ライフサイエンスコーパス: carpel

1 nt in many angiosperms during genesis of the carpel.

2 ate stigmatic crests are conspicuous on each carpel.

3 ns for ideas on the origin of the angiosperm carpel.

4 plants is the female reproductive organ, the carpel.

5 egg cells are buried within the ovary of the carpel.

6 edundantly with other factors in stamens and carpels.

7 meristems, and within developing stamens and carpels.

8 tem to be maintained after the production of carpels.

9 vity and promotes development of stamens and carpels.

10 ation of petals into sepals and stamens into carpels.

11 ng the homeotic conversion of lodicules into carpels.

12 nversions of petals to sepals and stamens to carpels.

13 l sepal-petal-stamens and fourth whorl sepal-carpels.

14 ), a gene that specifies abaxial identity in carpels.

15 ers consisting almost entirely of sepals and carpels.

16 irects leaf development towards formation of carpels.

17 uired for development of petals, stamens and carpels.

18 from a gynoecium that consists of two fused carpels.

19 per serrations, serrated petals and deformed carpels.

20 apacity to make branches and result in extra carpels.

21 ally to ensure proper differentiation of the carpels.

22 orls of stamens, and an indefinite number of carpels.

23 uced organs in place of petals, stamens, and carpels.

24 ivity also results in an increased number of carpels.

25 the formation of specific tissues in ectopic carpels.

26 e fourth (innermost) whorl is made up of two carpels.

27 gions produced flowers with more stamens and carpels.

28 ttern of 5 sepals, 5 petals 5 stamens, and 2 carpels.

29 rs have 4 sepals, 4 petals, 6 stamens, and 2 carpels.

30 3 double mutants have completely derepressed carpels.

31 adds robustness to the apical fusion of the carpels.

32 nd more than five spirally arranged separate carpels.

33 organs in inner whorls and complete loss of carpels.

34 hibit supernumerary stamens but usually lack carpels.

35 hundreds of individual apocarpous (unfused) carpels.

36 sions of petals into sepals and stamens into carpels.

37 stamens and staminodia were transformed into carpels.

38 nt in anthers and pollen tube passage in the carpels.

39 ls, and a perianth distinct from stamens and carpels.

40 e MADS-box gene, resulted in more petals and carpels.

41 three whorls of sepals surrounding abnormal carpels.

42 produces flowers comprising only sepals and carpels.

43 nto female gametophyte-bearing organs termed carpels.

44 Specifically, we show that carpel abortion acts downstream of organ identity and re

45 derived aspects of maize flower development: carpel abortion and floral asymmetry.

46 y, including the evolution of the angiosperm carpel and anatropous bitegmic ovule.

47 show that all VRS genes repress fertility at carpel and awn emergence in developing lateral spikelets

48 ling, globulin expression, fruit dehiscence, carpel and epidermal development.

49 Although the processes involved in carpel and fruit morphogenesis are not well understood,

50 y homology to CRABS CLAW, a gene involved in carpel and nectary development in Arabidopsis.

51 elopment and that SYD is required for proper carpel and ovule development.

52 red for organ formation during embryo, leaf, carpel and ovule development.

53 rm radiations are identified: (i) the closed carpel and showy radially symmetrical flower, (ii) the b

54 a new function in the differentiation of the carpel and the control of seed size, acting downstream o

55 ING TRACT (NTT), which play pivotal roles in carpel and transmitting tract development, are downregul

56 um majus, predominantly in vascular tissues, carpels and anthers.

57 that bnq3 mutants have pale-green sepals and carpels and decreased chlorophyll levels, suggesting tha

58 hat has homeotic conversions of stamens into carpels and lodicules into palea/lemma-like structures.

59 which produces flowers with greatly reduced carpels and other floral organs.

60 floral meristems, and in developing stamens, carpels and ovules.

61 s did show cell abnormalities in stamens and carpels and produced extremely small fruit-like organs d

62 Female flowers have five fused carpels and ten arrested stamen primordia.

63 pe with respect to both primary (stamens and carpels) and secondary (petals) sexual traits.

64 exclusively expressed in leaves, stamens and carpels, and briefly in petal primordia.

65 eed yield include short filaments, defective carpels, and dysfunctional pollen grains.

66 ired for abaxial identity in both leaves and carpels, and encodes a nuclear-localized protein in the

67 between stamens and staminodes compared with carpels, and provide insight into the process of FMT, wh

68 lant sexual reproduction because stamens and carpels are absent from ag mutant flowers.

