戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  blocking it with antibodies leads to cancer cell phagocytosis.
2 e alone is inefficient in stimulating glioma cell phagocytosis.
3 or I, thus eliciting highly selective cancer cell phagocytosis.
4 atelet and endothelial activation and immune cell phagocytosis.
5 demonstrated their ability to promote target cell phagocytosis.
6 O mice displayed enhanced rates of apoptotic-cell phagocytosis.
7  the efficiency of serum-dependent apoptotic cell phagocytosis.
8 ma-hemorrhage-induced suppression of Kupffer cell phagocytosis.
9 hich sublethal oxidative stress inhibits RPE cell phagocytosis.
10 gulation of the cytoskeleton during stellate cell phagocytosis.
11 tissues and a macrophage defect in apoptotic cell phagocytosis.
12 e are capable of infection and inducing host cell phagocytosis.
13 lls are deleted through apoptosis or Sertoli cell phagocytosis.
14 ot appear related to inactivation of Kupffer cell phagocytosis.
15 G) and N297D/S298A-IYG optimally drove tumor cell phagocytosis.
16  CRISPRi perturbations that can affect tumor cell phagocytosis.
17 ell-intrinsic mechanisms and increased tumor cell phagocytosis.
18 ent cell cytotoxicity and antibody dependent cell phagocytosis.
19 tor (CSF1Ri) to monitor the change of immune cells phagocytosis.
20  with GdCl3, which is known to block Kupffer cell phagocytosis and antigen processing, the spontaneou
21        We also demonstrate increased myeloid cell phagocytosis and degradation of tau aggregates link
22  and blockade of its function leads to tumor cell phagocytosis and elimination.
23 ges play a dual function, contributing to FL cell phagocytosis and FL cell survival through long-last
24 e will outline the consequences of apoptotic cell phagocytosis and illustrate how apoptotic cells cou
25 rew significantly slower with enhanced tumor cell phagocytosis and increased recruitment of M2-like t
26  receptor tyrosine kinase mediates apoptotic cell phagocytosis and modulates macrophage cytokine prod
27 l inhibition of CD47 and Gal3 enhances tumor cell phagocytosis and reprograms macrophages to overcome
28                                    Apoptotic cell phagocytosis and soluble TREM2 shedding were unalte
29 pha2 but not alpha1 AMPK was involved in RPE cell phagocytosis and that activation of alpha2 AMPK con
30 ta5 receptor is required for lens epithelial cell phagocytosis and that UV light treatment of lens ep
31 s a downregulatory signal that inhibits host cell phagocytosis, and CD47 therefore functions as a "do
32 or cells and induction of antibody-dependent cell phagocytosis as one of the primary mechanisms of ac
33                            Inhibition of RPE cell phagocytosis by activation of alpha2 AMPK was assoc
34                            Inhibition of RPE cell phagocytosis by AICAR was fully reversed by blockad
35                                       Tumour cell phagocytosis by antigen presenting cells (APCs) is
36      We find that androgen regulates Sertoli cell phagocytosis by controlling expression of miR-471-5
37    A distinct chiral probe stimulates tumour cell phagocytosis by covalently modifying Y387 in the re
38         Trauma-hemorrhage suppressed Kupffer cell phagocytosis by decreasing Fc receptor expression a
39 recruitment of C1q, which enhanced apoptotic cell phagocytosis by immature dendritic cells.
40 e" signal, which subsequently promotes tumor cell phagocytosis by macrophages and also triggers an an
41 lls in vitro with specificity, trigger tumor cell phagocytosis by macrophages, and efficiently clear
42 aired in their abilities to induce apoptotic cell phagocytosis by murine peritoneal macrophages.
43 a2 AMPK contributed to the inhibition of RPE cell phagocytosis by oxidative stress.
44 s dose-dependently impaired apoptotic Jurkat cell phagocytosis by primary rat or human AM, irrespecti
45  and new phagocyte functions after apoptotic cell phagocytosis demonstrates the enormity of ways to m
46                                    Apoptotic cell phagocytosis (efferocytosis) is mediated by specifi
47 nation nearly abolished macrophage apoptotic cell phagocytosis; elimination of Axl, Tyro3, or both, r
48                      The increase in Kupffer cell phagocytosis following hypoxemia was also prevented
49 ngs indicate that the suppression of Kupffer cell phagocytosis following trauma-hemorrhage is indepen
50 RFP)), microglia are effector cells of tumor cell phagocytosis in response to anti-CD47 blockade.
51 expression, and augmented mAb-mediated tumor cell phagocytosis in vitro However, only STINGa reversed
52 ntaining viral DNA also showed evidence of T cell phagocytosis in vivo, suggesting that their viral D
53              We investigated whether Kupffer cell phagocytosis is differentially regulated following
54                                              Cell phagocytosis is impaired in type 2 diabetes and req
55 cytoskeletal involvement, although apoptotic cell phagocytosis is not involved.
56 hagocytic receptor(s) responsible for tumour cell phagocytosis is(are) largely unknown.
57             The uptake of large particles by cells (phagocytosis) is an important factor in cell biol
58 mbospondin 1 (TSP1) indicated that mesangial cell phagocytosis of apoptotic cells involved an alpha(v
59             Hence, KIM-1-mediated epithelial cell phagocytosis of apoptotic cells protects the kidney
60 g the injury-limiting potential of mesangial cell phagocytosis of apoptotic cells.
61                                    Mesangial cell phagocytosis of apoptotic neutrophils in vitro was
62                   We conclude that mesangial cell phagocytosis of apoptotic neutrophils involves a no
63 etermined that IFN-gamma reduced endothelial cell phagocytosis of C. albicans by 41.3% compared with
64                            Since endothelial cell phagocytosis of C. albicans is required for damage
65                            Since endothelial cell phagocytosis of C. albicans is required for endothe
66                 Thus, inhibiting endothelial cell phagocytosis of C. albicans may be a mechanism by w
67  cause inactivation or activation of Kupffer cell phagocytosis of colloidal carbon.
68 dative stress dose-dependently inhibited RPE cell phagocytosis of photoreceptor outer segments (POS)
69 macological activator of AMPK, inhibited RPE cell phagocytosis of POS in a dose-dependent manner.
70  to C. albicans, suggesting that endothelial cell phagocytosis of the organism is required to induce
71 lm bacteria was mediated through mononuclear cell phagocytosis since treatment with cytochalasin B, w
72                             Increased tumour cell phagocytosis subsequently enhances antigen cross-pr
73 before transplantation, or LEW donor Kupffer cell phagocytosis was blocked with GdCl3 (7 mg/kg) on da
74 ation by GdCl3 inhibition of hepatic Kupffer cell phagocytosis was pivotal in preventing the developm
75 are revealing universal principles of immune-cell phagocytosis, while also dispelling misconceptions
76  Here, under conditions that maximize cancer cell phagocytosis within cohesive tumors, we uncover pat