ライフサイエンスコーパス: catabolite

1 as the export of their degradation products (catabolites).

2 lterations in hexoses, lysolipids and purine catabolites.

3 accumulation of immunosuppressive tryptophan catabolites.

4 rther narrow down the structure of the found catabolites.

5 ient deprivation due to decreasing glutamine catabolites.

6 bundance of several nitrogen-rich nucleotide catabolites.

7 ecific biomarkers of interest were oxidative catabolites.

8 When exposed to beta-lactams, however, PG catabolites (1,6-anhydroMurNAc-peptides) accumulate in t

9 at catalyzes oxidation of IAA to its primary catabolite 2-oxindole-3-acetic acid (oxIAA) remains unch

10 cation of AP at the N-terminus and the minor catabolite [29-270] by truncations of either side of the

11 ucuronide and the colonic microbiota-derived catabolite 3-(3'-hydroxy-4'-methoxyphenyl)hydracrylic ac

12 The major catabolite [3-285] was formed by truncation of AP at the

13 f drug-related material, whereas that of the catabolites [3-285] and [29-270] accounted for 66% and 9

14 Urinary excretion of MTX and its catabolite, 7-OH-MTX, was measured in 2 24-hour urine sp

15 In vitro, ACD2 can reduce red chlorophyll catabolite, a chlorophyll derivative.

16 Total urinary and fecal excretion of catabolites accounted for <5% of each of the administere

17 Another catabolite, acrylic acid, affects meristem development b

18 K, but no such relationship was observed for catabolite-activated genes, suggesting that large number

19 The catabolite activator protein (CAP) bends DNA in the CAP-

20 An E. coli catabolite activator protein (CAP) has been converted in

21 The catabolite activator protein (CAP) of Escherichia coli i

22 he binding of DNA to several variants of the catabolite activator protein (CAP) that differentially p

23 haracterize cyclic AMP (cAMP) binding to the catabolite activator protein (CAP), a transcriptional ac

24 tion complex comprising the Escherichia coli catabolite activator protein (CAP), RNA polymerase holoe

25 we apply it to a challenging test case: the catabolite activator protein (CAP).

26 n TTHB099 (TAP) [homolog of Escherichia coli catabolite activator protein (CAP)], T. thermophilus RNA

27 e cAMP-mediated allosteric transition in the catabolite activator protein (CAP; also known as the cAM

28 ynechocystis sp. adenylyl cyclase (Cya1) and catabolite activator protein (SYCRP1) mutants to differe

29 terial transcription factors, including four catabolite activator protein homologues.

30 The catabolite activator protein is a dimer that consists of

31 ion curves for cells with and without active catabolite activator protein.

32 ulations of 5 ns on protein-DNA complexes of catabolite-activator protein (CAP), lambda-repressor, an

33 nosine triphosphate (ATP) and its first five catabolites: adenosine diphosphate (ADP), adenosine mono

34 ements.We hypothesized that alpha-tocopherol catabolites alpha-carboxyethyl hydroxychromanol (alpha-C

35 These catabolites also directly stimulate human and mouse Trpa

36 work identifies C99 as the earliest betaAPP catabolite and main contributor to the intracellular bet

37 t niche-dependent manner, is known as carbon catabolite and nitrogen catabolite repression (CCR, NCR)

38 d by LC/MS to determine the alpha-tocopherol catabolites and alpha-carboxyethyl hydroxychromanol (alp

39 Both pigments are known porphyrin catabolites and are found in the eggshells in conjunctio

40 linker has multiple paths to produce active catabolites and that antibody and intracellular targets

41 h the accumulation of significantly more ABA catabolites and the complete restoration of normal wild-

42 Trp catabolites and their derivatives offer a new strategy f

43 ituation, a nomenclature system for phenolic catabolites and their human phase II metabolites is prop

44 e, decreased isovaleryl-carnitine (a leucine catabolite), and decreased tricarboxylic acid (TCA) cycl

45 ing hormone abscisic acid (ABA), three auxin catabolites, and cytokinins (26 isoprenoid and four arom

46 When colon-derived phenolic catabolites are included with flavanone glucuronide and

47 findings demonstrate that kynurenine pathway catabolites are involved in the generation of the more s

48 poorly absorbed in the small intestine, but catabolites are very efficiently absorbed after microbia

49 Gut microbiome catabolites as novel modulators of muscle cell glucose m

50 ure elucidation of tetrapyrrolic chlorophyll catabolites, as well as by complementary biochemical and

51 rabidopsis and specifically demethylates Chl catabolites at the level of FCCs in the cytosol.

