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1 response to MPP+ treatment, which led to the cell degeneration.
2      VH infusion did not induce dopaminergic cell degeneration.
3 el abolished the RD-associated photoreceptor cell degeneration.
4 asilar papilla at the earliest signs of hair cell degeneration.
5  which lacks auditory responses and has hair-cell degeneration.
6 s characterized by progressive photoreceptor cell degeneration.
7 xpression leads to photoreceptor and bipolar cell degeneration.
8 ssion of normal opsin leads to photoreceptor cell degeneration.
9 pture could be a direct trigger for the hair cell degeneration.
10 , atypical nuclei, perinuclear clearing, and cell degeneration.
11 of Bmp8b causes male infertility due to germ cell degeneration.
12 lear inclusion (NI) formation and late-onset cell degeneration.
13 ds gene mutations resulting in photoreceptor cell degeneration.
14 ed by ibogaine mediates excitotoxic Purkinje cell degeneration.
15 -onset, and relentless form of photoreceptor cell degeneration.
16 ormer being associated with evidence of beta-cell degeneration.
17 bogaine displayed clear evidence of Purkinje cell degeneration.
18 eless sufficient to induce bilateral granule cell degeneration.
19 ased basal autophagy, leading to progressive cell degeneration.
20 lutamine dependence and attenuating Purkinje cell degeneration.
21 be controlled with small molecules to reduce cell degeneration.
22 gical symptom onset and more severe Purkinje cell degeneration.
23 ted males demonstrated RPE and photoreceptor cell degeneration.
24 otoxic stress response, contributing to hair cell degeneration.
25    Iron-containing microglia showed signs of cell degeneration.
26 t contributes to aminoglycoside-induced hair cell degeneration.
27  mossy-fiber sprouting 5-6 weeks after mossy cell degeneration.
28 chromophore showed accelerated photoreceptor cell degeneration.
29 s of CCP1 in mice causes cerebellar Purkinje cell degeneration.
30 ease involving RPE atrophy and photoreceptor cell degeneration.
31                   With time this causes cone cell degeneration.
32 ults from retinal pigmented epithelium (RPE) cell degeneration.
33 m of SCA17 transgenic mice prior to Purkinje cell degeneration.
34 F3A loss results in very rapid photoreceptor cell degeneration.
35  dysmorphic stereocilia and progressive hair cell degeneration.
36 ve a therapeutic role in human photoreceptor cell degenerations.
37  enrichment, P = .041); and retinal ganglion cell degeneration (2-fold enrichment, Pcorr = .049).
38 -) mice lose hearing and manifest outer hair cell degeneration after systemic cisplatin treatment.
39 y is characterized by vascular smooth muscle cell degeneration and accumulation of diagnostic granula
40       Rapid and progressive postnatal acinar cell degeneration and acinar-to-ductal metaplasia occur,
41 entiviral knockdown of EZH2 rescued Purkinje cell degeneration and behavioral abnormalities in Atm(-/
42 etes mellitus at 30+ weeks of age, when beta-cell degeneration and both amorphous and amyloid deposit
43                        Significant levels of cell degeneration and cell debris, characteristic of nec
44 1 in mice causes severe ataxia with Purkinje cell degeneration and cerebellar atrophy, partially rese
45 e mutant channels, leading over time to hair-cell degeneration and contributing to their deafness.
46 ysiological potentials likely underlies hair cell degeneration and deafness in Va and Va(J) mice.
47  that alter channel properties, causing hair cell degeneration and deafness.
48 echanism for the terminal phases of neuronal cell degeneration and death in human neurodegenerative d
49 activation of this receptor induces neuronal cell degeneration and death via stimulation of caspases.
50 ermline mutant mice, including photoreceptor cell degeneration and defective ERG responses.
51 -finger transcription factors, leads to germ cell degeneration and defective sperm production in adul
52 following hearing onset did progressive hair cell degeneration and enhanced auditory neuron excitabil
53 tered proinsulin processing, as well as beta-cell degeneration and hyperstimulation.
