ライフサイエンスコーパス: cell cycle

1 n single fission yeast cells during a normal cell cycle.

2 oblasts, and it is maintained throughout the cell cycle.

3 may facilitate hepatocyte re-entry into the cell cycle.

4 ll lines, induced apoptosis and modified the cell cycle.

5 percentage of GSCs in M- and S-phase of the cell cycle.

6 particularly for periodic processes such as cell cycle.

7 maturation of rRNA and the regulation of the cell cycle.

8 tosine methylation dynamics during the plant cell cycle.

9 n completion of the malaria parasite asexual cell cycle.

10 ntial for mitotic exit and completion of the cell cycle.

11 zer operating largely in the G1 phase of the cell cycle.

12 opment by influencing the progression of the cell cycle.

13 with inflammation, extracellular matrix and cell cycle.

14 ith transcription, DNA damage repair and the cell cycle.

15 Cells confront DNA damage in every cell cycle.

16 steps of ciliogenesis in G1/G0 phase of the cell cycle.

17 lar asymmetry and differentiation during the cell cycle.

18 ely to regulate cardiomyocyte maturation and cell cycle.

19 ereas mCHG remains asymmetric throughout the cell cycle.

20 ropagation of DNA methylation throughout the cell cycle.

21 ired for Muller glia to progress through the cell cycle.

22 fine cell identity must be restarted in each cell cycle(2-5) but how this is accomplished is poorly u

23 different times as cells proceed through the cell cycle [4, 5].

24 In sum, we show how fluctuations of cell cycles across lineage trees help in understanding t

25 alate induced parallel changes in phenotype, cell cycle activity, and gene expression.

26 IL2RA promoted proliferation and cell-cycle activity and inhibited apoptosis in human AML

27 ing pathways that regulate the cardiomyocyte cell cycle and advances in stem cell technology, strateg

28 , Schizosaccharomyces pombe exit the mitotic cell cycle and become irreversibly committed to the comp

29 t cell populations, the custom design of the cell cycle and cleavage properties, the protein number p

30 e associated with plasticity/rigidity of the cell cycle and correlated with sensitivity to CDK4/6 inh

31 el role of E2F2 in cancer progression beyond cell cycle and could impact patient treatment.

32 We focus on the cell cycle and describe its dependence on pre-mRNA splic

33 ation to centrosomes is regulated during the cell cycle and developmentally.

34 inase 7 (CDK7) is a central regulator of the cell cycle and gene transcription.

35 We investigate the cell cycle and identify the nutrition supply as the ener

36 alysis revealed downregulation of matrisome, cell cycle and immune related gene sets in Lcn2(-/-) mic

37 Cell size varies during the cell cycle and in response to external stimuli.

38 They are known to participate in cell cycle and in signaling pathways involved in neurode

39 air pathways, during different stages of the cell cycle and in varied chromatin environments.

40 amage, occurs primarily in S/G2 phase of the cell cycle and is associated with replication forks.

41 prevalence of two subclones associated with cell cycle and primary immunodeficiency pathways identif

42 These genes are frequently enriched for cell cycle and proliferation pathways, indicating a role

43 on burden and multiple mutations in genes in cell cycle and receptor tyrosine signaling pathways.

44 tive than PD-1 blockade alone in enhancing T cell cycling and differentiation, expanding effector-mem

45 trains to construct a comprehensive atlas of cell-cycle and asexual development, revealing hidden sta

46 indings show that DZIP3 is a novel driver of cell-cycle and cancer progression via its control of Cyc

47 m in its stabilization of Cyclin D1 to drive cell-cycle and cancer progression.

48 ression or depletion of USP22, enrichment of cell-cycle and DNA repair pathways was observed in the U

49 nt|>= 0.9) with 91 up-regulated genes in the cell-cycle and Rho-GTPase pathways.

50 d with and counteracted EZH2 and SOX2 during cell-cycle and self-renewal regulation to restrain tumor

51 se to therapy, with a reciprocal decrease in cell-cycle and WNT signaling pathways in responding biop

52 is, cell metabolism, translation initiation, cell cycle, and antigen presentation.

53 a role in the progression of S-phase of the cell cycle, and both these functions require CW and cata

54 silon localize to centrosomes throughout the cell cycle, and in interphase cells to the nucleus, and

55 ivo assessments of urothelial proliferation, cell cycle, and ploidy status.

56 ion of gene expression, DNA replication, the cell cycle, and the DNA damage response.

