1 lular distribution of (64)Cu was measured by
cell fractionation.
2 ion, as determined by immunofluorescence and
cell fractionation.
3 sociated with microsomal membranes following
cell fractionation.
4 sed form of gB could be coisolated following
cell fractionation.
5 ablished both through immunofluorescence and
cell fractionation.
6 by indirect immunofluorescence as well as by
cell fractionation.
7 in primary neutrophils by flow cytometry and
cell fractionation.
8 also confirmed by coimmunoprecipitation and
cell fractionation.
9 eins in Escherichia coli was used along with
cell fractionation.
10 live cell imaging, immunocytochemistry, and
cell fractionation.
11 labelling, domain-specific biotinylation and
cell fractionation.
12 Cell fractionation according to lineage depletion and AL
13 -cell imaging, deconvolution microscopy, and
cell fractionation,
all concurred that exogenous rSPARC
14 found predominantly as a soluble protein by
cell fractionation,
all three proteins were found to loc
15 We show that by
cell fractionation,
almost all MPP10 was found in isolat
16 Microscopic and
cell fractionation analyses of parasites expressing one
17 Immunocytochemical staining and
cell fractionation analysis demonstrated that sentrin mo
18 pitope tag into NIH 3T3 cells and subsequent
cell fractionation analysis shows that the PATE protein
19 This was confirmed by
cell fractionation analysis.
20 A11 in cells by using immunofluorescence and
cell fractionation analysis.
21 2 in the nucleolar fraction as determined by
cell fractionation and by immnocytochemical analysis.
22 Using
cell fractionation and cell contact-dependent assay, we
23 Cell fractionation and confocal immunofluorescence micro
24 Cell fractionation and confocal localization of formin I
25 Cell fractionation and confocal microscope studies revea
26 Both
cell fractionation and confocal microscopic analyses of
27 In the present work,
cell fractionation and confocal microscopy studies demon
28 n null mice (Peri-/- MEF), we demonstrate by
cell fractionation and confocal microscopy that up to 50
29 Based on
cell fractionation and confocal microscopy, we find SH2-
30 gating enzyme 9 (Ubc9) in HCC cell nuclei by
cell fractionation and confocal microscopy; physical int
31 Cell fractionation and cytological experiments suggest t
32 degraded with a t1/2 of 2.9 h, were found by
cell fractionation and density-shift experiments to resi
33 Cell fractionation and electron-microscopic immunolabeli
34 Cell fractionation and extractions with MgCl(2) indicate
35 Using
cell fractionation and fluorescence methods, we demonstr
36 h most of the CAD in the cell was cytosolic,
cell fractionation and fluorescence microscopy showed th
37 underlying Tor-dependent signaling, we used
cell fractionation and immunoaffinity chromatography to
38 Cell fractionation and immunoblot analyses using polyclo
39 Cell fractionation and immunoblot analysis confirmed thi
40 A::TnphoA) mutants do not elicit the HR, but
cell fractionation and immunoblot analysis revealed that
41 Cell fractionation and immunoblotting showed mrnp 41 in
42 Cell fractionation and immunoblotting showed that PEB1a
43 their secretion phenotypes were assessed by
cell fractionation and immunochemistry.
44 Using
cell fractionation and immunocytochemical experimental a
45 Cell fractionation and immunocytochemistry indicated tha
46 Cell fractionation and immunocytochemistry show caveolae
47 Cell fractionation and immunocytochemistry studies demon
48 In this work we used
cell fractionation and immunocytochemistry to determine
49 taining RNA are all detected on membranes by
cell fractionation and immunoelectron microscopy.
50 Cell fractionation and immunofluorescence analyses indic
51 n of ERalpha to the nucleus as shown by both
cell fractionation and immunofluorescence microscopic st
52 ght into this process, we applied mechanical
cell fractionation and immunofluorescence microscopy to
53 venous etoposide administration, followed by
cell fractionation and immunofluorescence studies of var
54 By
cell fractionation and immunogold electron microscopy, w
55 Cell fractionation and immunogold labeling show that in
56 Cell fractionation and immunolocalisation studies reveal
57 as predominantly localized in the nucleus by
cell fractionation and immunostaining.
