ライフサイエンスコーパス: caudate-putamen

1 n (the core of the nucleus accumbens and the caudate putamen).

2 s dorsal to the VP (e.g., nucleus accumbens, caudate putamen).

3 and lower levels in the corpus callosum and caudate putamen.

4 r are colocalized in the cerebral cortex and caudate putamen.

5 +)-NeuN(+)-betaIII-tubulin(+) neurons to the caudate putamen.

6 the cingulate cortex, nucleus accumbens, and caudate putamen.

7 ing levels of both ligands were found in the caudate putamen.

8 n the occipital cortex, parietal cortex, and caudate putamen.

9 was also a prominent increase in GMD in the caudate putamen.

10 3/J mice SSTR-3 mRNA was not detected in the caudate putamen.

11 levation of SSTR-1 and -3 mRNAs in the mouse caudate putamen.

12 us, and cortex but not to the hippocampus or caudate putamen.

13 opamine-innervated, neuronal subtypes in the caudate-putamen.

14 Neuropeptide Y was elevated in the caudate-putamen.

15 Aergic DARPP-32+ medium spiny neurons in the caudate-putamen.

16 dala or the nucleus accumbens but not to the caudate-putamen.

17 increases in ubiquitin levels were unique to caudate-putamen.

18 at maintains extensive axonal projections to caudate-putamen.

19 ine uptake in both the nucleus accumbens and caudate-putamen.

20 mpus and cortex and smallest in thalamus and caudate-putamen.

21 allidal neurons which lie in the dorsomedial caudate-putamen.

22 of labelled varicosities in the dorsolateral caudate-putamen.

23 somedial accumbens along its border with the caudate-putamen.

24 rgent projections to multiple regions of the caudate-putamen.

25 the cortex and thalamus but disagree in the caudate-putamen.

26 choroid plexus, 0.29 ug . g(-1) +/- 0.05 in caudate-putamen, 0.26 ug . g(-1) +/- 0.05 in reticular n

27 otor cortices (18%, P<0.01) and dorsolateral caudate putamen (17%, P<0.01), but not in the ventrolate

28 s, all three drugs increased accumulation in caudate putamen 3-5 times, and indomethacin and probenec

29 naive rats, quinpirole decreased LCGU in the caudate/putamen (84% of control), lateral habenula (80%

30 pallidum (82% of control) as well as in the caudate/putamen (86% of control), lateral habenula (77%

31 the exception of cingulate cortex and caudal caudate-putamen, a significantly greater response was ob

32 [(18)F](R)-FIPCT uptake in the caudate/putamen achieved transient equilibrium at 75 min

33 ine in the nucleus accumbens relative to the caudate-putamen after systemic cocaine administration.

34 in several other brain areas, including the caudate-putamen, amygdala, and brainstem regions such as

35 Brain areas analyzed were caudate, putamen, amygdaloid complex, hippocampal format

36 S decreased 5-HT release in the dorsolateral caudate-putamen, an area implicated in the etiology of H

37 llosum, habenulae, septum, fornix, thalamus, caudate putamen and a few in fasciculus retroflexus and

38 l septum, from the posterior thalamus to the caudate putamen and cerebral cortex, and from the parabr

39 and D(4) receptors in nucleus accumbens and caudate putamen and D(2) receptors in medial prefrontal

40 C38, a marker for GABAergic neurons, in the caudate putamen and lateral shell of the accumbens.

41 nstrated that individual neurons in both the caudate putamen and lateral shell of the nucleus accumbe

42 , displayed down-regulated expression in the caudate putamen and NAc of morphine, but not cocaine, ab

43 ate presynaptic dopaminergic function in the caudate putamen and nucleus accumbens of adult male rats

44 cocaine administration, were measured in the caudate putamen and nucleus accumbens of C57BL/6J and 12

45 zation of the CXCL12 receptor, CXCR4, in the caudate putamen and nucleus accumbens of the adult rat b

46 The ability of terminals in the caudate putamen and nucleus accumbens to release and reg

47 sphorylation in the two striatal structures (caudate putamen and nucleus accumbens).