54  retinas against light-induced photoreceptor cell degeneration and inhibited photoreceptor apoptosis.
55                Mechanisms underlying this NK cell degeneration and its biological significance are no
56     Histopathological findings revealed beta-cell degeneration and loss consistent with the drop in t
57 or and an abnormal gait, as well as Purkinje cell degeneration and photoreceptor cell loss.
58 ole in the BR-mediated regulation of tapetal cell degeneration and pollen development in Solanum lyco
59 inase Cgamma (PKCgamma), leading to Purkinje cell degeneration and progressive cerebellar dysfunction
60 ntually hyperexcitability gives rise to dark cell degeneration and reduced cerebellar output.
61 ounding the SG neurons, leading to satellite cell degeneration and subsequent SG degeneration with a
62 hat loss of Dicer1 leads to early-onset cone cell degeneration and suggest that Dicer1 is essential f
63  gene expression is synchronal with nucellar cell degeneration and thus, nucellin may be involved wit
64 ly sufficient to delay the onset of Purkinje cell degeneration and to correct the ataxia.
65  which disruptions in NMNAT1 lead to retinal cell degeneration and would provide a resource for testi
66 ral defects in the retina (e.g. dopaminergic cell degeneration and/or lack of regeneration).
67 d extracellular matrix, medial smooth muscle cell degeneration, and adventitial fibrosis.
68 sing loss of conventional MET currents, hair cell degeneration, and deafness in mice.
69 sing into large vacuoles preceding satellite cell degeneration, and followed by SG degeneration.
70 l protein frataxin causes neural and cardiac cell degeneration, and Friedreich's ataxia.
71 ity that glial dysfunction leads to Purkinje cell degeneration, and generated mice that express ataxi
72 rebellum of wild-type mice leads to Purkinje cell degeneration, and Inpp5a overexpression decreases i
73 ine nucleus migration defects, cochlear hair cell degeneration, and profound hearing loss.
74 as knocked out leading to postnatal Purkinje cell degeneration, and standard delay eyeblink condition
75 ) is linked to pancreatic inflammation, beta-cell degeneration, and the pathogenesis of type 2 diabet
76  stress, lipid transport and catabolism, and cell degeneration are enriched for genes associated with
77 GSIS, suggest a molecular mechanism for beta-cell degeneration as a consequence of Ide deficiency, an
78 g a 6-OHDA lesion, it is evident that nigral cell degeneration begins much earlier.
79 overlapping phenotypes arising from Purkinje cell degeneration, cerebellar atrophy and varying degree
80  of Purkinje cells revealed findings of dark cell degeneration consistent with excitotoxic injury.
81 tion of sodium influx to development of dark cell degeneration (DCD) in Purkinje neurons (PNs) follow
82 -induced delayed neurotoxicity known as dark cell degeneration (DCD) that is expressed as cytoplasmic
83 days old), AMPA produced in addition to dark cell degeneration (DCD), an edematous type of toxicity (
84 erker, a recessive mutation that causes hair cell degeneration, deafness, and vestibular dysfunction.
85 bal Npc1 null mice, suggesting that Purkinje cell degeneration does not underlie these phenotypes.
86  week after radiation, with extensive acinar cell degeneration, dysfunction, and cholinergic denervat
87           Additional neurons undergoing dark cell degeneration exhibited none of these apoptotic feat
88  mild fatty degeneration and inflammation to cell degeneration, fibrosis, and cirrhosis with or witho
89 wever, their involvement in cochlear sensory cell degeneration following acoustic trauma is unknown.
90                                Photoreceptor cell degeneration follows a radial gradient, more severe
91 ial activation may be involved in the nigral cell degeneration in 6-OHDA induced parkinsonian mice.
92 urkinje cells, we documented severe Purkinje cell degeneration in 92Q SCA7 transgenic mice.
