ライフサイエンスコーパス: cell tumor

1 rmal tumor, and one desmoplastic small round cell tumor).

2 inal stromal tumor, leiomyosarcoma, granular cell tumor).

3 ally named PEComas (perivascular epithelioid cell tumors).

4 pathological features of tenosynovial giant-cell tumor.

5 fication of the specific Ig genes from the B-cell tumor.

6 f skin surface and dermis is related to mast cell tumor.

7 al events in patients with a history of germ cell tumor.

8 d in the aggressive desmoplastic small round cell tumor.

9 oach from the Brazilian GCT-99 study on germ cell tumors.

10 ma skin cancer (NMSC), particularly squamous cell tumors.

11 a potential target for the therapy of Leydig cell tumors.

12 rs, lung and mammary carcinomas, and spindle cell tumors.

13 echanism that controls cell growth in Leydig cell tumors.

14 with newly diagnosed non-germinomatous germ cell tumors.

15 with newly diagnosed non-germinomatous germ cell tumors.

16 RAS/LKB1-mutant adenocarcinomas and squamous cell tumors.

17 gene is elevated in most tenosynovial giant-cell tumors.

18 e inactivated in a majority of lung squamous cell tumors.

19 y in flies, worms, zebrafish, and human germ cell tumors.

20 tral nervous system tumors, or renal or germ-cell tumors.

21 rge B cell lymphoma (DLBCL) and additional B cell tumors.

22 the development of adenocarinoma and spindle cell tumors.

23 either seminomas or nonseminomas/mixed germ cell tumors.

24 the survival of mice with implanted human B-cell tumors.

25 ightly stronger for B-cell tumors than for T-cell tumors.

26 are at increased risk of developing Sertoli cell tumors.

27 ovarian serous adenocarcinomas and granulosa cell tumors.

28 the poor outcome reported in all MHC II(-) B-cell tumors.

29 ficacy should be examined in patients with B-cell tumors.

30 PIK3CA were previously described in squamous cell tumors.

31 ent of certain categories of testicular germ cell tumors.

32 omatography to membrane preparations of beta-cell tumors.

33 29% of clear cell samples and 8% of nonclear cell tumors.

34 rtility and are associated with ovarian germ cell tumors.

35 a key determinant of oncogenic fate in germ-cell tumors.

36 or misexpression of Chinmo results in blood cell tumors.

37 nd immunophenotype, such as small round blue cell tumors.

38 into how cell identity is lost in some germ cell tumors.

39 rom 0.669 for signet cell to 0.75 for goblet cell tumors.

40 a, mesothelioma, melanoma, gastric, and germ cell tumors.

41 transcriptional regulator in prostatic basal cell tumors.

42 e most prevalent molecular features of clear cell tumors.

43 s, systemic mastocytosis, and localized mast cell tumors.

44 typical prolonged washout observed in Leydig cell tumors (12 of 21 patients, P < .001 when compared w

45 y higher in adenocarcinomas than in squamous cell tumors (17.73 vs 11.46; P = .043).

46 umors (29%), predominantly in Sertoli-Leydig cell tumors (26 of 43, or 60%), including 4 tumors with

47 primitive neuroectodermal tumors, 57.8; germ cell tumors, 63.5; ependymoma or high-grade glioma, 69.8

48 h hepatoblastoma or desmoplastic small round cell tumor achieved a partial response; eight patients h

49 cells (Tregs) and myeloid-derived suppressor cells, tumor Ag-specific Tregs interfere with the detect

50 or is pathognomonic for adult-type granulosa cell tumors (AGCT) and a diagnostic marker for this tumo

51 Granulosa cell tumors also developed in G12V mice.

52 nce of FOXL2 and KRAS mutations in granulosa cell tumors and in mucinous tumors, respectively.

53 ductive system, including proliferative germ-cell tumors and uterine masses that express neuronal and

54 thyroid, thymic and lymph node lesions; germ cell tumors and vascular lesions.