48 ra and ventral tegmental area project to the caudate putamen and nucleus accumbens, where they modula

49 ra and ventral tegmental area project to the caudate putamen and nucleus accumbens/olfactory tubercle

50 1r immunoreactivity (gfD1r-IR), found in rat caudate putamen and rat retina were virtually identical

51 Persistent injury to the caudate putamen and thalamus at P1 was significantly cor

52 However, EM-induced FOSir was absent in the caudate putamen and the accumbens nucleus, both areas of

53 three subdivisions of the basal ganglia, the caudate, putamen and globus pallidus in brown capuchin m

54 ume and diffusivity changes in the thalamus, caudate, putamen and hippocampus and examined diffusion

55 patients had higher (11)C-CNS 5161 uptake in caudate, putamen and precentral gyrus compared to the pa

56 describe pharmacokinetic-occupancy curves in caudate, putamen and thalamus.

57 l dopamine transporter availability in ADHD (caudate, putamen and ventral striatum: +24%, p<0.01); wh

58 ry processes mediated by the hippocampus and caudate-putamen and (ii) bias the brain toward the use o

59 s rapidly eliminates the DA terminals in the caudate-putamen and causes cell bodies in the midbrain t

60 increase in mitochondrial DNA damage in the caudate-putamen and cerebellum.

61 II maximally inhibited 50% of binding in the caudate-putamen and had no effect on 125I-epibatidine bi

62 Neurotoxic injury of the caudate-putamen and lateral globus pallidus unilaterally

63 Optogenetic stimulation in the caudate-putamen and neocortex of "histaminergic" axonal

64 Therefore, we characterized the caudate-putamen and nucleus accumbens extracellular acet

65 Extracellular DA in the caudate-putamen and nucleus accumbens, evoked by electri

66 rizations along the dorsolateral edge of the caudate-putamen and sparsely-packed arborizations more m

67 disease (PD) including the cerebral cortex, caudate-putamen and substantia nigra pars compacta.

68 ay and dopaminergic neuronal degeneration in caudate-putamen and substantia nigra.

69 teral projections mainly to both the ventral caudate-putamen and the amygdala, but not to the dorsal

70 ue levels of DA were higher in the MPOA, the caudate-putamen and the bed nucleus of stria terminalis

71 rs (10-30 microg of MPEP) in the dorsomedial caudate-putamen and the similar cytological expression p

72 PPI - dorsolateral (dlCPu) and medial (mCPu) caudate/putamen and core (NACc) and medial shell (NACms)

73 he amygdala, and decreased metabolism in the caudate/putamen and medial geniculate nucleus.

74 ograde tracers, placed into the dorsolateral caudate/putamen and the nucleus accumbens, were used to

75 of the midbrain project to the dorsolateral caudate/putamen and to the ventromedially located nucleu

76 ns (cerebellum, cortex, substantia nigra and caudate-putamen) and skin fibroblasts from HD patients.

77 all spontaneously active neurons in dorsal (caudate-putamen) and ventral (accumbens, core) striatum;

78 NA expression levels in cortex, hippocampus, caudate putamen, and amygdala decreased during the secon

79 at brains were mostly located in the cortex, caudate putamen, and amygdala with an extremely low dens

80 ajor axonal pathways and in the hippocampus, caudate putamen, and cerebellum.

81 rs in the rostral nucleus accumbens, rostral caudate putamen, and layer I of the rostral cingulate co

82 ve neurons were consistently observed in the caudate putamen, and moderately or weakly labeled neuron

83 er types avoided the nucleus accumbens core, caudate putamen, and the globus pallidus.

84 n the olfactory tubercle, nucleus accumbens, caudate putamen, and the layer VI of the neocortex compa

85 as also increased in the olfactory tubercle, caudate putamen, and the nucleus accumbens of mu-opioid

86 mages showed an increased local field in the caudate, putamen, and globus pallidus of patients relati

87 Three basal ganglia structures (caudate, putamen, and globus pallidus) and one compariso

88 ologic features of the basal ganglia nuclei (caudate, putamen, and globus pallidus) in children with

89 The average sizes of the caudate, putamen, and globus pallidus, but not of the th

90 increases and decreases in subregions of the caudate, putamen, and hippocampus in 22q-dup relative to

91 ssexuals increased SERT binding in amygdala, caudate, putamen, and median raphe nucleus.