93                                              Cell degeneration in Alzheimer's disease is mediated by
94            The polygenic basis of outer hair cell degeneration in Beethoven mice provides a model sys
95 or comparable transneuronal retinal ganglion cell degeneration in both primate species and show that
96 t alter either clinical symptoms or Purkinje cell degeneration in Dpl transgenic mice.
97 ivatives (13n and 36a) caused adrenocortical cell degeneration in guinea pigs treated with these inhi
98  and potentially moderating progressive hair cell degeneration in hearing loss more generally.
99 such as presbycusis, that involve outer hair cell degeneration in humans.
100                                         Hair cell degeneration in mambo mice precedes endocochlear po
101  causes profound hearing loss and outer hair cell degeneration in mice.
102  pathogenesis of the disease and progressive cell degeneration in MLIV patients.
103 ms of PNH that include toxins, anterior horn cell degeneration in motor neurone disease and genetic d
104 e non-cell-autonomous nature of the Purkinje cell degeneration in our SCA7 mouse model indicates that
105 ntributes to the cascade leading to dopamine cell degeneration in Parkinson's disease (PD).
106 ia nigra and the specific patterns of nigral cell degeneration in Parkinson's disease.
107 al dysfunction in the pathogenesis of nigral cell degeneration in Parkinson's disease.
108  immune/inflammatory factors in dopaminergic cell degeneration in parkinsonian substantia nigra, we a
109 fied as a candidate gene to explain Purkinje cell degeneration in pcd mice, but its function remains
110 n, but unlike Nna1, neither rescues Purkinje cell degeneration in pcd mice, indicating that they do n
111 ately leading to loss of visual function and cell degeneration in PRPH2 mutant models and human patie
112 action contributes to the selective Purkinje cell degeneration in SCA1.
113 hate (IP(3)) levels and ameliorates Purkinje cell degeneration in SCA17 knock-in mice.
114 unctional changes that characterize ganglion cell degeneration in the glaucomatous eye, and following
115 brain tissue to investigate whether Purkinje cell degeneration in the neocerebellum is present in HD,
116  to 4.2mm posterior to the bregma to examine cell degeneration in the retrosplenial and piriform regi
117 y occasional necrotic cells and no extensive cell degeneration in the same brain regions.
118        Such rats showed no signs of neuronal cell degeneration in the striatum, but did show signific
119  causes a late onset of retinal dopaminergic cell degeneration in zebrafish.
120 t a novel signaling pathway for dopaminergic cell degeneration, in which MPP+ induces the up-regulati
121 cing of Map2k7, strongly attenuates Purkinje cell degeneration induced by LZK, but not DLK.
122  due primarily to a late-onset photoreceptor cell degeneration involving initially the rods but event
123             Hearing loss resulting from hair cell degeneration is a common disease that affects milli
124                                Photoreceptor cell degeneration is a feature common to these diseases,
125 raocular pressure-sensitive retinal ganglion cell degeneration is a hallmark of glaucoma, the leading
126                          In rats, TD-induced cell degeneration is accompanied by an accumulation of a
127                                However, germ cell degeneration is observed in 47% of adult homozygous
128 ze that the slowly progressive photoreceptor cell degeneration is secondary to abnormal function of t
129            By 12-14 months, retinal ganglion cell degeneration is usually very severe and essentially
130    Atg7(Deltapan) mice exhibit severe acinar cell degeneration, leading to pancreatic inflammation an
131 a (SNpc), a brain region where dopamine (DA) cell degeneration leads to Parkinson's disease (PD), exp
132 nic epilepsy, including hippocampal atrophy, cell degeneration, loss of parvalbumin-positive interneu
133 progressive retinal pigment epithelial (RPE) cell degeneration, mainly caused by an impaired antioxid
134 iably exhibit embryonic or larval lethality, cell degenerations, malformation of the posterior body,
135                                     Thus, NK cell degeneration may serve as a means evolved by the im
136 motor coordination in mildly ataxic Purkinje cell degeneration mice (pcd; Machado et al., 2015).
137 ses (CCP1-6) and is mutated in pcd (Purkinje cell degeneration) mice.
