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1 d specific (91%) but not sensitive (23%) for cartilage loss.
2 fundamentally different from those producing cartilage loss.
3 observed when posterior femur regions showed cartilage loss.
4 PRD can compromise this function and produce cartilage loss.
5 s in increased disability and progression of cartilage loss.
6 and ACL tears was associated with more rapid cartilage loss.
7 reased subchondral bone activity can predict cartilage loss.
8 us decrease in bacterial burden and prevents cartilage loss.
9 OA), a degenerative disease characterized by cartilage loss.
10 incident and progressive medial tibiofemoral cartilage loss.
11 raphic risk factors was predictive of future cartilage loss.
12 ic regression models were applied to predict cartilage loss.
13  were evaluated as possible risk factors for cartilage loss.
14 at might otherwise contribute to progressive cartilage loss.
15  disease, including protection from bone and cartilage loss.
16 ly associated with an increased risk of fast cartilage loss.
17  point to reflect osteoarthritis progression/cartilage loss.
18 rtilage loss, and only 20 (5.8%) showed fast cartilage loss.
19 ls were subregions in that same knee without cartilage loss.
20 bone marrow lesions (BMLs) have been tied to cartilage loss.
21 medial extension were related to ipsilateral cartilage loss.
22 nd reduced height also increased the risk of cartilage loss.
23 calcinosis was not associated with increased cartilage loss.
24 rong association between meniscal damage and cartilage loss.
25 this function has important consequences for cartilage loss.
26 ion was associated with an increased risk of cartilage loss.
27 with a higher baseline BML score had greater cartilage loss.
28 rging BMLs are strongly associated with more cartilage loss.
29  miss a substantial proportion of knees with cartilage loss.
30 owed a higher average rate of progression of cartilage loss (22%) than that seen in those who had int
31 s related predominantly to a reduced risk of cartilage loss (adjusted OR = 0.3, 95% CI 0.1-0.8).
32 ment, and laxity each predicted tibiofemoral cartilage loss after controlling for the other factors.
33 artment) and determine their relationship to cartilage loss and ACL tears.
34 ion between clinical outcome and severity of cartilage loss and bone marrow edema in the medial femor
35  APM was associated with greater severity of cartilage loss and bone marrow edema in the same compart
36                              Despite greater cartilage loss and bone sclerosis in medial condyles, th
37 iographically, patients experience continued cartilage loss and destructive bone changes as they age,
38 especially vitamin C, may reduce the risk of cartilage loss and disease progression in people with OA
39 e acetonide every 3 months on progression of cartilage loss and knee pain.
40 ective against trabecular bone and articular cartilage loss and markedly prevented neurogenesis decli
41 echanical stress can accelerate the onset of cartilage loss and progression to OA in transgenic mice.
42 degenerative joint disease, characterized by cartilage loss and subchondral bone remodeling in respon
43 nt swelling, histopathological signs of AIA, cartilage loss and suppressed TNFalpha induction.
44 or, pegsunercept, the number of osteoclasts, cartilage loss, and number of TNF-alpha and receptor act
45           Of 347 knees, 90 (25.9%) exhibited cartilage loss, and only 20 (5.8%) showed fast cartilage
46 tory of these lesions, their relationship to cartilage loss, and the association between change in th
47 ociation between change in these lesions and cartilage loss are unknown.
48 gions of the tibiofemoral joint, and defined cartilage loss as an increase in score (scale 0-4) at an
49 on of joint space narrowing is predictive of cartilage loss assessed on MRI, radiography is not a sen
50                           Using quantitative cartilage loss assessment, local factors that independen
51 p between baseline alignment and subregional cartilage loss at 2 years, adjusting for age, sex, body
52 ationship between severity of full-thickness cartilage loss at baseline and incident SCs.
53 reater quadriceps strength protected against cartilage loss at the lateral compartment of the patello
54 r quadriceps strength was protective against cartilage loss at the lateral compartment of the patello
55 e and at 15 and 30 months was used to assess cartilage loss at the tibiofemoral and patellofemoral jo
56 ater quadriceps strength had no influence on cartilage loss at the tibiofemoral joint, including in m
57  association between quadriceps strength and cartilage loss at the tibiofemoral joint.
58 ties in both early and chronic OA, including cartilage loss, bone edema, synovial enhancement, osteop
59 s with chondrocalcinosis had a lower risk of cartilage loss compared with knees without chondrocalcin
60                  It is unclear how articular cartilage loss contributes to pain in patients with knee
61  the medial compartment, 104 knees (46%) had cartilage loss detected by MRI.
62 d clinical features that clearly distinguish cartilage loss due to disuse atrophy and those due to os
63                                  To simulate cartilage loss from disease or injury, the top layers of
64 nd that changes in oxygen tensions following cartilage loss from injury or disease alter cartilage me
65 d in each plate, with progression defined as cartilage loss >2 times the coefficient of variation for
66                                              Cartilage loss in a subregion was defined as an increase
67 us alignment with a reduction in the risk of cartilage loss in certain lateral subregions.
68 d with a reduction in the risk of subsequent cartilage loss in certain medial subregions and neutral
69 4-expressing articular cartilage accelerates cartilage loss in DMM-induced OA.
