ライフサイエンスコーパス: cell attachment

1 tirely accounted for by the decrease in host cell attachment.

2 of effects on translocation from effects on cell attachment.

3 lity and mobility of PDL cells after initial cell attachment.

4 aracterized cs defect directly affected host cell attachment.

5 e, probably inhibiting infection by blocking cell attachment.

6 owed increased biofilm formation and cell-to-cell attachment.

7 nspecific protein, fibroblast, and bacterial cell attachment.

8 proved as good indicators of the rigidity of cell attachment.

9 recognize sialic acid as receptors for host cell attachment.

10 and normalized vessels with increased mural cell attachment.

11 esults in reduced virulence and impeded host cell attachment.

12 shows that these factors contribute to host cell attachment.

13 t embryos exhibit a failure in muscle-tendon cell attachment.

14 rin alpha6beta4, which often mediates stable cell attachment.

15 t-1 can be counteracted by integrin-mediated cell attachment.

16 nt and biofilm formation, thereby inhibiting cell attachment.

17 ar F-actin bundles, particularly at areas of cell attachment.

18 9 and an ELP (A99-ELP-R) show dose-dependent cell attachment.

19 ved in important cellular processes, such as cell attachment.

20 cytotoxicity while providing a substrate for cell attachment.

21 otein product, TGFBIp, mediates an effect on cell attachment.

22 ed that deleting the five TA systems reduced cell attachment.

23 s uniquely counteracted by integrin-mediated cell attachment.

24 tantiating their possible role in initiating cell attachment.

25 the Akt signaling pathway and enhanced PC-3 cell attachment.

26 s the hinged short tail fibers to facilitate cell attachment.

27 omain/receptor affinity and is necessary for cell attachment.

28 n the surface of EBOV mediates the EBOV-host cell attachment.

29 virus infectivity, presumably blocking host-cell attachment.

30 moted increased cell survival after 1 day of cell attachment.

31 a1-mediated adhesion, whereas Pyk2 opposes T-cell attachment.

32 ated increased cell survival after 3 days of cell attachment.

33 ults in a light-independent increase in cell-cell attachment, a response that requires both the conse

34 ns type and caused a significant increase in cell attachment across all groups.

35 t, whereas mutant rec-M2 and rec-M3 retained cell attachment activity but did not promote cell spread

36 scaffold and characterize the biological and cell attachment activity using human dermal fibroblasts

37 principle be the result of weak, incomplete cell attachment, adhesion is usually too strong for this

38 7 were intact and free-floating without host cell attachment, although the numbers of peritoneal CD20

39 n anti-HIV-1 mechanism that suppresses virus-cell attachment and a previously unappreciated process o

40 doses of the drug we observed inhibition of cell attachment and a reduction of focal adhesions.

41 cells, and that EPS inhibition reduces both cell attachment and antimicrobial tolerance.

42 ecombinant Scl-1 protein suggested a role in cell attachment and binding and inhibition of serum prot

43 ly overcomes WIN drug-mediated inhibition of cell attachment and capsid breathing.

44 acts with two similar proteins that regulate cell attachment and cell migration called paxillin (PXN)

45 bility of three titanium surfaces to promote cell attachment and cell motility of cells relevant to p

46 Analysis of cell attachment and cell proliferation revealed signific

47 Cell attachment and cell proliferation were quantified u

48 e seeded on root surfaces and quantified for cell attachment and cell proliferation.

49 Initial cellular activities such as cell attachment and cell spreading were concentration-de

50 d FAK tyrosine phosphorylation increase upon cell attachment and decrease upon detachment from extrac

51 ing RNAs for RhoD and WHAMM showed increased cell attachment and decreased cell migration.

52 el such as cell migration or the dynamics of cell attachment and detachment are also addressable by o

53 Ins(3,4,5)P(3) polarity to facilitate proper cell attachment and detachment during chemotaxis.

54 e bacterial cell that likely facilitate host cell attachment and DNA transfer.

55 tic polymer fibers provide guidance cues for cell attachment and elongation.

