ライフサイエンスコーパス: caval

1 portal-caval, and 5 H-shaped (H-type portal-caval)], 2 had portal-to-hepatic vein shunts (portohepat

2 unt [subdivided in 5 end-to-side-like portal-caval, 7 side-to-side-like portal-caval, and 5 H-shaped

3 The wide-open triangular caval anastomosis is easy to perform, allowing short imp

4 e liver graft was reduced and latero-lateral caval anastomosis was performed.

5 VC) stenosis in a patient with a "piggyback" caval anastomosis.

6 Extracorporeal bypass, including both caval and portal venous return, produced significant inc

7 Caval and pulmonary veins were occluded.

8 ike portal-caval, 7 side-to-side-like portal-caval, and 5 H-shaped (H-type portal-caval)], 2 had port

9 stance, and lowering cerebral, superior vena caval, and pulmonary blood flows.

10 This study describes the first use of caval-aortic access and closure to enable transcatheter

11 Caval-aortic access and tract closure were successful in

12 Caval-aortic access has been successful in animals.

13 Caval-aortic access refers to percutaneous entry into th

14 ibitive-risk patients who underwent TAVR via caval-aortic access.

15 There were no deaths attributable to caval-aortic access.

16 January 2014, 19 patients underwent TAVR via caval-aortic access; 79% were women.

17 l technique of CT-guided trans superior vena caval approach for 3 tricky deep-seated mediastinal lesi

18 witching from an inferior to a superior vena caval approach; 5) use of a 60-cm guiding sheath; 6) det

19 for dialysis, and unsuitability of the iliac-caval axis as a site for KT.

20 ity pressure during transient inferior, vena caval balloon occlusions.

21 the sheath in situ, the trans superior vena caval biopsy was performed under CT guidance, and multip

22 d tomography (CT)-guided trans superior vena caval biopsy, which we have performed in 3 consecutive p

23 was established postasystole via aortic and caval cannulation and maintained for 2 h.

24 ovenous bypass, portocaval decompression, or caval clamping in 11 recipients and describe the modific

25 ss was 500 mL, and no patient required total caval clamping.

26 ovenous bypass, portocaval decompression, or caval clamping.

27 In the present models, the caval connections were offset through a range of 0.0 to

28 Chronic thoracic inferior vena caval constriction (TIVCC) is a model of reduced cardiac

29 unloading produced by thoracic inferior vena caval constriction (TIVCC).

30 Fontan physiology includes knowledge of the caval contributions to right (RPA) and left (LPA) pulmon

31 cipal diagnosis of proximal or inferior vena caval deep vein thrombosis and treated with CDT from 200

32 donor vena cava was too short to bridge the caval defect for interposition.

33 ight ventricular function, and inferior vena caval diameters.

34 the common trunk allowed near completion of caval dissection with no prolonged inflow occlusion.

35 ing the technique of liver resection without caval excision (the piggyback technique).

36 nt percutaneous placement of a superior vena caval filter for prevention of PE.

37 ere was a fivefold increase in the number of caval filter implants.

38 Except for one randomized trial, the vena caval filter literature consists of case series or conse

39 Eleven patients received anticoagulants or caval filter placement as a result of CT findings.

40 Inferior vena caval filters (IVCFs) may prevent recurrent pulmonary em

41 ary therapy for venous thromboembolism, vena caval filters are an important alternative when anticoag

42 ndications to anticoagulation, inferior-vena-caval filters can be considered, but their use needs car

43 to establish the appropriate place for vena caval filters in the treatment of venous thromboembolic

44 Inferior vena caval filters provide protection from life-threatening P

45 Vena caval filters represent a potentially important but poor

46 safety, effectiveness, numbers, and types of caval filters.

47 (POH-DCM); they were compared to VOH (aorta-caval fistula).

48 Myocardial infarcted rats and aorto-caval fistulated rats were used as a low output HF model

49 After the method was validated, caval flow contributions were quantified in patients.

50 imaging enable in vivo quantification of the caval flow distribution to the PAs in patients with Font

51 All patients had persistent aorto-caval flow immediately post-procedure.

52 litates total hepatectomy with vena cava and caval flow preservation.

53 in the fetal heart that direct inferior vena caval flow towards the foramen ovale.

54 abdominal aortic blood flow and reduced vena caval flow which is only partially compensated for by in

55 by using primarily endobronchial forceps for caval fragments and snares for cardiac and pulmonary fra

56 epatic venous gas, and two had inferior vena caval gas.