57 receptor, integrin-beta1, to inhibit tubular cell cycle arrest and apoptosis in in vivo and in vitro

58 s a stress response that elicits a permanent cell cycle arrest and triggers profound phenotypic chang

59 H-SY5Y cells with PMA and it correlated with cell cycle arrest at G2/M, upregulation of tumor suppres

60 e found that overexpression of ABHD5 induces cell cycle arrest at the G1 phase and causes growth reta

61 s induced an antiproliferative response with cell cycle arrest at the G2/M phase.

62 to ERS and an attenuation of ERS-associated cell cycle arrest caused by WSPM and multiple prototypic

63 n of IL18RAP inhibited cell proliferation by cell cycle arrest in NKTCL cells.

64 in (IDP), regulates cell division by causing cell cycle arrest when bound in ternary complex with cyc

65 Physiologically, DJ34 induced apoptosis, cell cycle arrest, and cell differentiation.

66 At low passage NPCs (P1 to P3), we observed cell cycle arrest, apoptosis, progressive change to a gl

67 keletal-microtubule organization, leading to cell cycle arrest, genotoxic stress, and innate immunity

68 independent growth of ILC cell lines through cell cycle arrest.

69 which lead to premature differentiation and cell cycle arrest.

70 ces HIV-1 gene expression and induces (G2/M) cell cycle arrest.

71 gered by transient Snt1 phosphorylation upon cell cycle arrest.

72 provided resistance to dexamethasone-induced cell-cycle arrest and apoptosis, illuminating a new poss

73 proliferation, clonogenicity, induced G(2)/M cell-cycle arrest and caspase-mediated-apoptosis of CRC

74 so mouse and human cells from stress-induced cell-cycle arrest and cell death in a polymer length-dep

75 atal development when cardiomyocytes undergo cell-cycle arrest and polyploidization.

76 is a central tumor suppressor, which induces cell-cycle arrest and senescence.

77 roliferation by reducing checkpoint--induced cell-cycle arrest during interphase.

78 ture of antigen processing and presentation, cell-cycle arrest, and execution phase of apoptosis on t

79 compounds induce significant PEL apoptosis, cell-cycle arrest, and intracellular ceramide production

80 cells in different cell-cycle phases and in cell-cycle arrest.

81 in-3A, which otherwise predominantly induced cell-cycle arrest.

82 A may be targeted for demethylation when the cell cycle arrests.

83 were found in all three procedures included cell cycle associated genes (E2F1, CCND1, FOXM1, TP53, a

84 tumor growth and decreased the expression of cell cycle-associated genes, indicating that tumor growt

85 a spatial and lineage-specific separation in cell-cycling behaviour.

86 epends on progression through S phase of the cell cycle, but the molecular nature of this requirement

87 r cohesin, organize chromatin throughout the cell cycle by a process known as loop extrusion.

88 trols unidirectional progression through the cell cycle by marking key cell cycle proteins for protea

89 excessive cellular enlargement during slowed cell cycles by down-regulating translation capacity.

90 During late M-phase of the cell-cycle, Cdc6 binds to ORC and the ORC-Cdc6 complex l

91 but transcriptional CDKs (CDK7 and CDK9) and cell cycle CDKs (CDK4 and CDK6) as well.

92 t through eight cancer hallmarks: apoptosis, cell cycle, cell death, cell motility, DNA repair, immun

93 ults demonstrate that targeted inhibition of cell cycle checkpoint activation following ionizing radi

94 increased micronuclei formation utilizing a cell cycle checkpoint inhibitor to drive cell cycle prog

95 ough ataxia-telangiectasia mutated (ATM) and cell cycle checkpoint kinase 2 (CHK2), a DNA damage resp

96 n human cells but also adversely affects the cell cycle checkpoint, resulting in profound chromosomal

97 cer cells to override the p53-dependent G2/M cell-cycle checkpoint.

98 1 kinase inhibitor AZD1775 (WEE1i) overrides cell cycle checkpoints and is being studied in HNSCC reg

99 LMP2A affected apoptosis and cell-cycle checkpoints by dysregulating the expression o

100 ll as innate immunity, chronic inflammation, cell cycle, circadian rhythm, and olfactory functions.

