ライフサイエンスコーパス: cavernosum

1 tudied in vitro using isolated rabbit corpus cavernosum.

2 d the beneficial actions of RSV on aorta and cavernosum.

3 nt relaxation to acetylcholine for aorta and cavernosum.

4 hich causes cGMP to accumulate in the corpus cavernosum.

5 cle and endothelial subcluster in the corpus cavernosum.

6 was present in the endothelium of the corpus cavernosum.

7 antioxidant genes and proteins in the corpus cavernosum.

8 tric oxide is present in the diabetic corpus cavernosum.

9 r rosuvastatin (RSV) were examined on corpus cavernosum and aorta from streptozotocin-induced diabeti

10 osphate hydrolysis [corrected] in the corpus cavernosum and therefore increases the penile response t

11 O synthase is found in human clitoral corpus cavernosum and vagina, we hypothesized that human argina

12 ents underlie detumescent tone in the corpus cavernosum, and that modulation of this mechanism by the

13 Many tonic SMCs (vascular, airway and corpus cavernosum) are driven by a cytosolic Ca2+ oscillator th

14 A Rho-kinase inhibitor increases corpus cavernosum (CC) pressure in an in vivo rat model suggest

15 ernal pudendal artery (dIPA), and the corpus cavernosum (CC).

16 but not in rat primary corpus smooth muscle cavernosum cells; and (iv) in situ hybridization of glan

17 In corpus cavernosum defects in rabbits and pigs, implantation of

18 and this effect was reproduced in WT corpus cavernosum exposed to NOS inhibitors.

19 reactive oxygen species (ROS) in the corpus cavernosum have been implicated as a causative factor.

20 ion and a blood-gas analysis from the corpus cavernosum helps to distinguish between ischaemic and no

21 cally, we 3D printed a hydrogel-based corpus cavernosum incorporating a strain-limiting tunica albugi

22 In rabbit penile corpus cavernosum, intracellular cGMP was determined to be 18 +

23 The corpus cavernosum is the most important structure for penile er

24 dy provides an insight into the human corpus cavernosum microenvironment and a reference for potentia

25 g the eNOS gene (AdCMVeNOS) into the corpora cavernosum of the aged rat.

26 s, including the smooth muscle of the corpus cavernosum of the penis.

27 of heme oxygenase-1 increased in the corpus cavernosum of the PHZ group, but PDE5 protein expression

28 and 4-hydroxynonenal increased in the corpus cavernosum of the PHZ group, suggesting a state of oxida

29 ed blood flow into the penis, raising corpus cavernosum pressure to culminate in penile erection.

30 sm of Rho-kinase results in increased corpus cavernosum pressure, initiating the erectile response in

31 Increased corpus cavernosum relaxant responses to acetylcholine and EFS w

32 rical field stimulation (EFS)-induced corpus cavernosum relaxations in vitro were obtained.

33 Relaxation of arterial and corpus cavernosum smooth muscle (CCSM) is necessary to increase

34 membrane currents in isolated rabbit corpus cavernosum smooth muscle cells.

35 Therefore, we aimed to evaluate the corpus cavernosum smooth muscle relaxant function in a murine m

36 vascular hemolysis promotes increased corpus cavernosum smooth muscle relaxation associated with incr

37 bility to NO synthase in human penile corpus cavernosum smooth muscle, the inhibition of human argina

38 Corpus cavernosum strips were dissected free and placed in orga

39 oth muscle relaxation in human penile corpus cavernosum tissue that is required for erection.

40 t arginase is present in human penile corpus cavernosum tissue, and that the arginase inhibitor BEC c

41 functional role of BK channels in the corpus cavernosum utilizing a knock-out mouse lacking the Slo g

42 Relaxations of cavernosum were in the nondiabetic range following preve

43 noncholinergic nerve-mediated relaxations of cavernosum were reduced 25-33% by diabetes.

44 ures; and an implantable model of the corpus cavernosum, whose complex vascular network is critical f