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1 ell neoplasm that often expresses the CD4+ T cell surface marker.
2  cells has been difficult without a defining cell surface marker.
3  major glycoprotein 2 (GP2) as a PP-specific cell surface marker.
4 nto Th17 cells in vitro and in vivo via CD25 cell surface marker.
5 re limited by the lack of available specific cell surface markers.
6 e ISCs from mouse and human tissues based on cell surface markers.
7 al nerve, and immunofluorescence staining of cell surface markers.
8 4, KDR) and osteoblastic (osteocalcin [OCN]) cell surface markers.
9 her coagulation, inflammatory, or lymphocyte cell surface markers.
10 subpopulation characterized previously using cell surface markers.
11 arly-outgrowth colony-forming unit assay and cell surface markers.
12 ke cysts that selectively incorporate apical cell surface markers.
13 eased up-regulation of activation-associated cell surface markers.
14 xpression of both DC and monocyte/macrophage cell surface markers.
15 iling cytokines, intracellular molecules and cell surface markers.
16 st cells as shown by cellular morphology and cell surface markers.
17 an be identified by the presence of specific cell surface markers.
18 et been achieved due to the lack of specific cell surface markers.
19 ls that previously identified based on other cell surface markers.
20 y Ab responses due to an absence of specific cell surface markers.
21 using flow cytometry to determine lymphocyte cell surface markers.
22  protein assays for cytokine, chemokine, and cell surface markers.
23 as electrical activity, gene expression, and cell surface markers.
24  TNF-alpha(+)CD4(+) T cells expressing naive cell surface markers.
25 positive cells and increased stem/progenitor cell surface markers.
26 lliliter of blood and the fold expression of cell surface markers.
27 human fetal pancreatic differentiation using cell surface markers.
28 ytometry together with various hematopoietic cell surface markers.
29 he result of context-dependent expression of cell surface markers.
30 ined challenging due to the lack of specific cell surface markers.
31 oskeleton regulators and the localization of cell surface markers.
32 pol mRNA, intracellular p24 Gag protein, and cell surface markers.
33 ypically purified from the bone marrow using cell surface markers.
34 Fab:Fab interactions in targeting oligomeric cell-surface markers.
35 ession profiles and identification of unique cell-surface markers.
36 lacked mature arterial, venal, and lymphatic cell-surface markers.
37 mass cytometry revealed expression of unique cell-surface markers.
38 roid progenitor cells that express analogous cell-surface markers.
39 expression of APC (macrophages and dendritic cells) surface markers.
40 ngle-cell mass cytometric measurements of 14 cell surface markers, 20 signaling/cell cycle proteins,
41 unbiased manner a panel of all commonly used cell surface markers (280 genes) from individual cells.
42 ogenase-bright cells expressed CD34 or CD133 cell surface markers (57.0% and 27.1%, respectively), co
43                      We show herein that the cell surface marker 6-sulfo LacNAc (slan) can define sla
44 for viral haemagglutinin (HA) expression and cell surface markers 8-16 hours post infection.
45                         From the panel of 21 cell surface markers, 9 were overexpressed in fetal prog
46 nfection of DC with live 35000HP caused less cell surface marker activation than infection with heat-
47  also expressed higher levels of more mature cell surface markers, additionally linking inflammasome
48 ses were assessed by measuring expression of cell surface markers (adhesion molecules, fibrinogen-lik
49                        Additional cytokines, cell surface markers, adhesion molecules, and accessory
50 ated with respect to endocytosis properties, cell surface markers, allostimulatory activity, and cyto
51                                        These cell surface markers allowed direct isolation of rare ce
52 ice, csGRP78 co-localized with the mesangial cell surface marker alpha8-integrin.
53 ary, we identified GD2 as a new CSC-specific cell surface marker and GD3S as a potential therapeutic
54         In summary, we describe here a novel cell surface marker and targeting tools for tumor macrop
55 thelial cells expressed specific endothelial cell surface markers and also exhibited the capacity for
56 bpopulations of CSCs, characterized by their cell surface markers and colony morphology, which can se
57  stem cells to reveal the connection between cell surface markers and distinct cell phenotypes.