56 the utilization of conjugative plasmids for cell attachment and entry comprises independent evolutio

57 t heparan sulfate plays an important role in cell attachment and entry, although to various degrees,

58 Initial steps of transmission, including cell attachment and entry, likely occur in the presence

59 ), uses the viral spike (S) protein for host cell attachment and entry.

60 le, poorly studied coat responsible for host cell attachment and entry.

61 that Vp1 contains the major determinants for cell attachment and entry.

62 tein (RBP), SosV-RBP, which facilitates host-cell attachment and entry.

63 d/or host cell tropism at the level of virus-cell attachment and entry.

64 bacterial surface polysaccharides, enhancing cell attachment and environmental stability, potentially

65 FAs are integrin-associated sites of cell attachment and establish linkages to the cellular a

66 te cancer cells and, concordantly, increased cell attachment and focal adhesion formation.

67 ) surface glycoprotein (GP1,2) mediates host cell attachment and fusion and is the primary target for

68 th cells, suggesting a detrimental impact on cell attachment and growth when conditioning by this age

69 ipeptide, RGD, and experiments comparing the cell attachment and haptotactic migration-enhancing prop

70 chain, A99 (AGTFALRGDNPQG), promotes strong cell attachment and has demonstrated utility in cell cul

71 ne resulted in a remarkable reduction of HCV cell attachment and infection.

72 nce for a critical function of GAGs in CHIKV cell attachment and infection.IMPORTANCE Alphavirus infe

73 USF81 restored host cell attachment and invasion activities.

74 Here we show that epithelial cell attachment and invasion by T. forsythia are depende

75 es that were resistant to inhibition of host cell attachment and invasion by the compound.

76 F. nucleatum 12230 US1 was defective in host cell attachment and invasion in vitro, but it also exhib

77 d in increased anchorage-independent growth, cell attachment and invasion through Matrigel in vitro,

78 es, but not purified fibrin, supported tumor cell attachment and invasion.

79 portant roles in virulence by promoting host cell attachment and invasion.

80 es anchorage-independence by driving cell-to-cell attachment and matrix-independent integrin beta4/SH

81 protein (GP), responsible for mediating host-cell attachment and membrane fusion, contains a heavily

82 of the virus, NiV-G and NiV-F, mediate host-cell attachment and membrane fusion, respectively, and a

83 bdoviruses (e.g., rabies virus) mediate both cell attachment and membrane fusion.

84 utinin (HA) glycoprotein spike mediates host cell attachment and membrane fusion.

85 Regulation of epithelial cell attachment and migration are essential for normal d

86 Cell attachment and migration were examined for four cel

87 tually linked through mechanisms that govern cell attachment and migration.

88 form of DMP1 had greatly reduced ability for cell attachment and migration.

89 ort that depletion of GRASP proteins reduces cell attachment and migration.

90 s two weeks post-stimulation shows increased cell attachment and mineralization around microcracks an

91 as a scaffolding protein with activities in cell attachment and mitotic control, suggesting SRC and

92 Cell attachment and penetration rates, as well as the re

93 ysis showed that GelMA supported hDPSC/HUVEC cell attachment and proliferation and also provided atta

94 ior, WN-1 displays PMA-like behavior in U937 cell attachment and proliferation assays, as well as in

95 riments with PDL cells demonstrated improved cell attachment and proliferation in all samples coated

96 Cell attachment and proliferation were quantified using

97 sponses to cell adhesion--enhanced mammalian cell attachment and proliferation, and enhanced resistan

98 rbed to the culture surface, promoted H1 hES cell attachment and proliferation, as well as maintained

99 spectra with the motional characteristics of cell attachment and proliferation.

100 to test their ability to influence human PDL cell attachment and proliferation.

101 l-adhesive capability, effectively promoting cell attachment and proliferation.

102 NFAT-DsRed rat basophil leukemia cell attachment and retention during washing steps was a

103 en has the potential to improve both initial cell attachment and retention of endothelial cells on va

104 BSP is multifunctional, affecting cell attachment and signaling through an RGD integrin-bi

105 l surface and a corresponding enhancement of cell attachment and spreading on fibronectin-coated subs

106 e ability of syndecan-4 molecules to support cell attachment and spreading without the direct extrace

107 e, as expression of TNCEGFL delayed melanoma cell attachment and spreading.