52 found that extracellular NAD(+), but not its catabolites, caused cell death (half-maximal effective c

53 for direct drug-to-antibody ratio (DAR) and catabolite characterization of antibody-drug conjugates

54 Two PC catabolites, choline and glycine betaine (GB), were suff

55 ilms, indicating active uptake, and arginine catabolites citrulline, ornithine, and putrescine were d

56 C46A3 silencing led to relative increases in catabolite concentrations in the lysosome.

57 amyloid beta-protein precursor (APP) and its catabolites contribute to the impaired synaptic plastici

58 ies expand our appreciation of CcpA-mediated catabolite control and provide insight into potential in

59 and the contributions of the CcpA and LacD.1 catabolite control pathways to the regulation of this re

60 in Bacillus subtilis for recognition of the catabolite control protein (CcpA) and consequential repr

61 n Streptococcus mutans can be independent of catabolite control protein A (CcpA) and requires specifi

62 a mutation in the transcriptional regulator catabolite control protein A (CcpA) demonstrated signifi

63 spx gene expression, we assessed the role of catabolite control protein A (CcpA) in spx expression co

64 The LacI/GalR family regulator catabolite control protein A (CcpA) is a global regulato

65 Catabolite control protein A (CcpA) is a highly conserve

66 Catabolite control protein A (CcpA) is a master regulato

67 ed us to analyze the role in pathogenesis of catabolite control protein A (CcpA), a GAS ortholog of a

68 Here we report that the catabolite control protein A (CcpA), a highly conserved

69 elements (cre) important for binding by the catabolite control protein A (CcpA), a mediator of CCR i

70 e regulation (CCR) is mediated by the carbon catabolite control protein A (CcpA), a member of the Lac

71 ment (cre) and affected the binding of CcpA (catabolite control protein A), a key regulator of many c

72 ve binding of the maltose repressor MalR and catabolite control protein A.

73 ory networks such as RALPs, Rgg/RopB and the catabolite control protein CcpA.

74 Previously, we described a novel protein, catabolite control protein E (CcpE) that functions as a

75 aureus, we identified a LysR-type regulator, catabolite control protein E (CcpE), with homology to th

76 n A (CcpA) is the master regulator of carbon catabolite control, which ensures optimal energy usage u

77 all, our data indicate that a cellular prion catabolite could interfere with Abeta-associated toxicit

78 roup of distinct dioxobilin-type chlorophyll catabolites (DCCs) as the major breakdown products in wi

79 NCCs are formyloxobilin-type catabolites derived from chlorophyll by oxygenolytic ope

80 nsumption should use in vivo metabolites and catabolites detected in this investigation at physiologi

81 Acetaldehyde, an alcohol catabolite detoxified by ALDH2, precipitates similar eff

82 Dimethylsulfoniopropionate (DMSP) and its catabolite dimethyl sulfide (DMS) are key marine nutrien

83 no changes in epinephrine (Epi) or monoamine catabolites (DOPAC, 5-HIAA) at any ammonia concentration

84 bon scavenging from alpha-hydroxycarboxylate catabolites during the biochemical transition accompanyi

85 Concomitant formation of catabolites (e.g. 4-hydroxyphenylacetic acid) occurred w

86 signaling events propagated by the different catabolites enable the plant to execute a specific respo

87 was to identify and quantify metabolites and catabolites excreted in urine 0-24 h after the acute ing

88 sosomal ion homeostasis, membrane potential, catabolite export, membrane trafficking, and nutrient se

89 e of ABA homeostasis, as it is the major ABA catabolite exported from the cytosol.

90 transported out of the lysosome via specific catabolite exporters or via vesicular membrane trafficki

91 generated from their fluorescent chlorophyll catabolite (FCC) precursors by a nonenzymatic isomerizat

92 nverted to different fluorescent chlorophyll catabolites (FCCs) and nonfluorescent chlorophyll catabo

93 se from intermediary fluorescent chlorophyll catabolites (FCCs) by an acid-catalyzed isomerization in

94 entify bacteria expressing genes relevant to catabolite flow and to locate these genes within their e

95 timal utilization of androgen precursors and catabolites for DHT synthesis.

96 nt molecule while simultaneously identifying catabolites for recombinant fusion proteins.

97 3-acetic acid (oxIAA) is a major primary IAA catabolite formed in Arabidopsis thaliana root tissues.

