ライフサイエンスコーパス: cell strain

1 lear extracts from a normal human fibroblast cell strain.

2 mal human melanocytes compared to a melanoma cell strain.

3 poliovaccine was passaged in a human diploid cell strain.

4 ylation capacity during arrest than a normal cell strain.

5 types of polystyrene microparticles and four cell strains.

6 ast strain and two condensin-modified mutant cell strains.

7 is regulation is not apparent in all primary cell strains.

8 l human cell lines than in six untransformed cell strains.

9 ctors for recently established human sarcoma cell strains.

10 ses will replicate on approved primary human cell strains.

11 on of 716-730 are found in both XP-D and TTD cell strains.

12 ogated chemoresistance in all drug-resistant cell strains.

13 t genomic defects in CRISPR-Cas9 dnaE edited cell strains.

14 res lower applied stresses to achieve higher cell strains.

15 lar meshwork (MTM) tissues and establish MTM cell strains.

16 of TSTs on GRbeta-GRalpha ratios in human TM cell strains.

17 and 40) in human normal and glaucomatous TM cell strains.

18 ated proMMP-3 production in some but not all cell strains.

19 th either promoter in any of the four normal cell strains.

20 .1(-)-dtps1 and obtained more than 20 stable cell strains.

21 was not different from that of the parental cell strains.

22 nce in vitro for a number of different human cell strains.

23 air-deficient and repair-proficient isogenic cell strains.

24 H-1 cell line or by any of the tumor-derived cell strains.

25 gher level in tumor lines compared to normal cell strains.

26 in cultured normal human mammary epithelial cells (strains 5, 16, and 40), established from tissue t

27 posed of about 95 and 5% of highly pigmented cells (strain 531Ad) and cells with normal levels of pig

28 ity was not found in another furin-deficient cell strain, 7.P15, selected from the monkey kidney cell

29 or lines, the normal human breast epithelial cell strains AD074 and MCF10A rapidly metabolize [3H]ret

30 Some of the cell strains also coexpressed vimentin.

31 Two HTM cell strains and an alphavbeta3 integrin-overexpressing

32 similar biomolecular abundances in all four cell strains and between proliferating and senescent cel

33 whereas it is generally absent from primary cell strains and from many tumor adjacent tissue samples

34 to replicative senescence observed in normal cell strains and includes altered cell morphology and ge

35 tern blotting with multiple primary prostate cell strains and prostate cancer cell lines reveals that

36 he metabolism of [3H]retinol in normal human cell strains and SCC lines from the oral cavity and skin

37 dvanced-stage melanomas, using early-passage cell strains and snap-frozen tissues (n = 18 and n = 24,

38 among 2p, 11q, 12q and XpYp within the same cell strains and was dependent on the replicative kineti

39 was due to elastin mRNA stabilization in one cell strain, and here this behavior was confirmed in ski

40 ith the expression of LMP1 in EBV-positive B-cell strains, and its expression inhibited the apoptosis

41 Total RNA was isolated from TM cell strains, and mRNA expression for FST 317/344 isofor

42 Trp correlates with highly UV-sensitive TTD cell strains, and mutation of Arg683 to Trp correlates w

43 xpressed by HCMV permissive human fibroblast cell strains, and that TLR2 is a functional sensor in th

44 tening has been reported to be faster in WRN cell strains, and the length of telomeres in senescent W

45 on cytotoxic actions of anthrax LT that are cell-, strain-, and species-specific, which have not bee

46 A cell strain armed with the controller in co-culture enab

47 mRNA levels are elevated in some fibroblast cell strains at replicative senescence, through this reg

48 We studied four human fibroblast cell strains (BJ, MRC-5, IMR-90, and WI-38) from prolife

49 ts the c-NHEJ deficits of some XLF-deficient cell strains but not others, suggesting a variable requi

50 all insertions or deletions in five of these cell strains, but we could not identify the mutation in

51 article's companion, we updated the average cell strain by defining a correction factor ("strain fac

52 Rapid changes in cell strain by disruption of cytoskeletal and nuclear fi

53 ations and DNA repair characteristics of the cell strains can be correlated with the particular mutat

54 Different cell strains can be grown in parallel in the device unde

55 ack of arginase activity also in several CHO cell strains (CHO-DP12, CHO-S, and DUXB11) and other mam

56 lymerase chain reaction of rat aorta-derived cell strains confirmed the presence of VEGF165 and VEGF1

57 thymic mouse hosts, all of the tumor-derived cell strains consistently formed tumors.

