ライフサイエンスコーパス: cell morphology

1 sly characterize its supra-unit and sub-unit cell morphology.

2 , loss of astrocyte coupling, and changes in cell morphology.

3 oximately 15%, with only marginal changes to cell morphology.

4 lecules in multiple cells by normalizing for cell morphology.

5 Lon, intracellular localization of Lon, and cell morphology.

6 r both virus assembly and modulation of host cell morphology.

7 ormation on the three-dimensional epithelial cell morphology.

8 ith UM advancement, but has little effect on cell morphology.

9 sites and for the establishment of migratory cell morphology.

10 eletal structures that can dynamically alter cell morphology.

11 chondria, a landmark signature of eukaryotic cell morphology.

12 c1-dependent cytoskeletal dynamics, and thus cell morphology.

13 nd expression of the OtDUB(GEF) alone alters cell morphology.

14 orescence staining was conducted to evaluate cell morphology.

15 ociated beta-galactosidase activity and flat cell morphology.

16 g methods that match molecular expression to cell morphology.

17 molecule inhibitors and disruption of normal cell morphology.

18 ine kinase, KSHV-TK modulates signalling and cell morphology.

19 assify leukocytes and characterize red blood cell morphology.

20 ductase, abrogates pilus assembly and alters cell morphology.

21 has no effect on focal adhesion integrity or cell morphology.

22 synthesis and hydrolysis to maintain proper cell morphology.

23 tion of hippocampal LTP and on CA1 pyramidal cell morphology.

24 ely on qualitative observation of changes in cell morphology.

25 he Spag6-deficient MEFs rescued the abnormal cell morphology.

26 l bursting discharge, Ih currents, and islet cell morphology.

27 nd, thus, regulation of axonal stability and cell morphology.

28 can of peripheral blood smears for red blood cell morphology.

29 uminal spaces, and restored villi and goblet cell morphology.

30 e-associated beta-galactosidase activity and cell morphology.

31 arepsilon is sufficient to block mesenchymal cell morphology.

32 Piezo1 mechanotransduction to regulation of cell morphology.

33 dema within the submucosa and altered mucous cell morphology.

34 hrough changes in both cell biochemistry and cell morphology.

35 ibiotics, reducing growth rate, or deranging cell morphology.

36 erating stress fibers, and severe defects in cell morphology.

37 sure diminishes osteogenesis, independent of cell morphology.

38 he ratio of the nucleus to the cytoplasm and cell morphology.

39 host immune cells to major changes in fungal cell morphology.

40 vities of the Rac and Rho GTPases to control cell morphology.

41 hs, which is governed by fluctuations in the cell morphology.

42 Prom1, focussing primarily on its effects on cell morphology.

43 hosphotransferase-independent alterations in cell morphology.

44 ntly and is strongly associated with bipolar cell morphology.

45 microscopy (SEM) was utilized to understand cell morphology.

46 creased membrane fluidity and alterations of cell morphology.

47 um currents, and central, radial or vertical cell morphologies.

48 ear if or how myelodysplasia (abnormal blood cell morphology), a key MDS feature in humans, presents

49 odulus that produced striking differences in cell morphology, actin organization, and membrane dynami

50 l effector proteins implicated in regulating cell morphology, adhesion, and migration in various cell

51 not observed under these conditions, nor is cell morphology affected by alphavbeta6 expression.

52 d not affect cell viability; neither was the cells morphology affected as demonstrated by live cell i

53 The sample fixation methodology preserves cell morphology, allows analysis in the ultrahigh vacuum

54 ansparency, posing a challenge: how is their cell morphology, also critical for transparency, control

55 -1alpha-induced adhesion using shear stress, cell morphology alterations, and crawling on intercellul

56 Transcriptomic and cell morphology analyses of epicardial cells from epicar

57 Knockdown of TbeIF5A led to abnormal cell morphologies and detached flagella, suggesting that

58 aglpQ and DeltaphoD mutants revealed altered cell morphologies and effects on autolytic activity and

59 evolution and coordinate a diverse range of cell morphologies and migrations.

