ライフサイエンスコーパス: carotid artery occlusion

1 diet and modeled VCID via unilateral common carotid artery occlusion.

2 before and after ischemia induced by middle carotid artery occlusion.

3 ding condition that occurs in the setting of carotid artery occlusion.

4 agonists and pepducins was effective against carotid artery occlusion.

5 perfusion deficits even after release of the carotid artery occlusion.

6 pler probe, combined with ipsilateral common carotid artery occlusion.

7 bal ischemia was induced by bilateral common carotid artery occlusion.

8 anaesthetized rats with a unilateral common carotid artery occlusion.

9 e of stroke risk in patients with unilateral carotid artery occlusion.

10 middle cerebral artery and bilateral common carotid artery occlusion.

11 littermates after 3 min of bilateral common carotid artery occlusion.

12 zone 1-2 weeks after 10 min bilateral common carotid artery occlusions.

13 Rats were subjected to bilateral carotid artery occlusion (2-VO) or sham occlusion (No-VO

14 similar in HRG-deficient and wild-type mice, carotid artery occlusion after FeCl3 injury was accelera

15 cFvSCE5-scuPA (75 U/g body weight) prevented carotid artery occlusion after ferric chloride injury in

16 into Mrp14(-)/(-) mice decreased the time to carotid artery occlusion after injury, indicating that p

17 ts with symptomatic atherosclerotic internal carotid artery occlusion (AICAO) and hemodynamic cerebra

18 e present pooled analysis (120 with internal carotid artery occlusion and 360 with isolated middle ce

19 Ischemia consisted of 30 min of fetal carotid artery occlusion and 72 h of reperfusion.

20 obtained in a patient with a right internal carotid artery occlusion and an infarct in the middle ce

21 mg was given i.v. and 3 min later bilateral carotid artery occlusion and blood withdrawal-induced hy

22 natinib induced severe responses, leading to carotid artery occlusion and extensive vessel wall damag

23 l of surgical revascularisation for complete carotid artery occlusion and haemodynamic ischaemia, wer

24 ia was induced by 10 min of bilateral common carotid artery occlusion and hypotension in rats.

25 dult mice that consists of unilateral common carotid artery occlusion and hypoxia with tightly regula

26 gina, previous ipsilateral CE, contralateral carotid artery occlusion and other severe comorbid illne

27 rfusion was achieved with bilateral internal carotid artery occlusion and pharmacologically induced s

28 asted Wistar rats underwent bilateral common carotid artery occlusion and severe hypotension (50 mmHg

29 neuronal integrity in a model of unilateral carotid artery occlusion and systemic hypobaric hypotens

30 (PAI-1) in rat pups subjected to unilateral carotid artery occlusion and systemic hypoxia.

31 ) by hypoxia (FiO2 0.1), ischemia (bilateral carotid artery occlusion), and hypotension (mean arteria

32 ed in the AQP4(-/-) vs. AQP4(+/+) mice after carotid artery occlusion, as were blood-brain barrier (B

33 nd again at 2 h 30 min after 5 min bilateral carotid artery occlusion (BCAO) in the gerbil.

34 rat pups, which were subjected to bilateral carotid artery occlusion (BCAO) on postnatal day 1.

35 al ischemia produced by transient, bilateral carotid artery occlusion (BCAO).

36 male C57black6 mice after a 60-min bilateral carotid artery occlusion (BCAO).

37 ebral ischaemia in mouse is bilateral common carotid artery occlusion (BCCAo) and the neuropathologic

38 Bilateral common carotid artery occlusion (BCCAO) in a patient with Sjogr

39 sion and VaD was induced by bilateral common carotid artery occlusion (BCCAO) in adult male Sprague D

40 Chronic bilateral common carotid artery occlusion (BCCAO) in adult rats has been

41 Permanent bilateral common carotid artery occlusion (BCCAO) was used as a model of

42 term experimental permanent bilateral common carotid artery occlusion (BCCAO).