92 ed behavioral approach tendencies by biasing caudate, putamen, and nucleus accumbens but not amygdala

93 acial cues in reward-related regions such as caudate, putamen, and nucleus accumbens but not the amyg

94 Striatal volume (sum of caudate, putamen, and nucleus accumbens volumes) and ICV

95 actor analysis, and Pearson correlations for caudate, putamen, and pallidum (also correlated with age

96 patients in the amygdala, nucleus accumbens, caudate, putamen, and posterior ventral thalamus, while

97 The left and right SBRs for caudate, putamen, and striatum were evaluated with the m

98 he NHP experiment, binding potentials in the caudate, putamen, and substantia nigra (4.9, 4.9, and 1,

99 nding in the hippocampus, prefrontal cortex, caudate, putamen, and substantia nigra.

100 pical neuroleptic was associated with higher caudate, putamen, and thalamus volumes, whereas a higher

101 and N-acetylaspartate in the left and right caudate, putamen, and thalamus were scaled into concentr

102 e metabolic rates bilaterally in the ventral caudate, putamen, and thalamus.

103 Caudate, putamen, and total brain volumes were quantifie

104 l D2R availability compared with nonsmokers (caudate, putamen, and ventral striatum) and with ex-smok

105 easured in the subdivisions of the striatum (caudate, putamen, and ventral striatum) in addition to t

106 rsomedial and ventrolateral quadrants of the caudate-putamen, and in the rostrobasal cell cluster, ro

107 as seen in the piriform cortex, hippocampus, caudate/putamen, and cerebellar cortex.

108 the cerebral cortex, hippocampus, thalamus, caudate/putamen, and olfactory bulb, with lower levels i

109 levels for gb2 were virtually absent in the caudate/putamen, and significantly lower in the medial b

110 monoamine transporter type 2 density for the caudate, putamen,and substantia nigra were 21.50%, 58.20

111 verage SBRs and z scores for whole striatum, caudate, putamen, anterior putamen, and posterior putame

112 suggests that lateral sensorimotor areas of caudate putamen are important for responding based on ex

113 ising the nucleus accumbens core, shell, and caudate-putamen, are instrumental for a wide-range of fu

114 post-synaptic areas (frontal cortex, septum, caudate putamen), as well as significantly higher plasma

115 signals in both health and disease from the caudate-putamen, as well as possibly from other subcorti

116 sal ganglia, cortex above the diencephalon], caudate-putamen, basal forebrain, hypothalamus, hippocam

117 no effect on ethanol-induced Fos-IR changes (caudate putamen, bed nucleus of the stria terminalis, an

118 amphetamine, MK-801 did not increase FLI in caudate-putamen, bed nucleus of the stria terminalis, or

119 ts' cerebellum, cortex, substantia nigra and caudate-putamen brain regions.

120 ntration in the nucleus accumbens (NAcc) and caudate putamen but not the medial prefrontal cortex or

121 associated with a selective decrease in the caudate-putamen but not nucleus accumbens extracellular

122 ctive decrease in the dopamine response, the caudate-putamen but not nucleus accumbens extracellular

123 mine binge decreased acetylcholine levels in caudate-putamen, but had no effect on levels in nucleus

124 e show that inactivation of the dorsolateral caudate-putamen, but not other structures previously imp

125 gia after restoration of DA signaling in the caudate putamen by viral gene therapy.

126 sted in retinae of goldfish and rat, and rat caudate putamen, by using immunoblots and light microsco

127 ochemical manipulation of the hippocampus or caudate-putamen can bias an animal toward the use of a s

128 activated regions include accumbens nucleus, caudate putamen, claustrum, bed nucleus of the stria ter

129 rtex, olfactory tubercle, nucleus accumbens, caudate-putamen, claustrum, dorsal endopiriform nucleus,

130 In the dorsal portion of the caudate putamen, core and shell of the nucleus accumbens

131 lateral ventricle, in zones adjacent to the caudate putamen, corpus callosum, and lateral septal nuc

132 to a lesser extent, in the hippocampus (HP), caudate putamen (CP) and frontal cortex (FC).

