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1 effects on human health, including decreased cardiovascular function.
2 ygen-sensing machinery that underlies global cardiovascular function.
3 n obligate role for GR in maintaining normal cardiovascular function.
4 micals were screened for targeted effects on cardiovascular function.
5 hannels play a prominent role in controlling cardiovascular function.
6 p influence both cortical brain activity and cardiovascular function.
7 ctivating protein, is strongly implicated in cardiovascular function.
8 te cardiac RyR2 phosphorylation critical for cardiovascular function.
9 OR and that aberrant denitrosylation impairs cardiovascular function.
10 odulate food intake, motivation, stress, and cardiovascular function.
11 BNP in experimental HF resulted in improved cardiovascular function.
12 re to pravastatin did not affect fetal basal cardiovascular function.
13 ety of messengers that are key regulators of cardiovascular function.
14 lation and the effects of corticosteroids on cardiovascular function.
15 ough which ambient temperature may influence cardiovascular function.
16 a unique pathway for the redox regulation of cardiovascular function.
17 t is imperative to determine their impact on cardiovascular function.
18 rol of breathing and in autonomic control of cardiovascular function.
19 ication that may have significant effects on cardiovascular function.
20 imental effects on metabolic homeostasis and cardiovascular function.
21 lar linkage between the serum BDNF level and cardiovascular function.
22 plays an important role in the regulation of cardiovascular function.
23 nformation with ongoing autonomic control of cardiovascular function.
24 T-1 has important implications for renal and cardiovascular function.
25 t and selective Galpha(q) GAP that modulates cardiovascular function.
26 votal role of this enzyme in many aspects of cardiovascular function.
27 d CVLM might also play a role in integrating cardiovascular function.
28 s solitarii (NTS) participates in modulating cardiovascular function.
29 mittent hypoxia (CIH)-induced alterations in cardiovascular function.
30 onses with an underlying circadian rhythm in cardiovascular function.
31 uronal substances that subsequently modulate cardiovascular function.
32 he dietary isoflavones in soy protein affect cardiovascular function.
33 triculography to determine rest and exercise cardiovascular function.
34 at ACTH may play a role in the regulation of cardiovascular function.
35 ates effects of the Arg16Gly polymorphism on cardiovascular function.
36 te the sympathetic nervous system control of cardiovascular function.
37 termittent fasting on glucose regulation and cardiovascular function.
38 bryo development, postnatal growth and later cardiovascular function.
39 llular event in central Ang II regulation of cardiovascular function.
40 uction of the gas, thereby maintaining basal cardiovascular function.
41 ance to the regulation of blood pressure and cardiovascular function.
42 es or markers associated with hepatorenal or cardiovascular function.
43 an important mechanism in the regulation of cardiovascular function.
44 elective sites for pharmacological tuning of cardiovascular function.
45 important area in the central regulation of cardiovascular function.
46 of the beneficial effects that VD exerts on cardiovascular function.
47 n that estrogen produces negative effects on cardiovascular function.
48 e, among others, modulation of autonomic and cardiovascular function.
49 xygen, without any significant impairment in cardiovascular function.
50 o involved in regulating sleep, arousal, and cardiovascular function.
51 he nCom may play a role in the regulation of cardiovascular function.
52 sm of this important enzyme participating in cardiovascular function.
53 pressor area (CVLM) in regulating/modulating cardiovascular function.
54 used 8 noninvasive measures of autonomic and cardiovascular function.
55 relaxin should be considered a regulator of cardiovascular function.
56 ic, this review is focused on its effects on cardiovascular function.
57 ides a mechanism for the rapid regulation of cardiovascular function.
58 AG mediate the ability of the IL to regulate cardiovascular function.
59 , possibly critical, aspects with respect to cardiovascular function.
60 ecies have emerged as important molecules in cardiovascular function.
61 localized in areas of brain more related to cardiovascular function.
62 des in the central nervous system control of cardiovascular function.
63 r regulation of both breathing and autonomic cardiovascular function.
64 urrent and subsequently to the modulation of cardiovascular function.
65 in genes whose functions underlie mammalian cardiovascular function.
66 gic neurotransmission and central control of cardiovascular function.
67 the basolateral amygdala (BLA) in regulating cardiovascular function.
68 omponent of the NO-cGMP pathway, critical to cardiovascular function.
69 indicating dysregulation of post-exertional cardiovascular function.
70 nd norepinephrine, which are known to affect cardiovascular function.