55 a target of chromosomal translocations in T-cell tumors and was activated by retroviral vector inser

56 ion can be abrogated by exposing susceptible cells (tumor and mycobacteria-infected cells, or aminobi

57 serve as binding sites on the breast cancer cells/tumor and the LHRH-conjugated drugs inhibited the

58 nd paraganglioma, pericardial cyst, Purkinje cell tumor, and papillary fibroelastoma (n = 1, each).

59 lution in relation to retinoblastoma or germ cell tumors, and both cancers are rare, these findings r

60 sion was validated in Ewing sarcoma cells, B-cell tumors, and human keratinocytes where NOTCH dysregu

61 expression is deregulated in testicular germ cell tumors, and loss of imprinting occurs frequently in

62 ype-specific variations, chromophobes, clear-cell tumors, and oncocytomas were composed exclusively o

63 nocarcinomas, squamous cell carcinomas, mast cell tumors, and soft tissue sarcomas.

64 vel marker for this lymphoma across mature B-cell tumors, and support the distinction of NMZL as an i

65 SCN-like epithelial cases, small-round-blue cell tumors, and unexpectedly in hematological malignanc

66 Integrated analysis in H3.3K27M cells, tumors, and in vivo imaging in patients showed en

67 d-glucose (6-[(18)F]FDG) was studied in EMT6 cells, tumors, and muscle and correlated to GLUT1 and GL

68 ic antigen receptors (CAR) specific to the B cell tumor antigen CD19 can successfully eradicate syste

69 Endothelial cell tumors are the most common soft tissue tumors in in

70 Endothelial cell tumors are the most common soft tissue tumors in in

71 Leydig cell tumors are the most frequent interstitial neoplasms

72 Insulinomas (pancreatic islet beta cell tumors) are the most common type of functioning pan

73 ic astrocytoma (PA) is the most common glial cell tumor arising in children.

74 Follicular lymphoma (FL) is a B-cell tumor arising in germinal centers and retaining fea

75 te Phf7 expression, is also observed in germ cell tumors arising from the loss of bag of marbles (bam

76 OCT4 is commonly expressed in germ-cell tumors as well as putative cancer stem cells in sev

77 d-MyD88 infection of primary human dendritic cells, tumor-associated fibroblasts, and colorectal carc

78 Among tumor-infiltrating immune cells, tumor-associated macrophages (TAMs) take a center

79 ular immune modulators, such as regulatory T cells, tumor-associated macrophages, and myeloid-derived

80 PD-L1 is expressed on tumor cells, tumor-associated macrophages, and other cells in

81 ose proximity to colonies of prostate cancer cells, tumor-associated stromal cells and newly formed w

82 alterations, and gene expression and immune cell-tumor axis changes in a mouse model of oviductal se

83 noculated with 2 x 10(7) of Walker 256 tumor cells [tumor bearing (TB) rats].

84 therapeutics to manage various B cell and T cell tumors, because failure to open DNA hairpins and ac

85 the diagnosis of adrenomedullary chromaffin cell tumors can be jeopardized by physiologic uptake by

86 nal translocations, nearly all analyzed CP B-cell tumors carried clonal translocations.

87 e Pfn1's involvement in vascular endothelial cell tumor cell cross-talk.

88 antly increases CD20 levels in established B-cell tumor cell lines and primary malignant cells.

89 We analyzed three pairs of testicular germ cell tumor cell lines using Affymetrix expression microa

90 SPN knockout in AML cell lines facilitated T cell-tumor cell clustering and enhanced CD3 bsAb-mediate

91 type, supporting a critical role of direct T cell-tumor cell contact.

92 ubpopulations of tumor cells through stromal cell-tumor cell interactions.