58 Cell fractionation and in situ hybridization analysis of
59 Furthermore,
cell fractionation and in vitro methylation assays demon
60 Cell fractionation and labeling studies with [(3)H]palmi
61 nt Ras localization to the PM as measured by
cell fractionation and photobleaching.
62 Cell fractionation and PP1 catalytic subunit isolation s
63 Cell fractionation and protease accessibility experiment
64 Cell fractionation and qRT-PCR analysis indicated that L
65 Cell fractionation and quantitative RT-PCR revealed that
66 We have utilized
cell fractionation and ribosome profiling to obtain a ge
67 d improve protein quantitation, we optimized
cell fractionation and sample processing steps at both t
68 of FMR1 and the FXR proteins, we carried out
cell fractionation and sedimentation experiments with mo
69 mic Organellar Maps (DOMs) approach combines
cell fractionation and shotgun-proteomics for global pro
70 l subcellular localization were evaluated by
cell fractionation and Western blot analysis.
71 Cell fractionation and Western blotting demonstrated tha
72 We determined by use of
cell fractionation and Western blotting that, during in
73 Cell fractionation and western-blot analysis localized t
74 Protease protection,
cell fractionation,
and affinity purification experiment
75 In this report, using immunocytochemistry,
cell fractionation,
and chromatin immunoprecipitation an
76 2, RAC1, and RHOA were measured with G-LISA,
cell fractionation,
and immunoblots.
77 were assessed by arteriovenous measurements,
cell fractionation,
and immunofluorescence.
78 ing immunogold labeling/electron microscopy,
cell fractionation,
and protein analysis techniques, we
79 Genetic epistasis,
cell fractionation,
and quantitative live-cell imaging a
80 vidence using immunofluorescence microscopy,
cell fractionation,
and SNARE protein interaction studie
81 ked Golgi fraction was isolated by classical
cell fractionation,
and the protein complement (the Golg
82 alphas, using immunofluorescence microscopy,
cell fractionation,
and total internal reflection fluore
83 nce localization, sucrose density gradients,
cell fractionation,
and yeast two-hybrid experiments.
84 By
cell fractionation,
ANKRA is found both in the cytosol a
85 We previously reported a
cell fractionation approach that includes the selection
86 l using complementary immunofluorescence and
cell fractionation approaches and found that it exclusiv
87 However, using Tm4
cell fractionation,
AR stimulation decreased betaPix/AR
88 Cell fractionation as well as indirect immunofluorescenc
89 In the present study, we found by
cell fractionation assays and confocal microscopy that p
90 NMD substrates classified based on
cell fractionation assays as "nucleus associated" or "cy
91 Cell fractionation assays confirmed subcellular distribu
92 Cell fractionation assays localized p32 to the P100 frac
93 Developmental 2D Western blots,
cell fractionation assays, and analysis of a missense pr
94 d the associated nascent RNA are prepared by
cell fractionation,
avoiding immunoprecipitation or RNA
95 Unlike other
cell-fractionation-
based methodologies, our protocol min
96 r intracellular calreticulin released during
cell fractionation because it was expressed on circulati
97 Cell fractionation by sucrose gradient centrifugation in
98 These results suggest
cell fractionation can be used to study the uptake of mo
99 Using transmission electron microscopy,
cell fractionation,
cell wall sorting signal domain swap
100 ng immunohistochemistry/confocal imaging and
cell fractionation/
co-immunoprecipitation, we found that
101 This hypothesis was supported by
cell fractionation,
coimmunoprecipitation, and coimmunol
102 Cell fractionation,
coimmunoprecipitation, and immunogol
103 Cell fractionation combined with quantitative assays was
104 Cell fractionation confirmed the lysosomal localization
105 In addition,
cell fractionation data indicate that Cdc37 is found in
106 Cell fractionation demonstrated a predominant decrease i
107 OptiPrep gradient
cell fractionation demonstrated that BK channels were co
108 lular localization of the kinase activity by
cell fractionation demonstrated that it is enriched in c
109 immunofluorescence, immunogold labeling, and
cell fractionation demonstrated that rat slit diaphragms
110 A
cell fractionation experiment using cisplatin-treated He
111 Cell fractionation experiments also revealed that phosph
112 Strikingly,
cell fractionation experiments and confocal immunofluore
113 Cell fractionation experiments and cryoimmunoelectron mi
114 Cell fractionation experiments confirmed that macroH2A1
115 tron microscopy, using anti-MompA serum, and