69 These individual carpels are arranged in a spiral pattern on the subtendi

70 Carpels are essential for sexual plant reproduction beca

71 hich homeotic transformations from sepals to carpels are found in flowers derived from old terminatin

72 The margins of the two fused carpels are meristematic in nature and give rise to plac

73 ering plants, the sexual organs (stamens and carpels) are composed almost entirely of somatic cells,

74 ans (integuments) and a model that considers carpels as analogs of complex leaves.

75 unction during the maturation of stamens and carpels, as well as in their early development.

76 ith one additional petal, sepal, stamen, and carpel at each of the four whorls, respectively, thus un

77 nd found to be a ridge with the fourth whorl carpels at the summit and the first whorl transverse sep

78 it mosaic floral organs typified by multiple carpels at the total or partial expense of stamens.

79 are required to specify petals, stamens, and carpels because these organs are converted into sepals i

80 ressed early in floral development, controls carpel cell number, and has a sequence suggesting struct

81 , which also affects the formation of stamen-carpel chimera due to fon1 mutations.

82 ies were particularly enhanced in the warmer carpels compared with stamens during the cold night befo

83 grains that contain male gametes, while the carpels contain the ovules that when fertilized will pro

84 etals and stamens are missing and sepals and carpels develop in their place.

85 lopment, C-genes are required for stamen and carpel development and floral determinacy, and D-genes w

86 he expression of different genes involved in carpel development and phytohormonal pathways regulation

87 Activation of carpel development by STM is independent of LEAFY and WU

88 To advance our knowledge of carpel development in the absence of pollination, we cre

89 form crucial functions specifying stamen and carpel development in the flower and controlling late fr

90 (Ts6) and tasselseed4 (ts4) mutations permit carpel development in the tassel while increasing merist

91 ts shown here of light quality perception on carpel development lead us to speculate on the potential

92 t the phenotype is either independent of the carpel development program or that fdh-1 mutations activ

93 is provided by the restoration of wild-type carpel development to spt mutants by low red/far-red lig

94 terized rice gene that specifically controls carpel development under heat stress, ensuring plant fem

95 vules suggest that the molecular toolkit for carpel development was largely present in the last commo

96 that the two genes act linearly in leaf and carpel development, but synergistically in the developme

97 al function of CRC lies in the regulation of carpel development, it may have been co-opted as a regul

98 loral C-function, which specifies stamen and carpel development, played a pivotal role in the evoluti

99 The essential role for STM in carpel development, together with its previous reported

100 g approach provides a new tool for examining carpel development, which we hope will advance research

101 inhibited carpel fusion to complete loss of carpel development.

102 of the SAM, and have also been implicated in carpel development.

103 nome-wide expression during early stamen and carpel development.

104 SHP genes are responsible for AG-independent carpel development.

105 rms of key transcription factors involved in carpel development.

106 ich results in modified cell division during carpel development.

107 AG activity indicates that an AG-independent carpel-development pathway exists.

108 ets OsETTIN1 and OsETTIN2 redundantly ensure carpel differentiation.

109 tion under short days, adaxialize leaves and carpels, disrupt the phyllotaxis of the inflorescence, a

110 pically and transcriptionally similar to the carpel, due to the parasite hijacking underlying genetic

111 nitially perfect but abort either stamens or carpels during their development, indicating that sex de

112 fication of reproductive organs (stamens and carpels) during the early steps of flower development.

113 nct organ types (sepals, petals, stamens and carpels), each of which may be a modified leaf.

114 including defects in petal polar expansion, carpel elongation, and anther and ovule differentiation.

115 Angiosperms are defined by carpels enclosing ovules, a character demonstrated in th

116 Since carpel epidermal derivatives manifest both of these prop

117 In Arabidopsis thaliana, mutations in CARPEL FACTORY (CAF), a Dicer homolog, and those in a no

118 A new recessive mutant, carpel factory (caf), converts the floral meristems to a

119 The previously described roles of CARPEL FACTORY in the development of Arabidopsis embryos

120 N1, and demonstrate its identity to the CAF (CARPEL FACTORY) gene important for normal flower morphog

121 Mutation of an Arabidopsis Dicer homolog, CARPEL FACTORY, prevents the accumulation of miRNAs, sho

122 f carpel initiation, a phenocopy for loss of CARPEL FACTORY/DICER LIKE1, indicating that miRNA is cri

123 ponent of the spectacular diversification of carpel (flower and fruit) form and reproductive cycles i

124 Here, we characterized floral organs in carpels (foc), an Arabidopsis mutant with a Ds transposo

125 In addition, extra carpels form in female florets and ovule tissue prolifer

126 rise to a floral phenotype in which ectopic carpels form.

127 ed mechanisms in plant organs that, like the carpel, form within the shade of surrounding tissues.

128 ome overrides female development to suppress carpel formation and promote stamen development.

