ライフサイエンスコーパス: caseous

1 e tuberculous granulation tissue surrounding caseous and liquefied pulmonary foci and cavities, we fo

2 Echocardiographic images of caseous calcification are often heterogenous due to calc

3 However the appearance of caseous calcification can imitate that of abscess, tumor

4 Caseous calcification of mitral annulus is rather rare e

5 ary sarcoma, 2; myxoma, 4; fibroelastoma, 1; caseous calcification of mitral annulus, 3; and thrombus

6 y large, often with an ulcerated surface and caseous core.

7 to rabbit lung tissues containing a lesion (caseous granuloma) caused by tuberculosis infection.

8 g absence of tubercle lesions grossly and of caseous granulomas histologically.

9 Caseous granulomas were observed in the lung as early as

10 Only caseous granulomas were observed in the lungs at these e

11 Treated RMs retain caseous granulomas with high FDG uptake and incomplete C

12 r and local tissue environment, resulting in caseous granulomas with incomplete bacterial sterilizati

13 cell alpha-chemoattractant) within solid and caseous granulomas, and there was only minimal expressio

14 lli in low-oxygen microenvironments, such as caseous granulomas, has been hypothesized to have the po

15 and that the highest signal intensity was in caseous granulomas.

16 in a similar microenvironment to that of the caseous human lesion.

17 entrations in hard-to-treat lesions, such as caseous lesions and cavities.

18 xin production leads to development of large caseous lesions, and in infective endocarditis, increase

19 s within the nutrient-starved environment of caseous lung granulomas.

20 Caseous lymphadenitis is a chronic suppurative disease c

21 Caseous lymphadenitis was first reported for goats in th

22 ys a major role in the liquefaction of solid caseous material and in the subsequent cavity formation.

23 Although caseous mitral annular calcification is typically an inc

24 CT brought the suggestion of caseous mitral annular calcification.

25 in tuberculosis immunopathology, leading to caseous necrosis and compromising the immune response, r

26 ion may be a critical initial event, causing caseous necrosis as opposed to resulting from it.

27 from intense granulomatous inflammation with caseous necrosis for infection with type C to minimal in

28 l tenet of tuberculosis pathogenesis is that caseous necrosis leads to extracellular matrix destructi

29 egates of leukocytes and a greater degree of caseous necrosis than those from JH2-2-infected mice.

30 lar to TB in humans, namely, granulomas with caseous necrosis, liquefaction, and cavities.

31 ion of those from inbred rabbits showed more caseous necrosis, more visible bacilli, and fewer mature

32 Zebrafish tuberculous granulomas undergo caseous necrosis, similar to human tuberculous granuloma

33 n of human matrix metalloproteinase 1 causes caseous necrosis, the pathological hallmark of human tub

34 acellular matrix destruction map to areas of caseous necrosis.

35 surrounding bacteria, infected cells, and a caseous necrotic core.

36 was done and dilated bile ducts filled with caseous necrotic material were seen intra-operatively.

37 into preexisting granulomas, including their caseous (necrotic) centers, through specific mycobacteri

38 le adduct formation surrounding necrotic and caseous regions of pulmonary granulomas by immunohistoch

39 ly it was shown that PZA penetrates necrotic caseous TB lung lesions and kills nongrowing, drug-toler

40 d by the similar induction of MMP-9 in human caseous tuberculosis (TB) granulomas.

41 Expression of NPC2 was lower in caseous tuberculosis granulomas and M. tuberculosis-infe

42 Liquefaction of solid caseous tuberculous lesions and the subsequent cavity fo