58  We used a combination of flow cytometry for cell surface markers and enzyme-linked immunospot method
59 to different sub-populations on the basis of cell surface markers and examine their function in an in
60 comprised of distinct subsets with different cell surface markers and functional characteristics and
61 ntional dendritic cells (cDCs) with distinct cell surface markers and functions exist in mouse and hu
62 ancer cell line, which we first confirmed by cell surface markers and gene profiling to be highly enr
63 otypes, express telomerase activity, express cell surface markers and genes that characterize human E
64                                  Analysis of cell surface markers and immunoglobulin H gene rearrange
65 s include (1) the identification of distinct cell surface markers and other cellular properties in he
66 stinct subsets can be distinguished based on cell surface markers and pathophysiological function.
67                                   Changes in cell surface markers and patterns of gene expression are
68 d expression of CD80, CD86, CD40, and MHC-II cell surface markers and production of proinflammatory c
69 single skeletal stem cell population through cell surface markers and the development of single-cell
70 functional phenotypes characterized by their cell surface markers and their cytokine profiles.
71 this likeness extended into the non-ISCT MSC cell surface markers and trilineage differentiation, whi
72 ubsets based on their expression of specific cell surface markers and used them in our adoptive trans
73 s and healthy control donors share a similar cell-surface marker and gene expression profile.
74 l death (AICD) and expressed a unique set of cell-surface markers and gene profiles.
75  display significantly altered expression of cell-surface markers and produce increased inflammatory
76 ly, neutrophil morphology (nucleus shape and cell-surface markers) and functions (phagocytosis, degra
77 nology for measuring cell entity, evaluating cell surface marker, and peculiarly in the field of stem
78 xpress characteristic transcription factors, cell surface markers, and cytokines, including glycoprot
79 ion of apoptosis, acquisition of tumorigenic cell surface markers, and epithelial-mesenchymal transit
80 including those encoding effector cytokines, cell surface markers, and key transcription factors.
81 ptor (uPAR), a uniquely overexpressed cancer cell-surface marker, and facilitating the immune-mediate
82                                 Because many cell surface markers are shared between AML blasts and h
83                                        Tumor cell-surface markers are usually overexpressed or mutate
84 wed AME downregulates the expression of such cell surface markers as CD80, CD86, and major histocompa
85  interferon-gamma production, without losing cell surface markers associated with memory.
86 al cells in part by increasing the levels of cell surface markers associated with mesenchymal stem ce
87                                We identified cell surface markers associated with repression of p16(I
88  effector T cells, and caused an increase in cell surface markers associated with T(Regs) such as Fox
89 nsplants in regenerative medicine depends on cell-surface marker-based characterization and/or purifi
90 eration of reagents that specifically target cell-surface markers, because transmembrane proteins are
91 n NMR biosensor that can identify a specific cell surface marker by targeted (129)Xe MRI.
92  and FACS analyses demonstrate that specific cell surface markers can be used to discriminate prostat
93                                       As the cell surface marker CD133 identifies cancer-initiating c
94                                          The cell surface marker CD133 is frequently used to identify
95 ed high Wnt activity was associated with the cell surface markers CD133, CD166, and CD29, but not CD2
96                               Currently, the cell surface marker CD138 (SDC1) is used for this enrich
97 ive L3 for 48 h showed no alterations in the cell surface markers CD14, CD86, CD83, CD207, E-cadherin
98 ammatory T cells into tissues, or target the cell surface marker CD20 (rituximab; Rituxan for hematol
99 data in K562 leukemic cells, we identify the cell surface marker CD24 as co-varying with chromatin ac
100                     A new study shows that a cell-surface marker, CD27, identifies the first point of
101 s with great thermogenic potential using the cell surface marker CD29.
102  (MLN), spleen and thymus were labeled for T cell surface markers (CD3, CD4, CD8) and intracellular F
103                                          The cell surface marker CD34 marks mouse hair follicle bulge
104 including CTNND1 and the early hematopoietic cell surface marker CD34, resulted in reduced leukemic g
105                                        Using cell surface markers (CD34, CD133, kinase insert domain
106 nterferon-gamma with a unique combination of cell surface markers (CD4(+)CD25(-)CD44(hi)CD62L(lo)) an
107 s of breast cancer stem cells (BCSCs) is the cell surface marker CD44.
108 d stem cell-like characteristics express the cell surface marker CD44.