129 e KNOX gene SHOOT MERISTEMLESS (STM) induces carpel formation and promotes homeotic conversion of ovu

130 organogenesis and repress key regulators of carpel formation.

131 o decrease in the seed but fluctuated in the carpels from 10 to 30 days post-anthesis (DPA).

132 ding to the previously identified stamen and carpel functions for GRCD1 and GRCD2, two partially redu

133 HEC1 interacts with SPATULA (SPT) to control carpel fusion and that both transcription factors restri

134 formation of placental tissues and inhibited carpel fusion to complete loss of carpel development.

135 the post-anthesis viability of unpollinated carpels has been overlooked, despite its importance for

136 are small follicles formed from conduplicate carpels helically arranged.

137 patterns were examined in perianth, stamens, carpel, hypanthial tube and corona tissue.

138 ified previously as regulators of stamen and carpel identities and floral determinacy because the rec

139 required for the specification of stamen and carpel identities and for the proper termination of stem

140 Stamen and carpel identities are specified by the combinatorial act

141 dopsis floral meristem to specify stamen and carpel identity and to repress further proliferation of

142 , whose members typically promote stamen and carpel identity as well as floral meristem determinacy.

143 gene and as such ultimately depends upon the carpel identity gene AG for proper gene expression.

144 OsMAIL1 expression correlates with that of carpel identity genes and RNA-seq of osmail1-1 mutant co

145 showed that OsMAIL1 is required to activate carpel identity genes expression when floral meristems a

146 l feature IbAG appears to specify stamen and carpel identity, but is not sufficient to specify merist

147 ity, stunting or weak transformation towards carpel identity.

148 s to the development of petals, stamens, and carpels in addition to sepals and that it plays an impor

149 Carpels in maize undergo programmed cell death in half o

150 perianth in Nuphar, and between stamens and carpels in Persea.

151 ermination occurs through abortion of female carpels in the tassel and arrest of male stamens in the

152 the boundary between stamens in whorl 3 and carpels in whorl 4, as superman mutants exhibit supernum

153 roliferation of numerous petals, stamens and carpels indicating loss of floral determinacy.

154 In flowers, phv1 causes reiteration of carpel initiation, a phenocopy for loss of CARPEL FACTOR

155 uired for floral organogenesis, particularly carpel initiation, and acts through the auxin pathway in

156 r indicate a specific requirement for STM in carpel initiation, as further supported by the loss of l

157 based on cell division dynamics that precede carpel initiation.

158 Flowers in catkins from modified events had 'carpel-inside-carpel' phenotypes.

159 sulted in homeotic conversion of stamens and carpels into sepaloid organs and loss of flower determin

160 The carpel is the female reproductive organ of flowering pla

161 In Arabidopsis, congenital fusion of two carpels leads to the formation of an enclosed gynoecium.

162 pidermal cells were ectopically expressing a carpel-like program.

163 ls and petals, and conversion of sepals into carpel-like structures that grew into fruits and ripened

164 that preceded the origin of the true closed carpel, long styles, multiseeded ovaries, and, in monoco

165 gynoecium consists of two congenitally fused carpels made up of two lateral valve domains and two med

166 ere, we show that a mechanism that regulates carpel margin development in the model flowering plant A

167 r double mutants, failed to establish normal carpel margin meristem (CMM) and its derivative tissues,

168 nce meristem (IM), floral meristem (FM), and carpel margin meristem (CMM).

169 iptional insight into the development of the carpel margin meristem in Arabidopsis.

170 In Arabidopsis (Arabidopsis thaliana), the carpel margin meristem is a vital meristematic structure

171 of the medial ovary domain that contains the carpel margin meristem, a vital female reproductive stru

172 female reproductive tissues derived from the carpel margin meristem.

173 female reproductive tissues derived from the carpel margin.

174 r along a specifically developed junction at carpel margins.

175 een leaves and showed green anthers, central carpels, mature pods, and seeds during senescence.

176 ractions, GT1 and RA3 proteins colocalize to carpel nuclei in developing flowers.

177 ards differing significantly in others, like carpel number and petal length.

178 anscription factor (fasciated) that controls carpel number during flower and/or fruit development.

179 V3 expression was negatively correlated with carpel number in cucumber cultivars.

180 The carpel number is an important fruit trait that affects f

181 CsWUS, CsFUL1(A) and CsARF14 in determining carpel number variation in an important vegetable crop -

182 hermore, we found that auxin participated in carpel number variation in cucumber through interaction

183 n as a negative and a positive regulator for carpel number variation, respectively.

184 n of adult vegetative traits, an increase in carpel number, and produce abnormal spacing of flowers i

185 ii) AG homologs are expressed in stamens and carpels of most basal angiosperms, in agreement with the

186 On the other hand, the basipetal carpels of the 35S::XAL2 lines lose determinacy and main

187 e homeotic conversion of sepals into petals, carpels, or stamens, depending on the genetic context.