cell fractionation experiments confirmed the localizatio
116 N-terminus and site-directed mutagenesis and
cell fractionation experiments confirmed this protein is
117 Cell fractionation experiments demonstrated that LdK39 w
118 In the latter case,
cell fractionation experiments displayed that mitochondr
119 Subsequent immunocytochemistry and
cell fractionation experiments fully supported the concl
120 Cell fractionation experiments indicated that the change
121 Mutational analysis and
cell fractionation experiments indicated that the inv(16
122 Cell fractionation experiments on freshly isolated CD4+
123 TAF1 colocalizes with UBF in Hela cells, and
cell fractionation experiments provided further evidence
124 Cell fractionation experiments revealed a transcription-
125 Furthermore,
cell fractionation experiments revealed that significant
126 Confocal microscopy and
cell fractionation experiments revealed that upon LPS st
127 GFP fusion protein localization and
cell fractionation experiments show that this E3 ligase
128 Cell fractionation experiments showed that caspase-3 act
129 Pulse-chase
cell fractionation experiments showed that chitosome pro
130 Cell fractionation experiments showed that ExoT is trans
131 Cell fractionation experiments showed that it is localiz
132 Cell fractionation experiments showed that meningococcal
133 Our imaging and
cell fractionation experiments suggest nearly 30% of RIC
134 Cell fractionation experiments suggested that Inp53p ass
135 Cell fractionation experiments surprisingly revealed two
136 immunolocalization, immunoprecipitation, and
cell fractionation experiments, here we show association
137 AKT2 was localized to the cytoplasm by
cell fractionation experiments, immunocytochemistry, and
138 In agreement with the results of
cell fractionation experiments, immunofluorescence micro
139 In
cell fractionation experiments, more signal recognition
140 e microscopy, fluorescence spectroscopy, and
cell fractionation experiments, we found that depending
141 ntermediate was isolated in the periplasm in
cell fractionation experiments.
142 experiments to calculate the effective whole-
cell fractionation factors between water and organic mat
143 Cell fractionation,
fluorescence imaging and immunoelect
144 Subcellular localization using
cell fractionation followed by immunoblot detection, as
145 Cell fractionation followed by Western blot analysis ind
146 ncluding several attempts to isolate them by
cell fractionation from different cell types.
147 fluorescent protein-fused PIMT proteins and
cell fractionation-
immunoblot analysis revealed that apa
148 uclear localization of UL78 was confirmed by
cell fractionation,
immunofluorescence microscopy, and p
149 ear localization of UL78 was validated using
cell fractionation,
immunofluorescence microscopy, and p
150 bes catalyzed by transglutaminase 2 prior to
cell fractionation,
immunoprecipitation, and detection w
151 and function of beta-catenin was analysed by
cell fractionation,
immunoprecipitation, immunoblots, QR
152 Detailed
cell fractionation,
immunoprecipitation, microscopy, and
153 test this, we measured V-ATPase assembly by
cell fractionation in HEK293T cells treated with and wit
154 in EAAT4 and GluRdelta2 by protein blot and
cell fractionation in SCA5 autopsy tissue.
155 trafficking in control and BDL livers using
cell fractionation in the context of in vivo pulse-chase
156 riven by diverse methodologies, ranging from
cell fractionation,
in situ and proximity labeling, imag
157 Immunofluorescence and
cell fractionation indicate that Bud1p remains associate
158 Cell fractionation indicated specific loss of acidic lip
159 Cell fractionation indicated that Frmpd1 stabilizes AGS3
160 Cell fractionation into a cytoskeleton-unbound and a cyt
161 Cell fractionation localized both raf1CAAX and raf1(257L
162 Using
cell-fractionation methods, we observed that approximate
163 Cell fractionation of Arabidopsis leaves revealed that A
164 Crude
cell fractionation of infected cells using detergent lys
165 Using quantitative confocal microscopy and
cell fractionation of Nef-expressing cells and HIV-1-inf
166 Cell fractionation of SCP-2 overexpressing L-cells and W
167 mY RNAs are largely cytoplasmic in wild-type
cells, fractionation of knockout cells revealed that the
168 However, extraction of viable
cells, fractionation of lineages, and in vitro analysis
169 beta-Catenin signaling was assessed by
cell fractionation or immunoconfocal microscopy to detec
170 Refinement of the
cell fractionation procedure indicated that the completi
171 zed with double-label immunofluorescence and
cell fractionation procedures.