98 The main focus was on catabolites formed by proteolysis of the fusion protein

99 th tmax of anthocyanins and other polyphenol catabolites from 1.0h to 6.33h in plasma and urine.

100 as a direct transporter of maytansine-based catabolites from the lysosome to the cytoplasm, promptin

101 In addition, these lipid species and their catabolites function as secondary signalling molecules i

102 ion start and is thought to promote the CAP (catabolite gene activation protein)-directed transcripti

103 The binding of cAMP to the Escherichia coli catabolite gene activator protein (CAP) produces a confo

104 yclic AMP receptor protein (CRP, also called catabolite gene activator protein or CAP) plays a key ro

105 tive cell killing with a cysteine-VC(R)-MMAE catabolite generated by lysosomal catabolism.

106 n of soluble heat-labile toxin is subject to catabolite (glucose) activation, and three binding sites

107 sis in an aldol condensation of the unstable catabolites glyceraldehyde 3-phosphate and dihydroxyacet

108 either rHuPH20 nor its directly generated HA catabolites have inflammatory properties in the air pouc

109 mino acid pipecolic acid (Pip), a common Lys catabolite in plants and animals, as a critical regulato

110 Consistent with a role for biotin and its catabolites in modulating these cell signals, greater th

111 lism, mTORC2 responds to declining glutamine catabolites in order to restore metabolic homeostasis.

112 The high levels of ABA and its catabolites in the senescing breeding line under long-te

113 NK cells with indoleamine 2,3-dioxygenase 1 catabolites in vitro ablated IL-17 production in a dose-

114 is supported by biochemical measurements of catabolites in wild-type and mutant animals.

115 ich results in aberrant lysosomal storage of catabolites, including the subunit c of mitochondrial AT

116 yanins were bioavailable, microbial phenolic catabolites increased approximately 10-fold more than an

117 eptidoglycan derivatives and secreted chitin catabolites, induced migration.

118 both intact drug concentration and important catabolite information for this recombinant fusion prote

119 responsible for delivering noncleavable ADC catabolites into the cytoplasm.

120 Addition of the tryptophan catabolite kynurenine to DC cultures in which IDO activi

121 er, in the immune system, tryptophan and its catabolites, kynurenine and 3-hydroxyanthranilic acid (3

122 and a significant rise in the content of ATP-catabolites, malondialdehyde and ADP-ribose.

123 Some of these colon-derived catabolites may have a role in vivo in the potential pro

124 correlates with elevated levels of the toxic catabolite methylglyoxal.

125 immunomodulation by accumulating tryptophan catabolites, most notably kynurenine, appears to play an

126 representative of the energy state (ATP, ATP-catabolites), N-acetylaspartate (NAA), antioxidant defen

127 Nonfluorescent chlorophyll catabolites (NCCs) were described as products of chlorop

128 is broken down to nonfluorescent chlorophyll catabolites (NCCs).

129 olites (FCCs) and nonfluorescent chlorophyll catabolites (NCCs).

130 droxyvitamin D3 ((24R),25(OH)2D3) is a major catabolite of 25-hydroxyvitamin D metabolism and is an i

131 ncentrations of homovanillic acid (the major catabolite of dopamine) and the purine compound xanthine

132 ycolylglucosamine (GlcNGc) was shown to be a catabolite of NeuNGc.

133 Here, we show that a catabolite of the plant hormone abscisic acid (ABA), nam

134 sed production of kynurenine (Kyn) as a main catabolite of tryptophan (Trp) degradation is involved i

135 However, urinary excretion of 3 colonic catabolites of bacterial origin, most notably, 3-(3'-hyd

136 Branched-chain alpha-keto acids (BCKAs) are catabolites of branched-chain amino acids (BCAAs).

137 involved in the formation of dioxobilin-type catabolites of chlorophyll in Arabidopsis.

138 ne containing insulin and another containing catabolites of insulin.

139 Catabolites of Trp suppressed proliferation of myelin-sp

140 affect clearance dynamics and that access to catabolites only matters at low H2O2 concentrations.

141 hyrin-related molecules like red chlorophyll catabolite or exogenous protoporphyrin IX.

