ライフサイエンスコーパス: cardiovascular collapse

1 One control died from cardiovascular collapse.

2 vent perioperative adrenal insufficiency and cardiovascular collapse.

3 t the earliest signs of toxicity may prevent cardiovascular collapse.

4 in 5-min stages until the onset of impending cardiovascular collapse.

5 er body negative pressure (LBNP) to onset of cardiovascular collapse.

6 od pressure was a late indicator of imminent cardiovascular collapse.

7 ive production of inflammatory cytokines and cardiovascular collapse.

8 result of intense sympathetic activation of cardiovascular collapse.

9 acute lethal toxicity (ALT) characterized by cardiovascular collapse.

10 may induce severe neurological symptoms and cardiovascular collapse.

11 not significantly decrease the incidence of cardiovascular collapse.

12 , decreased lymphocyte count, and eventually cardiovascular collapse.

13 omplex constellation of findings with sudden cardiovascular collapse, acute left ventricular failure

14 Cardiovascular collapse also posed a serious threat, con

15 ite aggressive measures for the treatment of cardiovascular collapse and cardiac arrest, the hypotens

16 ralian box jellyfish) stings can cause acute cardiovascular collapse and death.

17 in the llama since their abolition leads to cardiovascular collapse and death.

18 ickle mouse model, intravenous hemin induced cardiovascular collapse and mortality within 120 minutes

19 Common causes of death included: PTLD, cardiovascular collapse, and infection; all occurred at

20 Leakage current causes cardiovascular collapse at levels below the VF threshold

21 The individual components of the cardiovascular collapse composite outcome did not differ

22 The primary outcome was cardiovascular collapse (defined as new or increased rec

23 ed in the intention-to-treat population, was cardiovascular collapse, defined as a new systolic blood

24 ks with repeated attempts, desaturation, and cardiovascular collapse during emergency airway manageme

25 Cardiovascular collapse during intubation occurred in 26

26 ion of an intravenous fluid bolus to prevent cardiovascular collapse during intubation of critically

27 The secondary outcome was cardiovascular collapse during intubation, defined by th

28 emic blood pressure and delayed the onset of cardiovascular collapse during severe hypovolemic hypote

29 pedance threshold device at the same time of cardiovascular collapse during sham breathing (102 +/- 3

30 us did not decrease the overall incidence of cardiovascular collapse during tracheal intubation of cr

31 wing infection, leading to organ failure and cardiovascular collapse if untreated.

32 o the duration of LBNP exposure required for cardiovascular collapse in each subject (i.e., LBNP maxi

33 heterodimeric cytolytic protein that induces cardiovascular collapse in humans and native predators.

34 hat only excessive iNOS-derived NO underlies cardiovascular collapse in shock, our data strongly supp

35 thways other than sGC signalling mediate the cardiovascular collapse in shock.

36 ient increases in blood pressure followed by cardiovascular collapse in wild-type mice, but U-46619 c

37 factors associated with an increased risk of cardiovascular collapse included multisystem organ failu

38 TD may provide short-term protection against cardiovascular collapse induced by orthostatic stress or

39 nflammatory response syndrome with resulting cardiovascular collapse, ischemic damage to vital organs

40 used loss of pulsatile arterial pressure and cardiovascular collapse (mean arterial pressure, 32+/-8

41 Cardiovascular collapse occurred in 113 patients (21.0%)

42 Cardiovascular collapse occurred in 33 (20%) of 168 pati

43 highlights the recognition and treatment of cardiovascular collapse or cardiopulmonary arrest in an

44 al anaesthesia may be attributable to sudden cardiovascular collapse precipitated by ventricular arrh

45 Cardiovascular collapse resulting from hemorrhage is unr

46 sures that are early indicators of impending cardiovascular collapse resulting from progressive reduc

47 ts in cardiac arrest, hemorrhagic shock, and cardiovascular collapse secondary to a number of life-th

48 Risk of death from cardiovascular collapse secondary to immunosuppression w

49 The cardiovascular collapse seen during the first 4 h of end

50 ere reductions in blood pressure, leading to cardiovascular collapse that can accompany septicemia.

51 marked severity and duration may progress to cardiovascular collapse unresponsive to volume replaceme

52 Central hypovolemia and impending cardiovascular collapse were induced in human volunteers

53 xiation from pulmonary edema and generalized cardiovascular collapse were the most likely pathogenic