ライフサイエンスコーパス: carotid atherosclerosis

1 symptomatic carotid plaques in patients with carotid atherosclerosis.

2 (CT) can assess both anatomy and biology of carotid atherosclerosis.

3 y for suspected NASH; 427 were evaluated for carotid atherosclerosis.

4 childhood to adulthood had increased risk of carotid atherosclerosis.

5 on to the clinical and ultrasound markers of carotid atherosclerosis.

6 ne correlated with the presence or extent of carotid atherosclerosis.

7 rticulate matter exposures on development of carotid atherosclerosis.

8 (n=362) in a prevalent case-control study of carotid atherosclerosis.

9 ded for primary prevention and management of carotid atherosclerosis.

10 tudy the pathogenesis and natural history of carotid atherosclerosis.

11 or the clinical assessment and management of carotid atherosclerosis.

12 ic core and intraplaque hemorrhage, in human carotid atherosclerosis.

13 ic information in patients with asymptomatic carotid atherosclerosis.

14 ients with both symptomatic and asymptomatic carotid atherosclerosis (163 for the derivation set and

15 Carotid atherosclerosis accounts for approximately 10% o

16 The potentially causal associations with carotid atherosclerosis and atherosclerotic risk factors

17 Genetic liability to both carotid atherosclerosis and atrial fibrillation was asso

18 was generally associated with higher risk of carotid atherosclerosis and clinical cardiovascular dise

19 We investigated the relationship between carotid atherosclerosis and CRP and their joint roles in

20 ured by SUA may indicate risk of subclinical carotid atherosclerosis and high 10-year ASCVD risk in m

21 Carotid atherosclerosis and microalbuminuria are associa

22 rdiovascular disease, with evidence of early carotid atherosclerosis and moderately elevated LDL chol

23 ment is associated with an increased risk of carotid atherosclerosis and stroke, determinants of cogn

24 cipal method for determining the severity of carotid atherosclerosis and the need for endarterectomy.

25 idence that auscultatory gaps are related to carotid atherosclerosis and to increased arterial stiffn

26 er PTSD symptoms were associated with higher carotid atherosclerosis and, among women who were APOEep

27 gh-quality epidemiological investigations on carotid atherosclerosis are needed to better address the

28 in elderly men and postmenopausal women with carotid atherosclerosis, as well as with risk of stroke

29 The main exposures were carotid atherosclerosis, assessed using carotid intima-m

30 These findings indicate excess coronary and carotid atherosclerosis at ABI values below 1.10 (men) a

31 eeded to better address the global burden of carotid atherosclerosis at finer levels.

32 endovascular techniques for the treatment of carotid atherosclerosis, beginning with balloon angiopla

33 Baseline carotid atherosclerosis but not atherosclerotic CVD risk

34 cholesterol were associated only weakly with carotid atherosclerosis but were associated strongly wit

35 -based studies that quantified prevalence of carotid atherosclerosis by means of increased carotid in

36 arotid endarterectomy underwent screening of carotid atherosclerosis by ultrasound and MR.

37 Estimation of the epidemiological burden of carotid atherosclerosis can serve as a basis for prevent

38 We evaluated the prevalence of carotid atherosclerosis compared with a control populati

39 In nine subjects with carotid atherosclerosis, double-oblique, contrast materi

40 Prevention and control of carotid atherosclerosis during middle age may positively

41 ctively collected patients with asymptomatic carotid atherosclerosis evaluated by duplex sonography.

42 The impact on carotid atherosclerosis evolution is not known.

43 A substantial global burden of carotid atherosclerosis exists.

44 pausal women and 1124 men (>/=45 years) with carotid atherosclerosis, from prospective population-bas

45 An association between fatty liver and carotid atherosclerosis has been established; however, i

46 plex roles of pre-existing disease (frailty, carotid atherosclerosis, hypertension, low diastolic blo

47 plasma fatty acid pattern is associated with carotid atherosclerosis in a direction generally consist

48 ation between antioxidant vitamin status and carotid atherosclerosis in a group of elderly persons.

49 onomic reward, and the 4-year progression of carotid atherosclerosis in a population-based sample of

50 eview and meta-analysis of the prevalence of carotid atherosclerosis in China.

51 osis is associated with a high risk of early carotid atherosclerosis in G1 CHC patients.

52 vational studies have reported less frequent carotid atherosclerosis in healthy women taking postmeno

53 erresponsivity to accelerated development of carotid atherosclerosis in human subjects and to exacerb

54 ter repeat polymorphism were associated with carotid atherosclerosis in humans, an effect that was ex

55 history of major depression and subclinical carotid atherosclerosis in middle-aged women.