58 used p53(-/-) and p21(-/-) human fibroblast cell strains constructed by gene targeting to investigat

59 p arm of chromosome 17 of the focus-derived cell strains containing the codon 215 mutation revealed

60 The cell strains continued to express both basal- and lumina

61 Normal prostate epithelial cell strains demonstrate no methylation.

62 Further inclusion of 18 additional cell strains demonstrated that this mRNA subset was pred

63 27kDa subunits whose activity is absent from cell strains derived from a subset of Xeroderma Pigmento

64 Fibroblast cell strains derived from either fetal or adult SCID dog

65 ays in human urothelial cells have relied on cell strains derived from invasive tumours or NHUC immor

66 light, we used a genetic approach involving cell strains derived from patients with inherited defici

67 arising TP53(wt) and TP53(mut) 4N cells from cell strains derived from premalignant Barrett's esophag

68 Cell strains derived from xeroderma pigmentosum group E

69 About 20% of the cell strains derived from XP-E patients lack a damaged D

70 Similar analysis of a separate series of cell strains, derived from foci induced by cobalt-60, re

71 nuclei to DOX, whereas chronic reduction of cell strain desensitize nuclei to DOX.

72 nd a subset of primary serous ovarian cancer cell strains (DFs).

73 e normal karyotype, and hTERT(+) endothelial cell strains do not exhibit a transformed phenotype.

74 o driving pressure fluctuations and variable cell strain due to cell size fluctuations therefore caus

75 G island in primary human mammary epithelial cell strains during escape from mortality stage 0 (M(0))

76 n various human diploid fibroblast and tumor cell strains during the period that precedes activation

77 hydrate diets have been associated with beta-cell strain, dyslipidemia, and endothelial dysfunction.

78 Furthermore, microsomal extracts of normal cell strains (e.g., OKF4) exhibited about 7-fold more pa

79 Confluent culture supernatants from three cell strains each of normal, SSc, and NSF dermal fibrobl

80 In fibronectin-cultured cells, strain elicited no increase in SM-1.

81 in stress (sigma), and resultant changes in cell strain (epsilon) were measured.

82 re that the cultured normal human epithelial cell strains esterified [3H]retinol to a much greater ex

83 an melanocytes with six independent melanoma cell strains from advanced lesions.

84 Tissue culture of immortal cell strains from diseased patients is an invaluable res

85 Investigation of four DDB(+) cell strains from patients previously assigned to XP-E,

86 tein (DDB) is absent from a subset, Ddb-, of cell strains from patients with xeroderma pigmentosum gr

87 ollection of normal human mammary epithelial cell strains from women ages 16 to 91 years, derived fro

88 ellular penetration was similar for all four cell strains, genetically modified cells exhibited a sig

89 ibed a genetically unstable human fibroblast cell strain (GM2995), isolated from normal appearing ski

90 Glaucomatous TM cell strains have a lower GRbeta-GRalpha ratio compared

91 The T+E2-induced cell strains have been designated BPH1(TETD)-A and -B an

92 th in vitro passaging of both WRN and normal cell strains; however, the rate of shortening has been r

93 he population doubling limit of their parent cell strains, hTERT(+) keratinocytes entered a slow grow

94 in such damage avoidance, a human fibroblast cell strain in which both error-prone translesion synthe

95 Three different SC cell strains in 14 independent experiments responded wit

96 olated from these tumors were established as cell strains in culture.