57 ess to the abdominal aorta by electrifying a caval guidewire and advancing it into a pre-positioned a

58 For all 3 caval inflow ratios, a positive correlation existed betw

59 of HFD in response to age-related changes in caval inflows (SVC:IVC, 2, 1, and 0.5 corresponded to ag

60 ive insights into the impact of the changing caval inflows on Fontan's long-term HFD, highlighting th

61 The effect of age-related changes in caval inflows on HFD was evaluated using cardiac magneti

62 erations (52%) which were performed with the caval interposition approach to liver transplantation, c

63 gery in managing these complex patients with caval involvement.

64 rterial pressure catheters and inferior vena caval (IVC) occluders; four had placement of thoracic ao

65 studied four dogs before and during inferior caval (IVC) occlusion at five different inotropic stages

66 epatic venous (HV), subhepatic inferior vena caval (IVC), and portal venous (PV) flow rates were meas

67 rior margin of the inferior vena cava (hilar-caval line) on lateral radiographs; this line correspond

68 er (n = 66) or anterior to (n = 2) the hilar-caval line.

69 Using the aorto-caval model of an AV fistula model in the rat, we demons

70 Transient inferior vena caval obstruction was used to determine PV relations.

71 bdominal compression, nitroglycerin, or vena caval obstruction).

72 plotted versus end-diastolic volume during a caval occlusion (preload-independent recruitable systoli

73 Vena caval occlusion (VCO) was used to reduce left ventricula

74 ystolic pressure-volume relationships during caval occlusion and was used as the gold standard of LV

75 iastolic pressure-volume relationship during caval occlusion at baseline, after sildenafil, and BNP i

76 The piggyback technique without caval occlusion is possible in the majority of patients.

77 We show that when performing caval occlusion is unfeasible, single-beat metrics can b

78 and outcome of a piggyback technique without caval occlusion or veno-venous bypass (VB), we retrospec

79 ants, and an animal with acute inferior vena caval occlusion to produce portal hypertension.

80 es (strain) from successive diastoles during caval occlusion were used to evaluate LV/RV diastolic me

81 Isolated Pringle maneuver and caval occlusion with Pringle maneuver produced significa

82 t increases in MAP and cardiac output during caval occlusion with Pringle maneuver, while atriocaval

83 after caval occlusion, Pringle maneuver, and caval occlusion with Pringle maneuver.

84 indices of cardiac contractility that avoid caval occlusion would offer considerable advantages for

85 nditions were altered by saline infusion and caval occlusion, and lusitropic state was changed by dob

86 For inferior vena caval occlusion, control biopsy specimens lost 1.23 g, w

87 recorded at baseline and at intervals after caval occlusion, Pringle maneuver, and caval occlusion w

88 ular P/Q, created by transient inferior vena caval occlusion, under basal and endotoxic conditions.

89 modified utilizing fluid administration and caval occlusion, whereas dobutamine and esmolol were use

90 Loading conditions were altered by caval occlusion, whereas lusitropic state was changed by

91 Preload independence during vena caval occlusions was achieved by preload adjustment (1/[

92 esults strongly suggest the incorporation of caval offsets in future total cavopulmonary connections.

93 Surgery may be complicated by superior vena caval or right upper pulmonary vein (RUPV) stenosis, sin

94 portal-caval shunts patients have a 1-stage caval partition, and the others have a 1-stage ligation.

95 reas the 5 others had a 1-stage longitudinal caval partition.

96 The first 2 side-to-side-like portal-caval patients had a successful 2-stage closure whereas

97 All 5 end-to-side-like portal-caval patients had a threadlike intrahepatic portal veno

98 vidence of IVC thrombosis, device migration, caval penetration, or pulmonary embolism.

99 ahepatic portosystemic shunts, H-type portal-caval, portohepatic, and patent ductus venosus patients

100 estimate the agreement between superior vena caval pressure (SVCP) and femoroiliac venous pressure (F

101 rterial pressure (AP) and intrathoracic vena caval pressure (VP).

102 here were trends toward higher superior vena caval pressure early after the operation and at follow-u

103 rences between right atrial or inferior vena caval pressures among the groups.

104 od pressure, right atrial, and inferior vena caval pressures were measured continuously.

105 Total caval pulmonary anastomosis was performed in 53 patients

106 the piggyback group (40.3%) compared to the caval replacement group (51.8%, P < 0.001).

