ライフサイエンスコーパス: carotid artery thrombosis

1 s, we also performed ferric chloride-induced carotid artery thrombosis.

2 times and impaired mesenteric arteriole and carotid artery thrombosis.

3 re protected from TF-dependent FeCl3-induced carotid artery thrombosis.

4 del of ferric chloride induced non-occlusive carotid artery thrombosis.

5 FeCl3 was used to induce carotid artery thrombosis.

6 ed decreased susceptibility to FeCl3-induced carotid artery thrombosis.

7 mostasis in a tail bleeding model and normal carotid artery thrombosis.

8 aluated in rat models of mural and occlusive carotid artery thrombosis.

9 -) mice have long bleeding times and delayed carotid artery thrombosis, 78 +/- 6.7 minutes, versus 31

10 l microscopy, we evaluated susceptibility to carotid artery thrombosis after FeCl3-induced injury in

11 beta3(Delta760-762) mice were protected from carotid artery thrombosis after vessel injury with FeCl(

12 UT7 activity was examined in mouse models of carotid artery thrombosis and collagen/epinephrine-induc

13 t aggregation/secretion, FeCl3-induced mouse carotid artery thrombosis and tail bleeding time.

14 ant in both the rat model of FeCl(2)-induced carotid artery thrombosis and the rabbit model of jugula

15 inal fragment protects against FeCI3-induced carotid artery thrombosis as well as cerebral infarction

16 d only a partial correction, but in an FeCl3 carotid artery, thrombosis assay correction was equivale

17 d VWF had defective hemostasis and defective carotid artery thrombosis, but experienced significant c

18 ce showed nearly complete protection against carotid artery thrombosis caused by low FeCl(3) injury.

19 ive vehicle or drug immediately after common carotid artery thrombosis (CCAT).

20 In a model of carotid artery thrombosis, FgaWT/EK and FgaEK/EK mice we

21 rosclerotic lesion area, and delayed time to carotid artery thrombosis in a photochemical injury mode

22 re resistant to the development of occlusive carotid artery thrombosis in the absence of systemic fib

23 Low-dose aspirin ameliorated carotid artery thrombosis in VFGp1ba and Jak2VF CH mice

24 ) mice showed retardation in FeCl(3)-induced carotid artery thrombosis in vivo.

25 -bleeding times and impaired FeCl(3)-induced carotid artery thrombosis in vivo.

26 large vessel thrombus formation, we studied carotid artery thrombosis in wild-type mice, mice lackin

27 arterioles (P<0.05) but not from accelerated carotid artery thrombosis induced by the HM/LF diet.

28 sis (P < .01) in an electrolytically induced carotid artery thrombosis model in dogs.

29 Finally, we showed with an in vivo carotid artery thrombosis model that genetic deletion of

30 Using a murine carotid artery thrombosis model we demonstrated CD36-dep

31 time) and in vivo (e.g. rat FeCl(2)-induced carotid artery thrombosis model).

32 n overt bleeding diathesis, but in a FeCl(3) carotid artery thrombosis model, all showed impaired thr

33 In a carotid artery thrombosis model, both AbetaPP(-/-) and A

34 In a mouse carotid artery thrombosis model, non-targ-CD39, although

35 In an FeCl3 injury-induced carotid artery thrombosis model, thrombus growth rate an

36 in is more potent than hirulog-1 in a murine carotid artery thrombosis model.

37 s antithrombotic effect examined in a rabbit carotid artery thrombosis model.

38 platelet VWF showed normal tail bleeding and carotid artery thrombosis, similar to wild-type mice.

39 ptor-deleted mice (Bdkrb2(-/-)) have delayed carotid artery thrombosis times and prolonged tail bleed

40 Susceptibility to carotid artery thrombosis was first examined in wild-typ

41 Carotid artery thrombosis was not accelerated in mice de

42 KIN59 significantly inhibited FeCl3-induced carotid artery thrombosis without affecting hemostasis.