133 olfactory bulb (MOB), cerebral cortex (CTX), caudate putamen (CP), hippocampus (HP), thalamus (TH), a

134 brain areas including the hippocampus (HI), caudate putamen (CP), the hypothalamic paraventricular n

135 ctive interfering particles (DIPs), into the caudate-putamen (CP) and scored for an innate immune res

136 ithin the SN, while loss of RN in the SN and caudate-putamen (CP) was slower and required > or =100 n

137 sponse included the nucleus accumbens (NAS), caudate-putamen (CP), hippocampus, and medial thalamus.

138 measured in the nucleus accumbens (NAc) and caudate putamen (CPu) after 3-day 'binge' pattern cocain

139 underlying basis for differences between the caudate putamen (CPu) and nucleus accumbens (NAc).

140 Membranes of rat caudate putamen (CPu) and thalamus or CHO-HA-rMOR cells

141 ucleus (MnPO), periventricular nucleus (Pe), caudate putamen (CPU) and the ependymal lining of the ve

142 stem in the cortex, nucleus accumbens (NAc), caudate putamen (CPu) and the region containing the subs

143 as restoration of dopamine production to the caudate putamen (CPu) does not fully restore the hyperlo

144 ), nucleus accumbens (NAc), hippocampus, and caudate putamen (CPu) in morphine-induced conditioned pl

145 and sends a glutamatergic projection to the caudate putamen (CPu) in rat.

146 oration of dopamine synthesis to the central caudate putamen (CPu) of DD mice rescues feeding and sur

147 abel dopaminergic axons and terminals in the caudate putamen (CPu) of rats 7 days prior to a neurotox

148 e effect of NBQX (0, 0.3 mug/0.3 mul) in the caudate putamen (CPu) on CS responding in the non-alcoho

149 In the hippocampus (HIP) and caudate putamen (CPu), pERK and FosB/DeltaFosB levels we

150 early genes (IEGs) c-Fos and JunB in the rat caudate putamen (CPu).

151 ny corresponding drug-induced changes in the caudate-putamen (CPu) after L-DOPA administration.

152 5 from the globus pallidus (GP), 98 from the caudate-putamen (CPu) and 23 from the central nucleus of

153 RTI-121-labeled dopamine transporters in the caudate-putamen (CPu) and cortex as well as depletion of

154 tyrosine hydroxylase immunoreactivity in the caudate-putamen (CPu) and decreases in CPu tissue dopami

155 possible mechanisms by which neurons in the caudate-putamen (CPu) and globus pallidus (GP) participa

156 and MKP-1 phosphorylation (p-MKP-1), in the caudate-putamen (CPu) and nucleus accumbens (NAc) of Fis

157 MOR activation was first detected in the caudate-putamen (CPU) at e12.5, and by e15.5, activity h

158 t, is induced in nucleus accumbens (NAc) and caudate-putamen (CPu) by repeated exposure to drugs of a

159 n blot, were found to be elevated in NAc and caudate-putamen (CPu) of FR relative to AL rats.

160 protein were induced in the hippocampus and caudate-putamen (CPu), accompanied by increased caspase-

161 ession in the nucleus accumbens core (NAcC), caudate-putamen (CPu), and subthalamic nucleus.

162 ine-induced c-Fos and JunB expression in the caudate-putamen (CPu), the mu receptor antagonist, beta-

163 Jun-B, in the dorsomedial portion of the rat caudate-putamen (CPu).

164 al pathways (substantia nigra, SN and dorsal caudate-putamen, CPu).

165 ctural MRI to measure concurrent declines in caudate/putamen D2 neuroreceptor binding and tissue volu

166 ride positron emission tomography to measure caudate/putamen D2 receptor binding.

167 in regional metabolism and network activity, caudate/putamen DAT binding, and Unified Parkinson's Dis

168 erminalis, septal nuclei, nucleus accumbens, caudate putamen, diagonal band, amygdala, hypothalamus,

169 behaviors characteristic of DA injections in caudate-putamen did not occur.

170 , we examined the effects of a) dorsolateral caudate putamen (dlCPu) lesions on cocaine self-administ

171 c to dorsomedial and ventromedial regions of caudate-putamen (dmCPu, vmCPu).