71 lopmental cardiac abnormalities and impaired cardiovascular function.
72 earchers and clinicians to better understand cardiovascular function.
73 processes ranging from neurotransmission to cardiovascular function.
74 nervous systems in regard to control of the cardiovascular function.
75 s been linked to beneficial effects on human cardiovascular function.
76 etal hypoxia, growth restriction and altered cardiovascular function.
77 eful tool for the quantitative evaluation of cardiovascular function.
78 y tuning heart rates is critical for regular cardiovascular function.
79 uption of sympathetic pathways that modulate cardiovascular function.
80 ical alterations, including perturbations in cardiovascular function.
81 gulation of metabolic homeostasis, governing cardiovascular function.
82 bles is converted into NO in vivo to improve cardiovascular function.
83 significant short- and long-term effects on cardiovascular function.
84 cle oxidative capacity and improving certain cardiovascular functions.
85 enes implicated in cell cycle, motility, and cardiovascular functions.
86 al and plays a role in the neural control of cardiovascular functions.
87 rs (5-HTRs) play critical roles in brain and cardiovascular functions.
88 la following transient focal ischemia affect cardiovascular functions.
89 gulation of insulin-regulated metabolism and cardiovascular functions.
90 where they were found in nuclei having known cardiovascular functions.
91 eight but only induce modest improvements in cardiovascular functions.
92 role in the regulation of blood pressure and cardiovascular functions.
93 tant regulator of body fluid homeostasis and cardiovascular functions.
94 ychosocial factors may promote healthy adult cardiovascular functioning.
96 emonstrated significantly less impairment of cardiovascular function after biphasic defibrillation.
97 oxygenated HBOC-201 rapidly restored viable cardiovascular function after exsanguinating cardiac arr
100 S), a tissue RAS having an important role in cardiovascular function also exists in the central nervo
102 here is an increasing need for understanding cardiovascular function and blood flow control in normal
104 lyl cyclase (sGC) plays an important role in cardiovascular function and catalyzes formation of cGMP.
105 APJ) system is involved in the regulation of cardiovascular function and central control of body home
106 atory cycle that is required for both normal cardiovascular function and circulatory adaptation to hy
108 metabolites in pathways that are integral to cardiovascular function and disease, including inflammat
109 erstanding of the diverse roles miRs play in cardiovascular function and disease, we applied high-thr
112 n injuries must quickly restore and maintain cardiovascular function and fluid balance while minimizi
114 tors and effectors of BMP signalling control cardiovascular function and how the dysregulated BMP sig
116 is a key hormonal system which regulates the cardiovascular function and is implicated in several aut
118 he effects of glucocorticoids on fetal basal cardiovascular function and on the fetal cardiovascular
120 s long-acting glycemic control with improved cardiovascular function and reduced tissue toxicity in m
121 exogenous ELA or Apelin-13 infusion improves cardiovascular function and survival after cecal ligatio
122 al treatment with dexamethasone alters basal cardiovascular function and the cardiovascular response
123 act with environmental variables to modulate cardiovascular function and the response to therapeutic
124 s and underscore their physiological role in cardiovascular function and their clinical relevance to
125 t phosphodiesterase-5 inhibitors may enhance cardiovascular function and thus exercise capacity in he
126 nicotine inhalation during pregnancy impairs cardiovascular function and uterine hemodynamics with co
127 by brain regions that are known to regulate cardiovascular function and which are activated by chron
128 e has been reported to restore the depressed cardiovascular functions and cell-mediated immune respon
129 y the autonomic nervous system in modulating cardiovascular functions and in controlling blood pressu
133 antisense RNA1, a noncoding gene related to cardiovascular function) and rs269868-C/Ser1067 in DUOX2
134 e been implicated as important modulators of cardiovascular function, and APJ receptor agonists may b
135 data into clinically relevant indicators of cardiovascular function, and estimate hemodynamic variab
136 rstanding of how mutations affect developing cardiovascular function, and few studies have been perfo
137 e is a well-known regulator of metabolic and cardiovascular functions, and signaling through thyroid
138 ts by which leptin regulates food intake and cardiovascular function are differentially influenced by
142 the notion that hypothalamic influences upon cardiovascular functions are in part mediated through hy
143 , where GPCRs regulate such core measures of cardiovascular function as heart rate, contractility, an
144 ardiovascular models represent a snapshot of cardiovascular function at a given post-transplant recov
145 erse consequences for NO bioavailability and cardiovascular function at adulthood, and whether neonat