107 vena cava preserving piggyback technique or caval replacement technique without veno-venous bypass o

108 Piggyback technique, compared with caval replacement, was associated with a reduced inciden

109 an ADV and an accompanying alternative porto-caval shunt between the right portal vein and inferior v

110 120 min (n=8) liver warm ischemia in splenic-caval shunt group survived for over 1 day, 6/8 for over

111 with or without portosystemic shunt (splenic-caval shunt).

112 s produced in 7-day-old rabbits via an aorto-caval shunt, after which, the rabbits were treated with

113 e rats with liver warm ischemia plus splenic-caval shunt.

114 er division of the bile duct without a porto-caval shunt.

115 have a 2-stage closure, side-to-side portal-caval shunts patients have a 1-stage caval partition, an

116 es are good provided end-to-side-like portal-caval shunts patients have a 2-stage closure, side-to-si

117 inated contrast-enhanced material showed the caval size to be within 3 mm in all 119 patients.

118 bypass in 11 operations (41%) performed with caval sparing (piggyback) surgical technique.

119 iation, simultaneous arterial, superior vena caval (SsvcO2), and pulmonary venous (SpvO2) oximetry wa

120 nts identified with late portal vein or vena caval stenoses or thromboses from a cohort of 524 grafts

121 Hepatic arterial fraction obtained with caval subtraction agreed well with those with fluorescen

122 strated between TLBF in humans measured with caval subtraction and direct inflow phase-contrast MR im

123 Conclusion Caval subtraction phase-contrast MR imaging is a simple

124 Thereafter, consistency of caval subtraction phase-contrast MR imaging-derived TLBF

125 Purpose To validate caval subtraction two-dimensional (2D) phase-contrast ma

126 Arterial (SaO(2)) and superior vena caval (SvO(2)) co-oximetry and cerebral oxygen saturatio

127 Unavailability of the iliac-caval system due to thrombosis or aberrant anatomy may p

128 When the iliac-caval system is unavailable, kidney graft implantation o

129 eed in the case of an adult with unsuspected caval system obliteration.

130 according to the ending of the shunt in the caval system.

131 nnels between the superior and inferior vena caval systems after bidirectional cavopulmonary anastomo

132 tions between the superior and inferior vena caval systems were identified and measured.

133 Robotic surgery for selected level I and II caval thrombi is feasible.

134 The incidence of caval thrombosis and DVT among patients with nonretrieve

135 d SNAD given for 7 days appeared to decrease caval thrombosis in this model of deep vein thrombosis.

136 The prevalence of observed post-filter caval thrombosis was 2.7%.

137 est this hypothesis a model of inferior vena caval thrombosis was used.

138 tal vein thrombosis, and 1 had inferior vena caval thrombosis.

139 A passive splenic and vena caval to jugular vein shunt with systemic heparinization

140 ding quantification of superior and inferior caval, total pulmonary artery, total pulmonary vein, asc

141 ) had complete closure of the residual aorto-caval tract.

142 tricle, atrioventricular septal defects, and caval vein abnormalities.

143 ur lineage analysis unequivocally shows that caval vein and atrial myocardium share a common origin a

144 tracing has suggested a distinct origin for caval vein myocardium, from a proposed third heart field

145 Inferior caval vein thrombosis was induced in cohorts of adult wi

146 pose tissue (PVAT) and induces it in venous (caval vein) PVAT.

147 caval veins, and a persistent left inferior caval vein.

148 egurgitant flow in the superior and inferior caval veins and tricuspid valve (adjusted r = 0.28-0.55;

149 are created by abnormal localization of the caval veins combined with ectopic pericardial cavity for

150 Respiratory-resolved analyses of caval veins showed significantly increased net and peak

151 y reduced sinus horn myocardium, hypoplastic caval veins, and a persistent left inferior caval vein.

152 ialization, alignment, and morphology of the caval veins.

153 us horn myocardium, and the alignment of the caval veins.

154 Vena caval venting decreased the release of potassium into th

155 conclude that portal vein flush without vena caval venting provided a lower incidence of PRS than any

156 The patients in groups 3 and 4 (vena caval venting) demonstrated smaller percentage increases

157 oup 4 (n=19), hepatic artery flush with vena caval venting.

158 group 3 (n=29), portal vein flush with vena caval venting; and group 4 (n=19), hepatic artery flush

159 : group 1 (n=31), portal vein flush, no vena caval venting; group 2 (n=21), hepatic arterial flush, n

160 up 2 (n=21), hepatic arterial flush, no vena caval venting; group 3 (n=29), portal vein flush with ve

161 fragments of polydioxanone suture within the caval wall at 32 weeks.