109  a Matrigel-based differentiation assay, and cell surface markers CD44 and CD24.
110 ese pancreatic cancer stem cells express the cell surface markers CD44, CD24, and epithelial-specific
111 on of pancreatic cancer cells expressing the cell surface markers CD44, CD24, and epithelial-specific
112  stage transitions, marked by changes in the cell-surface markers CD44 and ICAM1, and a Nanog-enhance
113 Here, we explored this hypothesis by using 2 cell surface markers, CD44 and CD137, to isolate antitum
114 hat express IL-10, as well as Tr1-associated cell surface markers, CD49b and LAG-3, and transcription
115 rences between these profiles, we identified cell surface markers, CD69 and CD36, whose genes were di
116 )/CCR4(+) T cells that also lack the usual T cell surface markers CD7 and/or CD26.
117 seq data, we defined a novel set of possible cell surface markers (Cd74 and Cd81) for these candidate
118 -surface protein also known as the leukocyte cell-surface marker CD82.
119 n the low nanomolar range, we identified the cell surface marker CD86 as a sensitive surrogate biomar
120 ls based on a variety of phenotypes, such as cell surface markers, cell proliferation and drug respon
121 B in vitro, as measured by the expression of cell surface markers, cellular signaling events, and cyt
122 man MDDCs exposed to galectin-1 up-regulated cell surface markers characteristic of DC maturation (CD
123 ubsets of HPCs examined, including HPCs with cell surface markers consistent with immature hematopoie
124 ssion signatures, morphological changes, and cell surface markers consistent with myeloid maturation.
125 e cells (CSCs) from DCIS.com cell line using cell surface markers (CS24(-)CD44(+)ESA(+)) and found th
126  within the tumor preferentially express the cell surface markers CTLA-4 and OX40.
127              Given the limited repertoire of cell-surface markers currently available for neural prog
128 oadly characterized by the expression of the cell surface marker CXCR4.
129  blood frequencies of MAIT cells, defined by cell surface markers, decline during tuberculosis (TB) d
130 etailed study has been hampered by a lack of cell surface markers defining tumor-specific dysfunction
131                                        Forty cell-surface markers, distinguishing all major leukocyte
132 is approach is that the presence of specific cell surface markers does not directly reflect the trans
133 umor cell (CTC) detection strategies rely on cell surface marker EpCAM and intracellular cytokeratins
134 XCL9, CXCL10, CXCL12, CXCL13 and CXCL16) and cell surface marker expression (CD3, CD4 and CXCR3) in p
135 eripheral blood, cutaneous mTregs had unique cell surface marker expression and cytokine production.
136                       Although not affecting cell surface marker expression and phagocytotic function
137 lar trilineage differentiation potential and cell surface marker expression as bone marrow hMSCs.
138 ted control iDCs to WT capsule did not alter cell surface marker expression but did elicit IL-8.
139 onal profiling, TCR repertoire analyses, and cell surface marker expression indicate that Dock2-defic
140  normal, displaying no obvious compromise in cell surface marker expression or antibody production ei
141 n in vivo, and except for CD138, plasmablast cell surface marker expression was unaffected.
142 tro proliferation responses, alloreactivity, cell surface marker expression, and antibody production.
143 T cells showed convergence in the pattern of cell surface marker expression, cytokine profiles, and g
144 n of MEK1/2 did not reduce CT-B induction of cell surface marker expression, it did attenuate CT-B-me
145 nd, when isolated from skeletal muscle using cell surface marker expression, these cells showed compa
146 ains the decrease in cytokine production and cell surface marker expression.
147 entiation for at least 25 d, as evidenced by cell surface marker expression.
