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1 and many of these presympathetic neurons are catecholaminergic.
2 k tyrosine hydroxylase (TH) and thus are not catecholaminergic.
3  beta-hydroxylase, indicating that they were catecholaminergic.
4 abeled neurons were either serotoninergic or catecholaminergic.
5 GABAergic, non-glycinergic and predominantly catecholaminergic (54%).
6 b) the dynamic properties of growth cones in catecholaminergic a-differentiated neuroblastoma cells.
7                Results indicate that AMPK in catecholaminergic A1-C1m neurons is activated by glucopr
8 r primary focus has been to interfering with catecholaminergic activity after fear acquisition or exp
9 k, is not always effective and has important catecholaminergic adverse effects.
10 ls in transgenic mice in which both types of catecholaminergic amacrine (CA) cells were labeled with
11  by reserpine indicate that these nerves are catecholaminergic and are required for functional inhibi
12                      Among these are several catecholaminergic and cholinergic cell groups, the peria
13                            The activation of catecholaminergic and hypothalamic-pituitary-adrenal axi
14                                         Both catecholaminergic and noncatecholaminergic neurons conta
15 rochemistry (using in situ hybridization) of catecholaminergic and noncatecholaminergic neurons which
16 e properties of unmodified human MSCs on rat catecholaminergic and serotonergic cell cultures exposed
17 TH(-) neurons but spared facial motoneurons, catecholaminergic and serotonergic neurons and the ventr
18                         BS-NSC graft-derived catecholaminergic and serotonergic neurons showed remark
19 and sparse projections were observed in many catecholaminergic and serotonergic nuclei, as well as th
20  may be expressed in a few subpopulations of catecholaminergic and serotoninergic neurons in the carp
21                      Brainstem serotonergic, catecholaminergic, and inflammatory adaptations during c
22 mportantly, analysis of GFP in the zebrafish catecholaminergic areas revealed the same expression pat
23 tilization, GFP began expressing in distinct catecholaminergic areas.
24           In the absence of aFGF+ChABC fewer catecholaminergic axons entered the graft, no axons exit
25 ns were counterstained with Cuprolinic Blue; catecholaminergic axons were stained immunohistochemical
26 s, allowing for robust growth, especially of catecholaminergic axons, through the graft and back into
27 Hydroxylase (Dbh), a gene encoding a crucial catecholaminergic biosynthetic enzyme.
28 m BAT combined with immunohistochemistry for catecholaminergic biosynthetic enzymes revealed the vent
29 ent of ventral medullary sympatho-excitatory catecholaminergic C1 neurons, using inhibitory Drosophil
30 ine hydroxylase (TH) to identify bulbospinal catecholaminergic (C1) neurons in sedentary and active r
31 Although the neuroanatomical distribution of catecholaminergic (CA) neurons has been well documented
32                                              Catecholaminergic (CA) neurons innervate sensory areas a
33         Developmental studies of the central catecholaminergic (CA) system are essential for understa
34                     We have investigated the catecholaminergic (CA) systems in the brain of two repre
35 Kalpha) in hindbrain micropunches containing catecholaminergic cell groups A1 through the middle regi
36                                              Catecholaminergic cell groups in the pons (LC) and medul
37        Additional assays were performed on a catecholaminergic cell line and animal models of iron de
38                           Because the murine catecholaminergic cell line CAD5 is susceptible to a div
39  novel plasminogen receptor, and its role in catecholaminergic cell plasminogen activation and regula
40 mary, Plg-R(KT) is present on the surface of catecholaminergic cells and functions to stimulate plasm
41             We discover neural crest-derived catecholaminergic cells associated with zebrafish pharyn
42                  Neurotransmitter release by catecholaminergic cells is negatively regulated by proho
43 r connectivity and alternating activity, the catecholaminergic cells may generate the rhythm.
44                           Distinct groups of catecholaminergic cells were observed in the olfactory b
45  the AMPK-alpha1 and/or AMPK-alpha2 genes in catecholaminergic cells, which compose the hypoxia-respo
46 al plasma membrane protein on the surface of catecholaminergic cells.
