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1 ence (G0) allows cycling cells to reversibly cease proliferation.
4 ype 1 (HTLV-1)-infected HeLa and SupT1 cells cease proliferation and become senescent immediately aft
5 tum (dpc), the stage in mice when germ cells cease proliferation and begin differentiation into prosp
6 cells in the external granule cell layer to cease proliferation and begin migration to the internal
8 essed at progressively higher levels as NPCs cease proliferation and differentiate into postmitotic n
10 e number of population doublings before they cease proliferation and enter a growth-arrest state term
11 sors only induce SPARC expression after they cease proliferation and migration, and transform into ma
16 keratinocytes increase intracellular Ca(2+), cease proliferation, and differentiate passively in resp
17 n the absence of nos-1 and nos-2, germ cells cease proliferation at the end of the second larval stag
19 understanding when and how these progenitors cease proliferation during development can have importan
20 RNA is initially detectable when osteoblasts cease proliferation, increasing during differentiation a
21 granulosa cells in p21(Cip1)-deficient mice ceased proliferation similarly to those in wild-type mic
22 tured RB1(-/-) calvarial osteoblasts fail to cease proliferation upon reaching confluence or followin
23 of reporter systems, we show that HeLa cells cease proliferation within one or two division cycles af
24 r transplantation, whereas adult hepatocytes ceased proliferation within the first month; 2) both the