142 gen species over-accumulation and phytotoxic catabolite over-buildup in the chlorophyll degradation p

143 The CouR dimer bound two molecules of the catabolite p-coumaroyl-CoA (Kd = 11 +/- 1 muM).

144 The catabolite PA was positively associated with neopterin a

145 dent on sufficient amounts of glutaminolysis catabolites particularly alpha-ketoglutarate, which are

146 pheophorbide a to a primary fluorescent chl catabolite (pFCC) and it is catalyzed by two enzymes: ph

147 ane diterpenoid dehydroabietinal, the lysine catabolite pipecolic acid, a glycerol-3-phosphate-depend

148 A number of the catabolites produced along this pathway show neurotoxic

149 inary flavanone metabolites and ring fission catabolites produced by the action of the colonic microb

150 s to falling levels of glucose and glutamine catabolites, promoting glutaminolysis and preserving the

151 acids (BCAAs) (Leu, Ile, and Val) and their catabolites, propionylcarnitine and butyrylcarnitine, we

152 In Gram-positive bacteria, carbon catabolite protein A (CcpA) is the master regulator of c

153 ect regulator of phoPR transcription, carbon catabolite protein A, CcpA.

154 transcription by glucose required the carbon catabolite protein CcpA via an indirect mechanism.

155 Two of the catabolites, raphanusamic acid and 3-butenoic acid, diff

156 : pheophorbide a oxygenase (PaO) and red chl catabolite reductase (RCCR).

157 In Gram-positive bacteria, carbon catabolite regulation (CCR) is mediated by the carbon ca

158 As a key component of carbon catabolite regulation, CcpA has been previously reported

159 apping pathways for global control of carbon catabolite regulation.

160 under negative control of the global carbon catabolite regulator CcpA.

161 n of respiration genes, and CcpA, the carbon catabolite regulator protein.

162 vation of Gln3 and transcription of nitrogen catabolite-repressed (NCR) genes whose products function

163 These enzymes are universally catabolite-repressed and are further regulated by a rich

164 In addition, we observe that nitrogen catabolite-repressed genes are upregulated by Nab3 deple

165 lation was noted between the dependencies of catabolite-repressible gene expression on CcpA and HprK,

166 Carbon catabolite repression (CCR) allows bacteria to alter met

167 s mannose and glucose, is involved in carbon catabolite repression (CCR) and regulates the expression

168 olite control protein (CcpA) mediates carbon catabolite repression (CCR) by controlling expression of

169 riptional regulator that accounts for carbon catabolite repression (CCR) control of the anaerobic cat

170 CcpA is the global mediator of carbon catabolite repression (CCR) in gram-positive bacteria, a

171 , two regulatory genes that carry out carbon catabolite repression (CCR) in staphylococci and other G

172 Carbohydrate catabolite repression (CCR) in Streptococcus mutans can

173 Carbon catabolite repression (CCR) is a regulatory phenomenon i

174 Carbon catabolite repression (CCR) is a regulatory phenomenon o

175 ilm formation were under some form of carbon catabolite repression (CCR), a regulatory network in whi

176 so includes inhibited glycolysis, and carbon catabolite repression (CCR)-mediated carbohydrate-depend

177 ated that CcpA plays a direct role in carbon catabolite repression (CCR).

178 ntegral to pneumococcus's strategy of carbon catabolite repression (CCR).

179 , is known as carbon catabolite and nitrogen catabolite repression (CCR, NCR), and has been shown to

180 tor (arcA and etrA [fnr homolog]) and carbon catabolite repression (crp and cya) proteins affect arse

181 circuit responsible for regulating nitrogen catabolite repression (NCR) in yeast.

182 e factors regulate transcription of nitrogen catabolite repression (NCR) sensitive genes when preferr

183 en use is mediated in large part by nitrogen catabolite repression (NCR), which results in the repres

184 of Gln3 and Gat1, the activators of nitrogen catabolite repression (NCR)-sensitive genes whose produc

185 ities is transcriptional control of nitrogen catabolite repression (NCR)-sensitive genes.

186 Gln3 intracellular localization and nitrogen catabolite repression (NCR)-sensitive transcription in S

187 Nitrogen catabolite repression (NCR)-sensitive transcription is a

188 Gln3, the major activator of nitrogen catabolite repression (NCR)-sensitive transcription, is

189 ogical response has been designated nitrogen catabolite repression (NCR).

190 s leads to an increase in succinate-mediated catabolite repression (SMCR).

191 carbon sources is termed succinate-mediated catabolite repression (SMCR).

192 implications for mechanisms of CRP-dependent catabolite repression acting in conjunction with a membe

193 ritical role for the PTS in CcpA-independent catabolite repression and induction of cel gene expressi

194 ssion of fruA is under the control of carbon catabolite repression and is induced by growth in fructa

195 itoring and use that information to regulate catabolite repression and related responses.