47 her with epinephrine and norepinephrine from catecholaminergic cells.
48                      Activation of medullary catecholaminergic centers might reflect the afferent and
49            Ventricular arrhythmias evoked by catecholaminergic challenge (caffeine/adrenaline) in S28
50  used for simultaneous detection of NOS with catecholaminergic, cholinergic, and serotonergic structu
51  via the aggregation of neural crest-derived catecholaminergic (chromaffin) cells already associated
52 We identified cholinergic, serotonergic, and catecholaminergic ciliomotor neurons.
53 al adaptations may be mediated by changes in catecholaminergic control systems in the brain.
54 ng neural pathways including both a distinct catecholaminergic "danger" pathway, and a possibly multi
55 mRNA and protein levels, and thereby central catecholaminergic-dependent behavioral pathways.
56 parse, and no reports have yet characterized catecholaminergic (dopamine [DA]- or norepinephrine-synt
57 nd noradrenaline) and review the efficacy of catecholaminergic drugs in treating post-traumatic brain
58           The findings support the notion of catecholaminergic dysfunction as a possible trait abnorm
59 pose of this study was to examine a possible catecholaminergic dysfunction in the pathogenesis of bul
60        Cortical TH cells lack all additional catecholaminergic enzymes, and many coexpress GABA and c
61 nsferase (Pnmt), which also encode essential catecholaminergic enzymes, were severely reduced in post
62 ACCs, we show here that less than 10% of all catecholaminergic exocytosis, measured by carbon fibre a
63 ytosis as they do in the case of spontaneous catecholaminergic exocytosis.
64 e of NPY coexpression within interneurons or catecholaminergic fibers with somatostatin and tyrosine
65 tors included calcium , drugs modulating the catecholaminergic fight-or-flight stress response such a
66  way to investigate the relationship between catecholaminergic function and psychiatric disorder has
67 model simulations indicating that increasing catecholaminergic gain should reduce the angular dispers
68   More striking, we find that nearly all non-catecholaminergic glutamatergic neurons of the ventrolat
69 e suprachiasmatic area was identified by its catecholaminergic groups and the lack of Otp, and could
70 l together, our results demonstrate that all catecholaminergic groups in zebrafish are either GABAerg
71 in line with a model in which stress-induced catecholaminergic hyperactivation alters phasic neuromod
72 eurons originates predominantly from PNMT-ir catecholaminergic (i.e., C1 cells).
73 2808A+/+ mice were protected against chronic catecholaminergic-induced cardiac dysfunction.
74                                              Catecholaminergic inotropes have a place in the manageme
75 tter neurons are part of a larger network of catecholaminergic input to neuroendocrine neurons in the
76 d the ventrolateral medulla as the source of catecholaminergic input to the rRPa and demonstrated tha
77 ritical and previously unidentified group of catecholaminergic interneurons that are apt to sense cha
78 own anole brain and demonstrate evidence for catecholaminergic involvement in appetitive and consumma
79 tic submandibular ganglion (PSG) express the catecholaminergic marker tyrosine hydroxylase (TH) in de
80 These data demonstrate a causal link between catecholaminergic modulation and the adjustment of the m
81 ehaviour, and helps refine current models of catecholaminergic modulation of motivated action.
82 stem components; it is therefore likely that catecholaminergic molecular components influence the eff
83 face density and decreased cell viability in catecholaminergic N2a cells.
84                   In NGF-differentiated PC12 catecholaminergic nerve cells, we show that de novo expr
85 t the hypothesis that they are shaped by the catecholaminergic neuromodulators norepinephrine and dop
86  Potential candidate factors are subcortical catecholaminergic neuromodulatory systems, such as the l
87 cholaminergic neurons and a few PPE positive catecholaminergic neuron but neurochemical codes were la
88 lrhodopsin-2 (ChR2) under the control of the catecholaminergic neuron-preferring promoter PRSx8 was i
89 rotein ChR2-mCherry under the control of the catecholaminergic neuron-selective promoter PRSx8 and ob
90                Rostral ventrolateral medulla catecholaminergic neurones (RVLM-C1) modulate sympatheti
91 echolaminergic rostral ventrolateral medulla catecholaminergic neurones (RVLM-C1) to both haemodynami
92 e hydroxylase, revealing the distribution of catecholaminergic neurons (dopaminergic, noradrenergic,
93 genetics in tissue slices, we show that RVLM catecholaminergic neurons activate the locus coeruleus a
94 se parkin, resulting in neurodegeneration of catecholaminergic neurons and a familial form of Parkins
95 on and migration of neuronal progenitors for catecholaminergic neurons and interneurons.