93 In an ex vivo neutrophil-NK cell-tumor cell tri-cell co-culture system, neutrophils

94 For nonfixed, viable cells, tumor cell recovery ranged from 72.5% to 93.9% wi

95 be killed by CD8(+)alphabetaT or gammadeltaT cells; tumor cell killing was partially restored by trea

96 acteria in tumors and proliferation of tumor cells, tumor-cell DNA damage, and an inflammatory respon

97 of PD-L1 expression in both tumor and immune cells (tumor cells: 22c3, 2.96; 28-8, 3.26; SP142, 1.99;

98 their proximity to potential immune effector cells, tumor cells grow aggressively on these immune agg

99 Unlike normal differentiated cells, tumor cells metabolize glucose via glycolysis und

100 The interplay between stromal cells, tumor cells, and migratory cells such as lymphocy

101 eurofibromas are benign nerve sheath Schwann cell tumors characterized by biallelic mutations in the

102 g NKG2D ligands had increased incidence of B cell tumors, confirming that the inability to clear NKG2

103 ffector T-cell activation and natural killer cell tumor cytotoxicity.

104 Among B-cell tumors, deletion of 7q and NOTCH2 mutations are alm

105 nografted with human EGFRvIII-expressing GBM cells, tumor-derived suPAR was detected in the plasma, a

106 Desmoplastic small round cell tumor (DSRCT) is a rare disease of children, adoles

107 Desmoplastic small round cell tumor (DSRCT) is a rare sarcoma in adolescents and

108 lar pathogenesis of desmoplastic small round cell tumor (DSRCT), a fatal malignancy occurring primari

109 Desmoplastic small round cell tumor (DSRCT), a rare sarcoma of adolescents/young

110 al translocation in desmoplastic small round-cell tumors (DSRCT), a rare but aggressive soft tissue s

111 ily tumors (EFT) or desmoplastic small round cell tumors (DSRCT).

112 c niche consisting of transformed epithelial cells, tumor-educated fibroblasts, endothelial cells, an

113 The extragonadal germ cell tumors (EGCTs) represent a unique entity, and as su

114 endocrine tumors (pNET), also known as islet cell tumors, exhibit a wide range of biologic behaviors

115 A subset of B-cell tumors exhibits recurrent translocations of Bcl-3,

116 veral human B cell cancer lines, and human B cell tumors expressing AID at high levels have genomic u

117 n cohort of patients with tenosynovial giant-cell tumors (extension study).

118 a critical role in the progression of Hut78 cell tumor formation in skin, thus providing a new thera

119 wn to be critically required for endothelial cell tumor formation.

120 Squamous cell tumors from patients treated with an RAF inhibitor

121 tivated by retroviral vector insertions in T-cell tumors from X-SCID patients in gene therapy trials.

122 rowth, were enriched in NMZL across mature B-cell tumors, functionally caused the loss of the phospha

123 rpose Patients with relapsed metastatic germ cell tumor (GCT) can be cured with second-line and even

124 in patients with metastatic testicular germ cell tumor (GCT) is of unknown prognostic significance.

125 Giant cell tumor (GCT) of bone is a histologically benign oste

126 re conducted on 488 North American male germ cell tumor (GCT) survivors in relation to cumulative cis

127 nicopathologic heterogeneity, malignant germ cell tumors (GCT) share molecular abnormalities that are

128 than the classic markers of testicular germ cell tumors (GCTs) and are applicable toward both semino

129 sitivity, most patients with metastatic germ cell tumors (GCTs) are cured with cisplatin-based chemot

130 Germ cell tumors (GCTs) are the most common cancer in men bet

131 The anterior mediastinal germ cell tumors (GCTs) are the most common EGCT.

132 are in children, with pineoblastoma and germ cell tumors (GCTs) being the most common.

133 or children with extracranial malignant germ cell tumors (GCTs) have increased significantly.

