeneration in response to cardiotoxin injury.
66 ubstantially delayed regeneration induced by cardiotoxin injury.
67                            Here, we injected cardiotoxin into gastrocnemius muscle of Hmox1(+/+) and
68 g healthy tissue recovery after injection of cardiotoxin into murine hindlimb muscle.
69  as a delay of muscle regeneration following cardiotoxin-mediated injury.
70                   Unlike acute injuries from cardiotoxin, mPI regenerated poorly with a lack of viabl
71 ed in a profibrotic ischemia reperfusion and cardiotoxin muscle injury model.
72                  Early ablation (day 1 after cardiotoxin) resulted in the smallest regenerated myofib
73 und delay in satellite cell activation after cardiotoxin treatment in alpha7 integrin-null animals wh
74 beta1 integrin plays in muscle regeneration, cardiotoxin was used to induce damage in the tibialis an
75              In an experimental mouse model, cardiotoxin was used to induce muscle injury and repair,