96                                      Not all catecholaminergic neurons are activated and other neuroc
97                        Thus, the majority of catecholaminergic neurons are gad1b/2-positive and coexp
98 ure rats revealed that microglia surrounding catecholaminergic neurons are in a "surveillance" state
99 nput to the rRPa and demonstrated that these catecholaminergic neurons are synaptically connected to
100            We tested the hypothesis that NTS catecholaminergic neurons attenuate psychological stress
101 ctively expressed ChR2(H134R) in rostral VLM catecholaminergic neurons by injecting Cre-dependent ade
102 vidence that the axons of rostral VLM (RVLM) catecholaminergic neurons contact locus coeruleus, A1, a
103 s of glutamate, PACAP, and microglia on RVLM catecholaminergic neurons during the cardiovascular resp
104                It may be possible to protect catecholaminergic neurons from reactive oxygen species-i
105 ated sensory information activates hindbrain catecholaminergic neurons in a rate-dependent manner.
106 cle was microinjected into the NTS to lesion catecholaminergic neurons in male Sprague-Dawley rats, a
107  a useful strategy to reduce degeneration of catecholaminergic neurons in Parkinson's disease.
108 wed us to ascribe a role for ghrelin-engaged catecholaminergic neurons in stress-induced eating.
109 mal taxa, we embarked on a study to identify catecholaminergic neurons in the CNS of the wolf spider
110 ir in common somatodendritic compartments of catecholaminergic neurons in the LC, and also revealed C
111 to be propagated via specific populations of catecholaminergic neurons in the NTS and VLM, and likely
112                    Surprisingly, we found no catecholaminergic neurons in the NTS, A5 or Locus Coerul
113                                          The catecholaminergic neurons in the rostral ventrolateral m
114 ENT C1 neurons are glutamatergic/peptidergic/catecholaminergic neurons located in the medulla oblonga
115                    The hypothalamus contains catecholaminergic neurons marked by the expression of ty
116              Also, signaling specifically in catecholaminergic neurons mediated not only ghrelin's or
117                    The data suggest that NTS catecholaminergic neurons normally inhibit the arterial
118 medium spiny neurons of the striatum and the catecholaminergic neurons of the substantia nigra and lo
119 iciency leads to the premature demise of the catecholaminergic neurons of the ventral midbrain in fam
120  dorsal motor nucleus of the vagus [DMN] and catecholaminergic neurons of the ventrolateral medulla [
121                                    Brainstem catecholaminergic neurons play key roles in the autonomi
122        These likely include PPNPY expressing catecholaminergic neurons projecting to vasopressinergic
123                              Degeneration of catecholaminergic neurons remains unchanged by PrP(C) re
124               We tested in male rats whether catecholaminergic neurons that project to the medial and
125            Both transcripts were detected in catecholaminergic neurons throughout the CNS.
126 , we confirmed that the projection from RVLM catecholaminergic neurons to the orexinergic neurons ori
127                                      RTN and catecholaminergic neurons were transduced.
128                                              Catecholaminergic neurons within the central nervous sys
129 ual immunostaining for tyrosine hydroxylase (catecholaminergic neurons) and c-Fos (marker of neuronal
130  of the VLM, including a large proportion of catecholaminergic neurons, but no colocalization of sst2
131 range of stimuli activate neurons, including catecholaminergic neurons, in the ventrolateral medulla.