70  as dichotomous predictors in each model) on cartilage loss in each of the 5 plates within a compartm
71  and their death by apoptosis contributes to cartilage loss in inflammatory joint diseases, such as r
72 hanism of the long-term alignment effects on cartilage loss in knee OA.
73 s is unrelated to the risk of joint space or cartilage loss in knee OA.
74 inal BMD loss is associated with progressive cartilage loss in knees with OA.
75 rategy that addresses both pain symptoms and cartilage loss in OA.
76                      MRI can detect interval cartilage loss in patients over a short period (<2 years
77 ber of basal progenitor cells accompanied by cartilage loss in Shh-Cre;Gpr177(loxp/loxp) mutants.
78 scured the rate and variability of articular cartilage loss in subjects with knee OA.
79                                    Articular cartilage loss in the bGH mice was associated with eleva
80 ion between clinical outcome and severity of cartilage loss in the lateral femoral condyle and latera
81                            A reduced risk of cartilage loss in the lateral subregions was associated
82 rsus valgus) knees each have reduced odds of cartilage loss in the lateral subregions.
83 ersus varus) knees each have reduced odds of cartilage loss in the medial subregions and that neutral
84                            A reduced risk of cartilage loss in the medial subregions was associated w
85 confidence interval 5.6-9.9, P < 0.0001) for cartilage loss in the same subregion compared with subre
86               The incidence of arthritis and cartilage loss in vaccinated DBA/1 mice was remarkably l
87 disability worldwide and is characterized by cartilage loss, inflammation, and pain.
88                                              Cartilage loss is a leading cause of disability among ad
89 rgoing great compressive stress and in which cartilage loss is inevitable.
90                                              Cartilage loss may be partial or complete, and it may af
91                                              Cartilage loss occurred frequently in the central region
92 exhibited two or more subregions with severe cartilage loss (odds ratio [OR], 16.5; 95% confidence in
93 t space narrowing on radiographic images and cartilage loss on MRI, using a generalized estimating eq
94 radiographic progression being predictive of cartilage loss on MRI.
95 amin D supplementation for preventing tibial cartilage loss or improving WOMAC knee pain in patients
96         Knees were also classified as having cartilage loss or osteophyte growth if their maximal joi
97         SBA may directly influence overlying cartilage loss or serve as a marker of an area undergoin
98 nce interval [95% CI] 2.2-16.2) but not with cartilage loss (OR 1.6, 95% CI 0.8-3.1), while medial ty
99 le medial type 2 BMLs were related to medial cartilage loss (OR 6.1, 95% CI 1.0-35.2).
100 tly or compared quantitative and qualitative cartilage loss outcomes.
101              We evaluated the association of cartilage loss over 30 months with the presence of basel
102                                              Cartilage loss over 6 months is rare, but may be detecte
103 t baseline had a predilection for more rapid cartilage loss (P <or= 0.93).
104          After adjustment for full-thickness cartilage loss, prevalent BMLs showed a strong and signi
105 ontribution of MCs to joint inflammation and cartilage loss remains poorly understood.
106 the BOKS, vitamin D levels were unrelated to cartilage loss seen on MRI.
107                    In the BOKS, we evaluated cartilage loss semiquantitatively, using the Whole-Organ
108 djustment for BMLs, prevalent full-thickness cartilage loss showed a significant but much less import
109 compartment showed more rapid progression of cartilage loss than cartilage lesions in the anterior an
110 l proportion of knees (80 of 189 [42%]) with cartilage loss visible on MRI when no radiographic progr
111                    Adjusted relative risk of cartilage loss was 2.03 (95% confidence interval [CI]: 1
112  for change in the number of subregions with cartilage loss was assessed using Poisson regression, wi
113                                         Fast cartilage loss was defined as a WORMS of at least 5 (lar
114                                              Cartilage loss was determined from knee radiographs take
115                                              Cartilage loss was graded in the anterior, central, and
116 ation of BML change with medial tibiofemoral cartilage loss was not significant after adjusting for a
117                                              Cartilage loss was present if the score in any region of
118 d synovitis or effusion) to the risk of fast cartilage loss were assessed by using a multivariable lo
119                Risk factors for tibiofemoral cartilage loss were baseline meniscal extrusion (adjuste
120                 Predictors of patellofemoral cartilage loss were effusion and prevalent cartilage dam
121                 Predictors of patellofemoral cartilage loss were effusion, with an adjusted odds rati
122                         Disease severity and cartilage loss were evaluated by histopathological analy
123                    Strong predictors of fast cartilage loss were high BMI (adjusted odds ratio [OR],
124                   Predictors of tibiofemoral cartilage loss were prevalent cartilage damage, bone mar
125 on, even after adjustment for full-thickness cartilage loss, which supports the bone contusion theory
126 ithin a knee were defined as subregions with cartilage loss, while controls were subregions in that s
127 also recorded in only one case of acetabular cartilage loss with both methods.
128 observers was found in four cases of femoral cartilage loss with both MRa and CTa.
129 ciation of prevalent BMLs and full-thickness cartilage loss with incident SCs in the same subregion w
130 oreover, there has been a tendency to equate cartilage loss with osteoarthritic degeneration.
131              SBA is strongly associated with cartilage loss within the same subregion of a knee.
132 re evaluated whether SBA was associated with cartilage loss within the same subregion of the knee.

 
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