134 granulosa cells causes metastatic granulosa cell tumors (GCTs) in female mice and phenocopies human

135 Some germ cell tumors (GCTs) in men develop into hematologic malig

136 alignant lesions that develop into granulosa cell tumors (GCTs) spontaneously later in life or follow

137 in two thirds of patients with advanced germ cell tumors (GCTs) who relapsed after first-line chemoth

138 mainstay of treatment for disseminated germ cell tumors (GCTs), is associated with venous thromboemb

139 In men with metastatic germ cell tumors (GCTs), risk-directed treatment is determine

140 Germ-cell tumors (GCTs), which arise from pluripotent embryon

141 py (HDCT) in the management of advanced germ cell tumors (GCTs).

142 agement of central nervous system (CNS) germ cell tumors (GCTs).

143 e-wide association study for testicular germ cell tumor, genotyping 298,782 SNPs in 979 affected indi

144 - and drug-resistant Emu-myc p53-/- lymphoma cell tumors grown in live mice.

145 Inhibiting Sod2 expression reduces OCCC ES-2 cell tumor growth and metastasis in a chorioallantoic me

146 y, chronic MAF overexpression enhanced MPNST cell tumor growth in vivo, correlating with elevated pS6

147 dicer and microRNA in promoting endothelial cell tumor growth in vivo.

148 otch or zfh1 are depleted in the mesenchymal cells, tumor growth is compromised.

149 t cells were injected along with WT parental cells, tumor growth was enhanced with mutant cells becom

150 inhibitor imatinib after injection of cancer cells; tumor growth was measured based on bioluminescenc

151 n study, 12 patients with tenosynovial giant-cell tumors had a partial response and 7 patients had st

152 spectrum of human neoplasms, including germ cell tumors, high-grade and low-grade carcinomas and ben

153 0.96), was lower after nonseminomatous germ cell tumor (HR, 0.58; 95% CI, 0.35 to 0.96) and decrease

154 cell clones were often observed in T- and NK-cell tumors in a percentage higher than observed in reac

155 BLV infection is strongly associated with B-cell tumors in cattle.

156 s), suggesting similar etiology between germ cell tumors in mouse and man.

157 Loss of nito activity results in stem cell tumors in the female germ line as well as female-to

158 al important developments in testicular germ cell tumors in the last year.

159 al important developments in testicular germ cell tumors in the past year.

160 al important developments in testicular germ cell tumors in the past year.

161 d slows the growth of these "addicted" pre-B-cell tumors in vivo, suggesting a promising therapeutic

162 jugated drugs inhibited the growth of breast cells/tumor in in vitro and in vivo experiments.

163 s in 1 of 14 nonseminomatous testicular germ-cell tumors, in 2 of 5 embryonal rhabdomyosarcomas, and

164 hydra analysis framework to small blue round cell tumors (including rhabdomyosarcoma, synovial sarcom

165 rated lymphocytes, low level of regulatory T-cell tumor-infiltrated lymphocytes, and low PD-L1 expres

166 ody ((89)Zr-Df-IAB22M2C) to monitor CD8(+) T-cell tumor infiltrates by PET.

167 ability of this tracer to quantify CD8(+) T-cell tumor infiltrates was evaluated in preclinical stud

168 Finally, we evaluated the immune cell tumor-infiltrating lymphocyte (TIL) score for corre

169 recognized that rapidly proliferating cancer cells, tumor-infiltrating lymphocytes, and vascular endo

170 guish them from mature lymphatic endothelial cells, tumor-infiltrating lymphoid cells, and tissue-res

171 DQB2, HLA-DMA, and HLA-DOA), increased CD8 T-cell tumor infiltration (P=7.6x10(-5)), and trans-regula

172 okine receptor 4 (CXCR4) blockade promotes T cell tumor infiltration and is synergistic with anti-PD-

173 environment and as a negative regulator of T-cell tumor infiltration and patient survival in diverse

174 breast TME, redirecting TAM activation and T cell tumor infiltration in vivo.