146 he ability to identify chemicals that target cardiovascular function at nonteratogenic concentrations
149 s to screen and identify chemicals affecting cardiovascular function at sublethal, nonteratogenic con
150 al benefits in exercise capacity ( VO2max ), cardiovascular function at submaximal and maximal exerci
153 Circadian rhythmicity of many aspects of cardiovascular function-blood pressure, coagulation and
154 or chronic intermittent cold stress modifies cardiovascular function both under resting conditions an
155 ates numerous biological processes including cardiovascular function, bowel motility, ejaculatory lat
156 ced hypertension, not only maintaining basal cardiovascular function, but also permitting blockade of
158 supports the actions of statins in improving cardiovascular function, but the mechanisms of their pro
159 as a neuropeptide to control food intake and cardiovascular functions, but its neural role in glucose
160 t regulate stress responses, metabolism, and cardiovascular function by activating either of two rela
161 nverting enzyme (ACE) has a critical role in cardiovascular function by cleaving the carboxy terminal
163 a pivotal role in the central regulation of cardiovascular function by regulating other renin-angiot
164 ers of inflammation, metabolic function, and cardiovascular function (C-reactive protein, hemoglobin
167 nitric oxide (NO.), is a novel regulator of cardiovascular function, combining concomitant positive
171 ation (AM/AMBP-1) after hemorrhage, improves cardiovascular function despite the increased levels of
172 im was to systematically evaluate changes in cardiovascular function during acute allergic reactions
173 ts a role for this K(ATP) channel subunit in cardiovascular function during conditions of stress.
174 e skeletal muscle pump and vasodilatation on cardiovascular function during exercise, we determined l
176 this substitution differentially influenced cardiovascular function during short duration (9 min) lo
178 heir proinflammatory secretome also improves cardiovascular function, enhances insulin sensitivity, a
181 chanism for the disrupted NE homeostasis and cardiovascular function evident in OI patients with the
182 n index of mood (hypohedonia; Experiment 1), cardiovascular function (Experiment 2), and plasma elect
183 e physiological processes of blood pressure, cardiovascular function, fluid secretion, and others: me
187 Adenosine, long known as a regulator of cardiovascular function, has recently been identified as
188 g enzyme (ACE)2 in brain regions controlling cardiovascular function; however, the role of ACE2 in bl
189 a mitigate the adverse effects of seizure on cardiovascular function in a rat model of temporal lobe
190 iations between ambient pollution levels and cardiovascular function in a repeated measures study inc
191 spring renin-angiotensin system activity and cardiovascular function in a sperm and/or seminal plasma
193 reatment improved circulating parameters and cardiovascular function in both models, and restored SER
194 n to increase NO bioavailability and improve cardiovascular function in both preclinical and clinical
196 s, allowing for detailed characterization of cardiovascular function in genetically engineered mice,
199 effects of phosphodiesterase 5 inhibitors on cardiovascular function in heart failure (HF) patients,
201 generation of reactive oxygen species alter cardiovascular function in individuals with elastin hapl
202 short episode of postnatal undernutrition on cardiovascular function in mice at the whole animal, org
203 the feasibility of determining parameters of cardiovascular function in mice noninvasively by high-te
205 maternal treatment with allopurinol on fetal cardiovascular function in ovine pregnancy in late gesta
207 pproach permits the monitoring of changes in cardiovascular function in response to pharmacologic int
208 that progesterone can improve the depressed cardiovascular function in sex steroid-deficient female
211 patient-specific models are used to predict cardiovascular function in the form of right and left ve
212 the mechanisms underlying the development of cardiovascular function in the horse fetus, with particu
214 We examined whether BDNF in nTS modulates cardiovascular function in vivo and regulates synaptic a
215 VEGF-A and PDGF signalling in vitro, but its cardiovascular function in vivo remains relatively unexp
216 ne in endothelial cells on NO production and cardiovascular function in vivo using NOSTRIN knockout m
218 the two TSP variants are likely to influence cardiovascular functions in distinct but yet pathogenic
219 resent treatments for the loss or failure of cardiovascular function include organ transplantation, s
220 rmined in vivo longitudinal changes in fetal cardiovascular function including parallel measurement o
223 n to exert a number of beneficial effects on cardiovascular function, including reduction in BP and i
224 variety of physiological processes including cardiovascular function, inflammatory responses, and noc
225 (SP), a peptide that has been implicated in cardiovascular function, inflammatory responses, and noc
227 vestigating sex differences in mechanisms of cardiovascular function is highlighted by the National I