196 in stable environments, with more stringent catabolite repression and slower transcriptional reprogr

197 metabolic phenotype depends on the level of catabolite repression and the metabolic state-dependent

198 ) operon of Escherichia coli is regulated by catabolite repression and tryptophan-induced transcripti

199 cpA), a highly conserved regulator of carbon catabolite repression and virulence in a number of gram-

200 In addition to catabolite repression by glucose, l-leucine acts by inhi

201 suggests that Pyk may participate in glucose catabolite repression by serving among all of the factor

202 Deletion of hprK in S. meliloti enhanced catabolite repression caused by succinate, as did an S53

203 3 in-frame deletion mutants show a relief of catabolite repression compared to the wild type.

204 is was apparently not due to a defect in the catabolite repression control (Crc) protein.

205 slocator (ArsB), superoxide dismutase (SOD), catabolite repression control protein (Crc), or glutathi

206 a xylose catabolic activation independent of catabolite repression control.

207 The cyclic AMP (cAMP)-dependent catabolite repression effect in Escherichia coli is amon

208 tein IIA(Glc) plays a key regulatory role in catabolite repression in addition to its role in the vec

209 ant physiological roles, ranging from carbon catabolite repression in bacteria to mediating the actio

210 se-pairing RNA Spot 42 plays a broad role in catabolite repression in Escherichia coli by directly re

211 ENR to the regulatory network behind carbon catabolite repression in Escherichia coli is presented.

212 gether with the Hfq protein, participates in catabolite repression in pseudomonads, helping to coordi

213 ne-22 residue, and that HPr-His22-P enhances catabolite repression in the presence of succinate.

214 n enteric bacteria, the key player of carbon catabolite repression is a component of the glucose-spec

215 In addition, the carbon catabolite repression is alleviated by protease-based in

216 In Sinorhizobium meliloti, catabolite repression is influenced by a noncanonical ni

217 Here, we show that this catabolite repression is relieved by mutations that weak

218 the parent H26 and glpK mutant strains, with catabolite repression more pronounced in the glycerol ki

219 Catabolite repression of galactose by glucose is one of

220 d growth phenotype was reflected in a strong catabolite repression of pauA promoter activation by CAD

221 and that this mechanism in part accounts for catabolite repression of sigma(L)-directed levD operon e

222 enes; (iii) CcpA plays little direct role in catabolite repression of the cel regulon, but loss of sp

223 on this enzymatic activity or the canonical catabolite repression pathway, but likely does require s

224 substitution into HPr alleviated the strong catabolite repression phenotypes of strains carrying Del

225 se or casamino acids, suggesting that carbon catabolite repression plays a role in regulating xynA.

226 al abscess formation, indicating that carbon catabolite repression presents an important pathogenesis

227 We linked this to indirect regulation of the catabolite repression protein Crc via the non-coding RNA

228 pts by forming a regulatory complex with the catabolite repression protein Crc.

229 in carbon source can favor different carbon catabolite repression strategies.

230 concluded that the components of the carbon catabolite repression system are essential to regulating

231 utations also increase glycerol-induced auto-catabolite repression that reduces glpK transcription in

232 PTS) of gram-positive bacteria and regulates catabolite repression through phosphorylation/dephosphor

233 nd uptake in Escherichia coli are subject to catabolite repression through the cyclic AMP (cAMP)-CRP

234 Sucrose-dependent catabolite repression was also evident in strains contai

235 oreover, consistent with a classical role in catabolite repression, a cAMP-CRP-dependent reporter sho

236 atabolism, chemotaxis, glycogen utilization, catabolite repression, and inducer exclusion.

237 ulating central carbon metabolism and carbon catabolite repression, and is a frequent target of metab

238 ng mechanisms of signal transduction, carbon catabolite repression, and quorum-sensing.

239 lted in partial relief of succinate-mediated catabolite repression, extreme sensitivity to cobalt lim

240 nes are substrate inducible and sensitive to catabolite repression, mediated through ArcR and CcpA, r

241 by negative feedback on glpK expression via catabolite repression, possibly to prevent methylglyoxal

242 as luxS and ompX and provide a link between catabolite repression, quorum sensing, and nitrogen assi

243 The other mutants display less stringent catabolite repression, resulting in leaky expression of

244 The nanATEK operon is controlled by catabolite repression, suggesting that diminished expres

245 t cyclic-di-GMP may play a role in mediating catabolite repression, thereby facilitating the expressi

246 -responsive) and TorC1-independent (nitrogen catabolite repression-sensitive and methionine sulfoximi

247 Gln3-Myc13 nuclear accumulation and nitrogen catabolite repression-sensitive transcription, generate

248 ernative carbon sources is shut down, due to catabolite repression.