108 d PrKX proteins are required for PMA-induced cell attachment and spreading.

109 are essential effectors of integrin-mediated cell attachment and spreading.

110 PS-containing liposomes blocked HCV cell attachment and subsequent HCV infection.

111 kout in Huh-7.5 cells remarkably lowered HCV cell attachment and subsequent HCV infection.

112 ts how they mediate in a unique fashion both cell attachment and the initiation of membrane fusion du

113 s were activated with fibronectin to mediate cell attachment and the silicon oxide background was pas

114 tracellular matrix environment that promotes cell attachment and tissue-specific differentiation lead

115 t to those of all other reported AAVs, AAV12 cell attachment and transduction do not require cell sur

116 These proteins facilitate host cell attachment and viral infectivity and are the target

117 show that epithelial-derived FN potentiates cell attachment and wound healing through epithelial-mat

118 FN enhances cell attachment and wound healing, which is dependent on

119 Further, FN potentiated Caco2-BBE cell attachment and wound healing, which was inhibited b

120 important virulence factors involved in host cell attachment and/or biofilm formation, key steps in e

121 erficial epithelial cells with disruption of cell attachments and cell shedding.

122 Human fibroblast cells attachment and proliferation on FGF-immobilized PX

123 acidic phosphoprotein with collagen-binding, cell attachment, and hydroxyapatite-nucleating propertie

124 T is involved directly in membrane dynamics, cell attachment, and motility.

125 ar activities, including transcription, cell-cell attachment, and regulation of the cytoskeleton.

126 tructural surface features can guide cardiac cell attachment, and the subsequent syncytial behavior c

127 Focal adhesions (FAs) play a key role in cell attachment, and their timely disassembly is require

128 ectly or indirectly required for maintaining cell attachment, and this may represent a common but not

129 PGER sequence prevented integrin binding and cell attachment as predicted from molecular dynamics stu

130 els of VE-cadherin expression, and increased cell attachment, as evidenced by Evans blue dye injectio

131 LDL) was responsible for the blockade of HCV cell attachment, as VLDL-depleted mouse serum lost HCV-i

132 gues evaluated in the U937 proliferation and cell attachment assays displayed phorbol ester-like and/

133 I was determined with the use of fluid-phase cell attachment assays in HSFs, human foreskin fibroblas

134 ior activity in opsonophagocytic killing and cell attachment assays, and confer significant protectio

135 oproteins, thereby facilitating Sertoli-germ cell attachment at a particular stage of spermatogenesis

136 Defects in spatial regulation of the cell attachment at the leading edge in rapGAP1- (null) c

137 Following the exposure, real-time cell attachment behavior was monitored for at least 120

138 he cell and the surface that dictate overall cell attachment behavior.

139 lence factors used by P. aeruginosa for host cell attachment, biofilm formation, and twitching motili

140 he biofilm lifecycle occurs in three stages: cell attachment, biofilm maturation, and biofilm dispers

141 on of Bit1 is inhibited by integrin-mediated cell attachment but not by many other antiapoptotic trea

142 ta1 or alpha2beta1 adhesions mediate initial cell attachment but over time are switched to alpha5beta

143 es but not DeltaIDR2 virus displayed reduced cell attachment, but altering sigma1 length or flexibili

144 ellular matrix components because preventing cell attachment by forced suspension culture markedly re

145 harides, which enhances virion stability and cell attachment by increasing binding to the viral recep

146 Here, we show that metastatic tumor cell attachment causes the induction of the endothelial

147 WCNT-induced cellular changes in relation to cell attachment, cell-cell interactions and cell viabili

148 otein interactions in cell-cell junction and cell attachment complexes and on interactions with cytos

149 ng molecules through which integrin-mediated cell attachment controls Bit1 activity and anoikis.

150 lls that exhibit growth factor-independence, cell attachment defects, and a more invasive fibroblasti

151 teins have been implicated in mediating cell-cell attachment during fusion, but their binding partner

152 units), which is solely responsible for host cell attachment, endosomal entry and membrane fusion.

153 ing that the epitopes are critical for viral cell attachment/entry.

154 ated for many years, is indeed a major serum cell attachment factor particularly for tumor cells.