148  isolation, cells were characterized through cell-surface marker expression and lineage-specific diff
149  and, regardless of methodology for harvest, cell-surface marker expression of CD73, CD90, CD105, and
150                                      We used cell-surface marker expression to purify from the satell
151 lectively increased polyploidization, mature cell-surface marker expression, and apoptosis of maligna
152                                              Cell surface markers' expression and chemotaxis were det
153  transmembrane glycoprotein, is an important cell surface marker for both stem cells and cancer stem
154                            CCR6 was the best cell surface marker for IL-17A+ cells when compared with
155                 Moreover, huEGFRt provides a cell surface marker for in vivo tracking of adoptively t
156 xpression of CD83 (previously described as a cell surface marker for mature dendritic cells) on CD4 T
157                      We further identified a cell surface marker for prospective isolation of iNCs, w
158                    MUC16 is a well-validated cell surface marker for serous adenocarcinomas of the ov
159 matic stem cells, and it is widely used as a cell surface marker for the isolation and characterizati
160                    Our identification of new cell surface markers for enriching mammosphere-initiatin
161                                     Existing cell surface markers for GSC are developed from embryoni
162     Our analysis identified 24 new/potential cell surface markers for murine fetal hepatic stem cells
163 ss I molecules offers unique cancer-specific cell surface markers for the identification and targetin
164               We further identify CORIN as a cell-surface marker for isolating the TBX5+NKX2-5+ subpo
165 ceptor (MC1R), which has been evaluated as a cell-surface marker for melanoma.
166 timulation during cancer immunotherapy and a cell-surface marker for pancreatic cancer.
167 nd proteomic approaches to identify specific cell-surface markers for cardiac PW1(+) cells and found
168      In this study, we attempted to identify cell-surface markers for leukemia-initiating cells in FA
169                                              Cell-surface markers for prospective isolation of stem c
170 of commercial antibodies, we have identified cell-surface markers for the separation of pancreatic ce
171 cently, a simple phenotype for HSCs based on cell surface markers from the signaling lymphocyte activ
172 ood, we acquired an immunological profile of cell-surface markers from healthy control and untreated
173 ytosis activity, viability and expression of cell-surface markers, from tens of thousands of single i
174 ssessment, nitro-blue tetrazolium reduction, cell-surface markers, genome-wide patterns of gene expre
175 h all the hallmarks of stem cells, including cell surface markers, global gene expression profiles, a
176 ent, both targeting the erythrocyte-specific cell surface marker glycophorin A.
177      However, lack of known unique mesangial cell surface markers has hampered this process.
178  allowing detection of increasing numbers of cell surface markers, has challenged the traditional tec
179 r functional phenotype, because few specific cell surface markers have been identified.
180 rious combinations of antibodies directed to cell surface markers have been used to isolate human and
181                                      Several cell-surface markers have been reported to identify cand
182  the basis of the differential expression of cell-surface markers, here we identify a mesenchymal str
183 ific membrane antigen (PSMA), a prototypical cell surface marker highly overexpressed in prostate can
184 utrophils, fibroblasts, and lymphocytes; and cell surface markers, ie, F4/80, CD11b, CD11c, and Ly-6C
185 ify and purify anergic T cells by a distinct cell surface marker in an autoimmune disease and paves t
186 he up-regulation of macrophage/hematopoietic cell surface markers in a large proportion of NIH 3T3 ce
187                                        Novel cell surface markers in adult progenitor cells included
188 e due to the impaired expression of relevant cell surface markers in Eklf(-/-) erythroid cells.
189 o studied expression of the identified novel cell surface markers in fetal rat liver progenitor cells
190     In this work, we studied progenitor/oval cell surface markers in the liver of rats subjected to 2
191                    Persistence of respective cell surface markers in vitro is confirmed both by flow
192 responses, alloreactivity, and expression of cell surface markers in vitro.
193  selection using an optimized combination of cell surface markers including CD30.
194 ells that exhibit differential expression of cell surface markers, including CD105 (or endoglin), Thy
195 ethod was reported, using the SLAM family of cell-surface markers, including CD150 (SlamF1), to offer
196                          Although exhibiting cell surface markers indicative of activation, the IL-10
197     The transcriptional profile identified 2 cell-surface markers, ITGA6 and NGFR, which can be used
198 n inflammatory cell numbers and cytokine and cell-surface marker levels on monocytes and macrophages.
199 gland stem cells (MaSCs) using combinatorial cell surface markers (Lin(-)CD24(+)CD29(h)CD49f(h)) has
200 lation based on the expression profiles of 2 cell-surface markers LNGFR (CD271) and THY-1 (CD90).
201 re the identification of TIM3 as an AML stem cell surface marker more highly expressed on multiple sp
202                             PSMA is a unique cell surface marker, negatively regulated by androgen an
203   We have isolated rare cells bearing the NK cell surface marker NK1.1, as well as the dendritic cell
204  rapidly upregulate the expression of the NK cell-surface marker NK1.1 in response to MSU crystals bu
205 ty in vitro but lacked expression of myeloid cell surface markers normally seen with MLL-CBP.