101 DK2), Cyclins D1 and D3, indicating that key cell cycle components mediate cell viability reduction.

102 age signalling over the course of the mother cell cycle constitutes the predominant control mechanism

103 ive genes predicted a cell's position on the cell cycle continuum to within 14% of the entire cycle a

104 ve cell envelope stabilization that includes cell cycle control and an expanded role for Ldts in cova

105 us studies demonstrated a role for SETD1A in cell cycle control and differentiation.

106 oreover, we use cell cycle tags to reinstall cell cycle control to a deregulated version of Yen1, sho

107 maging tool to facilitate in vivo studies of cell-cycle control in a wide-range of developmental cont

108 of cyclin-dependent kinases as core to these cell cycle controls.

109 ver, mtDNA replication is independent of the cell cycle creating a significant concern that toxicants

110 pointing to a feedback mechanism between the cell cycle, cytoskeleton, and ROP.

111 odulation of Aurora B in interphase leads to cell cycle defects often linked to aberrant chromosomal

112 sion through the cell cycle, suggesting that cell cycle delay is sufficient to disrupt Ace2 asymmetry

113 by reductions in activated AKT signaling, G1 cell cycle delay, and decreases in expression of mesench

114 Furthermore, G1 cell cycle delays are sufficient to increase chemotherap

115 cells show that the Hst2-Bmh1 interaction is cell cycle dependent, peaking in the M phase.

116 The centering mechanism is cell-cycle dependent and weakens considerably during int

117 a composite of numerous proteins subject to cell cycle-dependent oscillations in levels and organiza

118 Cell cycle-dependent redox changes can mediate transient

119 We demonstrate that CYP24A1 expression is cell cycle-dependent; it was higher in the G(2)-M phase

120 To determine whether cell-cycle-dependent gene regulation occurs in mycobacte

121 in vitro VDR-knockdown impaired myogenesis (cell cycling, differentiation and myotube formation).

122 Specifically, we find that genes related to cell cycle, DNA repair, cell death, the IGF1 pathway, an

123 of cytokinesis," "G1/S transition of mitotic cell cycle," "DNA recombination," and "telomere maintena

124 irectly linking replication origin assembly, cell cycle duration and embryo development in vertebrate

125 ong-range intra-generational correlations in cell-cycle duration, up to second cousins, seem paradoxi

126 activate neural differentiation and inhibit cell cycle during the transition, whereas epilepsy genes

127 se promoter features appear to correspond to cell-cycle-dynamic rather than tissue/cell-lineage origi

128 tified key regulatory enzymes that drive the cell cycle, elucidated structural components that underl

129 from which the different vital phases of the cell cycle emerge.

130 on has the potential to induce cardiomyocyte cell cycle entry and potentially cardiac tissue regenera

131 acking MYC-MIZ1 complexes displayed impaired cell cycle entry of positively selected GC B cells and r

132 th upregulates TNFR2 expression and promotes cell cycle entry of tubular epithelial cells.

133 Mammalian cells typically start the cell-cycle entry program by activating cyclin-dependent

134 mitotic chemotherapies as the former prevent cell-cycle entry, thus interfering with S-phase- or mito

135 tivators to inhibitors with size, triggering cell-cycle entry.

136 rmines the timing of Sic1 degradation, a key cell cycle event.

137 ion of Yap, Yap5SA, is sufficient to prevent cell cycle exit and to prolong sensory tissue growth.

138 Many cells only build a primary cilium upon cell cycle exit, in G0.

139 ion, while contact removal prevents CMs from cell cycle exit.

140 ntify immature cardiomyocytes that enter the cell cycle following injury and disappear as the heart l

141 entral energy metabolism regulates bacterial cell cycle functions is not well understood.

142 ated pathway, with 15 of 241 DEGs related to cell cycle functions.

143 rrogated the accessibility of chromatin in a cell cycle (G1, S, and G2/M)-specific manner using mamma

144 This cell cycle gating provides a temporal compartmentalizati

145 this manner, SCIRT induced transcription at cell-cycle gene promoters by recruiting FOXM1 through EZ

146 tor homeobox Protein (ADNP) and located near cell-cycle genes recruits TFIIIC, which alters their chr

147 th distant CTCF sites near promoter of other cell-cycle genes, which also become hyperacetylated at H

148 ed the molecular mechanisms and logic of the cell cycle, identified key regulatory enzymes that drive

149 the organ of Corti-progenitor cells exit the cell cycle in a coordinated wave between E12.5 and E14.5

150 mproved therapeutic strategies targeting the cell cycle in cancer.