172 ml g(-1)), followed by ventricular regions (caudate putamen, ependyma, hippocampus, 0.05-0.14 ml g(-

173 with ERalpha in the MeA (MeA-ERalpha) or the caudate-putamen (ERalpha control) or luciferase (MeA-sit

174 pear to diffuse readily or accumulate in the caudate-putamen even though there was some penetration a

175 tly upregulated in the lateral region of the caudate-putamen exclusively in postweanling mice after c

176 measurements in the test experiment for the caudate putamen, frontal cortex, cerebral cortex, hippoc

177 ptors was measured in the nucleus accumbens, caudate putamen, frontal cortex, olfactory tubercle and

178 e globus pallidus, cingulate cortex, insula, caudate, putamen, frontal cortex, temporal cortex, and t

179 from blood was increased in cerebral cortex, caudate putamen, globus pallidus, entopeduncular nucleus

180 throughout basal ganglia (nucleus accumbens, caudate-putamen, globus pallidus, substantia nigra) in t

181 reactivity included the cerebral cortex, the caudate-putamen, globus pallidus, the hippocampal format

182 binding in the cerebral cortex, cerebellum, caudate/putamen, globus pallidus, substantia nigra, and

183 phA4 protein revealed discrete expression in caudate/putamen, globus pallidus, substantia nigra, cere

184 nked as follows: cingulate cortex > insula > caudate/putamen > frontal cortex > temporal cortex > tha

185 overlying diencephalon, the olfactory bulbs, caudate-putamen, hippocampus, tectum, and lower brainste

186 ne and amphetamine in the prefrontal cortex, caudate-putamen, hypothalamus, or cerebellum.

187 cantly increased mean dopamine levels in the caudate putamen in the C57BL/6J mice (with a 3-h mean of

188 icant deficits throughout all aspects of the caudate-putamen in animals exposed to METH.

189 performance, and that lesions of the lateral caudate putamen increased choice response time for the S

190 f Parkinson's disease, density of binding in caudate-putamen increased at KA, but not NMDA or AMPA re

191 ion of apomorphine into the NAcc but not the caudate putamen induced partner preferences in the absen

192 Caudate putamen infarction never occurred without cardia

193 rculum in response to food intake and in the caudate, putamen, insula, thalamus, and orbitofrontal co

194 Medial (mCPu), lateral, and complete (CPu) caudate-putamen lesions affected speed and accuracy of s

195 e prominent differences in areas such as the caudate, putamen, locus coeruleus, medial habenula, and

196 Affected structures included the caudate putamen, medial geniculate nucleus, lateral geni

197 cal forebrain in Lhx8 mutants, including the caudate-putamen, medial septal nucleus, nucleus of the d

198 s treated with methadone exhibited increased caudate/putamen metabolism, whereas buprenorphine produc

199 t expression included thalamic relay nuclei, caudate-putamen, molecular layer of the dentate gyrus, a

200 ression of 5-HT(2A) receptors in the cortex, caudate putamen, NAc and hippocampus of rat brain.

201 al, two somatomotor, one cerebellar, and one caudate-putamen network), and four "higher-order" associ

202 glutamatergic projections within the cortex, caudate putamen nucleus (CPN), hippocampal formation, an

203 y terminate in patch-like regions within the caudate putamen nucleus (CPN).

204 in the striatal circuitry originating in the caudate putamen nucleus (CPN).

205 These include the caudate-putamen nucleus (CPN) and the globus pallidus, w

206 The dorsolateral caudate-putamen nucleus (CPN) and the nucleus accumbens

207 nt, delta-opioid receptors (DORs) in the rat caudate-putamen nucleus (CPN) appear later than mu-opioi

208 The patch compartments of the caudate-putamen nucleus (CPN) are enriched in mu-opioid

209 Prefrontal corticostriatal afferents to the caudate-putamen nucleus (CPN) have been implicated in mo

210 tion of delta-opioid receptors (DORs) in the caudate-putamen nucleus (CPN) produces regionally distin

211 ed, respectively, by 32%, 35% and 30% in the caudate putamen, nucleus accumbens and ventral pallidum

212 esions in four sites: the medial and lateral caudate putamen, nucleus accumbens, and olfactory tuberc

213 aminergic nerve terminal-rich brain regions (caudate putamen, nucleus accumbens, and ventral pallidum

214 est network-wide hyporeactivity of striatal (caudate, putamen, nucleus accumbens) and cortical (insul

215 rontal cortex, temporal cortex, hippocampus, caudate, putamen, nucleus accumbens, amygdala, thalamus,