132 ral wake-promoting systems (serotonergic and catecholaminergic neurons, orexinergic neurons) are also
133            In conclusion, activation of RVLM catecholaminergic neurons, predominantly C1 cells, by so
134  membrane and mitochondria of astrocytes and catecholaminergic neurons, suggesting that it plays a ro
135 hypoglycemia require hypothalamus-projecting catecholaminergic neurons, the majority of which origina
136 INSPAReDT ablates peripheral but not central catecholaminergic neurons, thus avoiding the Parkinson-l
137 where VMH efferents make close contacts with catecholaminergic neurons.
138 eversed by selective reactivation of GHSR in catecholaminergic neurons.
139 nt and survival of enteric, sympathetic, and catecholaminergic neurons.
140 s than 2-deoxyglucose but similar numbers of catecholaminergic neurons.
141 butyric acid (GABA)ergic, glutamatergic, and catecholaminergic neurons.
142                            Here, we examined catecholaminergic neurosecretory cells for expression, t
143 ess reward-effort integration in relation to catecholaminergic neurotransmission at the behavioral an
144                    Topographically organized catecholaminergic "nigrostriatal," "mesolimbic," "mesoco
145 on, of Phox2b-expressing, glutamatergic, non-catecholaminergic, noncholinergic neurons located in the
146 ignals from the NTS are communicated by both catecholaminergic [norepinephrine (NE), epinephrine (E)]
147 ulation of five heritable transcripts in the catecholaminergic pathway in young (6 weeks) SHRs.
148 to the fourth ventricle does not act through catecholaminergic pathways.
149 retrotrapezoid nucleus (RTN), a group of non-catecholaminergic Phox2b-expressing central respiratory
150 s the transcription factor Phox2b and is non-catecholaminergic (Phox2b(+)TH(-)).
151 sis (17%), including Long-QT syndrome (13%), catecholaminergic polymorphic ventricular tachycardia (4
152                                              Catecholaminergic polymorphic ventricular tachycardia (C
153                                              Catecholaminergic polymorphic ventricular tachycardia (C
154                                           In catecholaminergic polymorphic ventricular tachycardia (C
155 associated with atrial fibrillation (AF) and catecholaminergic polymorphic ventricular tachycardia (C
156                                              Catecholaminergic polymorphic ventricular tachycardia (C
157 d mutations in CaM cause arrhythmias such as catecholaminergic polymorphic ventricular tachycardia (C
158    Current mechanisms of arrhythmogenesis in catecholaminergic polymorphic ventricular tachycardia (C
159 ch as congenital long-QT syndrome (LQTS) and catecholaminergic polymorphic ventricular tachycardia (C
160                                              Catecholaminergic polymorphic ventricular tachycardia (C
161  present with divergent clinical features of catecholaminergic polymorphic ventricular tachycardia (C
162                  Long QT syndrome (LQTS) and catecholaminergic polymorphic ventricular tachycardia (C
163 (CASQ2) cause an autosomal recessive form of catecholaminergic polymorphic ventricular tachycardia (C
164                                              Catecholaminergic polymorphic ventricular tachycardia (C
165 ne receptor (RyR2) mutations associated with catecholaminergic polymorphic ventricular tachycardia (C
166                                              Catecholaminergic polymorphic ventricular tachycardia (C
167 4%]), Brugada syndrome (BrS) (n = 16 [14%]), catecholaminergic polymorphic ventricular tachycardia (C
168                                              Catecholaminergic polymorphic ventricular tachycardia (C
169 ced pluripotent stem cells (hiPSCs) model of catecholaminergic polymorphic ventricular tachycardia (C
170 lymorphic ventricular tachycardia in humans [catecholaminergic polymorphic ventricular tachycardia (C
171 es, we propose a molecular mechanism for the catecholaminergic polymorphic ventricular tachycardia (C
172 er biomarkers were assessed in patients with catecholaminergic polymorphic ventricular tachycardia (C
173 ing that point mutation R33Q leads to lethal catecholaminergic polymorphic ventricular tachycardia (C
174 e of arrhythmogenic Ca(2+) release events in catecholaminergic polymorphic ventricular tachycardia (C
175                                              Catecholaminergic polymorphic ventricular tachycardia (C
176               Mutations in RYR2 cause type 1 catecholaminergic polymorphic ventricular tachycardia (C
177                                              Catecholaminergic polymorphic ventricular tachycardia (C
178                                              Catecholaminergic polymorphic ventricular tachycardia (C
