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1 rrier associated with solvent relaxation and cavity formation.
2 aval veins combined with ectopic pericardial cavity formation.
3 es where metal coordination is necessary for cavity formation.
4  bacteria is sufficient to trigger localized cavity formation.
5 cal scenarios that could result in localized cavity formation.
6 uent collapse of both neurogenesis and nasal cavity formation.
7  spinal cord or brain injury often result in cavity formation.
8 GF receptor and is important in FGF2 central cavity formation.
9 giogenesis diminishes concurrent with cystic cavity formation.
10 solid caseous material and in the subsequent cavity formation.
11 ipal stress is important in the promotion of cavity formation.
12 on during occlusion, which is accompanied by cavity formation.
13 ely for the central hydrophobic phenomena of cavity formation and association of inert gas solutes.
14  post-implantation embryos triggers amniotic cavity formation and developmental progression.
15 radiation conditions, indicating significant cavity formation and growth.
16 solidation, pleural effusion, calcification, cavity formation and military nodules.
17                Hydrophilic surfaces suppress cavity formation and reduce the water-water correlation
18 seous tuberculous lesions and the subsequent cavity formation are probably the most dangerous process
19  are able to predict the free energy cost of cavity formation at the interface directly from the free
20 he balling and sparring effects, keyhole and cavity formation by attaining effective melt pool format
21 al and show simulation results demonstrating cavity formation by iron starvation.
22 f the origin of hydrophobicity, derived from cavity formations by Pratt and Pohorille and Madan and L
23 e carefully designed to evaluate the role of cavity formation/dispersive interaction on the chromatog
24   Hydraulic fracturing plays a major role in cavity formation during embryonic development, when pres
25  and Cl(-) in aqueous solutions increase the cavity formation energy cost and the polar interaction e
26 PRC2 promotes trophoblast-fate induction and cavity formation in human blastoids.
27 ical model of mushroom-like architecture and cavity formation in Pseudomonas aeruginosa biofilms.
28 vere WMD including rarefaction, necrosis and cavity formation in the corpus callosum, external and in
29 ated between 300 degrees C and 500 degrees C cavity formation in the crystals was observed most proba
30                                              Cavity formation in zonal occludens-1-depleted cells is
31 of CPEB or zonal occludens-1 impairs central cavity formation, indicating a loss of cell polarity.
32             Specifically, the probability of cavity formation is enhanced in the vicinity of hydropho
33  kcal/mol, the unfavorable thermodynamics of cavity formation may be the major factor in destabilizin
34  the epiblast and the resultant pro-amniotic cavity, formation of a bipolar embryonic sac, and specif
35  the epiblast and the resultant pro-amniotic cavity, formation of a bipolar embryonic sac, and specif
36              In contrast, the probability of cavity formation or the free energy of binding of hydrop
37 t relative to the energy of the mobile-phase cavity formation process.
38 d RH4 indicates that both steric overlap and cavity formation provide strong driving forces for oligo
39 ts "fatness." In solution, the extra work of cavity formation to accommodate a fluorocarbon, compared
40 t destabilizing, allowing the energy cost of cavity formation to be estimated.
41 halpy of solution and calculated enthalpy of cavity formation using the scaled particle theory.
42 ly understood machinery involved in epiblast cavity formation, we interrogated the proteomes of apica
43          Flesh breakdown, core breakdown and cavity formation were reduced at 3 degrees C.
44 ccessible surface area term, as an index for cavity formation, were correlated with the free energies
45                    There were differences in cavity formation with 34.20% in non-DCM, 9.09% DCM, and
46                    There were differences in cavity formation, with 34.20% in the non-DCM group, 9.09