249 s regulatory phenomenon is defined as carbon catabolite repression.

250 source, glucose, in a process called carbon catabolite repression.

251 g very weakly transcribed genes under strong catabolite repression.

252 others through a regulatory mechanism termed catabolite repression.

253 itrate fermentation was under the control of catabolite repression.

254 h gene, the DMML encoding gene is subject to catabolite repression.

255 ns with a hpr(H22A) allele exhibited relaxed catabolite repression.

256 rating that HPr-His22-P is needed for strong catabolite repression.

257 trol region cloned in E. coli was subject to catabolite repression.

258 scriptome appeared to be regulated by carbon catabolite repression.

259 r region showed that both CREs contribute to catabolite repression.

260 activity were shown to be refractory to such catabolite repression.

261 efficient microbial conversion due to carbon catabolite repression.

262 l a key physiological role of cAMP-dependent catabolite repression: to ensure that proteomic resource

263 wth on non-glucose substrates as part of the catabolite-repression response.

264 Carbon catabolite repressor (CCR)4 and CCR4-associated factor (

265 ex, containing the two key components carbon catabolite repressor 4 (CCR4) and CCR4-associated factor

266 The carbon catabolite repressor 4-CCR4 associated factor1 (CCR4-CAF

267 ging and revealed a major role of the carbon catabolite repressor CRE-1 in regulating the expression

268 thermore, the inclusion of a deletion of the catabolite repressor gene, cre-1, in the triple beta-glu

269 he eight-subunit deadenylase complex "carbon catabolite repressor protein 4 (CCR4)-negative on TATA-l

270 r that is homologous to the Escherichia coli catabolite repressor protein, is thought to be the major

271 The role of the carbon catabolite repressor transcription factor homologue Bbcr

272 Mutation of two putative catabolite response elements (CREs) within the arc promo

273 r and binds to more than 50 operators called catabolite response elements (cres).

274 epression (CCR) by controlling expression of catabolite responsive (CR) genes or operons through inte

275 al xynA1 gene show significant similarity to catabolite responsive element (cre) defined in Bacillus

276 R) genes or operons through interaction with catabolite responsive elements (cres) located within or

277 The mutation was in a catabolite-responsive element (cre) and affected the bin

278 A catabolite-responsive element (CRE), a binding site for

279 s consensus identified a number of potential catabolite-responsive elements (cre) important for bindi

280 a bioinformatic search for additional carbon catabolite-responsive regulators in S. aureus, we identi

281 nd reveal critical roles for amino acid- and catabolite-sensing pathways in controlling gene expressi

282 The catabolites showed lowered antioxidant activity and cell

283 This result could be obtained without catabolite specific ELISAs or quantitative LC-MS assays.

284 Hydrogen sulfide is an essential catabolite that intervenes in the pathophysiology of sev

285 sts unwanted cellular components to generate catabolites that are required for housekeeping biosynthe

286 idation, or oxidation) in vivo, resulting in catabolites that can have a direct impact on drug safety

287 s indicate that syn-DHCA and syn-DHFA, colon catabolites that could be present in systemic circulatio

288 ryptophan to kynurenine and other downstream catabolites that inhibit T-cell proliferation and interl

289 ric acid (GABA), in the GABA shunt generates catabolites that may enter the tricarboxylic acid cycle,

290 the sum of TN-ApoA1, along with its two main catabolites, the individual PK profiles of all three com

291 These catabolites were 3-(3'-methoxy-4'-hydroxyphenyl)propioni

292 The GC-MS analysis revealed that 8 urinary catabolites were also excreted in significantly higher q

293 Two pharmacologically active catabolites were identified with conserved fusion region

294 The drug and its catabolites were isolated from rabbit plasma by immunoca

295 The captured drug and catabolites were released from the streptavidin-coated m

296 gradation products, are abundant chlorophyll catabolites, which occur in fall leaves and in ripe frui

297 t, was also observed with OJ-derived colonic catabolites, which, after supplementation in the trained

298 Corresponding alterations in tryptophan catabolites with immunomodulatory properties in serum of

299 nurenine and quinolinic acid, two tryptophan catabolites with potent immunosuppressive and neurotoxic

300 inary flavanone metabolites and (poly)phenol catabolites with the use of high-pressure liquid chromat