60 stic and ecological links between individual cell morphologies and the emergent features of cell grou

61 H3R17 methylation results in defective glial cell morphology and a sensory defect in a subpopulation.

62 Nicotine induced changes in cell morphology and ablate tight junctions consistent wi

63 cue of auditory/vestibular behavior and hair cell morphology and activity.

64 ment, thereby failing to support the Sertoli cell morphology and adhesion protein complexes (e.g., oc

65 rchitecture that is dependent on filamentous cell morphology and advantageous in environments with ra

66 llular viability which was in agreement with cell morphology and apoptosis.

67 ac and Rho GTPases exert opposing effects on cell morphology and are stimulated downstream of Gbetaga

68 Removal of blocking cargo also restores cell morphology and attenuates the ER-stress response.

69 senescence in GBM as evidenced by changes in cell morphology and beta-galactocidase accumulation.

70 These data suggest a critical link between cell morphology and cell signaling and reconcile the dic

71 ated particular longevity-related changes in cell morphology and characteristics, including critical

72 y, these findings suggest that Prom1 affects cell morphology and contributes to chloride conductance.

73 rotein that is involved in the regulation of cell morphology and cytoskeletal organization.

74 tion of the MAPK cascade and variably impact cell morphology and cytoskeletal rearrangement.

75 TEPA, a copper chelator, inhibited EMT-like cell morphology and cytoskeleton arrangement triggered b

76 Moreover, Sj7170 changed cell morphology and cytoskeleton of U87 cells by the GTP

77 en species, mitochondrial health, as well as cell morphology and determine that the hMSCs are minimal

78 ved domain plays an important role in proper cell morphology and developmental regulation in represen

79 We observed altered cell morphology and disrupted organization of F-actin in

80 Rap1, a noncanonical activator, and controls cell morphology and EGF-induced membrane protrusion dyna

81 When plated onto substrates, cell morphology and even stem-cell differentiation are i

82 ls from frame-to-frame, and characterize the cell morphology and fluorescence.

83 often involving complex alterations in host cell morphology and function.

84 ensible, we use high-dimensional image-based cell morphology and gene expression profiles.

85 SLIP yields rich data sets on cell morphology and gene expression that illustrate the

86 Released DCF induced changes in cell morphology and glycolytic activity.

87 lternatively, the documentation of a typical cell morphology and immunophenotype on blood cells coupl

88 f Myo9b in HL-60-derived macrophages altered cell morphology and impaired cell migration.

89 he BMAL1(-/-) macrophages identified altered cell morphology and increased motility.

90 ontrast, AR deletion in luminal cells alters cell morphology and induces transient over-proliferation

91 cyte length, suggesting a connection between cell morphology and intrinsic relaxation behaviour.

92 f cavities around MHs correlates with Muller cell morphology and is consistent with the hypothesis of

93 measuring the kinetics of bacterial growth, cell morphology and lysis, cancer-related gene expressio

94 sly allows tracking the resulting changes in cell morphology and mechanics as well as measuring the f

95 her approaches for cancer diagnosis based on cell morphology and microscopy (biopsies) are too not co

96 The effects of ACM on tumor cell morphology and migration also depended on astrocyte

97 dition of exogenous TSP2 to WT cells induced cell morphology and migration rates that were similar to

98 sembly supplies the structural framework for cell morphology and migration.

99 nts revealed that local GAP activity rescues cell morphology and migration.

100 ting SN-mDA neurons and how it affects their cell morphology and migratory behavior.

101 ases that contributes to regulation of NSCLC cell morphology and motile activity.

102 se (ROCK) has an essential role in governing cell morphology and motility, and increased ROCK activit

103 ys in generating the characteristic hook-end cell morphology and motility, have not been elucidated.

104 trix to the actin cytoskeleton and regulates cell morphology and motility.

105 ulators of cancer invasion via regulation of cell morphology and motility.

106 where it is responsible for proper epidermal cell morphology and overall plant growth.

107 constraints can influence three-dimensional cell morphology and packing within epithelial tissues.

108 where they function as master regulators of cell morphology and pathogenesis.