172 ial patterning, we developed a novel ex vivo
cell fractionation/
reconstitution assay.
173 Immunoelectron microscopy and
cell fractionation reveal that both proteins travel thro
174 Cell fractionation revealed temporal gene regulation, in
175 ccessibility, TX-114 phase partitioning, and
cell fractionation revealed that Msp exists as distinct
176 Cell fractionation revealed that SecADelta11 and SecADel
177 Cell fractionation revealed that variation in quantity o
178 scence assay, surface proteolysis, and novel
cell fractionation schemes revealed that MOSP in TDE exi
179 Cell fractionation showed that adenosine or an ADORA2A o
180 Cell fractionation showed that approximately 80% of the
181 Consistent with these results,
cell fractionation showed that both the GFP-2a fusion an
182 Furthermore, immunofluorescence staining and
cell fractionation showed that erlotinib treatment led t
183 Liver
cell fractionation showed that macrophages and activated
184 Cell fractionation showed that Omega-3 fatty acids induc
185 Cell fractionation showed that the phosphorylated pool o
186 Cell fractionation showed that the viral preintegration
187 Cell fractionation shows that both EGF-Rs and cAbl are f
188 Cell fractionation shows that cyclins A, E, and Cdk2 are
189 on by both immunofluorescence microscopy and
cell fractionation shows that the export of Pma1p from t
190 ows little or no endo H resistance, although
cell fractionation still needs to be optimized for these
191 In addition, in
cell fractionation studies an estimated 25% of Mr 34000
192 Cell fractionation studies and confocal microscopy showe
193 Cell fractionation studies and green fluorescent protein
194 ce its plasma membrane localization based on
cell fractionation studies and immunoelectron microscopy
195 Biochemical
cell fractionation studies as well as confocal images of
196 Cell fractionation studies demonstrate that exogenous Ap
197 Cell fractionation studies demonstrate that PANCR is pri
198 Previous
cell fractionation studies demonstrated that chitin synt
199 Cell fractionation studies demonstrated that expression
200 Cell fractionation studies determined that IL-12 and TNF
201 However, immunofluorescence and
cell fractionation studies identified Bax activation and
202 The confocal microscopy and
cell fractionation studies in MDA-MB-231 breast cancer c
203 Cell fractionation studies indicated that NO donors caus
204 Cell fractionation studies localize the 47- and 49-kDa p
205 Cell fractionation studies localize the three additional
206 Furthermore, in
cell fractionation studies of mitotic cells, phospho-PP-
207 Cell fractionation studies of primary bovine articular c
208 Cell fractionation studies of the t(2;5) translocation-c
209 Cell fractionation studies revealed that approximately 9
210 Cell fractionation studies revealed that cytosolic SNAP-
211 Cell fractionation studies revealed that KSR formed a co
212 Cell fractionation studies revealed that membrane-associ
213 Cell fractionation studies revealed that the N terminus
214 rog ECA(CYC) per milligram (dry weight), and
cell fractionation studies revealed that these molecules
215 Immunocytochemical and
cell fractionation studies revealed that TM6SF2 was pres
216 Cell fractionation studies showed that active p35/CDK5 w
217 Biochemical and
cell fractionation studies showed that BfpB is a 58-kDa
218 Triton X-114 phase separation and
cell fractionation studies showed that dominant negative
219 Infected-
cell fractionation studies showed that insoluble fractio
220 Direct
cell fractionation studies showed that latent virus is p
221 erinuclear and cytosolic localization, while
cell fractionation studies showed that most of the p38(J
222 Cell fractionation studies showed that NF90 and its hete
223 Cell fractionation studies showed that Nox1 and Nox5 but
224 Cell fractionation studies showed that only a small amou
225 Cell fractionation studies showed that SPN binds prefere
226 Cell fractionation studies showed that the fluorescent M
227 Immunofluorescence and
cell fractionation studies showed that the major MV memb
228 In vivo cell labeling and
cell fractionation studies showed that the majority of W
229 Cell fractionation studies showed that the NAE amidohydr
230 Biochemical and
cell fractionation studies suggest caveolae contain func
231 sus coimmunoprecipitated (Shc-antibody), and
cell fractionation studies that suggested that the Shc.P
232 Immunocytological and
cell fractionation studies with a specific antibody reve
233 Cell fractionation studies with S. gordonii further corr
234 assessed by chemical cross-linking following
cell fractionation studies with VP40 transfected cells.