138 ion mutants, and light-induced photoreceptor cell degeneration models), the use of Tb(3+) or Eu(3+) d
139 re implanted into the cerebellum of Purkinje cell degeneration mutant mice (pcd/pcd), a model of adul
140 In RT-treated rats, progressive dopaminergic cell degeneration occurred in the SNc.
141                   Progressive base-apex hair cell degeneration occurs at older ages, corresponding wi
142              Given the putatively broad hair-cell degeneration of these individuals, the effects are
143 ring loss correlated with cell death in hair cells, degeneration of spiral neurons and increased macr
144 live ablation produced little or no Purkinje cell degeneration or glial activation.
145 ns in patients who suffer from photoreceptor cell degeneration or related retinal diseases.
146  nerve transection (ONTx) to induce ganglion cell degeneration, or in combination with an ionotropic
147 protected from glaucomatous retinal ganglion cell degeneration out to 14 months of age (oldest assess
148                      We report that Purkinje cell degeneration (pcd) mice exhibit a profound impairme
149                                     Purkinje cell degeneration (pcd) mice have a mutation within the
150          Analysis of visibly ataxic Purkinje cell degeneration (pcd) mice reveals that while differen
151                                 The Purkinje cell degeneration (pcd) mouse displays a dramatic phenot
152                                 The Purkinje cell degeneration (pcd) mouse has a disruption in the ge
153                                 The Purkinje cell degeneration (pcd) mouse is a recessive model of ne
154 ses, and its mutation underlies the Purkinje cell degeneration (pcd) mouse.
155        These animals, the so-called Purkinje Cell Degeneration (PCD) mutant mice, suffer from a progr
156                                 The Purkinje cell degeneration (pcd) mutant mouse is characterized by
157 ne have been shown to be related to Purkinje cell degeneration (pcd) phenotypes including ataxia in m
158 e classical recessive mouse mutant, Purkinje cell degeneration (pcd), exhibits adult-onset degenerati
159 autosomal recessive mouse mutation, Purkinje cell degeneration (pcd), was first identified through it
160 s have previously demonstrated that Purkinje cell-degeneration (pcd) mutant mice are impaired in eyeb
161 neurons, causes the same pattern of Purkinje cell degeneration, providing a clue to the mechanism of
162 s pathogenesis and mechanisms governing beta-cell degeneration/regeneration.
163 y which this mutation leads to photoreceptor cell degeneration remains unknown.
164 NOTCH3 and results in vascular smooth muscle cell degeneration, stroke, and dementia.
165 to its decreased expression following nigral cell degeneration suggests that it may play an important
166 ermine whether sustained waking causes brain cell degeneration, three parallel strategies were used.
167  rapid, marked by type I alveolar epithelial cell degeneration, type II alveolar epithelial cell hype
168 ANF ameliorates mutant TBP-mediated Purkinje cell degeneration via protein kinase C (PKC)-dependent s
169                                          The cell degeneration was associated with an accumulation of
170 after intense-light treatment, photoreceptor cell degeneration was estimated by determining the level
171                                     Purkinje cell degeneration was evaluated with a Fink-Heimer II st
172                              We show that NK cell degeneration was mediated by IL-21 derived from aut
173 scopic observations demonstrated morphologic cell degeneration well before lactate dehydrogenase rele
174                                              Cell degeneration, when present, extends transneuronally
175 lastoma cells resulted in vacuologenesis and cell degeneration, whereas ES treatment after FIG4-small
176 mal, the triple mutants exhibited acute hair cell degeneration which corresponded with impaired swimm
177 loping audiometric losses are caused by hair cell degeneration, while flat audiometric losses are cau
178 s exhibiting cerebellar atrophy and Purkinje cell degeneration whose subtypes arise from 31 distinct
179 ession and aggregation of hA might link beta-cell degeneration with diabetes onset and progression, b
180 llar autopsy material showed severe Purkinje cell degeneration, with abnormal intracellular accumulat

 
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