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1 lar ROS levels and the resulting increase in cell membrane permeability.
2 membrane potential preceded the increase in cell membrane permeability.
3 r or ionizable fragments to achieve in vitro cell membrane permeability.
4 s state-of-the-art performance in predicting cell membrane permeability.
5 id mechanical forces to transiently increase cell membrane permeability.
6 We report a method to engineer proteins with cell-membrane permeability.
9 , PI, USFA, U/S, and IUFA, displaying higher cell membrane permeability and more severe cell membrane
10 including 7e, 7f, 7g, and 9k, that addressed cell membrane permeability and other physicochemical iss
11 obubbles, can temporarily change vascular or cell membrane permeability and release or activate vario
12 egy for improving the proteolytic stability, cell membrane permeability and target binding affinity o
13 rmore, deletion of both genes did not change cell membrane permeability and the oxidative and heat st
14 sents a new method for rapidly assessing the cell membrane permeability and toxicity of candidate CPA
15 y and reactivity in vitro but also decreases cell-membrane permeability and biological activity, (b)
16 amage was investigated by cell lethality, by cell membrane permeability, and by protein inactivation.
18 idative stress and altered mitochondrial and cell membrane permeability appear to be critical factors
21 te Plasmodium falciparum increases red blood cell membrane permeability during infection to allow for
22 Amyloid oligomers and protofibrils increase cell membrane permeability, eventually leading to cell d
23 peractive heteromeric hemichannels increases cell membrane permeability, favoring ATP release and Ca(
25 duce sonoporation that transiently increases cell membrane permeability for localized delivery of DNA
26 is disease is the reversible increase in red cell membrane permeability generated by hemoglobin S pol
27 rthermore, the compounds exhibited excellent cell membrane permeability in living cells and a higher
28 lent Shigella caused a rapid increase in the cell membrane permeability of infected human monocyte-de
29 it was found that the peptide increased the cell membrane permeability of M. arachidicola, S. cerevi
30 te-derived macrophages (HMDM) but not in the cell membrane permeability of monocytes, as demonstrated
32 ing rapid degradation and limited tissue and cell membrane permeability, pose a challenge to their su
33 ese approaches include a method based on the cell membrane permeability properties of the hydrophobic
34 olyticus may be attributed to the changes in cell membrane permeability, protein synthesis activity,
35 d with microbubbles can efficiently increase cell membrane permeability resulting in enhanced tissue
36 This new targeting mechanism is named the cell membrane permeability targeting (CMPT) mechanism, w
37 ed targeting mechanism is introduced, termed cell membrane permeability targeting (CMPT), which impro
38 port, the porin OprF is expressed to improve cell membrane permeability, the cytotoxicity of which, h
39 ance drug delivery and effects by increasing cell membrane permeability through oscillation and burst
40 these inhibitors still suffered from too low cell membrane permeability to enter into CNS drug develo
42 e key aspects involved, where R-MDP enhanced cell membrane permeability, transfer ability, biofilm fo
43 used in medicine to transiently increase the cell membrane permeability via electroporation to delive
44 erations of new analogues aimed at improving cell membrane permeability while maintaining high in vit
45 els of riboflavin (2.4-3.6 muM) and improved cell membrane permeability, with a positive correlation