235 ldosterone is a known regulator of renal and cardiovascular function, its role as a regulator of canc
236 ial to influence parasympathetic outflow and cardiovascular function, likely through an enkephalinerg
237 ing in mnTS plays a tonic role in regulating cardiovascular function, likely via modulation of primar
238 iac vagal activity play significant roles in cardiovascular function, little is known about the synap
239 I have any salutary effects on the depressed cardiovascular function, liver damage, and mortality rat
240 important for the control of food intake and cardiovascular function, making them good candidates to
241 teenagers, these studies raise concern that cardiovascular function may be especially vulnerable dur
242 cts in adult offspring include impairment of cardiovascular function, metabolic derangement and accel
243 five centers studying longitudinal lung and cardiovascular function of infants from HIV-infected mot
246 valuation of the pharmacological response of cardiovascular function on model animals is important es
247 inically significant adverse drug effects on cardiovascular function or growth at the end of 2 years
248 protein diet had minimal effects on paternal cardiovascular function or renin-angiotensin system acti
249 lcNAc stimulation improves global animal and cardiovascular function outcomes associated with a resto
250 the roles carried out by these molecules in cardiovascular function, pain, epilepsy, migraine and de
251 nucleus (PVN), a critical neuroregulator of cardiovascular function, plays an important role in the
253 entricular valves corresponded with impaired cardiovascular function (reduced ventricular beat rate a
254 isms by which PKGI kinase activity regulates cardiovascular function remain incompletely understood.
256 Secondary outcomes included measures of cardiovascular function, renal function, resource use, a
257 ess, suggests that the maintenance of normal cardiovascular function requires elevated cardiac sympat
258 atic processes, such as endocrine secretion, cardiovascular function, salt intake, and nociception.
259 gulation for sympathetic activity to improve cardiovascular function.SIGNIFICANCE STATEMENT Disruptio
260 in the regulation of various cAMP-dependent cardiovascular functions, such as calcium handling and v
261 ave been implicated in regulating neural and cardiovascular functions, such as cell spreading and che
262 rn of distribution and the lack of effect on cardiovascular function support the concept that RASONs
263 ive protein, fibrinogen, IL-6, and albumin), cardiovascular functioning (systolic and diastolic blood
265 (Ang II) has powerful modulatory actions on cardiovascular function that are mediated by specific re
266 a potentially beneficial effect of COX-2 on cardiovascular function that could be negated by COX-2 i
267 pharmacological therapy to improve impaired cardiovascular function that occurs in the context of he
268 and AMPH produced dose-related increases in cardiovascular function that were of comparable magnitud
269 ity of exosomes containing a key receptor in cardiovascular function, the angiotensin II type I recep
270 mechanisms underlies the effects of aging on cardiovascular function, the most important being excess
271 in the regulation of salt-water balance and cardiovascular function: the renin-angiotensin-aldostero
272 (AP) as been implicated in the regulation of cardiovascular function, there is no consensus regarding
273 up is predicted to play a role in regulating cardiovascular function, these findings suggest that dev
274 he NTS, NO from eNOS in the NTS may modulate cardiovascular function through an interaction between a
275 and participate in EA-related modulation of cardiovascular function through an opioid or serotonergi
276 iovascular well-being, programming offspring cardiovascular function through both sperm and seminal p
277 contribute to blood pressure regulation and cardiovascular function through its effects on the vascu
278 ular mechanisms underlying the regulation of cardiovascular functions through baroreceptor function.
279 rticoid-driven maturational changes in fetal cardiovascular function under basal and stressful condit
280 rtical and subcortical brain regions control cardiovascular function via the sympathetic and parasymp
287 are important in the regulation of renal and cardiovascular function, we studied the cardiovascular c
288 f maternal allopurinol on maternal and fetal cardiovascular function were also investigated following
290 Craving, negative emotion, anxiety, and cardiovascular function were assessed at baseline, immed
293 quences of variations in natural BW on basal cardiovascular function were investigated in pigs at 3 m
295 o the dorsolateral PAG and did not influence cardiovascular function when injected into the vlPAG of
296 serve capacity involving multiple domains of cardiovascular function, which contribute in an integrat
297 gh-intensity exercise training on integrated cardiovascular function, which incorporates the dynamic
299 pressure (via radial arterial tonometry) and cardiovascular function with echocardiographic measures
300 frequently limited by an inability to assess cardiovascular function within the intact organism.