216 in four basal ganglia regions, including the caudate, putamen, nucleus accumbens, and globus pallidus

217 any of the brain regions studied (thalamus, caudate, putamen, nucleus accumbens, globus pallidus, an

218 e decreased at postnatal day 21 (P21) in the caudate-putamen, nucleus accumbens and frontal cortex bu

219 -2Rbeta induced marked increases in c-Fos in caudate-putamen, nucleus accumbens and prefrontal cortex

220 eus of the hypothalamus, olfactory tubercle, caudate-putamen, nucleus accumbens and substantia nigra

221 having a striatal composition, including the caudate-putamen, nucleus accumbens, and olfactory tuberc

222 Fourteen striatal subterritories in caudate-putamen, nucleus accumbens, and olfactory tuberc

223 rog), both groups displayed increased FLI in caudate-putamen, nucleus accumbens, bed nucleus of the s

224 cortical brain regions (cortex, hippocampus, caudate-putamen, nucleus accumbens, thalamus, and hypoth

225 A4 mRNA levels were prominent in the DA-rich caudate/putamen, nucleus accumbens and globus pallidus,

226 hetamine-induced monoamine reductions in the caudate-putamen occur rapidly, peak at 75-80% below cont

227 cytes in the cortex, septum, hippocampus and caudate putamen of Id4(-/-) adult brains were decreased,

228 her protein levels were also observed in the caudate putamen of iP rats compared with iNP rats.

229 ex area 1 and rostral subventricular zone of caudate putamen of isoflurane-preconditioned rats than r

230 e five somatostatin receptors (SSTRs) in the caudate putamen of male C57BL/6J and 129P3/J mice.

231 of DeltaFosB also occurs exclusively in the caudate putamen of periadolescent mice after amphetamine

232 erations in the NMDA channel activity in the caudate-putamen of adult rats, and may be responsible fo

233 male rats, maximal [3H]MK-801 binding in the caudate-putamen of female ISO rats was lower than female

234 ubiquitin levels particularly as seen in the caudate-putamen of HD patients.

235 Maximal [3H]MK-801 binding in the caudate-putamen of male ISO rats was 58% higher compared

236 hetamine (40 mg/kg s.c.) was investigated in caudate-putamen of male Sprague-Dawley rats.

237 firing in prefrontal cortical layers and the caudate-putamen of rhesus monkeys, trained in a spatial-

238 inase (RSK), and c-Fos protein levels in the caudate/putamen of Fischer rats.

239 ccumulated upon neurons in the neocortex and caudate/putamen of infected mice and interacted with nat

240 (17%, P<0.01), but not in the ventrolateral caudate putamen or any subfield of the hippocampus.

241 implanted with a CMA guide cannula into the caudate putamen or nucleus accumbens.

242 overy, dialysis probes were lowered into the caudate putamen or the nucleus accumbens and mice were p

243 nificant differences on P0, P7 or P14 in the caudate-putamen or frontal cortex, suggesting that cell

244 N-positive or NeuN-negative cells in the P21 caudate-putamen or frontal cortex.

245 responsivity does not involve changes in the caudate-putamen or nucleus accumbens extracellular dopam

246 ume changes in dorsal or ventral hippocampi, caudate-putamen, or thalamus were not detectible.

247 structures analyzed (P<0.05) except for the caudate putamen (P=0.09) and the globus pallidus (P=0.12

248 .002) in protocol 1 and specifically, in the caudate/putamen (P<0.01) in both protocols.

249 as intracranial volume, but larger bilateral caudate, putamen, pallidum and lateral ventricle volumes

250 olume and surface-based shape metrics of the caudate, putamen, pallidum, and nucleus accumbens in 53

251 bels to each voxel, including left and right caudate, putamen, pallidum, thalamus, lateral ventricles

252 l formation, temporal pole), striatum (i.e., caudate, putamen), precuneus, and cerebellum.