155 HBGAs function as cell attachment factors by binding to a surface-exposed

156 ls, and are recognized as susceptibility and cell attachment factors for gastric pathogens like Helic

157 blood group antigens (HBGAs), which are also cell attachment factors for this virus.

158 n of a cohesive passive layer, after initial cell attachment, followed by the formation of a metal co

159 ose that while CP-delta likely mediates host cell attachment for all three nematode viruses, addition

160 ndicated that delta plays important roles in cell attachment for this group of nematode viruses.

161 several critical virus functions, including cell attachment, host range susceptibility, and virulenc

162 ty modulates the efficiency of reovirus host cell attachment.IMPORTANCE Nonenveloped virus entry is a

163 otein that has been demonstrated to regulate cell attachment in a variety of cell types.

164 Therefore, identifying factors regulating cell attachment in the abdominal cavity is critical to t

165 nce of blue light dramatically enhances cell-cell attachment in the lovK-lovR overexpression backgrou

166 tle out-of-plane microtopographic cues alter cell attachment, increase biomechanical stresses, and in

167 Silencing of DLC2 in human ECs has reduced cell attachment, increased migration, and tube formation

168 ncentration-dependent inhibition of Jurkat T-cell attachment, inhibition of lymphocyte activation, an

169 gy of the virus with regard to receptor use, cell attachment, internalization, and intracellular traf

170 ens, filamentous growth is critical for host-cell attachment, invasion into tissues, and virulence.

171 otein important for T. gondii motility, host cell attachment, invasion, and egress.

172 ed disease in our model, perhaps by reducing cell attachment/invasion and dampening inflammation by m

173 However, significantly improved cell attachment is observed to silk-fibronectin alloy fi

174 hydrogels however hydrogel opacity and poor cell attachment limit their usefulness in downstream app

175 erobacteriaceae, has evolved a second target cell attachment mechanism.

176 PPy-chitosan did not reduce cell attachment, metabolism, or proliferation in vitro.

177 intricate protein complexes that facilitate cell attachment, migration, and cellular communication.

178 0 reference cells were analyzed by measuring cell attachment, migration, and chemotaxis in the presen

179 This peptide promoted cell attachment, migration, differentiation and minerali

180 cytoskeleton and mediate signals modulating cell attachment, migration, proliferation, differentiati

181 cate that BSG may act as a germ cell-Sertoli cell attachment molecule.

182 mmunoglobulin-like fold, frequently found in cell attachment molecules.

183 that play important roles in motility, host cell attachment, moving junction formation, and invasion

184 viruses interacts with Sia and whether their cell attachment necessarily involves sialoglycans.

185 Cell attachment of all three MB types was significantly

186 demonstrated that apolipoprotein E mediates cell attachment of hepatitis C virus (HCV) through inter

187 whereas rTGFBIp did not significantly affect cell attachment of HFFs (P = 0.50) or HCFs (P = 0.24) to

188 When produced, NanI can contribute to host cell attachment of human intestinal disease strains, sin

189 HBGA, providing a novel paradigm for initial cell attachment of human rotavirus.

190 As with many other viruses, the initial cell attachment of rotaviruses, which are the major caus

191 he Mam7 protein, which is implicated in host cell attachment of V. cholerae, associated normally with

192 riation of glycan recognition during initial cell attachment of viruses is a critical determinant of

193 In simple epithelial cells, attachment of microtubule-organizing centers (MTO

194 echanisms and their roles in modulating host cell attachment, oligomer assembly, and membrane perfora

195 aled significantly higher osteoblast and PDL cell attachment on EMD-coated surfaces when compared wit

196 Importantly, microRNA-7 decreased Caco2-BBE cell attachment on laminin-1, and CD98 overexpression re

197 sponses to laminin-332, while leaving stable cell attachment on laminin-332 intact.

198 d for stable, alpha6beta4 integrin-dependent cell attachment on laminin-332.

199 nt functional role for Pf12 in parasite-host cell attachment or invasion.

200 reduced PDL cell mingling, without altering cell attachment or motility.

201 uch as surface chemistry and properties like cell attachment or protein adsorption-in order to identi

202 hat this inhibition was not owing to reduced cell attachment or spreading.

