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1 eased with increasing grade of the articular cartilage lesion.
2 itional surgically confirmed focal articular cartilage lesion.
3 aps in the detection of surgically confirmed cartilage lesions.
4 l degeneration, is a predisposing factor for cartilage lesions.
5 utine MR arthrography protocol for depicting cartilage lesions.
6 in the ACL increased with the development of cartilage lesions.
7  in parameters between methods for detecting cartilage lesions.
8 e noncanonical signaling Wnt5a did not cause cartilage lesions.
9 he routine MR imaging protocol for detecting cartilage lesions.
10 ed over the same interval were evaluated for cartilage lesions.
11  method could thus be used for the repair of cartilage lesions.
12 luation with particular emphasis on bone and cartilage lesions.
13 her specificity (P < .01) for helping detect cartilage lesions (92.2% for VIPR-SSFP and 88.4% for rou
14 s with a KL score of 4 showed full-thickness cartilage lesions and bone marrow edema pattern.
15 opment of osteoarthritis and correlates with cartilage lesions and clinical osteoarthritis scores.
16      PF152 treatment of dogs with OA reduced cartilage lesions and decreased biomarkers of type II co
17 ignificantly lower specificity for detecting cartilage lesions and higher accuracy for grading cartil
18 curacy of both imaging methods for detecting cartilage lesions and meniscal tears were determined.
19 roducts are currently in clinical trials for cartilage lesions and meniscal tears, opening new avenue
20 ogic changes seen in OA, including articular cartilage lesions and osteophytes, were present in the m
21 f anterosuperior labral tear, anterosuperior cartilage lesion, and abnormal alpha angle was recorded.
22 ted significantly (P <.05) with the grade of cartilage lesions, and a substantially higher percentage
23 y two musculoskeletal radiologists to detect cartilage lesions, anterior and posterior cruciate ligam
24 tine MR imaging protocol in the detection of cartilage lesions, anterior cruciate ligament tears, and
25 R imaging at 3.0 T increased the accuracy of cartilage lesion assessment when compared with imaging a
26 t higher sensitivity (P = .73) for detecting cartilage lesions at 3.0 T than at 1.5 T.
27 int space narrowing and with the presence of cartilage lesions at baseline and worsening during follo
28 in sensitivity and specificity for detecting cartilage lesions between the two sequences.
29 ere obtained and analyzed by two readers for cartilage lesions, bone marrow edema pattern, and ligame
30                                              Cartilage lesions, bone marrow edema pattern, and menisc
31  MRI protocol increases sensitivity to early cartilage lesions but is time consuming.
32         Samples were regionally assessed for cartilage lesions by visual inspection using Outerbridge
33  a routine MR imaging protocol for detecting cartilage lesions, cruciate ligament tears, collateral l
34        Interobserver agreement for detecting cartilage lesions did not differ between the two techniq
35 mage 10 porcine knees in which 29 artificial cartilage lesions had been created.
36   Caspase inhibitors reduced the severity of cartilage lesions in experimental OA, suggesting that th
37  inflammatory factors and the development of cartilage lesions in OA animal models.
38 relationships with meniscal degeneration and cartilage lesions in osteoarthritis.
39 ore rapid progression of cartilage loss than cartilage lesions in the anterior and posterior portions
40 without OA risk factors and with more severe cartilage lesions in the group with risk factors.
41  of Nupr1 expression reduced the severity of cartilage lesions in this model.
42  chondrogenic precursor cells from repairing cartilage lesions, leading to accelerated cartilage degr
43 utine MR imaging protocol was used to detect cartilage lesions, ligament tears, meniscal tears, and b
44                                              Cartilage lesions located in the central region of the m
45                                     Although cartilage lesions occur in the ankles, osteoarthritis ra
46                                              Cartilage lesions of OA were significantly less severe i
47 cy causes an increased severity of articular cartilage lesions of OA without the bony lesions normall
48 ion of matrix turnover that is seen in early cartilage lesions of the ankle would appear to represent
49 gery to determine the presence or absence of cartilage lesions on each articular surface, first by us
50      Two weeks of loading produced articular cartilage lesions only at sites of maximal contact as ex
51 ol contralateral joints, including articular cartilage lesions, osteophyte formation, and pathologic
52                      Despite the presence of cartilage lesions, osteophytes and subchondral sclerosis
53 R had a higher accuracy in correctly grading cartilage lesion (P = .012-.013).
54 MAC scores were found only for the grades of cartilage lesions (P <.05).
55          Presence of labral tears, articular cartilage lesions, paralabral cysts, os acetabuli, and s
56 nges and to determine their correlation with cartilage lesion patterns at all stages of osteoarthriti
57 em enabled significantly higher detection of cartilage lesion progression than did WORMS or BLOKS sys
58                             For detection of cartilage lesions, sensitivity of projection reconstruct
59       However, spatial relationships between cartilage lesion severity (CLS) and microstructural chan
60  labral lesions, paralabral cysts, articular cartilage lesions, subchondral cysts, osteophytes, and s
61 lage lesions and higher accuracy for grading cartilage lesions than did a routine MR arthrography pro
62  system is a reproducible scoring system for cartilage lesions that yields an improved detection rate
63 ture knee joints most often causes articular cartilage lesions, this study was undertaken to characte
64   At the time of arthroscopy, each articular cartilage lesion was graded by using the Noyes classific
65 he sensitivity and specificity for detecting cartilage lesions was 74% and 77%, respectively, for IDE
66 ower specificity of IDEAL-SPGR for detecting cartilage lesions was not seen in experienced readers.
67                    The prevalence of hyaline cartilage lesions was particularly high (86% at baseline
68 cificity, and accuracy for detecting all 192 cartilage lesions were 68.5%, 92.6%, and 84.5% for IDEAL
69 ty, and accuracy of MR imaging for detecting cartilage lesions were 69.3%, 78.0%, and 74.5% at 1.5 T
70 vity and specificity in the detection of 351 cartilage lesions were 74.6% and 97.8%, respectively, fo
71 he 1.5- and 3.0-T MR protocols for detecting cartilage lesions were determined by using arthroscopy a
72 the two techniques for detecting and grading cartilage lesions were determined.
73                                              Cartilage lesions were found in 95 (79.0%) of 120 knees,
74  MR imaging provides superior delineation of cartilage lesions when compared with two other sequences
75                               Progression of cartilage lesions with each scoring system was compared
76 safe and feasible for the treatment of focal cartilage lesions with promising preliminary evidences o
77 0 T improved sensitivity in the detection of cartilage lesions within the knee joint from 74.6% to 88