109 the effects of acidity on mammary carcinoma cell morphology and phenotype have not been thoroughly c

110 yze fluorescence intensity and localization, cell morphology and proliferation as well as other descr

111 ring which period they retained their normal cell morphology and proliferation rates.

112 ere reversible and accompanied by changes in cell morphology and pronounced reduction in both cell/ce

113 It contributes to the cell morphology and provides cell wall integrity against

114 uit analysis as well as analyzed the role of cell morphology and receptor internalization.

115 known to be important for ribosome assembly, cell morphology and resistance to cell wall-targeting an

116 ters (cell membrane capacitance referring to cell morphology and seal resistance referring to adhesio

117 est, Rb dephosphorylation, flat and enlarged cell morphology and senescence-associated beta-galactosi

118 imensional (3D) microscopy enable imaging of cell morphology and signaling with unprecedented detail.

119 signaling exhibit defects in endothelial tip cell morphology and sprouting.

120 luding cell type distributions, variation in cell morphology and stomatal depth, differentiation of h

121 ents (IFs) are key players in the control of cell morphology and structure as well as in active proce

122 al model benchmarked on the observations, on cell morphology and swimming traits.

123 report highlights that CK5 actively remodels cell morphology and that blockade of CK5-beta-catenin in

124 al spindle position is determined largely by cell morphology and that spindles consistently center th

125 Cell morphology and the abundance of cell-cell junction

126 Furthermore, cell morphology and the arrangement of epidermal cell la

127 cal simulations, we predicted that the large cell morphology and the high cellularity of tumor within

128 rescence (IF) microscopy adds information on cell morphology and the microenvironment that are not ob

129 Changes in cell morphology and the physical forces that occur durin

130 It also rapidly affected CPE cell morphology and tight junction protein levels.

131 Cell death introduces alterations in cell morphology and tissue micro-structures that cause m

132 critical component in regulating endothelial cell morphology and vascular network formation.

133 2-dependent secretion is required for normal cell morphology and virulence in L. monocytogenes; howev

134 ns in illumination, staining, imaging noise, cell morphology, and cell clustering.

135 ets have activities that influence survival, cell morphology, and cell-to-cell interaction.

136 arameters, such as gene expression profiles, cell morphology, and cytoskeleton arrangement, we demons

137 8 inhibited transferrin endocytosis, altered cell morphology, and decreased cell viability.

138 ular regulation influencing gene expression, cell morphology, and function.

139 ability were vacuolization, other changes in cell morphology, and increased macropinocytosis.

140 owed growth, altered DNA content, changes in cell morphology, and increased sensitivity to DNA damagi

141 Specifically, we measure traction forces, cell morphology, and invasiveness of MDA-MB 231 breast c

142 cell wall is one major determinant of plant cell morphology, and is an attractive bioresource.

143 TMIGD1 controls cell migration, cell morphology, and protects renal epithelial cells fro

144 ffector of EphB2/ephrinB signaling, controls cell morphology, and thereby cell repulsion.

145 The mutation in divK additionally affected cell morphology, and this effect was complementable by a

146 w that neither channel noise nor a realistic cell morphology are responsible for the rate dependent s

147 nonbulky proteins, ER stress, and defective cell morphology are secondary consequences of bulky carg

148 f CcfM on magnetosome chain organization and cell morphology are supported by the finding that CcfM i

149 SICM can be used to analyze cell morphology at nanoscale, follow membrane dynamics,

150 on caused reduced cell growth and defects in cell morphology, both of which were suppressed by overex

151 not impact microtubule network integrity or cell morphology but contributed to microtubule stabiliza

152 e the matrilin-1 knockdown had no effects on cell morphology, but increased cell death was observed.

153 en verified by correlating the data with the cell morphologies by scanning electron microscopy and th

154 the marginal band, that flattens the overall cell morphology by pushing on the cell cortex.

155 he shape change of polymer sheet, as well as cell morphology, calcium (Ca(2+)) influx, and focal adhe

156 Moreover, BrdU(+) cells with plasma cell morphology can be detected for 10 years after vacci

157 , shedding light on how protein function and cell morphology can evolve and diversify.