206 e attenuates acute loss of the developing OL cell surface marker O1 and the mature OL marker MBP (mye
207 ir expression of CD4, CD8, naive, and memory cell-surface markers, occupy distinct homeostatic compar
208                   CD44 is commonly used as a cell surface marker of cancer stem-like cells in epithel
209 xpression of CD146, a hypoxia down-regulated cell surface marker of human BM-MSCs.
210 e DLL1(+) cluster, revealed it to be a novel cell surface marker of human epidermal stem cells.
211  cell cycle progression and can be used as a cell surface marker of myofibroblasts.
212 11 receptor alpha subunit (IL-11Ralpha) as a cell surface marker of tumor progression that correlates
213 ets showed that effector memory pathways and cell surface markers of activation and proliferation in
214 ukocytes (VLC), due to sharing functions and cell surface markers of both dendritic cells and endothe
215  protein resulted in increased expression of cell surface markers of DC maturation and an increase in
216  that a group of immature myeloid cells with cell surface markers of Gr-1(+) CD11b(+) are highly enri
217 ion, cytokine production, proliferation, and cell surface markers of immune cells between GA-treated
218 ry CD8 T cells from naive old mice expressed cell surface markers of memory in addition to receptors
219 MSCs) were established and characterized for cell surface markers of mesenchymal stem cell origin in
220  also known as CD143), a recently identified cell-surface marker of adult human hematopoietic stem ce
221             CD30, originally identified as a cell-surface marker of Reed-Sternberg and Hodgkin cells
222                In addition, the loss of CD28 cell surface markers on CD8 + T cells, an indicator of T
223                   The expression of lymphoid cell surface markers on PBMC and splenocytes of mice hom
224 S, and microglial activation was assessed by cell surface marker or phospho-MAPK immunofluorescence.
225 eomic or flow cytometric characterization of cell surface markers or adoptive transfer.
226 ls (CSCs) can be identified by expression of cell surface markers or enzymatic activity, but these me
227 intercellular adhesion, without knowledge of cell-surface markers or intracellular proteins.
228                 Flow cytometry utilizing the cell surface markers p75 and HNK-1 was used to isolate l
229 haracteristics such as cell cycle status and cell surface marker phenotype, they respond to different
230     These results demonstrate that CD96 is a cell surface marker present on many AML-LSC and may serv
231 ese results demonstrate that IL-3Ralpha is a cell-surface marker present on FA-AML leukemia-initiatin
232 vity was confirmed by examining cytokine and cell surface marker production in bone-marrow-derived de
233 n cytokine-supplemented medium changed their cell surface marker profile and gene expression pattern
234 sms are cells characterized by a CD44+/CD24- cell surface marker profile.
235  over 24 h, as indicated by up-regulation of cell surface marker proteins CD40, CD80, and CD86.
236 OV-1 antigen, a6 integrin, and connexin 43), cell surface markers recently identified by us (CD44, CD
237  in rat tissue, they distinctively express a cell surface marker recognized by the SE-1 antibody.
238 ation; SP) in conjunction with canonical HSC cell-surface markers (Sca-1, c-Kit, and lineage markers)
239           When combined with high-throughput cell surface marker screening, this approach facilitates
240         This study identifies FolR1 as a new cell surface marker selectively expressed in mesDA proge
241     The antennal lobe neuropil expressed the cell surface marker semaphorin 1a.
242                                  Analysis of cell surface markers showed an age-dependent increase in
243 identical sets of T(EM)-associated genes and cell surface markers shown to be associated with latency
244                        These MDSCs express a cell surface marker signature (CD11b(+) Gr-1(+) Ly6C(+))
245 n is reliant on the presence of well-defined cell surface markers specific for diverse progenitor pop
246                          We identified novel cell surface markers specific for hepatic progenitor/ova
247  cells as demonstrated by expression of stem cell surface markers such as aldehyde dehydrogenase 1, s
248 m is complicated by the shared expression of cell surface markers such as CD11c.