175 growth, increased antigen-specific CD8(+) T-cell tumor infiltration, and increased median survival,

176 BL-8040 increased CD8(+) effector T cell tumor infiltration, decreased myeloid-derived suppr

177 enes, impaired IFN gene expression, anemic T cell tumor infiltration, poor tumor immunity, and shorte

178 type and in facilitating CD4(+) and CD8(+) T-cell tumor infiltration.

179 zation of cancer in which only a fraction of cells, tumor-initiating cells (TICs), can sustain tumor

180 ith menin knockdown, in MEN1-associated beta cell tumors (insulinomas), and also in human sporadic in

181 n oncogenic factor in insulin-secreting beta-cell tumors (insulinomas).

182 have implications for bidirectional CD4(+) T-cell:tumor interactions within the TME.

183 idence interval (CI): 2.65, 3.50), with germ cell tumors (IRR = 5.19, 95% CI: 2.67, 9.41), retinoblas

184 etween chlordane isomers and testicular germ cell tumors, it is reasonable to assume that chlordanes

185 However, chemotherapy of germ-cell tumors led to the induction of anti-OCT4 immunity i

186 n treatment of TRAF3-deficient B cells and B cell tumor lines with c-Myc inhibitors enhanced their se

187 g peptide that specifically recognizes tumor cells, tumor lymphatics, and tumor-associated macrophage

188 tramer-positive CD8 T cells, increased CD8 T-cell tumor lytic activity, augmented antigen-specific IF

189 iota of 11 dogs affected by spontaneous mast cell tumor (MCT), using skin contralateral sites as intr

190 tumor-associated myeloid-derived suppressor cells (tumor-MDSCs).

191 Effects against mitochondria and general cell tumor metabolism were noted at higher concentration

192 s with intermediate-risk (IR) malignant germ cell tumors (MGCT) if the administration of cisplatin, e

193 o better define the interaction between mast cell tumors, microbiota and host immune response.

194 ting of a genetic disease, the B-cell/plasma cell tumor microenvironment (TME) contributes significan

195 spontaneous translocations in other mouse B-cell tumor models, CP B-cell tumor translocations were n

196 ic characteristics of SED against H1650 stem cell tumor models.

197 vival outcomes in metastatic testicular germ cell tumor (MT-GCT), but how the initial risk changes ov

198 own as HHV8) is the causative agent of two B cell tumors, multicentric Castleman disease (MCD) and pr

199 tivation of genes expressed in retinal glial cells, tumor necrosis factor-alpha, and transforming gro

200 kers to differentiate non-germinomatous germ cell tumors (NGGCTs) from germinomas are critical, as th

201 nce (AS) for testicular nonseminomatous germ cell tumors (NSGCT) is widely used.

202 thologic stage (PS) IIA nonseminomatous germ cell tumors (NSGCTs) is 10%-20% but increases to >= 50%

203 osteosarcoma and, less frequently, in giant cell tumor of bone (GCT).

204 The neoplastic stromal cells of giant cell tumor of bone (GCTB) carry a mutation in H3F3A, lea

205 d villonodular synovitis (dtPVNS), and giant cell tumor of bone (GCTB).

206 PURPOSE OF REVIEW: Giant cell tumor of tendon sheath and pigmented villonodular s

207 -year-old man with a history of a mixed germ cell tumor of the testis presented with acute-onset, rig

208 ndings are mirrored in G34W-containing giant cell tumors of bone where patient-derived stromal cells

209 In contrast, in 92% (49/53) of giant cell tumors of bone, we found histone H3.3 alterations e

210 that has been implicated in testicular germ cell tumors of mouse and human.

211 The majority of small-cell tumors of the gynecologic tract will require system

212 Germ cell tumors of the ovary constitute less than one percen

213 Validation in a bigger cohort of clear-cell tumors of the ovary is warranted.

214 acteristics of large-cell calcifying Sertoli cell tumors of the testes (LCCSCTs).

