ライフサイエンスコーパス: carotid sinus

1 susceptible waveform mimicking flow in human carotid sinus.

2 lation of the aortic arch and/or ipsilateral carotid sinus.

3 ve terminals innervating the aortic arch and carotid sinus.

4 10) developed atherosclerotic lesions in the carotid sinuses.

5 e coronary arteries, the aortic arch and the carotid sinuses.

6 Glomus cells and carotid sinus afferents are anatomically connected, and

7 otid baroreceptors via a vascularly isolated carotid sinus and anodal block of baroreceptor afferent

8 forms to those of central projections of the carotid sinus and aortic depressor nerves.

9 ng pressure increments (80-100 mm Hg) in the carotid sinus and by electrical stimulation (stimulus in

10 es primary afferent projections from aortic, carotid sinus and cardiac receptors in the cat.

11 it and pressures distending the aortic arch, carotid sinus and coronary artery baroreceptors were con

12 Large changes in carotid sinus and coronary pressures decreased vascular

13 two distinct regions of the carotid artery (carotid sinus and distal internal carotid artery) that a

14 g muscle contraction following disruption of carotid sinus and vagus nerves it is proposed that: (1)

15 espiratory input following disruption of the carotid sinus and vagus nerves significantly blunted, bu

16 nt of cardiorespiratory input transmitted by carotid sinus and vagus nerves.

17 ulation of baroreceptors in the aortic arch, carotid sinuses and coronary arteries, stimulation of re

18 ing baroreceptor regions in the aortic arch, carotid sinus, and right subclavian artery, as well as i

19 opulmonary bypass, reflexogenic areas of the carotid sinuses, aortic arch and coronary arteries and t

20 r signals from arterial baroreceptors in the carotid sinuses are processed within the brain and contr

21 f interaction between pulmonary arterial and carotid sinus baroreceptor reflexes; physiological and p

22 nesthetized healthy male rats, the bilateral carotid sinus baroreceptor regions were surgically isola

23 Selective pressure activation of carotid sinus baroreceptors in an isolated sinus or sele

24 of the total sensory input conveyed from the carotid sinus baroreceptors to the dlNTS is mediated by

25 ative feedback reflex mediated by aortic and carotid sinus baroreceptors when systemic arterial press

26 n MSNA was secondary to loading/unloading of carotid sinus baroreceptors.

27 nous oxyradicals produced in atherosclerotic carotid sinuses contribute to baroreceptor dysfunction.

28 In six intact and nine carotid sinus denervated (CSD) fetal sheep (125-128 days

29 Carotid sinus denervation blocked the Fos response to BC

30 l syncope (227 [35%]); other causes included carotid sinus hypersensitivity (37 [5.8%]), and a group

31 falls in older adults with cardioinhibitory carotid sinus hypersensitivity (CSH).

32 Carotid sinus hypersensitivity should be considered in a

33 ion for non-life-threatening situations (eg, carotid sinus hypersensitivity), most device advisories

34 Of the 10 studies, 4 addressed patients with carotid sinus hypersensitivity, and the remaining 6 addr

35 receptors in the wall of the aortic arch and carotid sinus initiates autonomic reflexes to change hea

36 subjects with a positive response to either carotid sinus massage (CSM) or lower body negative press

37 influenced by the method of execution of the carotid sinus massage and the coexistence of the cardioi

38 ch patient underwent a head-up tilt test and carotid sinus massage during continuous electrocardiogra

39 ion, and predominant cardioinhibition during carotid sinus massage or prolonged electrocardiogram mon

40 Carotid sinus massage reproduced spontaneous symptoms in

41 Duration of asystole during carotid sinus massage was similar in both groups (5.1 vs

42 nts (9.5%) had significant ECG pauses during carotid sinus massage.

