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1 ponse is necessary for EC regeneration after carotid artery injury.
2 an occlusive thrombus after FeCl(3)-induced carotid artery injury.
3 well as neointima formation, after ZO common carotid artery injury.
4 lets, formed unstable thrombi in response to carotid artery injury.
5 ically upregulated following balloon-induced carotid artery injury.
6 ation and neointimal thickening after murine carotid artery injury.
7 hrombus formation on ferric chloride-induced carotid artery injury.
8 tiplatelet agent developed stroke related to carotid artery injuries.
10 d with permanent cranial nerve morbidity nor carotid artery injury, although 4 patients (all Knosp 3-
11 2) (PAMAM G-3) prevents thrombosis following carotid artery injury and pulmonary thromboembolism in m
12 ng from murine liver wounds, swine liver and carotid artery injuries, and the human radial artery pun
13 endothelial matrix, mice were subjected to a carotid artery injury assay in which ferric chloride adm
14 d with higher risk of stroke due to internal carotid artery injuries, but monitoring was not useful f
15 FVIII had no effect after longer (3-minute) carotid artery injury, but it shortened the TTO after sh
19 hrombosis was measured in vivo after FeCl(3) carotid artery injury, endothelial COX1 deletion acceler
21 g for ipsilateral and contralateral internal carotid artery injury grade (adjusted risk ratio, 2.91;
35 d markedly impaired thrombus formation after carotid artery injury or inferior vena cava ligation.
36 ient mice have reduced thrombus growth after carotid artery injury relative to conventionally raised
37 e found that after mesenteric arteriolar and carotid artery injury, Slit2 delayed vessel occlusion ti
38 By using a ferric chloride (FeCl3)-induced carotid artery injury thrombosis model, we found time to
41 The time to occlusion after FeCl(3)-induced carotid artery injury was delayed (11.8 +/- 3.6 minutes,
44 able occlusive thrombi after ferric chloride carotid artery injury, whereas the majority of wild-type
45 Recognition of brain injury in asymptomatic carotid artery injuries with conventional methods can be