158 s an environmentally relevant example of how cell morphology can impact bacterial fitness.

159 ed with collagen matrices including stellate cell morphologies, cell-mediated realignment of fibres,

160 other aspects of cellular behavior, such as cell morphology, cell mechanics, cell motility, cell sig

161 mall GTPases, functions in the regulation of cell morphology, cell migration, and actin cytoskeleton

162 A (Arl4A), an Arf small GTPase, functions in cell morphology, cell migration, and actin cytoskeleton

163 composite pictures of molecular expression, cell morphology, cellular interactions, proliferative st

164 eas beta-lactams were responsible for strong cell morphology changes (spheroplast with imipenem, fila

165 framework to a large-scale imaging screen of cell morphology changes induced by diverse drugs and the

166 nalling also reversed the AnxA8 loss-induced cell morphology changes, and induced beta-catenin transl

167 to detect single-cell binding events, track cell morphology changes, and monitor cell motility.

168 including membrane traffic, yet its role in cell morphology changes, such as the budding to filament

169 ed to the plasma membrane to orchestrate the cell morphology changes.

170 Cell morphology comparison and viability tests showed th

171 Cell morphology complexity assayed by fractal dimension

172 led to a more rounded, less mesenchymal-like cell morphology, consistent with decreased metastatic pr

173 We propose that these changes in ganglion cell morphology could impact the function of individual

174 nstrated the ability for inducing changes in cell morphology, cytoskeletal fiber orientation and chan

175 for neuronal differentiation and for normal cell morphology, cytoskeletal organization, proliferatio

176 al axis, which correlate with alterations in cell morphology, cytoskeleton and cell-cell contacts in

177 ition of STRIPAK phosphatase activity causes cell morphology defects in mitosis and impairs epithelia

178 e screening data sets on nuclear and mitotic cell morphologies demonstrates that CellCognition Explor

179 These dramatic changes in cell morphology depend on the auto-phosphorylation of ty

180 res that distinguish this model are variable cell morphology described by a collection of particles c

181 Loss of capzb affects cell morphology, differentiation and neural crest migrat

182 seven bacterial strains with a wide range of cell morphology, dimension, and surface characteristics.

183 d myosin-IIA can suitably couple nuclear and cell morphology downstream of matrix mechanics.

184 aracterized by abnormal cortical lamination, cell morphology (e.g., cytomegaly), and cellular polarit

185 ular protrusions are a ubiquitous feature of cell morphology, e.g., filopodia and microvilli, serving

186 high insulin-like growth factor-1, elongated cell morphology), even following exposure to Alzheimer's

187 utilize to generate the variety of bacterial cell morphologies found across the bacterial domain.

188 e describe a method for extracting realistic cell morphologies from fluorescence microscopy images to

189 his was associated with a dramatic change in cell morphology from one with scant cytoplasm and a glob

190 a readout (e.g., fluorescence, luminescence, cell morphology) from each cell in the population.

191 o or in vivo resulted in a loss of granulosa cell morphology, function, and gene expression.

192 hort hairpin RNA and investigated changes in cell morphology, gene expression, and behavior.

193 mellipodia formation, and membrane ruffling, cell morphologies generated by active Rac1.

194 Our work suggests that cell morphology has a strong impact within microbial com

195 Mammalian cell morphology has been linked to the viscoelastic prop

196 These data indicate that cell morphology has different requirements on cytoskelet

197 positive cells did not display the typical B cell morphology, having in general a more dendritic cell

198 Concomitant changes in cell morphology highlight the opportunity to study the r

199 While a range of cell morphologies in the EGL has long been known, how th

200 nt to stabilize focal adhesions and maintain cell morphology in infected cells and cells infected wit

201 es, including proper development of regional cell morphology in Kupffer's vesicle and the establishme

202 ing protein 4, and assumption of a dendritic cell morphology in response to anti-CD40 plus IL-4 were

203 liferation, TGF-beta expression, and altered cell morphology in SSCT cells.