179 es associated with sudden death-predisposing catecholaminergic polymorphic ventricular tachycardia (C
180  cause the highly lethal familial arrhythmia catecholaminergic polymorphic ventricular tachycardia (C
181                                              Catecholaminergic polymorphic ventricular tachycardia (C
182 yanodine Receptor gene (RYR2) cause dominant catecholaminergic polymorphic ventricular tachycardia (C
183 ed with severe forms of long QT syndrome and catecholaminergic polymorphic ventricular tachycardia (C
184 sm of therapeutic efficacy of flecainide for catecholaminergic polymorphic ventricular tachycardia (C
185 cardiac RyR (RyR2) mutations associated with catecholaminergic polymorphic ventricular tachycardia (C
186                Patients with LQTS (N=40) and catecholaminergic polymorphic ventricular tachycardia (N
187  spectrum of arrhythmogenic disease included catecholaminergic polymorphic ventricular tachycardia (n
188 rillator (ICD) therapy for the management of catecholaminergic polymorphic ventricular tachycardia (V
189 rdiac arrest, including long-QT syndrome and catecholaminergic polymorphic ventricular tachycardia (V
190 ence (10; 21%) and disease factors (18; 38%; catecholaminergic polymorphic ventricular tachycardia [6
191 netic form of exercise-induced sudden death (catecholaminergic polymorphic ventricular tachycardia [C
192 ed to exercise-induced sudden cardiac death (catecholaminergic polymorphic ventricular tachycardia [C
193 ias are long QT syndrome, short QT syndrome, catecholaminergic polymorphic ventricular tachycardia an
194 rited and acquired cardiac diseases, such as catecholaminergic polymorphic ventricular tachycardia an
195 Our data highlighted the predominant role of catecholaminergic polymorphic ventricular tachycardia an
196         The main etiologies established were catecholaminergic polymorphic ventricular tachycardia an
197 entified in the third proband diagnosed with catecholaminergic polymorphic ventricular tachycardia an
198 fied in individuals with clinically definite catecholaminergic polymorphic ventricular tachycardia ar
199 (2/10) and 50% of long QT syndrome (1/2) and catecholaminergic polymorphic ventricular tachycardia fa
200 ism of Ca(2+) release dysfunction underlying catecholaminergic polymorphic ventricular tachycardia ha
201                          Flecainide prevents catecholaminergic polymorphic ventricular tachycardia in
202                                              Catecholaminergic polymorphic ventricular tachycardia is
203                        The recessive form of catecholaminergic polymorphic ventricular tachycardia is
204 ardiac calsequestrin-2 gene; this variant of catecholaminergic polymorphic ventricular tachycardia is
205                                              Catecholaminergic polymorphic ventricular tachycardia is
206                                              Catecholaminergic polymorphic ventricular tachycardia is
207                                              Catecholaminergic polymorphic ventricular tachycardia is
208                                Patients with catecholaminergic polymorphic ventricular tachycardia ma
209  specific arrhythmogenic mutation, as in the catecholaminergic polymorphic ventricular tachycardia mi
210 e-threatening arrhythmias in CASQ2-defective catecholaminergic polymorphic ventricular tachycardia mi
211 ed properties not previously identified in a catecholaminergic polymorphic ventricular tachycardia mo
212 ntials and diastolic contractions (DCs) in a catecholaminergic polymorphic ventricular tachycardia mo
213  cause severe cardiac arrhythmias, including catecholaminergic polymorphic ventricular tachycardia or
214 y multicenter, retrospective cohort study of catecholaminergic polymorphic ventricular tachycardia pa
215 injection, analogous to what was observed in catecholaminergic polymorphic ventricular tachycardia pa
216 tions contributing to the SAN dysfunction in catecholaminergic polymorphic ventricular tachycardia pa
217 patients diagnosed with long QT syndrome and catecholaminergic polymorphic ventricular tachycardia re
218 nelopathies (short and long QT, Brugada, and catecholaminergic polymorphic ventricular tachycardia sy
219 DEP and sudden cardiac death cases linked to catecholaminergic polymorphic ventricular tachycardia th
220                                              Catecholaminergic polymorphic ventricular tachycardia ty
221                  A specific genetic test for catecholaminergic polymorphic ventricular tachycardia wa
222 orecard, the pretest clinical probability of catecholaminergic polymorphic ventricular tachycardia wa
223 ogenic right ventricular cardiomyopathy, and catecholaminergic polymorphic ventricular tachycardia we
224 ic arrhythmia syndromes (e.g., flecainide in catecholaminergic polymorphic ventricular tachycardia).