215 ciplinary approach to the management of germ cell tumors of the testis has resulted in survival rates

216 e the surgical management of metastatic germ cell tumors of the testis, highlighting the indications

217 ratio (OR) = 1.05; 95% CI: 1.01, 1.10]; germ cell tumors (OR = 1.16; 95% CI: 1.04, 1.29), particularl

218 only associated with endometrioid and clear cell tumors (P-het </= .01).

219 riplatin in the National Cancer Institute 60-cell tumor panel screen revealed a spectrum of activity

220 We extracted nascent plasma cell tumor (PCT) cells from within inflammatory oil gran

221 ecapitulating human perivascular epithelioid cell tumors (PEComas) from patients with TSC.

222 PURPOSE Perivascular epithelioid cell tumors (PEComas) represent a family of mesenchymal

223 nesis, leading to either an agametic or germ cell tumor phenotype.

224 f the resected tissue showed metastatic germ cell tumor predominantly consisting of a yolk sac elemen

225 Importantly, single-cell tumor profiles reveal highly defined cell hierarchi

226 on factors and miRNAs coordinate cancer stem cell tumor-propagating capacity are unclear.

227 Mice susceptible to plasma cell tumors provide a useful model for human multiple my

228 NK group 2D ligands in the impairment of NK cell tumor recognition and killing.

229 Upon direct recognition of cancer cells, tumor-recognizing CD4(+) T cells (TR-CD4) potentl

230 induction promoted drug resistance in MCF-7 cells, tumor regeneration in Du145 cells and, most impor

231 postorchiectomy early-stage nonseminoma germ cell tumors remains a topic of debate.

232 s a partner in recurrent translocations in B cell tumors, resulting in deregulated expression.

233 ably, hydra analysis of all small blue round cell tumors revealed similar subtypes, characterized by

234 Human germ cell tumors show a strong sensitivity to genetic backgro

235 ve been reported with IFNgamma-producing Th1 cells, tumor-specific Th2 cells have been largely neglec

236 d antimetastasis activity in two-dimensional cells, tumor spheroids, subcutaneous transplantation mou

237 y broad and high expression levels in plasma cell tumors such as multiple myeloma (MM).

238 These alterations might affect NK-cell tumor surveillance.

239 We also found that ovarian clear cell tumors susceptible to both genetic and pharmacologi

240 on of human carcinomas, e.g. diffuse large B cell tumors, T cell lymphomas, etc.

241 hepatoblastomas (HBs), 1 transitional liver cell tumor (TCLT), 1 hepatocellular carcinoma, and 2 mal

242 necrosis (20%), viable nonteratomatous germ cell tumor +/- teratoma (41%), and secondary somatic-typ

243 Patients with testicular germ cell tumor (TGCT) are at increased risk of developing a

244 lex genetic factors underlie testicular germ cell tumor (TGCT) development.

245 association (GWA) studies of testicular germ cell tumor (TGCT) have identified 18 susceptibility loci

246 Testicular germ cell tumor (TGCT) is the most common cancer in young men

247 aggressive fibromatosis, tenosynovial giant cell tumor (TGCT) or diffuse-type pigmented villonodular

248 molecular pathogenesis of tenosynovial giant cell tumor (TGCT) or pigmented villonodular synovitis (P

249 conventional inheritance for testicular germ cell tumor (TGCT) risk both in humans and mice implicate

250 yzed population-wide data on testicular germ-cell tumor (TGCT) status in 1,135,320 two-generational N

251 the most potent modifiers of testicular germ cell tumor (TGCT) susceptibility in mice and rats.

252 inct susceptibility loci for testicular germ cell tumor (TGCT).

253 new susceptibility loci for testicular germ cell tumor (TGCT).