235 surprisingly, immunofluorescence microscopy,
cell fractionation studies, and studies with enhanced gr
236 Using confocal microscopy and
cell fractionation studies, butyrate pretreatment of a h
237 Data from recent
cell fractionation studies, however, predominantly those
238 ivo, together with coimmunoprecipitation and
cell fractionation studies, provide compelling evidence
239 In
cell fractionation studies, the 46-kDa protein cofractio
240 udged by two-hybrid, immunofluorescence, and
cell fractionation studies.
241 in antibody staining, which was confirmed by
cell fractionation studies.
242 d supported by early electron microscopy and
cell fractionation studies.
243 nucleoids and/or mitochondrial ribosomes in
cell fractionation studies.
244 eolin-1 by immunofluorescence microscopy and
cell fractionation studies; and (iii) a caveolin-3-deriv
245 Cell-fractionation studies showed that it is the monocyt
246 Immunohistochemistry and
cell-fractionation studies suggested that mutant beta-ca
247 itor treatments either prior to or following
cell fractionation suggested the presence of a cell enve
248 Cell fractionation suggests that the overexpressed prote
249 to the plasma membrane as assessed by either
cell fractionation,
surface biotinylation, or the plasma
250 By
cell fractionation,
surface rTPO fractionated distinctly
251 fraction was substantiated by two different
cell fractionation techniques.
252 microsurgery as well as isolation by common
cell fractionation techniques.
253 We now show by
cell fractionation that folate receptors also must be cl
254 Herein we show, with morphology and
cell fractionation,
that all the components of a mitogen
255 By immunofluorescence and
cell fractionation,
the exocyst subunits were found to s
256 hIP-chip), ChIP-quantitative PCR (qPCR), and
cell fractionation,
the stable association of RBF1 with
257 By
cell fractionation,
these signaling molecules cosediment
258 o microscopy, immunoelectron microscopy, and
cell fractionation to enter transferrin-positive REs wit
259 nd immunogold electron microscopy as well as
cell fractionation to identify the intracellular localiz
260 ntal approaches they pioneered, ranging from
cell fractionation to immunoprecipitation to structural
261 ed a reliable and rapid method for mammalian
cell fractionation,
tuned for such proteomic analyses.
262 Cell fractionation,
two-phase partitioning, and detergen
263 tagging (LOPIT), which combines biochemical
cell fractionation using density gradient ultracentrifug
264 Cell fractionation was done to analyze this further.
265 Using confocal microscopy and
cell fractionation,
we demonstrated that up to 40% of en
266 g co-localization with organelle markers and
cell fractionation,
we determined that COMMD1 is located
267 Using ChIP-sequencing data and
cell fractionation,
we have compared the genomic distrib
268 ting the subcellular distribution of Chk1 by
cell fractionation,
we observed that around 20% of it lo
269 gy transfer, super-resolution microscopy and
cell fractionation,
we show that FFA4 localizes to intra
270 Using
cell fractionation,
we show that PLCgamma2-IP(3)-Ca(2+)
271 Also,
cell fractionation/
Western blot studies revealed that a
272 cytochemical findings have been supported by
cell fractionation,
which demonstrated that full-length
273 membrane, cytoplasm, and poles by combining
cell fractionation with deep-sequencing (Rloc-seq).
274 We coupled
cell fractionation with stable isotope labeling with ami