56 man subjects and to exacerbated coronary and carotid atherosclerosis in monkeys.

57 e used to identify a subset of patients with carotid atherosclerosis in need of intensified medical t

58 strogen use has been associated with reduced carotid atherosclerosis in observational studies, but th

59 is associated with the burden and quality of carotid atherosclerosis in older age suggesting that the

60 t of RRR-alpha-tocopherol supplementation on carotid atherosclerosis in patients with stable coronary

61 RA inflammation contribute significantly to carotid atherosclerosis in RA, and may modify one anothe

62 the extent to which ACS are associated with carotid atherosclerosis in RA.

63 levels in women with and without significant carotid atherosclerosis in the Atherosclerosis Risk in C

64 lence, number of cases, and risk factors for carotid atherosclerosis in the general population global

65 f antioxidant vitamins and either measure of carotid atherosclerosis in the women.

66 entify the lipid-rich necrotic core in human carotid atherosclerosis in vivo with high sensitivity an

67 us caps from thin and ruptured caps in human carotid atherosclerosis in vivo.

68 abstinence are associated with lower odds of carotid atherosclerosis in women.

69 The effect of ezetimibe on carotid atherosclerosis is indeterminate, but ezetimibe

70 nopause and hormone replacement therapy with carotid atherosclerosis is limited.

71 lus statins showed that modest regression of carotid atherosclerosis is possible in individuals with

72 CH status in combination with the extent of carotid atherosclerosis jointly predict long-term mortal

73 A total of 37 subjects with carotid atherosclerosis (mean age, 69.9 years; age range

74 6 was negatively associated to the extent of carotid atherosclerosis measured as intima-media thickne

75 uneck study with long-term follow-up data on carotid atherosclerosis measured by high-resolution dupl

76 Carotid atherosclerosis, measured as carotid intima-medi

77 ing endarterectomized patients with advanced carotid atherosclerosis (n=92) and myocardial infarction

78 SUA was associated with higher odds of carotid atherosclerosis (odds ratio [OR], 1.77; 95% CI,

79 llele was associated not only with prevalent carotid atherosclerosis (odds ratio [OR]: 1.46 [95% conf

80 ant of FABP4 on cardiovascular morbidity and carotid atherosclerosis on a population level (n=7491) a

81 g similar pathogenesis for both coronary and carotid atherosclerosis, one could interpret risk factor

82 pertrophy, systolic dysfunction, coronary or carotid atherosclerosis, or other vascular abnormalities

83 Association of baseline RF status with carotid atherosclerosis (overall mean carotid wall thick

84 Carotid atherosclerosis plays a fundamental part in the

85 The prognostic value of CH in patients with carotid atherosclerosis remains to be evaluated.

86 Carotid atherosclerosis, SLE duration, damage index, ser

87 0, 10.7) after adjustment for age, race, and carotid atherosclerosis status.

88 released February 1991) and the Asymptomatic Carotid Atherosclerosis Study (ACAS clinical alert relea

89 erectomy Trial (NASCET) and the Asymptomatic Carotid Atherosclerosis Study (ACAS) demonstrated the ef

90 Although the Asymptomatic Carotid Atherosclerosis Study (ACAS) reported that carot

91 erectomy Trial (NASCET) and the Asymptomatic Carotid Atherosclerosis Study (ACAS) showed significant

92 The Asymptomatic Carotid Atherosclerosis Study (ACAS) showed that carotid

93 osterone and SHBG to be inversely related to carotid atherosclerosis, suggesting their potential impo

94 produced significantly greater regression of carotid atherosclerosis than an LDL-C target of 90 to 11

95 rd have significantly greater progression of carotid atherosclerosis than more advantaged men.

96 cholesterol diet and in humans with unstable carotid atherosclerosis, the expression of miR-223 is in

97 Addition of CRP or carotid atherosclerosis to conventional risk factors mod

98 We measured carotid atherosclerosis using high-resolution ultrasound

99 Carotid atherosclerosis was assessed by ultrasound, wher

100 Carotid atherosclerosis was assessed in consecutively re

101 Carotid atherosclerosis was common in Burkina Faso (82 o

102 The prevalence and severity of carotid atherosclerosis was greatest in those with the l

103 stroke prevention strategy in patients with carotid atherosclerosis, we tested whether circulating I

104 Two measures of subclinical carotid atherosclerosis were assessed using B-mode ultra

105 Eight patients with symptomatic carotid atherosclerosis were imaged using 18FDG-PET and

106 plaque material from patients with advanced carotid atherosclerosis were performed using polarizatio

107 pite the significant associations of CRP and carotid atherosclerosis with CVD, these measures modestl