43 level of blood flow reduction was induced by carotid artery occlusion (CAO) over a similar 3 week tim

44 rmore, PKCdY311F mice exhibit longer time to carotid artery occlusion compared with WT control using

45 aroreceptor afferent inactivation, caused by carotid artery occlusion, elicit similar patterns of c-F

46 ongolian gerbils were subjected to bilateral carotid artery occlusion, exposed to nesting material an

47 n ovariectomized rats after transient middle carotid artery occlusion followed by a 24-h reperfusion.

48 erbils by a 5-min period of bilateral common carotid artery occlusion followed by reperfusion for 6 d

49 2/3(-/-)) were subjected to bilateral common carotid artery occlusion, followed by reperfusion and tr

50 displayed a significantly prolonged time to carotid artery occlusion following Rose Bengal administr

51 pressure of 30-35 mm Hg and bilateral common carotid artery occlusion for 8 min).

52 ischemia was induced in gerbils by bilateral carotid artery occlusion for a period of 5 minutes.

53 ere ischemia induced by reversible bilateral carotid artery occlusion, for either 1 or 25 hours.

54 angiograms in four patients showed internal carotid artery occlusion in one.

55 Transient bilateral carotid artery occlusion in the Mongolian gerbil is a wi

56 nt of hippocampal injury following bilateral carotid artery occlusion in this species.

57 uent stroke risk in patients with unilateral carotid artery occlusion, in order to define better the

58 method to monitor retinopathy in a bilateral carotid artery occlusion-induced ocular ischemia, we obs

59 od to demonstrate that 14 days of unilateral carotid artery occlusion induces increases in the calibe

60 cerebrovascular reserve in patients in whom carotid artery occlusion is contemplated.

61 ation and brain infarct injury in the middle carotid artery occlusion ischemia/reperfusion model.

62 l ischemia induced by middle cerebral/common carotid arteries occlusion (MCA/CCAo) induced up-regulat

63 tective properties in vivo (gerbil bilateral carotid artery occlusion model: BCAO) and in vitro (isch

64 ed using data from patients with symptomatic carotid artery occlusion (n = 68).

65 cally confirmed Moyamoya (n = 8) or internal carotid artery occlusions (n = 10).

66 focal ischemia in a filament model of middle carotid artery occlusion nor caused postthrombotic hemor

67 le cerebral artery +/- intracranial internal carotid artery occlusions on baseline computed tomograph

68 rwent 10 min of global ischemia by bilateral carotid artery occlusions plus hypotension.

69 in of global forebrain ischemia by bilateral carotid artery occlusions plus systemic hypotension.

70 sis and treatment of stroke in patients with carotid artery occlusion remains controversial.

71 sham operation (n=6) or 15 min of bilateral carotid artery occlusion resulting in global cerebral is

72 ediated cerebrovascular adaptation to common carotid artery occlusion resulting in hypoperfusion.

73 induced a dose-dependent prolongation of the carotid artery occlusion time in HCII(+/+) mice that was

74 Bleeding time and carotid artery occlusion time were significantly prolong

75 PRCP(gt/gt) mice demonstrate shorter carotid artery occlusion times in 2 models, and their pl

76 Klkb1(-/-) mice have delayed carotid artery occlusion times on the rose bengal and fe

77 chloromethylketone or PKSI 527 also shortens carotid artery occlusion times.

78 ected to global ischemia by bilateral common carotid artery occlusion under controlled ventilation fo

79 middle cerebral artery or terminal internal carotid artery occlusion using computed tomographic angi

80 After 45 mins, carotid artery occlusion was released, FiO2 was reset to

81 scle contraction) or baroreceptor unloading (carotid artery occlusion) were sensitive to SP knockdown

82 hemic injury was induced by bilateral common carotid artery occlusion, whereas severe focal stroke in

83 ebral ischemia (tGCI) using bilateral common carotid artery occlusion with hypotension.

84 raphically proven asymptomatic left internal carotid artery occlusion with normal CT after a gunshot