249                            Identification of cell surface markers sufficient to purify Treg cells exp
250 ed by a remarkable up-regulation of specific cell surface markers, suggesting that LPS stimulation le
251 n of MSC with both endothelial and pericytic cell surface markers suppresses the homing of cancer cel
252 olobus purpureas agglutinin (TPA) as a novel cell surface marker that allows for such delineation.
253           These data suggest that AA4.1 is a cell surface marker that can identify the earliest lymph
254                                  CD20 is a B cell surface marker that is expressed in various stages
255 eceptor superfamily member OX40 (CD134) is a cell surface marker that is highly specific for activate
256        We identified 33 transcripts encoding cell surface markers that are differentially expressed b
257 e and chemokine responses, and expression of cell surface markers that are related to T cell activati
258 nitive endoderm with the goal of identifying cell surface markers that can be used to track the devel
259 t LSC, one potential strategy is to identify cell surface markers that can distinguish LSC from norma
260 ssociated with subclonal mutations, and find cell surface markers that could be used to purify subclo
261                       We sought to delineate cell surface markers that could distinguish NK cells tha
262               In monocytes, we identify host cell surface markers that enable enrichment of latent ce
263 acteristics, as well as the precise panel of cell surface markers that uniquely define this newly des
264 ha,and IL-2, and up-regulation of numerous T cell surface markers that would promote T-T Ag presentat
265                       These results reveal a cell-surface marker that delineates functional activity
266 , zymosan, heat-killed or live bacteria, and cell-surface markers that coexpress with FR were identif
267                                  We identify cell-surface markers that delineate a series of stress e
268 ptor type I and II (CD121a/CD121b) as unique cell-surface markers that distinguish activated Tregs fr
269 lls have been hampered by a lack of suitable cell-surface markers that specifically enable their puri
270  cell subsets that display either CD4 or CD8 cell surface markers, the leukemic cell is exclusively o
271 ltiple defects in the expression of effector cell surface markers, the synthesis of cytokines, and in
272                Upon recognizing the targeted cell-surface marker, the APH enters the host cell via en
273 D(2) receptor, CRTH2, the best selective Th2-cell surface marker to date.
274 op an assay based on loss of expression of a cell surface marker to monitor epigenetic instability at
275 crophages, while alphaCD47 blocks CD47 tumor cell surface marker to promote phagocytosis.
276 ling core-2 O-glycosylation and identified a cell surface marker to quantify Notch signals in multipl
277             Further, we identified potential cell surface markers to allow for future identification
278 ins on fibroblast cells, which are potential cell surface markers to differentiate endothelial and fi
279 tential in part independent of commonly used cell surface markers to discriminate effector and memory
280                        We have used specific cell surface markers to examine the association of NG2 c
281 nhanced green fluorescent protein (eGFP) and cell surface markers to FACS-isolate DeltaSox2-eGFP(+) G
282  is due, in part, to the difficulty of using cell surface markers to identify CD4(+)CD25(+) T reg cel
283  we combine H2B-GFP-based pulse-chasing with cell-surface markers to distinguish quiescent from proli
284 ave used flow cytometry and a defined set of cell-surface markers to identify and subsequently isolat
285 ly carried out by quantification of multiple cell surface markers, transcription factors and cytokine
286                            Human trophoblast cell-surface marker (Trop-2) is a surface glycoprotein o
287 eries of heterogeneous subpopulations in its cell surface markers, tumorigenicity, invasion and metas
288               The cells lacked expression of cell surface markers typically expressed by germinal cen
289 ver based on the expression of CD4 and other cell surface markers uniquely expressed by pDCs.
290                   They discovered a panel of cell surface markers useful for isolating living hair fo
291                                Expression of cell surface markers was assessed by flow cytometry.
292           The specificity of progenitor/oval cell surface markers was confirmed by ISH and double IF
293                                            A cell-surface marker was identified by comparing the mRNA
294                                   Additional cell surface markers were also used to quantify differen
295 he differential expression patterns of these cell surface markers were dependent on Ly49H recognition
296                                              Cell surface markers were screened for their ability to
297 cently identified myeloid lineage restricted cell surface marker, which is overexpressed in over 90%
298 Cs and a germline seminoma that share a CD38 cell-surface marker, which collectively defines likely p
299                                    CX3CR1, a cell surface marker whose expression is associated with
300 r-, costimulation-, or activation-associated cell surface markers yet were associated with lower Ly6C

 
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