43 climatization, which leads to an increase in carotid sinus nerve (CSN) activity and ensuing hypervent

44 to examine the effects of DIO and leptin on carotid sinus nerve (CSN) activity and the role of Trpm7

45 ne carotid body (CB) preparation to evaluate carotid sinus nerve (CSN) activity using physiologically

46 e responses to electrical stimulation of the carotid sinus nerve (CSN) and steady-state relationships

47 The carotid sinus nerve (CSN) conveys electrical signals fro

48 r, Ba2+ (3 and 5 mM) significantly increased carotid sinus nerve (CSN) discharge over baseline firing

49 lotrimazole and miconazole), we measured the carotid sinus nerve (CSN) discharge using an in vitro pe

50 e have traced the central projections of the carotid sinus nerve (CSN) in the cat by utilizing the tr

51 Directly stimulating the carotid sinus nerve (CSN) is known to potentially reduce

52 Supressing carotid body signalling through carotid sinus nerve (CSN) modulation may offer a therape

53 Carotid sinus nerve (CSN) stimulation with 0.5-0.8 s ele

54 a by increasing chemosensory activity in the carotid sinus nerve (CSN), causing hyperventilation and

55 t inputs, steady-state relationships between carotid sinus nerve (electrical) stimulation frequency a

56 We found that leptin enhanced carotid sinus nerve activity at baseline and in response

57 Leptin increased carotid sinus nerve activity at baseline and in response

58 e to stimuli that evoked robust increases in carotid sinus nerve activity in five out of five control

59 Carotid sinus nerve activity is augmented by adenosine b

60 m transients in glomus cells, and stimulates carotid sinus nerve activity through Olfr78.

61 x failure is most often due to damage of the carotid sinus nerve because of neck surgery or radiation

62 Stimulation of the carotid sinus nerve causes an increase in inspiratory (I

63 Four hours after injection of zymosan carotid sinus nerve chemoafferent discharge assessed in

64 ated SHRs and aged-matched sham-operated and carotid sinus nerve denervated Wistar rats.

65 Carotid sinus nerve denervation (CSD) has recently been

66 We have previously shown that carotid sinus nerve denervation (CSD) reduces arterial b

67 Successful carotid sinus nerve denervation (CSD) was confirmed by t

68 Electrical activity of the carotid sinus nerve did not change in five out of five h

69 ese mechanisms are as follows: (1) increased carotid sinus nerve discharge rate to the respiratory ce

70 Baroreceptor activity was measured from the carotid sinus nerve during pressure ramps in isolated ca

71 Some carotid sinus nerve fibers and tyrosine hydroxylase-posi

72 that TRPC1/3/4/5/6 proteins localize to the carotid sinus nerve fibers, some of which were immunorea

73 Carotid sinus nerve fibers, which originate from periphe

74 pecific electrode, direct stimulation of the carotid sinus nerve in anesthetized human subjects cause

75 In conclusion, carotid sinus nerve inputs from the carotid body are, in

76 oreceptors, or electrical stimulation of the carotid sinus nerve or brainstem mid-line.

77 s were attenuated by either resection of the carotid sinus nerve or propranolol.

78 Isolated carotid body/carotid sinus nerve preparations were used to assess per

79 tor function was examined by recording whole carotid sinus nerve responses to cessation of ventilatio

80 ure, an effect that was attenuated following carotid sinus nerve section (CSNX).

81 e in normoxia, which is attenuated following carotid sinus nerve section.

82 g and was reduced by 75% following bilateral carotid sinus nerve section.

83 n sham-operated animals but not in bilateral carotid sinus nerve sectioned (CSNX) animals.

84 y chemodenervated animals (vagi, aortic, and carotid sinus nerve sectioned).

85 ng serotonin receptor antagonism or episodic carotid sinus nerve stimulation (CSNS).

86 These studies support the concept that brief carotid sinus nerve stimulations cause a transient outwa

87 respiratory oscillator's phase responses to carotid sinus nerve stimulations in cats to the phase re

88 nerve, brief repeated carotid occlusions and carotid sinus nerve stimulations were performed to deter

89 block of baroreceptor afferent fibers in the carotid sinus nerve to examine the medullary projections

90 and transmit this sensory information in the carotid sinus nerve to the brain via neurons in the petr

91 y responses to electrical stimulation of the carotid sinus nerve were enhanced by CIH (p < 0.05).

