ライフサイエンスコーパス: ceca

1 intestine and longer-term clearance from the ceca.

2 o showed a reduced ability to colonize chick ceca.

3 erial killing via an ion channel activity of CecA.

4 thway is preferentially expressed within the ceca.

5 nd fermentation is mostly in the jejunum and ceca.

6 pyrotag sequencing and compare that with the ceca.

7 diversity, and predicted function in chicken ceca.

8 ogical changes to the midgut and the hepatic ceca.

9 ceca, whereas herbivorous species have long ceca.

10 Avian ceca, a pair of blind sacs arising from the junction of

11 est as the ENCC wavefront passes through the ceca, a pair of pouches at the midgut-hindgut junction i

12 at the time of peak disease severity in the ceca and colons of all mice infected with a high-virulen

13 were increased approximately 2.5-fold in the ceca and colons of H. bilis-inoculated mice.

14 quantitatively detect H. hepaticus in mouse ceca and feces using the ABI Prism 7700 sequence detecti

15 gly expressed around ENCCs as they enter the ceca and hindgut.

16 Disruption of the hepatic ceca and midgut epithelial tissues implied digestive fun

17 display specific metabolomic profiles in the ceca and plasma during infection compared to mice with m

18 cytoplasm of epithelial cells of the gastric ceca and posterior stomach.

19 ow decreased commensal colonization of chick ceca and reduced colonization of BALB/cByJ mice compared

20 From ceca and water, potential ESBL E. coli isolates were onl

21 ical EF-G1 in bacteria harvested from murine ceca and, unlike EF-G1, specifically accumulates during

22 centration of the enzyme was observed in the ceca, and a 7.5-fold concentration occurred in the excre

23 biosis caused elongated small intestines and ceca, and B16-F10 melanoma and Lewis lung carcinoma prog

24 s to this trend, however, suggest that avian ceca are functionally complex and may have additional ro

25 dgut epithelial cells as well as the hepatic ceca are key target organs.

26 sults support a mechanism of action in which CecA binds to and covers the membrane surface, thereby c

27 eads to reduction in C. jeuni numbers in the ceca by nine weeks post infection.

28 ied throughout the midgut and within hepatic ceca cells, coinciding with tissue damages.

29 ese results reveal an essential role for the ceca during hindgut ENS formation and highlight an impor

30 des, and lipids were decreased in the turkey ceca early after BMD administration.

31 of the insect antibiotic peptide cecropin A (CecA) in the phospholipid bilayer membrane was determine

32 lly, ET3, which is strongly expressed in the ceca, inhibits the chemoattraction of NCC to glial-deriv

33 Removal of the ceca leads to hindgut aganglionosis, suggesting that the

34 -infected mice were colonized at high rates: ceca of 50/50 wild-type mice and 168/170 IL-10(-/-) mice

35 Helicobacter spp. were cultured from the ceca of 62 of 79 mice.

36 been isolated from the inflamed stomachs and ceca of adult Syrian hamsters.

37 portion of a Proteus mirabilis strain in the ceca of animals not pre-treated with antibiotics.

38 Salmonella more rapidly clear the ceca of birds administered the modified probiotic than o

39 oreover, miR-27a-5p was overexpressed in the ceca of C. difficile-infected mice, correlating with int

40 Ceca of C. jejuni 11168-infected mice were colonized at

41 ncentrations of Salmonella Gallinarum in the ceca of chicks treated with any form of phage were signi

42 ncentrations of Salmonella Gallinarum in the ceca of chicks treated with the mixture of unprotected a

43 C. albicans depleted simple sugars in the ceca of gnotobiotic mice but required oxygen to grow on

44 At 16 wpi, the ceca of H. bilis-infected Rag2(-/-) mice treated with co

45 Analysis of the gene expression in ceca of H. hepaticus infected mice revealed 25 up-regula

46 e gamma-H2AX was significantly higher in the ceca of H. saguini-infected gnotobiotic mice than in the

47 , but IFN-gamma expression was normal in the ceca of IL-23p19-deficient mice during serotype Typhimur

48 nalysis of C. jejuni localization within the ceca of infected mice determined that the primary differ

49 athology scores was observed in the ilea and ceca of mice infected with the ibeA mutant.

50 tion of gamma interferon was observed in the ceca of mice infected with the ibeA mutant.

51 While H2 levels were greatly reduced in ceca of mice treated with antibiotics, both the Deltahyc

52 s Bacteroidetes populations long term in the ceca of mice, but the presence of C. albicans during cef

53 ells against microbial Ags isolated from the ceca of normal animals was observed.

54 At 18 h postchallenge, the ceca of resistant C57BL/6 mice were histologically unrem

55 causes an acute inflammatory reaction in the ceca of streptomycin-pretreated mice that involves T-cel

56 causes an acute inflammatory reaction in the ceca of streptomycin-pretreated mice.

57 cing microbial richness and diversity in the ceca of stressed mice.

58 in the commensal E. coli harvested from the ceca of the stressed mice.

59 thetaiotaomicron was then harvested from the ceca of these hosts during the suckling period (postnata

60 Furthermore, histology of the ceca revealed that mice administered IL-12 antisera fail

61 tion of the beta-glucanase isolated from the ceca testified to its origin from the transgenic barley.

62 as little effect on bacterial numbers in the ceca, the main site of colonisation, where C. jejuni per

63 ortantly, increased fibrin deposition in the ceca was not associated with increased plasma fibrin whe

64 sures, with faunivorous species having short ceca, whereas herbivorous species have long ceca.

65 s ineffective in clearing C. jejuni from the ceca within the production lifetime of chickens, althoug