203 To define sites for single cell attachment, PEG hydrogel microwells (20 microm diam

204 small oscillatory motions within the primary cell attachment plane, rather than perpendicular to it,

205 n actin-binding protein that participates in cell attachment, plays important additional roles in sig

206 extract (CSE) at different times during the cell attachment process.

207 ve/Dead cytotoxic assay and displayed higher cell attachment, proliferation and mineralization than t

208 de effects on in vitro measures of fibrosis: cell attachment, proliferation and viability, and ECM de

209 gest that 1) EMD enhances osteoblast and PDL cell attachment, proliferation, and differentiation on N

210 g inhibitory effects of JSM6427 on human RPE cell attachment, proliferation, and migration is probabl

211 sEphB4 inhibits PDGF-induced RPE cell attachment, proliferation, and migration.

212 helium (RPE) and its ability to modulate RPE cell attachment, proliferation, migration, and F-actin c

213 stratum compliance and/or Lat-B treatment on cell attachment, proliferation, surface area, aspect rat

214 The cell attachment promoted by highly phosphorylated OPN co

215 we present new evidence indicating that the cell attachment properties of sigma1 are influenced by t

216 or, adhesion, and antimicrobial and specific cell-attachment properties.

217 ng the central region of mu1 in altering the cell attachment property of reovirus.

218 on, but rather with depressed expression the cell attachment protein FAK, accompanied by increased se

219 that requires coordinated activities of host cell attachment, protein secretion, and motility by the

220 ons of herpes simplex virus with its initial cell attachment receptor induce a rapid and highly effic

221 that expression of the Epstein-Barr virus B-cell attachment receptor, CD21, in B cells that lack thi

222 ein receptor(s) required for AAV entry after cell attachment remains unknown.

223 adaptable drop-plating method for selective cell attachment, removal of myelin debris, and expansion

224 Although T99K and WT poliovirus cell attachment, replication, and pathogenesis in mice a

225 Analysis of cell attachment revealed no significant differences amon

226 o different substrates were determined using cell attachment screening kits.

227 In this work, dual electrochemical pH and cell-attachment sensor arrays were developed for the rea

228 Further supporting a role for NanI in host cell attachment, sialidase inhibitors reduced F4969 adhe

229 at HEF1 activity impacts division as well as cell attachment signaling events.

230 pecies footprint analyses predicted a unique cell attachment site for HRV-Cs.

231 signals are recorded and analyzed (i) during cell attachment, spreading and differentiation of initia

232 Cell attachment studies demonstrate that alpha3beta1-med

233 These modified Scl2 proteins also acted as cell attachment substrates for fibroblast, endothelial,

234 support cell proliferation and provide good cell attachment suggesting them as potentially good cand

235 s retain a uniform mass sensitivity over the cell attachment surface.

236 MMP20 would result in tight ameloblast cell-cell attachments that may cause maturation-stage enamel

237 o-glycolic) acid (PLGA) particles to enhance cell attachment, the attachment procedure to avoid clump

238 ins of these proteins physically block virus-cell attachment, the inhibitory effect of PSGL-1 require

239 cal adhesion-associated protein that reports cell attachment through a RhoA-dependent mechanosensory

240 A role for talin in cell-cell attachment through cadherin has never been demonstr

241 nt on high-titer viremia; however, efficient cell attachment through HS binding can increase virulenc

242 Integrins are receptors that mediate cell attachment through multivalent binding to peptide s

243 hanotransductive response for integrin-based cell attachments through our elastomeric membrane-based

244 HPV) infection involves multiple steps, from cell attachment, through endocytic trafficking towards t

245 ve an important role in bacterial epithelial cell attachment, through ILK-bacterial OspE binding.

246 , PDL cell survival increased with increased cell attachment time to plastic.

247 gene, lovR, results in severe attenuation of cell attachment to a glass surface under laminar flow, w

248 ed by p190RhoGAP following integrin-mediated cell attachment to allow sampling of new adhesive enviro

249 tin stress fibers, is inhibited upon initial cell attachment to allow sampling of the new adhesive en

250 Mesangial cell attachment to collagen I led to increased Hic-5 exp

251 c-5 expression increases following mesangial cell attachment to collagen I, associated with increased

252 induction by tumor necrosis factor-alpha or cell attachment to collagen IV.