225 /18 (72%); long QT syndrome, 3/18 (17%); and catecholaminergic polymorphic ventricular tachycardia, 2
226 s (long QT syndrome, 9; Brugada syndrome, 8; catecholaminergic polymorphic ventricular tachycardia, 3
227  practicing cardiologists: long QT syndrome, catecholaminergic polymorphic ventricular tachycardia, a
228 ac calsequestrin (CASQ2) genes are linked to catecholaminergic polymorphic ventricular tachycardia, a
229 thal cardiac arrhythmia syndromes, including catecholaminergic polymorphic ventricular tachycardia, c
230                                           In catecholaminergic polymorphic ventricular tachycardia, e
231 es have been used to study long QT syndrome, catecholaminergic polymorphic ventricular tachycardia, h
232 l and structural properties of wild-type and catecholaminergic polymorphic ventricular tachycardia-as
233                         One exception is the catecholaminergic polymorphic ventricular tachycardia-ca
234 AN [Ca(2+)](i) handling in mice carrying the catecholaminergic polymorphic ventricular tachycardia-li
235 iac myocytes of heterozygous mice carrying a catecholaminergic polymorphic ventricular tachycardia-li
236 tibility genes (KCNQ1, KCNH2, and SCN5A) and catecholaminergic polymorphic ventricular tachycardia-su
237  phenotype and lengthy delay to diagnosis in catecholaminergic polymorphic ventricular tachycardia.
238 o define treatment outcomes in children with catecholaminergic polymorphic ventricular tachycardia.
239  reduces risk in long-QT syndrome (LQTS) and catecholaminergic polymorphic ventricular tachycardia.
240 t cardiac sympathetic denervation in LQTS or catecholaminergic polymorphic ventricular tachycardia.
241 rrhythmias in genetically modified mice with catecholaminergic polymorphic ventricular tachycardia.
242 r in heart failure, cardiac hypertrophy, and catecholaminergic polymorphic ventricular tachycardia.
243 rt diseases, such as the long-QT syndrome or catecholaminergic polymorphic ventricular tachycardia.
244  a variety of cardiac arrhythmias, including catecholaminergic polymorphic ventricular tachycardia.
245 ch as long QT syndrome, Brugada Syndrome, or Catecholaminergic Polymorphic Ventricular Tachycardia.
246 o provide a new arrhythmogenic mechanism for catecholaminergic polymorphic ventricular tachycardia.
247  syncope in his brother raised suspicion for catecholaminergic polymorphic ventricular tachycardia.
248 receptor-2 mutation that was consistent with catecholaminergic polymorphic ventricular tachycardia.
249 ng neighboring myocytes, and correlated with catecholaminergic polymorphic ventricular tachycardia.
250 ethylenedioxymethamphetamine, marijuana, and catecholaminergic polymorphic ventricular tachycardia.
251 ctric dysfunction in an established model of catecholaminergic polymorphic ventricular tachycardia.
252 me, short QT syndrome, Brugada syndrome, and catecholaminergic polymorphic ventricular tachycardia.
253  at position 307 in CASQ2 has been linked to catecholaminergic polymorphic ventricular tachycardia.
254 phenotype-enhanced variant classification in catecholaminergic polymorphic ventricular tachycardia.
255 ther with left ventricular noncompaction and catecholaminergic polymorphic ventricular tachycardia.