92 ATP to activate chemoafferent fibres of the carotid sinus nerve which transmit this information to t

93 e in the number of unmyelinated axons in the carotid sinus nerve, compared with age-matched normoxic

94 In vitro CB chemosensory (carotid sinus nerve, CSN) responses to hypoxia (PO(2)=35

95 during anodal block of large A-fibers in the carotid sinus nerve.

96 itter release and electrical activity of the carotid sinus nerve.

97 bolished following interruption of vagal and carotid sinus nerves (from 0.301 +/- 0.012 to 0.311 +/-

98 nated by prior bilateral transections of the carotid sinus nerves or by prior inhibition of neurones

99 (NMDA) but without altering responses of the carotid sinus nerves to intracarotid cyanide.

100 The carotid sinus nerves were surgically denervated under ge

101 roreceptor innervation (aortic depressor amd carotid sinus nerves) was intact, rhythms correlated to

102 ythm was not altered after sectioning of the carotid sinus nerves.

103 ll totally dependent on the integrity of the carotid sinus nerves.

104 body stimulation on SND in rats with intact carotid sinus nerves.

105 ated atherosclerotic plaque formation at the carotid sinus of Adamts13(-/-)/ApoE(-/-) mice compared w

106 Exposure of the carotid sinus of normal rabbits to exogenous free radica

107 inus nerve during pressure ramps in isolated carotid sinuses of anesthetized rabbits.

108 agonist, unilateral pressure changes in the carotid sinus, or occlusion of the descending aorta in 1

109 agonist, unilateral pressure changes in the carotid sinus, or occlusion of the descending aorta in t

110 +/- 2.2 impulses s(-1)) whereas increases in carotid sinus pressure (CSP) induced significant decreas

111 o subsystems of the arterial baroreflex: the carotid sinus pressure (CSP)-sympathetic nerve activity

112 t to peak sensitivity) and the corresponding carotid sinus pressure (equivalent to 'set point').

113 In Series 1, an increase in carotid sinus pressure (from 8 to 26 kPa) during constan

114 Increased coronary or carotid sinus pressure induced a significant vasodilatat

115 on of baroreflex activity through electrical carotid sinus stimulation influences insulin sensitivity

116 Hypotension due to carotid sinus stimulation is frequent after CAS with bal

117 nd mean arterial pressure (MAP) responses to carotid sinus stimulation were used to develop reflex fu

118 ho responded to treatment with an electrical carotid sinus stimulator.

119 atic hypotension (OH, 3 vs. 23%, P < 0.001), carotid sinus syndrome (CSS, 0.6 vs. 9%, P < 0.001), and

120 Two groups of 34 consecutive patients with carotid sinus syndrome as the sole cause of falls and sy

121 Cardioinhibitory carotid sinus syndrome causes syncope, and symptoms resp

122 Patients with carotid sinus syndrome have similar rates of witnessed l

123 Assessment of the vasodepressor reflex in carotid sinus syndrome is influenced by the method of ex

124 Carotid sinus syndrome presents with both falls and sync

125 Syncopal subjects with carotid sinus syndrome were more likely to be older male

126 he clinical characteristics of patients with carotid sinus syndrome who presented with falls with tho

127 r loss of consciousness seen in fallers with carotid sinus syndrome.

128 nsitive nerve endings in the aortic arch and carotid sinus that play a critical role in acute regulat

129 Exposure of the carotid sinus to the free-radical scavengers superoxide

130 Acute changes in carotid sinus transmural pressure were evoked using 5 s

131 Acute changes in carotid sinus transmural pressure were evoked using brie

132 lates blood pressure during acute changes in carotid sinus transmural pressure.

133 d dogs, the splenic pedicle was tied and the carotid sinuses were vascularly isolated and perfused at