188 horl of sepals surrounding a fourth whorl of carpels, or three whorls of sepals surrounding abnormal

189 ncreased fruit size chiefly due to increased carpel ovary cell number.

190 l somatic sectors were observed in siliques, carpels, petals and sepals while stemlike organs (filame

191 face metabolites of different flower organs (carpels, petals, and sepals) were profiled for the first

192 kins from modified events had 'carpel-inside-carpel' phenotypes.

193 controlling cell proliferation in stamen and carpel primordia and in ovules during flower development

194 two congenitally fused, laterally positioned carpel primordia bisected by two medially positioned mer

195 AGL1 expression at the tip of the growing carpel primordia, along the margins of the ovary valves

196 ained in stamen primordia, but excluded from carpel primordia, as well as vegetative tissues.

197 S-box family that is expressed in stamen and carpel primordia.

198 o a complex pattern within petal, stamen and carpel primordia.

199 Carpels produce heat with sugars transported from leaves

200 er development, specification of stamens and carpels requires the AGAMOUS gene.

201 to be one half of a sepal and one half of a carpel, respectively.

202 tamens are partially converted to sepals and carpels, respectively.

203 n may be involved in evolutionary control of carpel shape.

204 Female gymnosperm cones and angiosperm carpels share conserved genetic features, which may be a

205 t program or that fdh-1 mutations activate a carpel-specific developmental program downstream of the

206 -dependent manner, suggesting that AGL1 is a carpel-specific gene and as such ultimately depends upon

207 of CsWUS resulted in more sepals, petals and carpels, suggesting that CsCLV3 and CsWUS function as a

208 chromosome that is genetically linked to the carpel-suppressing locus.

209 d identify the gene that was affected by the carpel-suppressing mutation that was involved in the evo

210 etermining loci on the Y chromosome, one for carpel suppression, one for early stamen development, an

211 To identify additional regulators of carpel suppression, we performed a gt1 enhancer screen a

212 stem suppression, later recruited to mediate carpel suppression.

213 ve similar phenotypes, with more stamens and carpels than either fon1 or clv single mutants.

214 lorets of drl1 ears are sterile with unfused carpels that fail to enclose an expanded nucellus-like s

215 The evolution of the carpel, the defining feature of angiosperms, remains a f

216 ch leads to the specification of stamens and carpels, the reproductive organs of flowers.

217 ne expression patterns between the seeds and carpels, the two tissues showed a cooperative relationsh

218 aled possible genomic bases for petal color, carpel thermogenesis and domestication in lotus.

219 metabolism and transportation contributed to carpel thermogenesis.

220 member, display ectopic formation of adaxial carpel tissues only when the functions of other genes, s

221 s the reproductive tract, develop within the carpel to facilitate the journey of the pollen tube.

222 ble mutant had similar numbers of petals and carpels to bre.

223 ls that grow over long distances through the carpel toward the ovules, where double fertilization is

224 hape this variation, we examined 22 seed and carpel transcriptomes from 3 varieties of sesame with hi

225 g three whorls of sepals surrounding fertile carpels, two whorls of sepals with a diminished third wh

226 nd promoted the development of supernumerary carpels under water-stress conditions.

227 We show that the unpollinated carpel undergoes a well-defined initial growth phase, fo

228 nd, frequently, the premature rupture of the carpel valves.

229 of ovules depends on EPFL2 expression in the carpel wall and in the inter-ovule spaces, where it acts

230 rom the ERECTA (ER) family that act from the carpel wall and the placental tissue.

231 pathway controlled by EPFL9 acting from the carpel wall through the LRR-receptor kinases ER, ERL1, a

232 earing microsporangia of the anthers and the carpel walls of the gynoecium, which enclose the ovules.

233 differentiation of adaxial cell types in the carpel walls, and in the establishment of the correct nu

234 nd promotes homeotic conversion of ovules to carpels when ectopically expressed in flowers, as previo

235 entified transcripts are found in stamens or carpels, whereas few genes are predicted to be expressed

236 a3 single mutants have partially derepressed carpels, whereas gt1;ra3 double mutants have completely

237 3-RNAi led to increased number of petals and carpels, whereas overexpression of CsWUS resulted in mor

238 in, is involved in maintenance of the stamen/carpel whorl boundary (the boundary between whorl 3 and

239 In the sepal and carpel whorls the smallest sectors of marked and unmarke

240 embryos resulting from crosses of wild-type carpels with PRL::GUS pollen do not stain and are phenot

241 entric whorls of sepals, petals, stamens and carpels, with each of these floral organ types having a