151 zole 2l 1) caused significant effects on the cell cycle in PC3 cells, with the vast majority of treat

152 d end joining, which occurs during the first cell cycle in the zygote, leading to embryos with non-mo

153 Centrosomes themselves duplicate once per cell cycle, in a process that is controlled by the serin

154 The cell cycle, in particular, is thought to be a key driver

155 riptional repressor of key regulators of the cell cycle, in turn influencing contact inhibition and t

156 However, possible cell cycle-independent mechanisms behind its oncogenicit

157 atin is dynamically regulated throughout the cell cycle, indicating that CENP-A stability is also con

158 lar mechanism for how surface sensing drives cell-cycle initiation in Caulobacter crescentus We ident

159 naling input that couples surface contact to cell-cycle initiation via the second messenger cyclic di

160 a novel computational model of human mitotic cell cycle, integrating diverse cellular mechanisms, for

161 glycan synthesis is regulated throughout the cell cycle is poorly understood(5,6).

162 ted Arabidopsis leaves, on the breast cancer cell cycle, is associated with Cell Division Cycle 6 (CD

163 lls the functional space of other eukaryotic cell-cycle kinases controlling DNA replication.

164 s enhanced neurogenesis but with deficits in cell cycle kinetics of proliferating progenitors in the

165 int proteins such as WEE1, together with the cell cycle master regulator, CDK7.

166 egulation of key pathways regulating myeloid cell cycle, maturation and regenerative function of the

167 sts and triggered significant alterations in cell cycle, metabolic, and protein translation processes

168 Cell cycle mutants in the budding and fission yeasts hav

169 ce of engineered carbon nanomaterials on the cell cycle of PANC-1 and AsPC-1 cancer cell lines.

170 P2X7 stimulation affected cell cycling of effector T cells and resulted in generat

171 ical roles in working out how the eukaryotic cell cycle operates and is controlled.

172 eatment had no effect on cell proliferation, cell cycle or apoptosis in the PDXC as well as other CCA

173 els of diversity at many levels-ranging from cell cycle organization to chromosome ploidy to replicat

174 tains the stability, speed, and coherence of cell cycle oscillation, from which the different vital p

175 ll four strands show cooperativity, reducing cell cycle pathways and inhibiting lung cancer cell prol

176 h as the receptor tyrosine kinases, TP53 and cell-cycle pathways, and IDH1 R132.

177 y, cooperating alterations, and signaling or cell-cycle pathways.

178 Using a live-cell reporter of cell cycle phase and long-term imaging, we monitored sin

179 While we found that the cell cycle phase at the time of cisplatin addition was n

180 Our data and predictor of cell cycle phase can directly help future studies to acc

181 cle proteins, and/or shortening of the G0/G1 cell cycle phase leading to cell size reduction.

182 of Cyclin D1 mRNA and protein stability in a cell-cycle phase-dependent manner.

183 ryo segmentation, using the FUCCI transgenic cell-cycle-phase marker, revealed a spatial and lineage-

184 s increased during mitosis relative to other cell cycle phases suggests that redox modifications coul

185 arison of protein functionality at different cell cycle phases.

186 compare motility between cells in different cell-cycle phases and in cell-cycle arrest.

187 k1, the major mitotic kinase that drives the cell cycle, phosphorylates the Ask1 component of the Dam

188 Collectively, these results suggest that the cell cycle plasticity, which enables tumor models to eva

189 ith the environment, genetic background, and cell-cycle position.

190 structural components that underly essential cell cycle processes, and influenced our thinking about

191 Cells must couple cell-cycle progress to their growth rate to restrict the

192 isingly, IRX3/5 are required for appropriate cell cycle progression and chromatid segregation during

193 tween the molecular machineries that control cell cycle progression and EHT.

194 ween chromatids has the potential to disrupt cell cycle progression and genome integrity, so it is hi

195 hat loss of MCM10 function leads to impaired cell cycle progression and induction of DNA damage-respo

196 In parallel, we investigated cell cycle progression and levels of p53, p21 and c-Myc

197 adation at mitotic exit accelerates G1-phase cell cycle progression and perturbs global DNA methylati

198 gh matriglycan up-regulation does not affect cell cycle progression and proliferation of the cancer c

199 dian clock in comparison to their effects on cell cycle progression and tumorigenesis.

200 Although the major events in prokaryotic cell cycle progression are likely to be coordinated with

201 O-Vanillin also affected networks related to cell cycle progression as well as connective tissue deve

202 ly impact the synchrony of cell division and cell cycle progression during diel growth.