253 The effect of rTGFBIp (50 microg/mL) on cell attachment to collagen type I was determined with t

254 cells at the vascular endothelium, melanoma cell attachment to endothelial cells was significantly d

255 ivated and localized to focal adhesions upon cell attachment to extracellular matrix.

256 the anoikis function of Bit1, we found that cell attachment to fibronectin inhibits PKD activity.

257 a1 antagonist, significantly inhibited hfRPE cell attachment to fibronectin, but not laminin, or coll

258 c of alpha5-integrin to the cell surface and cell attachment to fibronectin.

259 Moreover, C3G promoted colon cancer cell attachment to fibronectin.

260 LS174T cell attachment to HUVEC was entirely E-selectin-depende

261 ell as preventing inappropriate inflammatory cell attachment to LECs, D6 is specifically involved in

262 A dose-dependent increase in oTr cell attachment to LGALS15 was found that could be inhib

263 , whereas multiple integrins are involved in cell attachment to Matrigel.

264 function of polar development is to maximize cell attachment to surfaces and to improve nutrient upta

265 complexes formed at the cytoplasmic side of cell attachment to the ECM and is activated after force

266 The first step is tumor cell attachment to the ECM.

267 se results suggest the operation of specific cell attachment to the electropolymerized films via the

268 Cell attachment to the extracellular matrix (ECM) requir

269 dimeric transmembrane receptors that mediate cell attachment to the extracellular matrix and are crit

270 ently controls integrin-mediated endothelial cell attachment to the extracellular matrix and migratio

271 perform the critical function of signalling cell attachment to the extracellular matrix or to other

272 Focal adhesions are specialized sites of cell attachment to the extracellular matrix where integr

273 Involvement in cell attachment to the extracellular matrix, motility, a

274 ll death by wild-type Bit1 following loss of cell attachment to the extracellular matrix.

275 cellent outgrowth of CNS neurons in vitro by cell attachment to the high density of arginine-glycine-

276 collection of the cells without the need for cell attachment to the microraft surface.

277 as observed on the cell viability during the cell attachment to the surface of immune-reactive biochi

278 tly, we demonstrated that bile salts enhance cell attachment to the target cell and increase the intr

279 d trypsinization revealed firm protein-based cell attachment to the underlying extracellular matrix f

280 difference in the degree of lens epithelial cell attachment to the various types of intraocular lens

281 gy is induced upon loss of integrin-mediated cell attachments to the surrounding extracellular matrix

282 Endothelial cell attachment upon treatment with foam was quantified

283 and strains of CPV differed in the levels of cell attachment, uptake, and infection in canine and fel

284 erythroblasts and macrophages, mediates cell-cell attachments via homophilic binding.

285 We observed cell attachment, viability, and microstructures by elect

286 from purified virions had no effect on virus-cell attachment, virus-cell fusion, particle infectivity

287 Fractionation of human serum revealed that cell attachment was confined to the fractions that had f

288 Cell attachment was more prominent in the acrylic lenses

289 Cell attachment was significantly inhibited by function-

290 sed nuclear accumulation of Twist1 following cell attachment was suppressed by treatment with an inhi

291 itions for coating glass plates and time for cell attachment were established.

292 Additionally, HCV infection and cell attachment were inhibited by PS but not by phosphat

293 ponse regulators, and genes for invasion and cell attachment were prominent among the differentially-

294 LADRAD decreased its ability to promote oTr cell attachment, whereas mutation of the CRD had little

295 n remarkable reductions of HCV infection and cell attachment, whereas SDC-3 and SDC-4 knockouts did n

296 to use virally encoded envelope proteins for cell attachment, which is the very first step of virus i

297 A549-luc cells treated before cell attachment with a single dose of GRN163L only weakl

298 s inhibited by blocking PVRL4-driven cell-to-cell attachment with monoclonal antibodies, demonstratin

299 balance, we next compared the time course of cell attachment with the washed films and demonstrated t

300 es demonstrated that mouse serum blocked HCV cell attachment without significant effect on HCV replic