254 -wide association studies of testicular germ cell tumor (TGCT; 3,558 cases and 13,970 controls) to id

255 Testicular germ cell tumors (TGCT) are considered a paradigm of chemosen

256 Testicular germ cell tumors (TGCT) are the most frequently diagnosed sol

257 Testicular germ cell tumors (TGCT) generally respond well to chemotherap

258 Susceptibility to testicular germ cell tumors (TGCT) has a significant heritable component

259 Testicular germ cell tumors (TGCT) originate from germ cells.

260 Testicular germ cell tumors (TGCT) represent the most common malignancy

261 Tenosynovial giant cell tumors (TGCT), are rare colony stimulating factor-1

262 actor for the development of testicular germ cell tumors (TGCT), but the initiating event linking the

263 tworks in two major types of testicular germ cell tumors (TGCT): seminoma (SE) and non-seminoma (NSE)

264 Testicular germ cell tumors (TGCTs) are classified into two main subtype

265 Testicular germ cell tumors (TGCTs) are highly responsive to and curable

266 e enhances susceptibility to testicular germ cell tumors (TGCTs) in mice, in part by interacting epig

267 , and risk stratification of testicular germ cell tumors (TGCTs) in the past year.

268 al important developments in testicular germ cell tumors (TGCTs) over the past year.

269 Testicular germ cell tumors (TGCTs) share germline ancestry but diverge

270 tanding of susceptibility to testicular germ cell tumors (TGCTs), but much of the heritability remain

271 so are misregulated in human testicular germ cell tumors (TGCTs), suggesting similar etiology between

272 Testicular germ cell tumors (TGCTs), the most common neoplasms of young

273 ed with an increased risk of testicular germ cell tumors (TGCTs).

274 er drug for the treatment of testicular germ cell tumors (TGCTs).

275 verage type and were slightly stronger for B-cell tumors than for T-cell tumors.

276 l zone lymphoma (NMZL) is a rare, indolent B-cell tumor that is distinguished from splenic marginal z

277 agnosis and therapy of malignant small round cell tumors that affect children, particularly in rhabdo

278 Insulinomas are beta-cell tumors that cause hypoglycemia through inappropriat

279 slowly proliferating, insulin-producing beta cell tumors that provide a molecular "recipe" or "roadma

280 is, leading to the accumulation of granulosa cell tumors that reactivates the epithelial program from

281 rtoli cells also caused testicular granulosa cell tumors that showed gene expression patterns that pa

282 preclinical canine study of spontaneous mast cell tumors, the treatment led to a 67% response rate (t

283 protein in primary human colonic epithelial cells, tumor tissues, and colon cancer cell lines.

284 with the widespread expression in epithelial cells, tumor tissues, and macrophages detected using BTN

285 e to the hypersensitivity of testicular germ cell tumors to cisplatin, are discussed.

286 ons in other mouse B-cell tumor models, CP B-cell tumor translocations were not recurrent and did not

287 ere indeed found in 7% of 43 testicular germ cell tumor trios; this percentage exceeds background CNV

288 s expression levels in human testicular germ cell tumors using patient tissues, model cell lines, and

289 glucopyranosyl lipid A (GLA), using a whole-cell tumor vaccine.

290 r the interaction between RRV and human host cells (tumor versus nontumor) in vitro.

291 importance of versican in this smooth muscle cell tumor, we used versican-directed siRNA to knock dow

292 ociated with lymphomas and some ovarian germ cell tumors, we present a case of calcitriol overproduct

293 Lung epithelial TC1 cell tumors were 84% greater in mice subjected to IH for

294 ts with a history of or newly diagnosed germ cell tumors were evaluable.

295 Malignant germ cell tumors were identified in 2.8% (31 of 1097) of boys

296 treatment of patients with nonseminoma germ cell tumor, whereas radiotherapy, as a standard treatmen

297 my, and pathology showed a 1.5-cm mixed germ cell tumor with 85% embryonal, 10% yolk sac tumor, and 5

298 skin developed significantly fewer squamous cell tumors with a delayed onset when compared with wild

299 Treatment of tenosynovial giant-cell tumors with PLX3397 resulted in a prolonged regress

300 529 was highly active against subcutaneous B-cell tumor xenografts in severe combined immunodeficient