203 First, size-dependent cell cycle progression ensures that smaller cells delay

204 g a cell cycle checkpoint inhibitor to drive cell cycle progression following ionizing radiation.

205 nt of APOBEC3 family proteins, and regulates cell cycle progression in HIV-infected cells.

206 In all orders of life, cell cycle progression in proliferating cells is depende

207 ed the role of miR-181c in the regulation of cell cycle progression in relation to HCV infection.

208 component of this response, which regulates cell cycle progression in the face of replication stress

209 (Cdk)1, one of the master regulators of G2/M cell cycle progression in U. maydis, interacts and contr

210 back loop between appressorium formation and cell cycle progression in U. maydis, which serves as a "

211 rowth, and the extent of growth required for cell cycle progression is proportional to growth rate.

212 , our method goes beyond this and quantifies cell cycle progression on a continuum.

213 at impaired growth is due to perturbation of cell cycle progression rather than induction of apoptosi

214 progression ensures that smaller cells delay cell cycle progression to accumulate more biomass than l

215 Ultimately, cell cycle progression was impaired at the G1/S and G2/M

216 PRANCR regulates keratinocyte proliferation, cell cycle progression, and clonogenicity.

217 inds to Rb, releasing it from E2F to promote cell cycle progression, and inducing ubiquitination of R

218 wo lesion types to the signals that regulate cell cycle progression, DNA replication, and cell surviv

219 cal role that this pathway has in regulating cell cycle progression, inhibiting CDK4/6 is an attracti

220 gressively accumulates in the nucleus during cell cycle progression, where it interacts with class I

221 2 (CDK2) and cyclin-A expression, arresting cell cycle progression, whereas overexpression of miR-18

222 t hepatocytes show a severe dysregulation of cell cycle progression, with incomplete mitoses, and a p

223 e developed a novel approach to characterize cell cycle progression.

224 he MDM2 locus that suppresses p53 levels and cell cycle progression.

225 and associated effects on DNA synthesis and cell cycle progression.

226 erely inhibited CD4 T cell proliferation and cell cycle progression.

227 phocytes that are associated with control of cell-cycle progression and genomic stability as well as

228 essing either miRNA showed downregulation of cell-cycle progression and mitosis-associated proteins.

229 The chaperone protein SmgGDS promotes cell-cycle progression and tumorigenesis in human breast

230 ion, we find that surface contact stimulates cell-cycle progression by demonstrating that surface-sti

231 SVC112 increased cell-cycle progression delay and slowed DNA repair follo

232 ric transcription factor NF-Y is crucial for cell-cycle progression in various types of cells.

233 ay crucial roles in cell differentiation and cell-cycle progression, and kinase dysregulation is asso

234 inst aberrant hypoxic signaling and abnormal cell-cycle progression.

235 ion and tumor growth, highlighting a role in cell-cycle progression.

236 -promoting genes, including those regulating cell cycle, proliferation, and metabolism, yet the roles

237 offer multiple applications: cell detection, cell cycle property determination, biomarker detection,

238 regulation of HNF4alpha and cyclin D1, a key cell cycle protein in the liver.

239 ession through the cell cycle by marking key cell cycle proteins for proteasomal turnover.

240 lerated proliferation, altered expression of cell cycle proteins, and/or shortening of the G0/G1 cell

241 Such modular architecture is common among cell-cycle proteins; thus, the WW-PPIase domain cross-ta

242 Finally, our half-cell cycling protocol also offers a method for evaluatin

243 er cell transcription through constraints on cell cycle re-entry of quiescent hepatocytes.

244 angiogenesis, inflammatory suppression, and cell cycle reentry as agrin's mechanisms of action.

245 We demonstrate that Nek8445 localization is cell cycle regulated and this kinase has a role in regul

246 ulates the ubiquitination of cohesin and its cell cycle regulated interaction with chromatin.

247 rom Escherichia coli, Caulobacter crescentus cell cycle-regulated DNA methyltransferase (CcrM), the M

248 ono-ubiquitination by BARD1/BRCA1 during the cell cycle regulates S phase progression to maintain gen

249 both skeletal muscle insulin metabolism and cell cycle regulation in endocrine pancreas (miR-15a and

250 c/apoptotic fate switch was mediated through cell-cycle regulation by Growth Arrest and DNA Damage 45

251 ular organization, cellular development, and cell-cycle regulation.

