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1 , ATO inhibits mitochondrial complex III and cell respiration.
2 L prevented the rotenone-induced decrease in cell respiration.
3 ced the endogenous mutant mtDNA and improved cell respiration.
4 enone addition were quantified by monitoring cell respiration.
5 erencing O2 electrode to monitor single beta-cell respiration.
6 nditions, notably as a negative regulator of cell respiration.
7     ATP synthase is likewise a key enzyme of cell respiration.
8                                However, both cell respiration and ATP synthesis were preserved in cel
9 p-trifluoromethoxyphenylhydrazone-stimulated cell respiration and caused respiratory failure in the p
10 tidrug efflux, the tricarboxylic acid cycle, cell respiration and cell division, were identified to h
11        Rotenone inhibition studies of intact cell respiration and pyruvate-malate oxidation in permea
12 spiratory complexes I, III and IV, decreased cell respiration and raised superoxide levels.
13         A ferricyanide-based electrochemical cell respiration assay was adapted for use in broad-spec
14 ation between the level of CI impairment and cell respiration, cell growth, free radical production,
15 ir prevents HCV protein-mediated decrease of cell respiration, collapse of mitochondrial membrane pot
16   The efficiency of ATP synthesis coupled to cell respiration, commonly referred to as the P/O ratio,
17                                          The cell respiration defect results from an alteration in th
18  ROS with resveratrol partially prevents the cell respiration defect.
19                                           As cell respiration genes are required for A. baumannii fit
20 found that Bcl-x(L) expression can stimulate cell respiration in cells with mitochondrial DNA.
21 plexes, which are involved in the process of cell respiration in mitochondria.
22                However, saNOR contributed to cell respiration in this assay once growth had resumed,
23 d the importance of COX3, a gene involved in cell respiration, in hypoxia adaptation.
24  use diverse terminal electron acceptors for cell respiration, including carbon dioxide, enabling a v
25 ein synthesis in mammalian cells and perturb cell respiration, leading to a time- and dose-dependent
26                                       During cell respiration, liberated electrons reduce PMS, which
27           Mitochondrial functions, including cell respiration, mitochondrial content, and bioenergeti
28 8E or T58I Cytc showed a reduction in intact cell respiration, mitochondrial membrane potential (Delt
29 t the aegA gene product is not essential for cell respiration or fermentation or for the utilization
30 ortantly, we show that Lpar5 regulates CD8 T cell respiration, proton leak, and reactive oxygen speci
31 s' shelf life at room conditions by reducing cell respiration rate and water evaporation.
32                                      The per-cell respiration rates of this community are about 2 ord
33 n C2C12 myotubes resulted in impaired intact cell respiration, reduced complex I/NADH oxidase activit
34   CD36 silencing in cultured LECs suppresses cell respiration, reduces VEGF-C-mediated VEGFR2/AKT pho
35 ncentrations of ADP and ATP directly mediate cell respiration remains unclear, however.
36 xicity in a variety of cancer types if their cell respiration was obstructed by hypoxia or by chemica
37  showed that in yeast cells expressing 103Q, cell respiration was progressively reduced after 4-6 h o
38                  Further, changes in CD4 + T cell respiration were associated with changes in naive C
39 acellular changes of dissolved oxygen due to cell respiration were monitored, with gene gun injected
40 in mitochondrial RNA (mtRNA) translation and cell respiration were reversed in two human disease line
41        Effects of direct nitrite exposure on cell respiration were studied in cultured human primary
42                 mBCl progressively decreased cell respiration, with no effect on mitochondrial proton