204 ron microscopy (SEM) revealed abnormal fiber cell morphology in Tdrd7-/- lenses.

205 e genetic methods to study neuronal or glial cell morphology in the mammalian brain.

206 reprogramming of acinar cells and regulates cell morphology in vivo and in vitro.

207 on neuronal proteostasis and maintenance of cell morphology in vivo.

208 skeletal organelles involved in establishing cell morphology, including the flagella connector, flage

209 y of mammalian cells and profound changes in cell morphology, including the loss of a single leading

210 ls specific alterations in cell motility and cell morphology indicating that the MG200-MG491 interact

211 f cell biological effects, including altered cell morphology, inhibition of cell growth and, in some

212 g machine learning, to analyze and integrate cell morphology, intracellular organization, gene expres

213 In addition, bacterial cell morphology is adaptable to changes in environmental

214 he cellular and molecular control of a lobed cell morphology is currently thought to involve PIN-FORM

215 y, high-throughput microscopy indicated that cell morphology is relatively insensitive to mild knockd

216 One of the most fundamental changes in cell morphology is the ingression of a plasma membrane f

217 ocyte injury results in a dramatic change in cell morphology known as foot process effacement.

218 rocess where a drastic change of endothelial cell morphology leads to the formation of blood stem and

219 direct link between drug similarities on the cell morphology level and the distance of their respecti

220 HMDMs were investigated by analyzing the cell morphology, LPS-induced cytokine profile, surface m

221 which results in irreversible disruption of cell morphology, mechanics, and function.

222 Furthermore, the role of Hfq in the cell morphology, metabolism, cell wall integrity, resist

223 hesion size and distribution, thus affecting cell morphology, migration and ultimately localization.

224 Included are cell morphology, migration, and specialised responses, s

225 nformation about individual cells, including cell morphology, molecular content and local cell densit

226 tion is frequently accompanied by changes in cell morphology (morphodynamics) on a range of spatial a

227 plicable to study complex phenotypes such as cell morphology, motility and biofilm formation over ext

228 ting effect, and displays altered colony and cell morphology, motility, antagonism against other micr

229 te that cyanobacterial CCRPs are involved in cell morphology, motility, cytokinesis and colony integr

230 Furthermore, cell morphology must be monitored in the host, as many f

231 hing and quantitatively characterizing brain cell morphologies noninvasively.

232 explore the possibility of quantifying brain cell morphology noninvasively.

233 hat acute METH treatment neither altered the cell morphology nor killed the cells, which echoed with

234 Our results demonstrate changes in cell morphology, observed as cytoskeleton protrusions-i.

235 Changes in S/W associated with mesophyll cell morphology occurred earlier than changes in S(c) /S

236 Here we visualize the dynamic cell morphologies of tangentially migrating SN-mDA neuro

237 nts to grow in nude mice with characteristic cell morphology of anaplastic thyroid cancer (ATC).

238 Fluorescent microscopy was used to assess cell morphology of different cell types which were stain

239 specific changes to the refractive index and cell morphology of individual cells concomitant with enh

240 r rapid and accurate reconstruction of whole cell morphology of large neuronal populations in densely

241 The cell morphology of rod-shaped bacteria is determined by

242 eening to identify molecules that affect the cell morphology of tobacco BY-2 cells.

243 cells in confluent samples, handles various cell morphologies, offers algorithms for quantitative an

244 es focused on developmental organization and cell morphology often use this layered stratification to

245 To systematically interrogate the impact of cell morphology on bacterial physiology, we used fluores

246 er, these results highlight the influence of cell morphology on fate determination processes.

247 hroughput, they produce no information about cell morphology or spatial resolution offered by microsc

248 nked to specific phenotypic changes, such as cell morphology or virulence.

249 es the 'morphome', a multivariate dataset of cell morphology parameters.

250 n motility (myosin light chain 1, myosin A), cell morphology (PhIL1), and host cell invasion (apical

251 , nor SSV2 induced any detrimental effect on cell morphology, plasma membrane and mitochondrial funct

252 features modulate cell behaviour, including cell morphology, proliferation and differentiation.