253 ated with decreased uptake of (3)H-NE in the caudate putamen (R=0.436, p=0.033) and locus coeruleus (

254 iring in the localized prefrontal layers and caudate-putamen region exhibited similar location prefer

255 njection of Cre recombinase into the central caudate putamen restores feeding and normalizes locomoti

256 Restoration of DA production within the caudate putamen restores feeding on regular chow and nes

257 to express Cre recombinase into the central caudate putamen restores normal Th gene expression to th

258 placing [3H]WIN 35 428 binding at DAT in rat caudate putamen revealed that the 4'-azido-3'-iodophenyl

259 8 binding at the dopamine transporter in rat caudate-putamen revealed that aromatic substitutions on

260 je cells, with high levels also in thalamus, caudate putamen, septal nucleus, nucleus accumbens, amyg

261 Reductions in caudate-putamen serotonin began earlier and were ultimat

262 showed increases in CBF and MUA, whereas the caudate-putamen showed decreased CBF with increased MUA.

263 However, the caudate-putamen showed fMRI, CBV, and CBF decreases desp

264 However, AMPH injections into dorsal control caudate putamen sites produced a modest, dose-dependent

265 (TRAP) to conduct transcriptomic analyses of caudate/putamen (striatal) cell type-specific gene expre

266 r with primary neuronal pathology within the caudate-putamen (striatum).

267 onounced in the basal ganglia, including the caudate-putamen, striatum and substantia nigra.

268 os expression in the ventral tegmental area, caudate putamen, substantia nigra pars reticulata, entor

269 d nuclear (beta-polymerase) targets from the caudate-putamen, substantia nigra and cerebellum regions

270 d ventricles and periventricular structures (caudate, putamen, thalamus, hippocampus).

271 N in the ventrolateral thalamic nucleus, the caudate putamen, the accumbens nucleus, the medial septu

272 The dopamine (DA) inputs to the caudate putamen, the nucleus accumbens, and the amygdala

273 ere injected into discrete subregions of the caudate-putamen, the nucleus accumbens, or the amygdala.

274 the bed nuclei of the stria terminalis; the caudate-putamen; the globus pallidus; the lateral septum

275 ) release in the nucleus accumbens (NAc) and caudate-putamen through an indirect mechanism that invol

276 for displacement of [(3)H]WIN 35 428 in rat caudate putamen tissue.

277 he displacement of [(3)H]WIN 35 428 from rat caudate putamen tissue.

278 ine uptake inhibition (IC(50) values) in rat caudate putamen tissue.

279 mined by inhibition of [3H]WIN 35,428 in rat caudate putamen tissue.

280 2.1]heptane derivatives were measured in rat caudate-putamen tissue and found to be 100-3000-fold les

281 gene expression in the nucleus accumbens and caudate-putamen, two structures known to be involved in

282 binding than patients without fatigue in the caudate, putamen, ventral striatum and thalamus.

283 Regions of interest for the caudate, putamen, ventral striatum, SN, and cerebellum w

284 Regions of interest for the caudate, putamen, ventral striatum, substantia nigra (SN

285 croprobes were used, the average BPND in the caudate putamen was 0.94, and no significant changes in

286 sity of Ang II receptor binding sites in the caudate putamen was 407+/-26 fmol/g, in the nucleus accu

287 owever the tissue content of dopamine in the caudate putamen was decreased, representing a diminution

288 Only the caudate/putamen was examined in protocol 2.

289 The caudate/putamen was rich in alpha1, alpha2, alpha5, all

290 l levels of dopamine in the dialysate of the caudate putamen were 4.2+/-0.2 nM in C57BL/6J mice and 5

291 Extracellular concentrations of NE in the caudate putamen were significantly decreased in response

292 te NMDA, AMPA and KA receptor binding in rat caudate-putamen were examined by quantitative in vitro r

293 ssue contents of dopamine within the NAc and caudate-putamen were not significantly different in ko c

294 ality in saline controls and the dorsomedial caudate-putamen, where Fos was correlated with respondin

295 amine uptake (IC50 range = 10-371 nM) in rat caudate putamen, whereas ligands with a nonbasic nitroge

296 ormally contribute to the medial part of the caudate-putamen, whereas late-born striatal cholinergic

297 ncrease in DNA damage in mitochondria of the caudate-putamen while there was no significant DNA damag

298 croglial activation and apoptosis, including caudate/putamen, white matter, and, in juvenile-onset ca

299 binding from the dopamine transporter in rat caudate putamen with K(i) values ranging from 13.9 to 47

300 1.0 nmol) activated both ERK 1/2 and CREB in caudate-putamen with no difference between feeding group