97 rs formed in athymic mice by MSU-1.1-derived cell strains independently transformed in culture showed

98 mation of this xeroderma pigmentosum group C cell strain involves the progressive acquisition of muta

99 Golabi Behmel Syndrome (SGBS) pre-adipocyte cell strain is widely considered to be a representative

100 on the kinetics of propagative IIF, and even cell strains lacking the gap junction protein connexin-3

101 of nine additional human mammary epithelial cell strains/lines were determined.

102 ing to the suggestion that senescence in WRN cell strains may not be exclusively associated with telo

103 olved, two populations of infinite life span cell strain MISU-1.1, differing only in level of O6-alky

104 origenic infinite life span human fibroblast cell strain MSU-1.1 and an isogenic fibrosarcoma-derived

105 The nontumorigenic parental cell strain, MSU-1.1, expresses high levels of this mRNA

106 with those from HeLa cells and a fibroblast cell strain, MSU-1.2, for the ability to replicate a UV-

107 In one cell strain of immortal LFS cells, P53(mut/-) , containi

108 duced capacity to replicate in primary human cell strains or in cells of rodent derivation and also e

109 secretion compared to their controls in all cell strains (P<0.05).

110 All human TM cell strains produced detectable basal amounts of proMMP

111 s expressed CCR2 and CXCR2 in 55% and 66% of cell strains, respectively.

112 nt or rabbit cell lines and in primary human cell strains (restrictive cells) but not in HEp-2 or Ver

113 expression of a telomerase transgene in WRN cell strains results in lengthened telomeres and replica

114 Early passage cell strains showed high consistency between patient and

115 nsible macrophage/CNS-tropic strains require cell/strain-specific approaches.

116 in the three normal human mammary epithelial cell strains suggests that, in addition to hormonal effe

117 -cultured with a pure rat cholangiocarcinoma cell strain (TDECC) derived from the same ICC type as TD

118 in response to E(2) and the MCF-7 derived C4 cell strain that has lost PgR expression as a model syst

119 e that cell-free extracts prepared from XP-E cell strains that either lacked or contained DDB activit

120 urprising that the amount of hTR was high in cell strains that lacked telomerase activity, and the le

121 er analysis showed that all 15 focus-derived cells strains that lacked p53 transactivating activity c

122 have used this methodology in several human cell strains to reduce expression of the Prkdc (DNA-PKcs

123 an tumor cell lines and two human fibroblast cell strains to the demethylating agent, 5-aza-2'-deoxyc

124 ce analysis of the ERCC2 cDNA from three TTD cell strains (TTD1V1, TTD3VI, and TTD1RO) revealed mutat

125 mation of this xeroderma pigmentosum group C cell strain, we examined the expression/function of seve

126 senescent cells; however, in the four aging cell strains, we found common molecular differences domi

127 Using two independent human keratinocyte cell strains, we found that exogenous telomerase express

128 Using multiple primary keratinocyte cell strains, we verified a delay in p16INK4a induction

129 ession of eight early-passage human melanoma cell strains were compared with newborn and adult melano

130 ization to the primary tumors from which the cell strains were derived.

131 EFA-deficient cell strains were supplied with one of four different fa

132 Four cell strains were tested and showed increased IL-8 relea

133 Five human TM cell strains were treated with selected compounds.

134 ndent experiments in which five different TM cell strains were used.

135 min B1 is lost from primary human and murine cell strains when they are induced to senesce by DNA dam

136 resistance in majority of the drug-resistant cell strains whereas DNMT1 showed heterogeneous results.

137 nto a primary human embryonic lung (HEL-299) cell strain, which is known to respond to the type I IFN

138 These cell strains, which are all derived from a common nontum

139 ver, in a mutant Chinese hamster ovary (CHO) cell strain with defective SPT activity due to a lack of

140 g658 to either His or Cys correlate with TTD cell strains with intermediate UV-sensitivity, mutation

141 evels were normal or reduced by 50% in three cell strains with undetectable levels of pol eta protein