256 : 14 Brugada syndrome; 4 long-QT syndrome; 1 catecholaminergic polymorphic ventricular tachycardia; a
257  retrospective review of young patients with catecholaminergic polymorphic VT and ICDs from 5 centers
258                              ICD efficacy in catecholaminergic polymorphic VT depends on arrhythmia m
259  17% of long-QT syndrome patients and 13% of catecholaminergic polymorphic VT patients.
260 edian (interquartile range) ages at onset of catecholaminergic polymorphic VT symptoms and ICD implan
261                                              Catecholaminergic polymorphic VT was diagnosed if epinep
262                                  Testing for catecholaminergic polymorphic VT was positive in 7% and
263 ncluding long QT syndrome, Brugada syndrome, catecholaminergic polymorphic VT, and short QT syndrome.
264 R4496C+/-) by crossbreeding PLN-KO mice with catecholaminergic polymorphic VT-associated RyR2-R4496C
265 s and stress-induced VTs in a mouse model of catecholaminergic polymorphic VT.
266  of cardiac ryanodine-receptor (RyR2)-linked catecholaminergic polymorphic VT.
267 had taken place, using L-Dopa, primarily, as catecholaminergic precursor.
268                          We conclude that 1) catecholaminergic projections to the hypothalamus provid
269 ular tachycardia and sudden cardiac death on catecholaminergic provocation by caffeine/epinephrine or
270                                              Catecholaminergic responses may be partially genetic and
271                                  To evaluate catecholaminergic responses without systemic counterregu
272 s study was to determine the contribution of catecholaminergic rostral ventrolateral medulla catechol
273 ctivation of rostral ventrolateral medullary catecholaminergic (RVLM-CA) neurons e.g., by hypoxia is
274 hether these rostral ventrolateral medullary catecholaminergic (RVLM-CA) neurons use glutamate as a t
275          We used an adenoviral vector with a catecholaminergic-selective promoter (AVV-PRS) to retrog
276 resulted in some degree of H3S28p, sustained catecholaminergic stimulation almost entirely failed to
277 Whereas, in CaMKII-deficient myocytes, acute catecholaminergic stimulation resulted in some degree of
278 acilitation of "top-down" control, excessive catecholaminergic stimulation, and subcortical imbalance
279 ) release channel and greatly exacerbated by catecholaminergic stimulation, with the development of a
280 eart rate and contractile responses to acute catecholaminergic stimulation.
281  first comprehensive and detailed map of the catecholaminergic structures in the brain of two represe
282 ry analysis of postmortem samples that human catecholaminergic substantia nigra and locus coeruleus n
283 ons, including glutamatergic, GABAergic, and catecholaminergic subtypes.
284 th norepinephrine and caffeine to simulate a catecholaminergic surge, Scn8a(N1768D/+) mice showed ven
285 owever, pharmacological manipulations of the catecholaminergic system have been scarce, and their pri
286 er, these findings provide evidence that the catecholaminergic system influences fear expression in h
287                                    Since the catecholaminergic system, especially the neurotransmitte
288    The results suggest novel links among the catecholaminergic system, oxidative pathways, and system
289 associated with dysregulation of the central catecholaminergic system.
290 iated neural circuits reveals defects in the catecholaminergic system.
291  the potential to identify disruption to the catecholaminergic systems and to provide a direct measur
292                                              Catecholaminergic systems in the brain are among the mos
293 ing the structure and function of a person's catecholaminergic systems is likely to allow more refine
294 appears to be a regular feature of zebrafish catecholaminergic systems.
295                                  Transiently catecholaminergic (TC), neural crest-derived precursors
296 vested effort involves mechanisms related to catecholaminergic transmission and a suppression of DMN
297 und in chromaffin granules, we conclude that catecholaminergic transmitter systems have the potential
298  genetic murine model of arrhythmic disease (catecholaminergic ventricular tachycardia; CPVT).
299 atory or indirect GABAergic pathway onto the catecholaminergic VTA/SNc homologs and serotonergic raph
300 PPPACAP, and PPNPY mRNAs, which were largely catecholaminergic, were activated by hydralazine but not

 
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