253 The hPSC-CFs resemble native heart CFs in cell morphology, proliferation, gene expression, fibrobl

254 reveal new roles for SPAG6 in modulation of cell morphology, proliferation, migration, and ciliogene

255 ective concentrations, UNC0638 did not alter cell morphology, proliferation, or erythroid differentia

256 e cell state characterized by differences in cell morphology, proliferative kinetics, and tumor-initi

257 o gain a deeper understanding of how helical cell morphology promotes host colonization by H. pylori,

258 Here we reported that MSI1 alters cell morphology, promotes cell migration, and increases

259 es of their microenvironment that can affect cell morphology, protein levels and localization, gene e

260 odology to quantify the relationship between cell morphology, pulse frequency, and electroporation re

261 Revealing VIP+ cell morphologies, receptive fields and synaptic connect

262 of integrin beta1 led to alterations in beta-cell morphology, reduced insulin gene expression, and en

263 ionally, betaIII-tubulin suppression altered cell morphology, reduced tumor spheroid outgrowth, and i

264 keleton), which links the magnetoskeleton to cell morphology regulation in Magnetospirillum gryphiswa

265 adosome under anaerobic conditions regulates cell morphology, resulting in Ecoli MG1655 cell filament

266 DM96 with the rates for a routine red blood cell morphology scan.

267 alyzed by CellaVision and microscopy for red cell morphology scans.

268 e, including root branching, root epithelial cell morphology, seed germination, and leaf conductance.

269 ssion rescues aberrant Rho3 localization and cell morphologies seen at the restrictive temperature in

270 ochastic, multicolor labeling for individual cell morphology studies.

271 derstand the regulation of some prototypical cell morphologies such as that of rod-shaped Escherichia

272 Simple plant cell morphologies, such as cylindrical shoot cells, are

273 prior literature linking GTPase activity to cell morphology, support the hypothesis that GTPase sign

274 ession leads to defective cortical pyramidal cell morphology, synaptic plasticity deficits, and alter

275 This generates a distinctive repair cell morphology that is favorable for the formation of t

276 We showed light-inducible control of cell morphology that resulted in a substantial increase

277 ation is involved in invasion, motility, and cell morphology, the proteins that utilize this PTM rema

278 plicitly model the cytoskeleton or resulting cell morphologies, these results, along with prior liter

279 We show that unlike the individual cell morphology, this population dynamics does not depen

280 Despite similarities in neuromast hair cell morphology, three classes of these cells can be dis

281 t achieves excellent transparency; preserves cell morphology, tissue architecture, and reporter molec

282 present a platform to relate the effects of cell morphology to gene expression induced by nanotopogr

283 trate stiffness, focal adhesion density, and cell morphology to show that the total amount of work a

284 ,25-(OH)2D3 at a time-dependent manner alter cell morphology towards osteoblast-associated characteri

285 We compete cells with different cell morphologies under a range of conditions and ask ho

286 ntration of free endotoxin released, and the cell morphology under light microscope.

287 Altered cell morphology was assigned to the viral M25 gene.

288 cytofluorescence, auto-fluorescence (AF) and cell morphology was determined.

289 Endothelial cell morphology was evaluated by subjective scoring (ran

290 The cell morphology was imaged by confocal microscopy and an

291 A striking change of cell morphology was observed with a rounded phenotype ar

292 the effect of these on phospholipidosis and cell morphology was studied.

293 YAP activation with smaller and more rounded cell morphologies were induced in hMSCs.

294 PIP5K6 expression on pollen tube growth and cell morphology were attenuated by coexpression of MPK6

295 Early, specific changes in endothelial cell morphology were found to "announce" an upcoming all

296 An alc deletion mutant showed modified cell morphology when grown under enriched CO(2) and impa

297 , opsin distribution in the target cell, and cell morphology, which affect the spatial selectivity of

298 roduce three complementary models of rounded cell morphologies with a prescribed excess surface area.

299 ctive motility but a fundamentally different cell morphology with highly flexible snake-like swarming

300 unique among bacteria based on their helical cell morphology with hook-shaped ends and the presence o