252 evels, increased Tnf expression, decreased a cell cycle regulator (Ccnb1), and increased apoptotic fa

253 endent manner, stimulating expression of the cell-cycle regulator AIL1 and suppressing BRANCHED1 expr

254 s, suppressing p53-induced expression of the cell-cycle regulator p21 and enhancing p53-induced up-re

255 Polo-like kinase 1 (PLK1) is an essential cell-cycle regulator that is frequently overexpressed in

256 t stimulates proliferation and expression of cell cycle regulators and stemness-associated genes, but

257 that ensures the faithful ubiquitination of cell cycle regulators during mitosis.

258 eomics profiles reveal a deregulation of key cell cycle regulators in lincNMR-depleted cells like the

259 Protein and transcript levels of cell cycle regulators were examined in breast cancer cel

260 clei define such pacemakers by concentrating cell cycle regulators.

261 Expression of the cell cycle regulatory gene CDK6 is required for Philadel

262 numerous studies have explored cardiomyocyte cell cycle regulatory mechanisms to enhance myocardial r

263 Cell-cycle regulatory proteins (p21(Cip1) /p27(Kip1) ) i

264 h tumors enriched the cell proliferation and cell cycle related gene sets in GSEA.

265 directly help future studies to account for cell cycle-related heterogeneity in iPSCs.

266 sion of genes involved in DNA metabolism and cell cycle-related processes, as well as downregulation

267 Cyclin proteins related to eumetazoan cell-cycle-related cyclins A, B, D, G/I and Y, and trans

268 ated in the regulation of DNA metabolism and cell-cycle-related gene expression during nitrogen (N) d

269 to prior results, HML and HMR had identical cell-cycle requirements for silencing establishment, wit

270 tical in the regulation of antiviral ISG and cell cycle responses that permit ZIKV to persistently in

271 ells fail to arrest at G(1)-S in response to cell-cycle restriction point signals, this information h

272 ses in proteins involved in DNA replication, cell cycle, RNA processing, and chromosome processing.

273 of a number of mRNAs involved in hypoxia and cell-cycle signaling.

274 Our data show that cell cycle, signaling, and differentiation are coordinat

275 We also demonstrate that half-cell cycling stability is consistent with full cell desa

276 gh standard methods assign cells to discrete cell cycle stages, our method goes beyond this and quant

277 meric chromatin and kinetochores at distinct cell-cycle stages, revealing extensive reorganization of

278 The foundation of bacterial cell cycle studies relies on two interconnected dogmas t

279 o overaccumulation of negative regulators of cell cycle such as Wee1-like protein kinase (WEE1).

280 utations that affect progression through the cell cycle, suggesting that cell cycle delay is sufficie

281 dependent kinases at distinct stages of the cell cycle, suppresses S-phase entry and promotes progre

282 Here, we present an advanced toolbox of cell cycle tag constructs in budding yeast with defined

283 Moreover, we use cell cycle tags to reinstall cell cycle control to a der

284 of this strategy in clinical exploration of cell cycle-targeting therapies in brain cancers.

285 ion, known as endocycle, is a variant of the cell cycle that differs from mitosis and occurs in speci

286 During interphase of the eukaryotic cell cycle, the microtubule (MT) cytoskeleton serves as

287 Our data suggest that the cell-cycle timing asynchrony of the early embryonic deve

288 It typically occurs in synchrony with the cell cycle to ensure that a complete copy of the genetic

289 egulating the transition from the end of one cell cycle to the beginning of the next.

290 were required for ZIKV NS5 to regulate hBMEC cell cycle transcriptional responses.

291 is essential for accurate timing of the G1-S cell cycle transition and is regulated by the correspond

292 ncrease in mitochondrial fusion and a G(1)/S cell cycle transition, both of which are linked to incre

293 ut not catalytic inhibitors, blocks the G1-S cell cycle transition.

294 ion of T-PLL cells evoked higher-than-normal cell-cycle transition and profiles of cytokine release t

295 Cell cycle transitions are generally triggered by variat

296 s in interphase, they are able to arrest the cell cycle until the breaks are repaired before entering

297 nly be initiated at the G1/early S phases of cell cycle upon the treatment onset, resulting in hetero

298 exploited genome-protective control for the cell cycle, we show Aurora B phosphorylation at S227 by

299 Cell viability, proliferation, and cell cycle were evaluated by the MTS assay, Ki-67 staini

300 e to the bulk of the population, including a cell cycle with a very low G1 fraction and a metabolomic